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International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2010, Volume 78, Issue 1, pp. 127 - 135
Purpose To examine the risk of subsequent primary malignancies (SPMs) in women diagnosed with endometrial cancer. Methods and Materials The National Cancer... 
Radiology | Hematology, Oncology and Palliative Medicine | Radiation-induced malignancies | HNPCC | Endometrial cancer | Second primary malignancies | SURGERY | LYNCH-SYNDROME | NONPOLYPOSIS COLORECTAL-CANCER | 2ND PRIMARY CANCERS | RISK | CERVICAL-CANCER | RADIATION-THERAPY | UTERINE | ONCOLOGY | OVARIAN | CARCINOMA | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | United States - epidemiology | Colonic Neoplasms - genetics | Follow-Up Studies | Humans | Middle Aged | Bone Neoplasms - epidemiology | Pharyngeal Neoplasms - etiology | Esophageal Neoplasms - epidemiology | Incidence | Vaginal Neoplasms - genetics | Young Adult | Neoplasms, Radiation-Induced - genetics | Endometrial Neoplasms - genetics | Lung Neoplasms - etiology | Aged, 80 and over | Intestine, Small - radiation effects | Bone Neoplasms - genetics | Neoplasms, Second Primary - genetics | Mouth Neoplasms - genetics | SEER Program | Risk Assessment | Vaginal Neoplasms - etiology | Mouth Neoplasms - etiology | Neoplasms, Second Primary - epidemiology | Mouth Neoplasms - epidemiology | Pharyngeal Neoplasms - genetics | Vaginal Neoplasms - epidemiology | Adolescent | Pharyngeal Neoplasms - epidemiology | Endometrial Neoplasms - radiotherapy | Esophageal Neoplasms - etiology | Age Factors | Neoplasms, Radiation-Induced - etiology | Urinary Bladder Neoplasms - genetics | Adult | Female | Intestinal Neoplasms - genetics | Urinary Bladder Neoplasms - epidemiology | Colonic Neoplasms - etiology | Lung Neoplasms - genetics | Urinary Bladder Neoplasms - etiology | Colonic Neoplasms - epidemiology | Intestinal Neoplasms - etiology | Intestinal Neoplasms - epidemiology | Bone Neoplasms - etiology | Neoplasms, Second Primary - etiology | Lung Neoplasms - epidemiology | Esophageal Neoplasms - genetics | Aged | Neoplasms, Radiation-Induced - epidemiology | Genetic aspects | Research | Oncology, Experimental | Cancer | Index Medicus | LARGE INTESTINE | GASTROINTESTINAL TRACT | ORAL CAVITY | ESOPHAGUS | DIGESTIVE SYSTEM | LUNGS | MEDICINE | PHARYNX | THERAPY | RADIOLOGY AND NUCLEAR MEDICINE | BLADDER | RADIOTHERAPY | RADIOLOGY | SMALL INTESTINE | NEOPLASMS | UTERUS | INTESTINES | NUCLEAR MEDICINE | FEMALE GENITALS | ORGANS | URINARY TRACT | DISEASES | RESPIRATORY SYSTEM | BRACHYTHERAPY | BODY
Journal Article
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 01/2018, Volume 110, Issue 1, pp. 109 - 120
Background: Cancer-associated fibroblasts (CAFs) are tumor-promoting and correlate with poor survival in many cancers, which has led to their emergence as... 
