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JNCI : Journal of the National Cancer Institute, ISSN 0027-8874, 01/2018, Volume 110, Issue 1, pp. 109 - 120
Background: Cancer-associated fibroblasts (CAFs) are tumor-promoting and correlate with poor survival in many cancers, which has led to their emergence as... 
CELL LUNG-CANCER | PHASE-II TRIAL | OXIDATIVE STRESS | ONCOLOGY | NAB-PACLITAXEL | ACTIVATION PROTEIN | DEPENDENT INVASION | PANCREATIC-CANCER | MONOCLONAL-ANTIBODY | DIFFERENTIATION | STROMAL CELLS | Lung Neoplasms - drug therapy | Up-Regulation | Pyrazoles - therapeutic use | Reactive Oxygen Species - metabolism | Cell Count | Humans | Middle Aged | Male | Oropharyngeal Neoplasms - pathology | Head and Neck Neoplasms - chemistry | RNA Interference | Cancer-Associated Fibroblasts - chemistry | Aged, 80 and over | NADPH Oxidases - genetics | Adenocarcinoma - chemistry | Esophageal Neoplasms - chemistry | Lung Neoplasms - chemistry | NADPH Oxidases - antagonists & inhibitors | Head and Neck Neoplasms - drug therapy | Carcinoma, Squamous Cell - chemistry | Survival Rate | Adenocarcinoma - drug therapy | Disease Progression | Phenotype | Carcinoma, Squamous Cell - drug therapy | Head and Neck Neoplasms - genetics | Mice | Neoplasm Transplantation | Carcinoma, Non-Small-Cell Lung - chemistry | Carcinoma, Squamous Cell - genetics | Colorectal Neoplasms - chemistry | Cell Transdifferentiation - genetics | Myofibroblasts - chemistry | Actins - analysis | Cell Transdifferentiation - drug effects | Adult | Female | Adenocarcinoma - genetics | Cancer-Associated Fibroblasts - physiology | Pyridines - therapeutic use | Lung Neoplasms - genetics | Myofibroblasts - pathology | Carcinoma, Non-Small-Cell Lung - genetics | NADPH Oxidases - analysis | Mouth Neoplasms - chemistry | NADPH Oxidase 4 | Cancer-Associated Fibroblasts - pathology | Animals | Esophageal Neoplasms - genetics | Mouth Neoplasms - pathology | Oropharyngeal Neoplasms - chemistry | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Colorectal Neoplasms - pathology | Esophageal Neoplasms - drug therapy
Journal Article
Digestive Diseases and Sciences, ISSN 0163-2116, 2/2017, Volume 62, Issue 2, pp. 491 - 501
Gastrointestinal (GI) tumor bleeding can vary from occult bleeding to massive hemorrhage and can be the presenting sign of malignancy.Our primary aims were to:... 
