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Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 7, pp. 678 - 684
Recovery from blood loss requires a greatly enhanced supply of iron to support expanded erythropoiesis. After hemorrhage, suppression of the iron-regulatory... 
OVERLOAD | HOMEOSTASIS | DIFFERENTIATION FACTOR 15 | BETA-THALASSEMIA-INTERMEDIA | ERYTHROPOIESIS | GENETICS & HEREDITY | MOUSE-LIVER | MICE | HEPCIDIN EXPRESSION | ANEMIA | HYPOXIA | Hepcidins - metabolism | beta-Thalassemia - pathology | Liver - pathology | Iron Overload - drug therapy | Molecular Sequence Data | Erythropoiesis - drug effects | Male | Gene Expression Profiling | Iron Overload - pathology | Iron Overload - metabolism | Epoetin Alfa | Erythropoietin - metabolism | Real-Time Polymerase Chain Reaction | Anemia - etiology | Disease Models, Animal | Recombinant Proteins - metabolism | Hormones - pharmacology | Enzyme-Linked Immunosorbent Assay | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Hemorrhage - complications | Hemoglobins - metabolism | Hemorrhage - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Iron - metabolism | Blotting, Western | Erythropoiesis - physiology | Mice, Knockout | Anemia - metabolism | Animals | Mice | beta-Thalassemia - metabolism | Physiological aspects | Genetic research | Genetic aspects | Iron in the body | Hormones | Research | Genetic regulation | Identification and classification | Flow cytometry | Plasma | Phosphorylation | Bone marrow | Homeostasis | Iron | Grants | Metabolism | Gene expression | Experiments | Hemorrhage | Index Medicus
Journal Article
Investigative ophthalmology & visual science, ISSN 0146-0404, 2012, Volume 53, Issue 6, pp. 2968 - 2977
PURPOSE. Retinopathy of prematurity (ROP) is a leading cause of blindness in children and is, in its most severe form, characterized by uncontrolled growth of... 
RETINAL NEOVASCULARIZATION | BLOCKERS | MOUSE MODEL | RAT RETINA | OPHTHALMOLOGY | INFANTILE HEMANGIOMAS | FACTOR EXPRESSION | PREMATURITY | INFANCY | ENDOTHELIAL GROWTH-FACTOR | Up-Regulation | Cell Proliferation | Angiopoietin-2 - genetics | Humans | Gene Expression Profiling | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | Angiopoietin-1 - metabolism | Vascular Endothelial Growth Factor A - genetics | Angiopoietin-2 - metabolism | Dose-Response Relationship, Drug | Receptors, Vascular Endothelial Growth Factor - metabolism | Retina - cytology | Propranolol - administration & dosage | Injections, Subcutaneous | Retinal Neovascularization - prevention & control | Erythropoietin - metabolism | Receptors, Vascular Endothelial Growth Factor - genetics | Drug Evaluation, Preclinical | Receptors, Adrenergic, beta - metabolism | Adrenergic beta-Antagonists - administration & dosage | Infant, Newborn | Disease Models, Animal | RNA - metabolism | Animals, Newborn | Oxygen - toxicity | Enzyme-Linked Immunosorbent Assay | Administration, Oral | Injections, Intraperitoneal | Reverse Transcriptase Polymerase Chain Reaction | Angiopoietin-1 - genetics | Erythropoietin - genetics | Animals | Retinopathy of Prematurity - prevention & control | Mice | Retinopathy of Prematurity - metabolism | In Vitro Techniques | Endothelial Cells - drug effects | Index Medicus | Disease Models | Other Medical Sciences | Endothelial Cells/drug effects | Oxygen/toxicity | Drug Evaluation | Vascular Endothelial Growth Factor A/genetics/metabolism | Infant | Propranolol/administration & dosage | Adrenergic | Oral | Intraperitoneal | RNA/metabolism | Receptors | Retinopathy of Prematurity/metabolism/prevention & control | beta/metabolism | Preclinical | Adrenergic beta-Antagonists/administration & dosage | Drug | Administration | Retinal Neovascularization/metabolism/prevention & control | Subcutaneous | Angiopoietin-2/genetics/metabolism | Erythropoietin/genetics/metabolism | Retina/cytology | Angiopoietin-1/genetics/metabolism | Injections | Newborn | Annan medicin och hälsovetenskap | Animal | Dose-Response Relationship | Vascular Endothelial Growth Factor/genetics/metabolism
Journal Article
Journal Article
Cellular Signalling, ISSN 0898-6568, 04/2013, Volume 25, Issue 4, pp. 898 - 909
The effect of differential signalling by IL-6 and leukaemia inhibitory factor (LIF) which signal by gp130 homodimerisation or LIFRβ/gp130 heterodimerisation on... 
