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Chemico-Biological Interactions, ISSN 0009-2797, 06/2015, Volume 234, pp. 309 - 319
Estrogens have important roles in the pathogenesis of endometrial cancer. They can have carcinogenic effects through stimulation of cell proliferation or... 
Aromatase pathway | Sulfatase pathway | Phase I and phase II estrogen metabolism | Aldo–keto reductase 1C3 (AKR1C3) | 17β-Hydroxysteroid dehydrogenases (17β-HSDs, HSD17B) | Catechol-O-methyl transferase (COMT) | 17b-Hydroxysteroid dehydrogenases | (17b-HSDs HSD17B)Aldoketo reductase 1C3 (AKR1C3) | 17 beta-Hydroxysteroid dehydrogenases (17 beta-HSDs, HSD17B) | DEHYDROGENASES | QUINONES | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROGESTERONE ACTION | 17-BETA-ESTRADIOL | CARCINOGENESIS | OVARIAN ENDOMETRIOSIS | Aldo-keto reductase 1C3 (AKR1C3) | ENZYMES | PHARMACOLOGY & PHARMACY | TOXICOLOGY | PHASE-I | CARCINOMA | PRE-RECEPTOR REGULATION | Sulfatases - genetics | Progesterone Reductase - metabolism | Humans | Endometrial Neoplasms - metabolism | Aromatase - metabolism | Catechol O-Methyltransferase - genetics | Hydroxyprostaglandin Dehydrogenases - metabolism | Androstenedione - metabolism | Catechol O-Methyltransferase - metabolism | Aromatase - genetics | Endometrial Neoplasms - genetics | Sulfatases - metabolism | Estrogens - genetics | 3-Hydroxysteroid Dehydrogenases - metabolism | Female | Sulfotransferases - metabolism | Estrogens - metabolism | Quinones - pharmacology | Glutathione S-Transferase pi - genetics | Sulfotransferases - genetics | Estrone - genetics | Quinone Reductases - metabolism | Androstenedione - genetics | Glutathione S-Transferase pi - metabolism | Estrogens - biosynthesis | RNA, Messenger - genetics | Transcriptome - genetics | Progesterone Reductase - genetics | Arylsulfotransferase - genetics | Hydroxyprostaglandin Dehydrogenases - genetics | Estrone - analogs & derivatives | Aldo-Keto Reductase Family 1 Member C3 | Estrone - metabolism | Cell Line, Tumor | Quinone Reductases - genetics | 3-Hydroxysteroid Dehydrogenases - genetics | Arylsulfotransferase - metabolism | Physiological aspects | Endometrial cancer | Sulfates | Genes | Estrogen | Steroids | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, pp. e0172244 - e0172244
Intrinsic or acquired drug resistance is a major impediment to the successful treatment of women with breast cancer using chemotherapy. We have observed that... 
CANCER-CELLS | DOXORUBICIN RESISTANCE | NEOADJUVANT CHEMOTHERAPY | MULTIDISCIPLINARY SCIENCES | ALDO-KETO REDUCTASES | GROWTH-FACTORS | ENDOCRINE THERAPY | MCF-7 CELLS | DRUG-RESISTANCE | RECEPTOR-ALPHA | ER-ALPHA | 3-Hydroxysteroid Dehydrogenases - biosynthesis | Cyclin D1 - metabolism | Gene Expression Regulation, Enzymologic - drug effects | Aldehyde Reductase - genetics | Humans | Breast Neoplasms - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Estrogens - genetics | Female | Gene Expression Regulation, Neoplastic - drug effects | Estrogens - metabolism | Gene Expression Regulation, Neoplastic - genetics | Aldehyde Reductase - biosynthesis | Hydroxyprostaglandin Dehydrogenases - biosynthesis | Signal Transduction - genetics | Breast Neoplasms - drug therapy | Hydroxyprostaglandin Dehydrogenases - genetics | Drug Resistance, Neoplasm - genetics | Breast Neoplasms - genetics | Cyclin D1 - genetics | Signal Transduction - drug effects | Aldo-Keto Reductase Family 1 Member C3 | Breast Neoplasms - pathology | Gene Expression Regulation, Enzymologic - genetics | 3-Hydroxysteroid Dehydrogenases - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | Care and treatment | Anthracyclines | Estrogen | Physiological aspects | Breast cancer | Research | Diagnosis | Drug resistance | Health aspects | Cell proliferation | Drugs | Health sciences | Phosphorylation | Growth rate | Transcription | Bcl-2 protein | Toxicity | Estrogens | Cytotoxicity | Hormones | Kinases | Cyclin D1 | Cancer therapies | Doxorubicin | Proteins | Signal transduction | Cell activation | Alterations | Cell growth | Synthesis | Pathways | Protein folding | Cell cycle | Epirubicin | Xenobiotics | Hydroxylation | Tumor cells | Detoxification | Endocrine therapy | siRNA | Metabolism | Gene expression | Aldo-keto reductase | Signaling | Chemotherapy | Sensitivity | Anthracycline | Breast | Estrone | Endocrinology | Apoptosis | Steroids | Tumors | Cancer | Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 05/2012, Volume 36, Issue 5, pp. 769 - 781
Journal Article
Genes and Development, ISSN 0890-9369, 11/2016, Volume 30, Issue 22, pp. 2551 - 2564
Journal Article
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 06/2004, Volume 96, Issue 12, pp. 936 - 945
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, pp. e97448 - e97448
G1-phase cell cycle defects, such as alterations in cyclin D1 or cyclin-dependent kinase (cdk) levels, are seen in most tumors. For example, increased cyclin... 