CELL LUNG-CANCER | PHASE-II TRIAL | OXIDATIVE STRESS | ONCOLOGY | NAB-PACLITAXEL | ACTIVATION PROTEIN | DEPENDENT INVASION | PANCREATIC-CANCER | MONOCLONAL-ANTIBODY | DIFFERENTIATION | STROMAL CELLS | Lung Neoplasms - drug therapy | Up-Regulation | Pyrazoles - therapeutic use | Reactive Oxygen Species - metabolism | Cell Count | Humans | Middle Aged | Male | Oropharyngeal Neoplasms - pathology | Head and Neck Neoplasms - chemistry | RNA Interference | Cancer-Associated Fibroblasts - chemistry | Aged, 80 and over | NADPH Oxidases - genetics | Adenocarcinoma - chemistry | Esophageal Neoplasms - chemistry | Lung Neoplasms - chemistry | NADPH Oxidases - antagonists & inhibitors | Head and Neck Neoplasms - drug therapy | Carcinoma, Squamous Cell - chemistry | Survival Rate | Adenocarcinoma - drug therapy | Disease Progression | Phenotype | Carcinoma, Squamous Cell - drug therapy | Head and Neck Neoplasms - genetics | Mice | Neoplasm Transplantation | Carcinoma, Non-Small-Cell Lung - chemistry | Carcinoma, Squamous Cell - genetics | Colorectal Neoplasms - chemistry | Cell Transdifferentiation - genetics | Myofibroblasts - chemistry | Actins - analysis | Cell Transdifferentiation - drug effects | Adult | Female | Adenocarcinoma - genetics | Cancer-Associated Fibroblasts - physiology | Pyridines - therapeutic use | Lung Neoplasms - genetics | Myofibroblasts - pathology | Carcinoma, Non-Small-Cell Lung - genetics | NADPH Oxidases - analysis | Mouth Neoplasms - chemistry | NADPH Oxidase 4 | Cancer-Associated Fibroblasts - pathology | Animals | Esophageal Neoplasms - genetics | Mouth Neoplasms - pathology | Oropharyngeal Neoplasms - chemistry | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Colorectal Neoplasms - pathology | Esophageal Neoplasms - drug therapy
Journal Article
Nature medicine, ISSN 1546-170X, 2017, Volume 23, Issue 6, pp. 664 - 673
Neuroendocrine (NE) cancers are a diverse group of neoplasms typically diagnosed and treated on the basis of their site of origin... 
MOLECULAR CHARACTERIZATION | MEDICINE, RESEARCH & EXPERIMENTAL | PANCREATIC ENDOCRINE NEOPLASMS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GASTROINTESTINAL-TRACT | AUTOCHTHONOUS PROSTATE-CANCER | T-ANTIGEN | CELL BIOLOGY | CELL LUNG-CANCER | TRANSGENIC ADENOCARCINOMA | MOUSE MODEL | N-MYC | MUTATIONAL LANDSCAPE | Lung Neoplasms - drug therapy | Colonic Neoplasms - genetics | Carcinoma, Small Cell - genetics | Colonic Neoplasms - drug therapy | Epigenesis, Genetic | Humans | Male | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Tumor Suppressor Protein p53 - genetics | Ovarian Neoplasms - genetics | Neoplasms, Glandular and Epithelial - genetics | Prostatic Neoplasms - genetics | Urinary Bladder Neoplasms - genetics | Female | Cell Differentiation | Retinoblastoma Binding Proteins - genetics | Ovarian Neoplasms - drug therapy | Prostatic Neoplasms - drug therapy | Triazoles - therapeutic use | Lung Neoplasms - genetics | Antibodies, Monoclonal, Humanized - therapeutic use | Carcinoma, Neuroendocrine - drug therapy | Carcinoma, Small Cell - drug therapy | Carcinogenesis - genetics | Head and Neck Neoplasms - drug therapy | Carcinoma, Neuroendocrine - genetics | Uterine Cervical Neoplasms - drug therapy | Urinary Bladder Neoplasms - drug therapy | Uterine Cervical Neoplasms - genetics | Disease Progression | Azepines - therapeutic use | Neuroendocrine Tumors - genetics | Proto-Oncogene Proteins c-met - genetics | Cell Lineage | Cell Plasticity - genetics | Esophageal Neoplasms - genetics | Pyrimidines - therapeutic use | Neuroendocrine Tumors - drug therapy | Head and Neck Neoplasms - genetics | Proto-Oncogene Proteins c-myc - genetics | Ubiquitin-Protein Ligases - genetics | Benzodiazepines - therapeutic use | Esophageal Neoplasms - drug therapy | Proteins | Research | Health aspects | Carcinogenesis | Gastrin
Journal Article
Nature communications, ISSN 2041-1723, 2016, Volume 7, Issue 1, p. 11111
.... Most, but not all, good responders pass through genetic bottlenecks, a feature associated with higher mutation burden pre-treatment... 