Glycoprotein130 | Cardiomyocyte | Interleukin-6 | MYOCARDIAL-INFARCTION | TRANSCRIPTION 3 | INDUCED APOPTOSIS | HEART-FAILURE | TRANSDUCER | CARDIAC MYOCYTE SURVIVAL | CELL BIOLOGY | ERYTHROPOIETIN RECEPTOR | SIMULATED ISCHEMIA | EXPRESSION | Phosphorylation | Cytokine Receptor gp130 - genetics | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Janus Kinases - metabolism | Leukemia Inhibitory Factor - genetics | Phosphatidylinositol 3-Kinases - metabolism | Recombinant Proteins - biosynthesis | Recombinant Fusion Proteins - metabolism | Receptors, Erythropoietin - genetics | Leukemia Inhibitory Factor - metabolism | STAT1 Transcription Factor - metabolism | Flavonoids - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-6 - metabolism | Reperfusion Injury - metabolism | STAT3 Transcription Factor - metabolism | Cell Survival - drug effects | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | Myocytes, Cardiac - cytology | Reperfusion Injury - pathology | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Cells, Cultured | Rats | Imidazoles - pharmacology | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Leukemia Inhibitory Factor - pharmacology | Animals | Myocytes, Cardiac - drug effects | Interleukin-6 - pharmacology | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Myocytes, Cardiac - metabolism | Recombinant Fusion Proteins - genetics | Mice | Pyridines - pharmacology | Nitric Oxide - metabolism | Receptors, Erythropoietin - metabolism | STAT3 Transcription Factor - antagonists & inhibitors | Hypertrophy | Mitogen-Activated Protein Kinase 1 - metabolism | Nitric Oxide Synthase Type II - metabolism | Interleukins | Nitric oxide | Heart cells | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 03/2016, Volume 127, Issue 10, pp. 1325 - 1335
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2017, Volume 19, Issue 6, pp. 626 - 638
Advances in genomic profiling present new challenges of explaining how changes in DNA and RNA are translated into proteins linking genotype to phenotype. Here... 
MESSENGER-RNAS | HEMATOPOIETIC STEM-CELLS | MOUSE MODEL | TRANSCRIPTION | DIAMOND-BLACKFAN ANEMIA | GENE-EXPRESSION | L-LEUCINE IMPROVES | MASS-SPECTROMETRY | MTOR | SEQ | CELL BIOLOGY | Erythropoiesis | Mitochondria - enzymology | Protein Biosynthesis | TOR Serine-Threonine Kinases - metabolism | Humans | Multiprotein Complexes - genetics | Mitochondrial Proteins - genetics | RNA, Messenger - metabolism | Receptors, Erythropoietin - genetics | Mechanistic Target of Rapamycin Complex 1 | RNA - genetics | DNA-Binding Proteins - metabolism | Multiprotein Complexes - metabolism | Organelle Biogenesis | TOR Serine-Threonine Kinases - genetics | Transfection | RNA Interference | Mitochondrial Proteins - metabolism | Acetylation | Proteomics - methods | Repressor Proteins - metabolism | RNA - metabolism | PTEN Phosphohydrolase - genetics | High Mobility Group Proteins - metabolism | Signal Transduction | RNA, Messenger - genetics | Cells, Cultured | PTEN Phosphohydrolase - metabolism | Repressor Proteins - genetics | Gene Expression Profiling - methods | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mice, Knockout | Transcription Factors - metabolism | Phenotype | Animals | Histones - metabolism | Receptors, Erythropoietin - metabolism | Hematopoietic Stem Cells - enzymology | High Mobility Group Proteins - genetics | Post-transcription | Deregulation | Transcription factors | Profiling | Transcription | Gene regulation | Genomes | Biosynthesis | Defects | Proteins | Signal transduction | Mitochondria | Deoxyribonucleic acid--DNA | Translation | Pharmacology | Metabolism | Gene expression | Erythroid cells | Ribonucleic acid--RNA | Signaling | Stem cells | Cells (biology) | In vivo methods and tests | In vitro methods and tests | Index Medicus
Journal Article
FASEB Journal, ISSN 0892-6638, 12/2005, Volume 19, Issue 14, pp. 2026 - 2028
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2002, Volume 22, Issue 23, pp. 10291 - 10301
In an in vitro model of cerebral ischemia (oxygen glucose deprivation, OGD) we investigated whether erythropoietin (EPO) plays a critical role in ischemic... 