CDK6 | ETS2 TRANSCRIPTION FACTOR | DEHYDROGENASE ISOFORMS | MULTIDISCIPLINARY SCIENCES | ALDO-KETO REDUCTASES | MAST-CELLS | REVEALS ROLES | RECEPTOR | PROLIFERATION | STEROID-HORMONE | G PHASE | Cyclin D1 - metabolism | Cytochrome P-450 CYP1B1 - genetics | Cell Proliferation | Cyclin-Dependent Kinase 4 - genetics | Humans | Gene Expression Regulation, Neoplastic | Aromatase - metabolism | Cytochrome P-450 CYP1B1 - metabolism | Mammary Glands, Human - metabolism | Hydroxyprostaglandin Dehydrogenases - metabolism | Aromatase - genetics | 3-Hydroxysteroid Dehydrogenases - metabolism | Female | Hydroxysteroid Dehydrogenases - metabolism | G1 Phase - genetics | Estrogens - metabolism | Estradiol - metabolism | 20-Hydroxysteroid Dehydrogenases - genetics | Signal Transduction | Cyclin-Dependent Kinase 6 - genetics | Mammary Glands, Human - pathology | Cyclin-Dependent Kinase 6 - metabolism | Hydroxysteroid Dehydrogenases - genetics | 20-Hydroxysteroid Dehydrogenases - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Hydroxyprostaglandin Dehydrogenases - genetics | Cyclin D1 - genetics | Aldo-Keto Reductase Family 1 Member C3 | Estradiol Dehydrogenases - genetics | Estrone - metabolism | Estradiol Dehydrogenases - metabolism | Cell Line, Tumor | 3-Hydroxysteroid Dehydrogenases - genetics | Genes | Estrogen | Cytochrome P-450 | Physiological aspects | Phenols | Breast cancer | Small and medium sized companies | Phosphotransferases | Cytochrome | Cell proliferation | Cell culture | Pediatrics | Phosphorylation | Transcription | Estrogens | Homology | Biology | Kinases | Cyclin D1 | Experiments | Estradiol | Cyclin-dependent kinase 4 | Proteins | Cyclin-dependent kinase | Alterations | Cell growth | Xenoestrogens | Rodents | Aromatase | Cell cycle | Enzymes | Tumor cells | Cyclin-dependent kinases | Cytochrome P450 | Tumor cell lines | Metabolism | Gene expression | Aldo-keto reductase | Sex hormones | Gene amplification | Breast | Estrone | Tumors | Cancer | Index Medicus
Journal Article
Breast Cancer Research and Treatment, ISSN 0167-6806, 4/2014, Volume 144, Issue 2, pp. 249 - 261
Estrogen receptor (ER) is essential for estrogen-dependent growth, and its level of expression is considered a crucial determinant of response to endocrine... 
FOXA1 | Medicine & Public Health | Oncology | Breast cancer | GATA3 | Progesterone receptor | Ki67 | p53 | LIGAND-BINDING ASSAY | CELLS | PREDICTING RESPONSE | TAMOXIFEN | ENDOCRINE THERAPY | ALPHA | HORMONE | ONCOLOGY | LETROZOLE | IMMUNOHISTOCHEMISTRY | Estradiol - blood | GATA3 Transcription Factor - genetics | Postmenopause - blood | Prognosis | Tumor Suppressor Protein p53 - biosynthesis | Humans | Middle Aged | Premenopause - blood | Estrone - blood | GATA3 Transcription Factor - biosynthesis | Progesterone - blood | Receptors, Progesterone - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Hepatocyte Nuclear Factor 3-alpha - biosynthesis | Tumor Suppressor Proteins - genetics | Aged, 80 and over | Adult | Female | Trefoil Factor-1 | Testosterone - blood | Gene Expression | Receptors, Estrogen - genetics | Hepatocyte Nuclear Factor 3-alpha - genetics | RANK Ligand - genetics | Disease-Free Survival | Breast Neoplasms - genetics | Ki-67 Antigen - genetics | Breast Neoplasms - pathology | Breast Neoplasms - blood | Receptors, Estrogen - biosynthesis | Ki-67 Antigen - biosynthesis | Aged | Receptors, Progesterone - biosynthesis | Immunohistochemistry | Testosterone | Analysis | Menopause | Estrogen | Phenols | Genetic research | Postmenopausal women | Progesterone | Tumor proteins | Index Medicus
Journal Article