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Toxicology Letters, ISSN 0378-4274, 04/2014, Volume 226, Issue 1, pp. 81 - 89
Journal Article
Cell, ISSN 0092-8674, 2007, Volume 130, Issue 5, pp. 811 - 823
Estrogen prevents osteoporotic bone loss by attenuating bone resorption; however, the molecular basis for this is unknown. Here, we report a critical role for... 
HUMDISEASE | DEVBIO | SIGNALING | ANDROGEN RECEPTOR | APOPTOSIS | RESORPTION | DISEASES | BIOCHEMISTRY & MOLECULAR BIOLOGY | MASS | MECHANISMS | DEFICIENT MICE | OSTEOPETROSIS | EXPRESSION | OSTEOPOROSIS | CELL BIOLOGY | Fas Ligand Protein - metabolism | Male | fas Receptor - metabolism | Bone Remodeling - drug effects | Selective Estrogen Receptor Modulators - therapeutic use | Estrogen Receptor alpha - metabolism | Female | Integrases - metabolism | Mice, Inbred DBA | Cell Differentiation | Estradiol - metabolism | Osteoclasts - pathology | Bone Density | Cathepsin K | Ovariectomy | Signal Transduction | Bone Diseases, Metabolic - prevention & control | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Osteoclasts - metabolism | Estrogen Receptor alpha - deficiency | Mice, Knockout | Bone Diseases, Metabolic - metabolism | Estrogen Receptor alpha - drug effects | Phenotype | Animals | Estrogen Receptor alpha - genetics | Cathepsins - genetics | Cathepsins - metabolism | Tamoxifen - therapeutic use | Tamoxifen - pharmacology | Mice | Bone Diseases, Metabolic - physiopathology | Fas Ligand Protein - genetics | Integrases - genetics | Selective Estrogen Receptor Modulators - pharmacology | Osteoclasts - drug effects | Apoptosis | Cathepsins | Antigens, CD95 | Estradiol | Life Sciences | Estrogen Receptor alpha | Biochemistry, Molecular Biology | Tamoxifen | Bone Diseases, Metabolic | Integrases | Fas Ligand Protein | Selective Estrogen Receptor Modulators | Osteoclasts | Molecular biology | Bone Remodeling
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 10/2013, Volume 272, Issue 1, pp. 127 - 136
Journal Article
Toxicology Letters, ISSN 0378-4274, 08/2017, Volume 277, pp. 54 - 63
Phthalate plasticizers have been reported to exert adverse effects activation of the estrogen receptors ERα and ERβ and inhibition of the androgen receptor AR... 
Estrogen receptor | PPAR activation | Phthalate | Androgen receptor | BIOMONITORING DATA | PROLIFERATOR-ACTIVATED RECEPTORS | ESTROGENIC ACTIVITY | DUPLICATE DIET | CHILDREN | REPORTER | IN-VITRO | ESTERS | TOXICOLOGY | DUISBURG BIRTH COHORT | EXPOSURE | Transcription, Genetic - drug effects | PPAR alpha - drug effects | PPAR gamma - drug effects | Phthalic Acids - toxicity | Humans | PPAR gamma - metabolism | Estrogen Receptor beta - drug effects | Dose-Response Relationship, Drug | Estrogen Receptor beta - metabolism | Endocrine Disruptors - urine | Estrogen Receptor beta - genetics | Phthalic Acids - urine | Transfection | Basic Helix-Loop-Helix Transcription Factors - metabolism | HEK293 Cells | Receptors, Aryl Hydrocarbon - metabolism | Estrogen Receptor alpha - metabolism | Basic Helix-Loop-Helix Transcription Factors - drug effects | Genes, Reporter | PPAR gamma - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Metabolic Detoxication, Phase II | Receptors, Aryl Hydrocarbon - genetics | Glucuronides - urine | PPAR alpha - genetics | Receptors, Aryl Hydrocarbon - drug effects | Glucuronides - toxicity | Plasticizers - toxicity | Endocrine Disruptors - toxicity | Estrogen Receptor alpha - drug effects | Gene Expression Regulation - drug effects | Estrogen Receptor alpha - genetics | PPAR alpha - metabolism | Index Medicus
Journal Article
Neuroscience, ISSN 0306-4522, 2009, Volume 159, Issue 2, pp. 883 - 895
Journal Article
Toxicology, ISSN 0300-483X, 2009, Volume 262, Issue 3, pp. 184 - 191
Journal Article
Neuropharmacology, ISSN 0028-3908, 09/2012, Volume 63, Issue 3, pp. 349 - 361
Ginsenoside Rg1 (Rg1) acts as a neuroprotective agent against various insults, however, the underlying mechanism has not been fully elucidated yet. Here, we... 
β-Amyloid | Ginsenoside Rg1 | ERK1/2 phosphorylation | Receptor nuclear translocation | Protein tyrosine nitration | ACTIVATION | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | KINASE | ISCHEMIA | NEUROSCIENCES | SIGNALING PATHWAY | ENDOTHELIAL-CELLS | beta-Amyloid | PHARMACOLOGY & PHARMACY | NF-KAPPA-B | EXPRESSION | PARKINSONS-DISEASE | Immunohistochemistry | Neuroprotective Agents | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | NF-kappa B - metabolism | Ginsenosides - pharmacology | Cerebral Cortex - cytology | Cerebral Cortex - metabolism | Female | Cerebral Cortex - drug effects | Neurons - drug effects | Receptors, Glucocorticoid - drug effects | Real-Time Polymerase Chain Reaction | Cell Survival - drug effects | Amyloid beta-Peptides - toxicity | Reactive Nitrogen Species - metabolism | Cells, Cultured | Rats | Rats, Sprague-Dawley | Blotting, Western | Estrogen Receptor alpha - drug effects | Pregnancy | Amyloid beta-Peptides - antagonists & inhibitors | Animals | Mitogen-Activated Protein Kinases - physiology | Signal Transduction - drug effects | RNA, Small Interfering | Index Medicus | Tyrosine | Cell culture | Neuroprotection | Nitration | Phosphorylation | Glucocorticoids | Neurons | Cortex | Estrogen receptors | Extracellular signal-regulated kinase | siRNA | Neuroprotective agents | Nitric-oxide synthase | Nuclear transport | Presenilin 2 | Nitric oxide | ginsenosides | Injuries | Apoptosis
Journal Article