Neuron | Bad | Janus kinase-2 | Phosphoinositol-3 kinase | Ischemic preconditioning | Oxygen glucose deprivation | Astrocyte | Hypoxia-inducible factor-1 | oxygen glucose deprivation | MOUSE-BRAIN | INDUCIBLE FACTOR-I | CULTURED NEURONS | ischemic preconditioning | neuron | ERYTHROID-CELLS | FACTOR-KAPPA-B | NEUROSCIENCES | CORTICAL-NEURONS | MESSENGER-RNA | hypoxia-inducible factor-1 | phosphoinositol-3 kinase | SIGNALING CASCADES | astrocyte | TRANSCRIPTION FACTOR | CEREBRAL-ISCHEMIA | Erythropoietin - pharmacology | Rats, Wistar | Protein-Tyrosine Kinases - metabolism | Neurons - cytology | Phosphatidylinositol 3-Kinases - metabolism | RNA, Messenger - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit | Brain - metabolism | DNA-Binding Proteins - metabolism | Dose-Response Relationship, Drug | Janus Kinase 2 | Brain - blood supply | Paracrine Communication - physiology | Neurons - metabolism | Proto-Oncogene Proteins | Erythropoietin - metabolism | Phosphorylation - drug effects | Neurons - drug effects | Hypoxia-Ischemia, Brain - metabolism | Astrocytes - cytology | Brain - cytology | Hypoxia-Inducible Factor 1 | Cells, Cultured | Ischemic Preconditioning | Rats | Nuclear Proteins - metabolism | Antibodies - pharmacology | Erythropoietin - genetics | Glucose - deficiency | Animals | Carrier Proteins - metabolism | Signal Transduction - drug effects | Models, Biological | Paracrine Communication - drug effects | Glucose - metabolism | Signal Transduction - physiology | Transcription Factors | Receptors, Erythropoietin - metabolism | Receptors, Erythropoietin - antagonists & inhibitors | bcl-Associated Death Protein | Apoptosis | Astrocytes - metabolism | Index Medicus
Journal Article
Journal Article
Aging Cell, ISSN 1474-9718, 04/2013, Volume 12, Issue 2, pp. 280 - 291
Testosterone administration increases hemoglobin levels and has been used to treat anemia of chronic disease. Erythrocytosis is the most frequent adverse event... 
steroids | aging | sex hormones | antiaging | endocrinology | Aging | Sex hormones | Antiaging | Endocrinology | Steroids | HEMOGLOBIN | ERYTHROPOIETIN | MECHANISM | SUPPRESSION | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | THERAPY | METABOLISM | INFLAMMATION | MEN | ANEMIA | EXPRESSION | Transcription, Genetic - drug effects | Humans | Receptors, Androgen - metabolism | Male | Spleen - drug effects | Hepatocytes - metabolism | Antimicrobial Cationic Peptides - metabolism | Testosterone - pharmacology | Bone Morphogenetic Proteins - metabolism | Hepatocytes - cytology | Liver - drug effects | Hepcidins | Erythrocytes - cytology | Female | Erythropoietin - metabolism | Hemoglobins - biosynthesis | Antimicrobial Cationic Peptides - genetics | Bone Morphogenetic Proteins - genetics | Hepatocytes - drug effects | Liver - metabolism | Mice, Transgenic | Smad Proteins - genetics | Iron - metabolism | Erythrocytes - drug effects | Gene Expression Regulation - drug effects | Erythropoietin - genetics | Animals | Receptors, Androgen - genetics | Signal Transduction - drug effects | Spleen - metabolism | Erythrocytes - metabolism | K562 Cells | Mice | Transferrin - metabolism | Smad Proteins - metabolism | Testosterone | Flutamide | Erythropoietin | RNA | Hemoglobin | Bone morphogenetic proteins | Iron | Hemoglobin synthesis | Genetic engineering | Genetic transcription | Androgens | Blood | Index Medicus
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 06/2010, Volume 106, Issue 11, pp. 1722 - 1730