Biochemistry, general | Neoplasm | Mass | Melena | Medicine & Public Health | Hepatology | Gastroenterology | Oncology | Ulcer | Hemorrhage | Hematemesis | Transplant Surgery | MORTALITY | GASTROENTEROLOGY & HEPATOLOGY | CANCER | Adenocarcinoma - pathology | Gastrointestinal Neoplasms - mortality | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Male | Jejunal Neoplasms - mortality | Lymphoma - mortality | Duodenal Neoplasms - complications | Esophageal Neoplasms - pathology | Neoplasm Metastasis | Gastrointestinal Stromal Tumors - mortality | Duodenal Neoplasms - pathology | Ileal Neoplasms - pathology | Stomach Neoplasms - complications | Endoscopy, Gastrointestinal | Hematemesis - etiology | Adenocarcinoma - secondary | Disease Progression | Ileal Neoplasms - complications | Carcinoma, Squamous Cell - complications | Stomach Neoplasms - mortality | Gastrointestinal Neoplasms - secondary | Prognosis | Duodenal Neoplasms - mortality | Gastrointestinal Neoplasms - complications | Stomach Neoplasms - pathology | Gastrointestinal Neoplasms - pathology | Esophageal Neoplasms - complications | Carcinoma, Squamous Cell - mortality | Gastrointestinal Stromal Tumors - pathology | Jejunal Neoplasms - complications | Esophageal Neoplasms - mortality | Female | Lymphoma - pathology | Retrospective Studies | Melena - etiology | Colonic Neoplasms - mortality | Ileal Neoplasms - mortality | Cross-Sectional Studies | Adenocarcinoma - complications | Colonic Neoplasms - complications | Gastrointestinal Hemorrhage - etiology | Colonic Neoplasms - pathology | Lymphoma - complications | Carcinoma, Squamous Cell - secondary | Gastrointestinal Stromal Tumors - complications | Aged | Adenocarcinoma - mortality | Jejunal Neoplasms - pathology | Medical colleges | Colon cancer | Gastrointestinal bleeding | Gastrointestinal diseases | Mortality | Gastrointestinal system | Metastasis | Cancer
Journal Article
British journal of cancer, ISSN 0007-0920, 02/2018, Volume 118, Issue 3, pp. 428 - 434
Background: Periodontal pathogens have been linked to oral and gastrointestinal (orodigestive) carcinogenesis. However, the exact mechanisms remain unknown.... 
chymotrypsin-like proteinase | Treponema denticola | periodontopathogen | COLLAGENASE-2 MMP-8 | PORPHYROMONAS-GINGIVALIS | PANCREATIC-CANCER | TISSUE INHIBITORS | PERIODONTAL-DISEASE | METALLOPROTEINASES | ONCOLOGY | gastrointestinal cancer | ALPHA-1-ANTITRYPSIN | EXPRESSION | oral cancer | HUMAN NEUTROPHIL | PROGRESSION | Digestive System Neoplasms - metabolism | Pancreatic Neoplasms - chemistry | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Treponema denticola - enzymology | Mouth Neoplasms - metabolism | Stomach Neoplasms - pathology | Tonsillar Neoplasms - chemistry | Complement C1q - metabolism | Colonic Neoplasms - metabolism | Head and Neck Neoplasms - metabolism | Esophageal Neoplasms - pathology | Tonsillar Neoplasms - metabolism | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Tissue Inhibitor of Metalloproteinase-2 - metabolism | Head and Neck Neoplasms - chemistry | Matrix Metalloproteinase 9 - metabolism | Adenocarcinoma - metabolism | Matrix Metalloproteinase 8 - metabolism | Esophageal Neoplasms - metabolism | Digestive System Neoplasms - pathology | Colonic Neoplasms - chemistry | Stomach Neoplasms - chemistry | Adenocarcinoma - chemistry | Esophageal Neoplasms - chemistry | Tonsillar Neoplasms - pathology | Pancreatic Neoplasms - pathology | Carcinoma, Squamous Cell - chemistry | Mouth Neoplasms - chemistry | Head and Neck Neoplasms - pathology | Cell Transformation, Neoplastic - immunology | Digestive System Neoplasms - chemistry | alpha 1-Antichymotrypsin - metabolism | Colonic Neoplasms - pathology | Chymases - analysis | Mouth Neoplasms - pathology | Molecular Diagnostics
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 1998, Volume 279, Issue 5350, pp. 577 - 580
Gastrointestinal stromal tumors (GISTs) are the most common mesenchymal tumors in the human digestive tract, but their molecular etiology and cellular origin... 