HETEROGENEITY | EXOME | WHOLE-GENOME | COLORECTAL CANCERS | MICROSATELLITE INSTABILITY | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | ARCHITECTURE | MUTATIONS | PROGRESSION | REVEALS | Cyclin D2 - genetics | TATA-Binding Protein Associated Factors - metabolism | Cyclin D2 - metabolism | Adenocarcinoma - pathology | DNA, Neoplasm - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Histone Acetyltransferases - genetics | Neoplasm Recurrence, Local - drug therapy | Male | Antineoplastic Agents - therapeutic use | Transcription Factor TFIID - metabolism | Neoplasm Proteins - metabolism | Phosphoproteins - metabolism | Esophageal Neoplasms - pathology | Exome | Neoplasm Recurrence, Local - pathology | Transcription Factor TFIID - genetics | Ribonucleoprotein, U2 Small Nuclear - metabolism | Adenocarcinoma - metabolism | Esophageal Neoplasms - metabolism | Histone Acetyltransferases - metabolism | DNA Copy Number Variations - drug effects | Ribonucleoprotein, U2 Small Nuclear - genetics | Adult | Female | Neoadjuvant Therapy | Adenocarcinoma - genetics | Neoplasm Proteins - genetics | Neoplasm Recurrence, Local - metabolism | Clonal Evolution - drug effects | RNA Splicing Factors | Phosphoproteins - genetics | TATA-Binding Protein Associated Factors - genetics | Adenocarcinoma - drug therapy | Sequence Analysis, DNA | Genetic Heterogeneity | Mutation - drug effects | Esophageal Neoplasms - genetics | Neoplasm Recurrence, Local - genetics | Aged | DNA, Neoplasm - genetics | Esophageal Neoplasms - drug therapy
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 2014, Volume 106, Issue 5
Journal Article
Clinical cancer research, ISSN 1557-3265, 2006, Volume 12, Issue 19, pp. 5777 - 5785
Purpose: The DNA methylation paradox, manifested as derepression of cancer-testis antigens, and silencing of tumor suppressors during malignant transformation,... 
lung cancer | clinical trial | esophageal cancer | mesothelioma | epigenetics | 5-aza-2′-deoxycytidine | RESPONSES | HISTONE DEACETYLASE INHIBITOR | DNA METHYLATION | ONCOLOGY | IMMUNOTHERAPY | DEREPRESSION | EPIGENOME | INFUSION | ANTIGENS | INDUCTION | REFRACTORY SOLID TUMORS | Lung Neoplasms - drug therapy | Genes, p16 - physiology | Pleural Neoplasms - genetics | DNA Modification Methylases - antagonists & inhibitors | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Humans | Lung Neoplasms - metabolism | Middle Aged | Transcriptional Activation | Male | Neoplasm Proteins - metabolism | Pleural Neoplasms - drug therapy | Pleural Neoplasms - metabolism | Esophageal Neoplasms - metabolism | Antigens, Neoplasm - metabolism | Adult | Antimetabolites, Antineoplastic - pharmacology | Female | Membrane Proteins - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Lung Neoplasms - genetics | Antigens, Neoplasm - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Membrane Proteins - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Azacitidine - analogs & derivatives | Mesothelioma - genetics | Mesothelioma - drug therapy | Azacitidine - pharmacology | Maximum Tolerated Dose | Carcinoma, Squamous Cell - drug therapy | Mesothelioma - metabolism | Esophageal Neoplasms - genetics | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Esophageal Neoplasms - drug therapy
Journal Article
Journal of clinical oncology, ISSN 1527-7755, 2016, Volume 34, Issue 23, pp. 2721 - 2727
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2014, Volume 124, Issue 12, pp. 5145 - 5158
Patients with gastric and esophageal (GE) adenocarcinoma tumors in which the oncogene ERBB2 has been amplified are routinely treated with a combination of... 
BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | GROWTH-FACTOR RECEPTOR | LUNG-CANCER | GASTRIC-CANCER | TYROSINE KINASE | RESISTANCE | GENOMIC ALTERATIONS | COPY-NUMBER ALTERATION | HER2 | KINASE ACTIVATION | Receptor, Epidermal Growth Factor - genetics | Adenocarcinoma - pathology | Receptor, ErbB-2 - genetics | Humans | Receptor, ErbB-2 - metabolism | Drug Resistance, Neoplasm | Esophageal Neoplasms - diet therapy | Male | Stomach Neoplasms - pathology | Phosphatidylinositol 3-Kinases - metabolism | Antineoplastic Agents - administration & dosage | Esophageal Neoplasms - pathology | Receptor, Epidermal Growth Factor - metabolism | Antibodies, Monoclonal, Humanized - administration & dosage | Biomarkers, Tumor - metabolism | Female | Adenocarcinoma - genetics | Stomach Neoplasms - enzymology | Stomach Neoplasms - genetics | Adenocarcinoma - enzymology | Stomach Neoplasms - drug therapy | Adenocarcinoma - drug therapy | Gene Amplification - drug effects | Phosphatidylinositol 3-Kinases - genetics | Esophageal Neoplasms - enzymology | Esophageal Neoplasms - genetics | Biomarkers, Tumor - genetics | Gene Amplification - genetics | Trastuzumab | Physiological aspects | Genetic aspects | Research | Drug therapy | Esophageal cancer | Oncogenes | Studies | Cloning | Response rates | Breast cancer | Genomes | Kinases | Cancer therapies | Patients | Tumors
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2010, Volume 107, Issue 5, pp. 2177 - 2182
The stromal compartment is increasingly recognized to play a role in cancer. However, its role in the transition from preinvasive to invasive disease is... 
Adenocarcinoma | Datasets | Metaplasia | Medical research | Genes | Fibroblasts | Inflammation | Digestive system diseases | Signatures | Cancer | gene set enrichment | CELLS | intraepithelial neoplasia | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | BARRETTS-ESOPHAGUS | FIBROBLASTS | BREAST-CANCER | microarray | GASTROESOPHAGEAL-REFLUX | inflammation | CHRONIC-PANCREATITIS | Barrett's esophagus | DIFFERENTIATION | EXPRESSION | PROGRESSION | Inflammation - pathology | Adenocarcinoma - pathology | Prognosis | Stromal Cells - pathology | Humans | Digestive System Neoplasms - immunology | Esophageal Neoplasms - pathology | Barrett Esophagus - pathology | Neoplasm Invasiveness - pathology | Digestive System Neoplasms - pathology | Serine Endopeptidases - genetics | Trans-Activators - genetics | Precancerous Conditions - pathology | Adenocarcinoma - genetics | Nuclear Proteins - genetics | Cytokines - genetics | Digestive System Neoplasms - genetics | Receptors, Cytokine - genetics | Barrett Esophagus - genetics | Oncogenes | Membrane Proteins - genetics | Adenocarcinoma - immunology | Stromal Cells - immunology | Inflammation - immunology | DNA-Binding Proteins - genetics | Esophageal Neoplasms - immunology | Precancerous Conditions - genetics | Precancerous Conditions - immunology | Esophageal Neoplasms - genetics | Gelatinases - genetics | Barrett Esophagus - immunology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Inflammation - genetics | Neoplasm Invasiveness - genetics | Proto-Oncogene Proteins c-bcl-6 | Genetic aspects | Research | Biological Sciences | Barrett’s esophagus
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 151, Issue 5, pp. 860 - 869.e7
Background & Aims Some patients develop multiple squamous cell carcinomas (SCCs) in the upper aerodigestive tract, attributed to field cancerization; alcohol... 