Complementary DNA | Lymphocytes | Cell lines | Reports | Mice | Mast cells | Genetic mutation | Small intestine | Summarization | Endothelial cells | Tumors | LIGAND-INDEPENDENT ACTIVATION | HUMAN SMALL-INTESTINE | W-LOCUS | POSSIBLE INVOLVEMENT | CONSTITUTIVE ACTIVATION | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | MAST-CELLS | SUBMUCOSAL BORDER | RECEPTOR TYROSINE KINASE | INTERSTITIAL-CELLS | Sequence Deletion | Antigens, CD34 - analysis | Phosphorylation | Gastrointestinal Neoplasms - genetics | Humans | Molecular Sequence Data | Stomach Neoplasms - metabolism | Stomach Neoplasms - pathology | Proto-Oncogene Proteins c-kit - metabolism | Esophageal Neoplasms - pathology | Phosphotyrosine - metabolism | Gastrointestinal Neoplasms - pathology | Transfection | Esophageal Neoplasms - metabolism | Stem Cell Factor - pharmacology | Proto-Oncogene Proteins c-kit - genetics | Intestinal Neoplasms - genetics | Stomach Neoplasms - genetics | Amino Acid Sequence | Cell Line | Recombinant Proteins - pharmacology | Proto-Oncogene Proteins c-kit - analysis | Intestinal Neoplasms - pathology | Gastrointestinal Neoplasms - chemistry | Animals | Esophageal Neoplasms - genetics | Cell Transformation, Neoplastic | Mice, Nude | Ligands | Mutation | Intestinal Neoplasms - chemistry | Proto-Oncogene Proteins c-kit - chemistry | DNA, Complementary | Digestive System - cytology | Mesentery | Research | Gastrointestinal tumors | Proteins | Digestive system | Deoxyribonucleic acid | DNA
Journal Article
Mayo Clinic proceedings, ISSN 0025-6196, 2012, Volume 87, Issue 5, pp. 430 - 436
Abstract Objective To evaluate the relationship between the use of zolpidem and subsequent cancer risk in Taiwanese patients. Methods We used data from the... 
Internal Medicine | INSOMNIA | MEDICINE, GENERAL & INTERNAL | SLEEP | VIRUS | PREVALENCE | EPIDEMIOLOGY | Confidence Intervals | Hypnotics and Sedatives - adverse effects | Lung Neoplasms - chemically induced | Humans | Middle Aged | Liver Neoplasms - chemically induced | Male | Hypnotics and Sedatives - therapeutic use | Esophageal Neoplasms - epidemiology | Young Adult | Liver Neoplasms - epidemiology | Mouth Neoplasms - chemically induced | Pyridines - adverse effects | Neoplasms - chemically induced | Adult | Female | Odds Ratio | Urinary Bladder Neoplasms - epidemiology | Breast Neoplasms - epidemiology | Pyridines - therapeutic use | Esophageal Neoplasms - chemically induced | Risk Factors | Mouth Neoplasms - epidemiology | Lung Neoplasms - epidemiology | Taiwan - epidemiology | Sex Distribution | Urinary Bladder Neoplasms - chemistry | Breast Neoplasms - chemically induced | Sleep Initiation and Maintenance Disorders - drug therapy | Population Surveillance | Neoplasms - epidemiology | Cohort Studies | Zolpidem | Complications and side effects | Research | Risk factors | Cancer | NHRI, National Health Research Institute | CI, confidence interval | NHI, National Health Insurance | HR, hazard ratio | CIN, cervical intraepithelial neoplasia | CIS, carcinoma in situ | Original | NHIRD, National Health Insurance Research Database | ICD-9-CM, International Classification of Diseases, Ninth Revision, Clinical Modification
Journal Article
Nature medicine, ISSN 1546-170X, 2017, Volume 23, Issue 6, pp. 664 - 673
Neuroendocrine (NE) cancers are a diverse group of neoplasms typically diagnosed and treated on the basis of their site of origin... 
MOLECULAR CHARACTERIZATION | MEDICINE, RESEARCH & EXPERIMENTAL | PANCREATIC ENDOCRINE NEOPLASMS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GASTROINTESTINAL-TRACT | AUTOCHTHONOUS PROSTATE-CANCER | T-ANTIGEN | CELL BIOLOGY | CELL LUNG-CANCER | TRANSGENIC ADENOCARCINOMA | MOUSE MODEL | N-MYC | MUTATIONAL LANDSCAPE | Lung Neoplasms - drug therapy | Colonic Neoplasms - genetics | Carcinoma, Small Cell - genetics | Colonic Neoplasms - drug therapy | Epigenesis, Genetic | Humans | Male | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Tumor Suppressor Protein p53 - genetics | Ovarian Neoplasms - genetics | Neoplasms, Glandular and Epithelial - genetics | Prostatic Neoplasms - genetics | Urinary Bladder Neoplasms - genetics | Female | Cell Differentiation | Retinoblastoma Binding Proteins - genetics | Ovarian Neoplasms - drug therapy | Prostatic Neoplasms - drug therapy | Triazoles - therapeutic use | Lung Neoplasms - genetics | Antibodies, Monoclonal, Humanized - therapeutic use | Carcinoma, Neuroendocrine - drug therapy | Carcinoma, Small Cell - drug therapy | Carcinogenesis - genetics | Head and Neck Neoplasms - drug therapy | Carcinoma, Neuroendocrine - genetics | Uterine Cervical Neoplasms - drug therapy | Urinary Bladder Neoplasms - drug therapy | Uterine Cervical Neoplasms - genetics | Disease Progression | Azepines - therapeutic use | Neuroendocrine Tumors - genetics | Proto-Oncogene Proteins c-met - genetics | Cell Lineage | Cell Plasticity - genetics | Esophageal Neoplasms - genetics | Pyrimidines - therapeutic use | Neuroendocrine Tumors - drug therapy | Head and Neck Neoplasms - genetics | Proto-Oncogene Proteins c-myc - genetics | Ubiquitin-Protein Ligases - genetics | Benzodiazepines - therapeutic use | Esophageal Neoplasms - drug therapy | Proteins | Research | Health aspects | Carcinogenesis | Gastrin
Journal Article
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e103882
Background: Activating transcription factor 4 (ATF4) is a stress response gene that is involved in homeostasis and cellular protection. However, its expression... 
SURVIVAL | RESECTION | MULTIDISCIPLINARY SCIENCES | REINITIATION | TRANSLATION | HYPOXIA | CANCER | EXPRESSION | Multivariate Analysis | Neoplasm Transplantation | Prognosis | Lung Neoplasms - mortality | Carcinoma, Squamous Cell - metabolism | Humans | Lung Neoplasms - metabolism | Middle Aged | Male | Liver Neoplasms - mortality | Esophageal Neoplasms - pathology | Carcinoma, Squamous Cell - mortality | Lung Neoplasms - secondary | Matrix Metalloproteinase 7 - metabolism | Esophageal Neoplasms - metabolism | Biomarkers, Tumor - metabolism | Esophageal Neoplasms - mortality | Female | Liver Neoplasms - secondary | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Kaplan-Meier Estimate | Proportional Hazards Models | Animals | Mice, Nude | Liver Neoplasms - metabolism | Cell Line, Tumor | Carcinoma, Squamous Cell - secondary | Aged | Mice, Inbred BALB C | Activating Transcription Factor 4 - physiology | Cell Movement | Squamous cell carcinoma | Metastasis | Esophageal cancer | Transcription factors | Homeostasis | Oncology | Matrix metalloproteinase | Kinases | Stomach cancer | Metastases | Proteins | Surgery | Gastroenterology | Metalloproteinase | Lymphatic system | Cell survival | Activating transcription factor 4 | Breast cancer | Gene expression | Patients | Esophagus | Medical prognosis | Biomarkers | Hypoxia | Matrilysin
Journal Article
Journal Article
Digestive Diseases and Sciences, ISSN 0163-2116, 2/2019, Volume 64, Issue 2, pp. 532 - 543
Journal Article