Gastroenterology and Hepatology | Drinking Alcohol | Genetics Methods | Risk Factor | Carcinogenesis | ENDOSCOPIC MUCOSAL RESECTION | SUBMUCOSAL DISSECTION | CANCER | REGION | ALDEHYDE DEHYDROGENASE GENOTYPES | PATTERN | LUGOL-VOIDING LESIONS | MEN | GASTROENTEROLOGY & HEPATOLOGY | ASSOCIATION | Esophageal Neoplasms - etiology | Optical Imaging | Prospective Studies | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Male | Carcinoma, Squamous Cell - diagnostic imaging | Neoplasms, Multiple Primary - diagnostic imaging | Esophageal Neoplasms - epidemiology | Esophageal Neoplasms - pathology | Esophagoscopy | Incidence | Neoplasm Grading | Head and Neck Neoplasms - epidemiology | Aged, 80 and over | Adult | Esophagus - diagnostic imaging | Female | Esophageal Squamous Cell Carcinoma | Neoplasms, Multiple Primary - etiology | Carcinoma, Squamous Cell - epidemiology | Head and Neck Neoplasms - diagnostic imaging | Risk Factors | Kaplan-Meier Estimate | Proportional Hazards Models | Alcohol Abstinence | Neoplasms, Multiple Primary - epidemiology | Neoplasms, Multiple Primary - pathology | Alcohol Drinking - adverse effects | Head and Neck Neoplasms - pathology | Neoplasms, Second Primary - epidemiology | Neoplasms, Second Primary - etiology | Carcinoma, Squamous Cell - etiology | Esophagus - pathology | Head and Neck Neoplasms - etiology | Aged | Neoplasms, Second Primary - diagnostic imaging | Longitudinal Studies | Neoplasms, Second Primary - pathology | Esophageal Neoplasms - diagnostic imaging | Smoking Cessation | Medical colleges | Squamous cell carcinoma | Oncology, Experimental | Research | Endoscopy | Drinking of alcoholic beverages | Esophageal cancer | Risk factors | Cancer
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 149, Issue 7, pp. 1700 - 1715
... locations5,6 and is affected by environmental factors (alcohol consumption and tobacco use) and genetic factors (mutations in enzymes that metabolize alcohol). Only 15... 
Gastroenterology and Hepatology | Field Cancerization | Genetic Polymorphism | Squamous Differentiation | Aldehyde Dehydrogenase 2 | Acetaldehyde | DNA-ADDUCTS | ENDOSCOPIC MUCOSAL RESECTION | HUMAN-PAPILLOMAVIRUS | PHASE-III TRIAL | GROWTH-FACTOR RECEPTOR | TO-MESENCHYMAL TRANSITION | EPITHELIAL-CELLS | NEOADJUVANT CHEMOTHERAPY | CISPLATIN PLUS FLUOROURACIL | DEFINITIVE CHEMORADIOTHERAPY | GASTROENTEROLOGY & HEPATOLOGY | Smoking - adverse effects | Predictive Value of Tests | Life Style | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Carcinoma, Squamous Cell - virology | Humans | Esophageal Neoplasms - epidemiology | Esophageal Neoplasms - pathology | Cell Transformation, Neoplastic - genetics | Esophageal Neoplasms - metabolism | Biomarkers, Tumor - metabolism | Papillomavirus Infections - virology | Esophageal Neoplasms - therapy | Esophageal Squamous Cell Carcinoma | Genetic Predisposition to Disease | Signal Transduction | Carcinoma, Squamous Cell - epidemiology | Papillomavirus Infections - epidemiology | Risk Factors | Carcinoma, Squamous Cell - therapy | Treatment Outcome | Combined Modality Therapy | Alcohol Drinking - adverse effects | Cell Transformation, Neoplastic - metabolism | Esophageal Neoplasms - virology | Animals | Esophageal Neoplasms - genetics | Alcohol Drinking - epidemiology | Smoking - epidemiology | Biomarkers, Tumor - genetics | Cell Transformation, Neoplastic - pathology | Neoplasm Staging | Medical research | Enzymes | Squamous cell carcinoma | PET imaging | Genomics | Clinical trials | Cell differentiation | Genetic polymorphisms | Fanconi's anemia | Chemotherapy | Metabolites | Medicine, Experimental | Endoscopy | Tumor proteins | Health aspects | Esophageal cancer | Cancer | Index Medicus | Abridged Index Medicus
Journal Article