Neuron, ISSN 0896-6273, 2005, Volume 46, Issue 4, pp. 533 - 540
Postsynaptic AMPA receptor (AMPAR) trafficking mediates some forms of synaptic plasticity that are modulated by NMDA receptor (NMDAR) activation and...
NSF | ACTIVATION | EXOCYTOSIS | LONG-TERM DEPRESSION | REFLECTION FLUORESCENCE MICROSCOPY | NITRIC-OXIDE | TRAFFICKING | GLUR2 | SYNAPTIC PLASTICITY | NEUROSCIENCES | CULTURED HIPPOCAMPAL-NEURONS | Cerebellum | Immunoprecipitation | Embryo, Mammalian | Vesicular Transport Proteins - metabolism | Nerve Tissue Proteins - deficiency | Penicillamine - analogs & derivatives | Recombinant Fusion Proteins - metabolism | Adenylyl Imidodiphosphate - pharmacology | Drug Interactions | Epoxy Compounds - pharmacology | Transfection - methods | Nitric Oxide Synthase Type I | Time Factors | Aldehydes - pharmacology | Protein Binding - drug effects | Ethylmaleimide - pharmacology | Cysteine - metabolism | Neurons - metabolism | Nitric Oxide Donors - pharmacology | Neurons - drug effects | Receptors, AMPA - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Blotting, Western - methods | Penicillamine - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Gene Expression Regulation - physiology | Rats | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization - methods | Excitatory Amino Acid Antagonists - pharmacology | Enzyme Activation - drug effects | Hippocampus - cytology | N-Ethylmaleimide-Sensitive Proteins | Mice, Knockout | Gene Expression Regulation - drug effects | Diagnostic Imaging | Animals | Nitric Oxide Synthase - deficiency | Sulfhydryl Reagents - pharmacology | Mice | Mutagenesis - physiology | Dizocilpine Maleate - pharmacology | Nitric Oxide - metabolism | Methyl aspartate | Neurosciences | Neurons | Nitric oxide | Proteins | Microscopy | Experiments | Rodents
NSF | ACTIVATION | EXOCYTOSIS | LONG-TERM DEPRESSION | REFLECTION FLUORESCENCE MICROSCOPY | NITRIC-OXIDE | TRAFFICKING | GLUR2 | SYNAPTIC PLASTICITY | NEUROSCIENCES | CULTURED HIPPOCAMPAL-NEURONS | Cerebellum | Immunoprecipitation | Embryo, Mammalian | Vesicular Transport Proteins - metabolism | Nerve Tissue Proteins - deficiency | Penicillamine - analogs & derivatives | Recombinant Fusion Proteins - metabolism | Adenylyl Imidodiphosphate - pharmacology | Drug Interactions | Epoxy Compounds - pharmacology | Transfection - methods | Nitric Oxide Synthase Type I | Time Factors | Aldehydes - pharmacology | Protein Binding - drug effects | Ethylmaleimide - pharmacology | Cysteine - metabolism | Neurons - metabolism | Nitric Oxide Donors - pharmacology | Neurons - drug effects | Receptors, AMPA - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Blotting, Western - methods | Penicillamine - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Gene Expression Regulation - physiology | Rats | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization - methods | Excitatory Amino Acid Antagonists - pharmacology | Enzyme Activation - drug effects | Hippocampus - cytology | N-Ethylmaleimide-Sensitive Proteins | Mice, Knockout | Gene Expression Regulation - drug effects | Diagnostic Imaging | Animals | Nitric Oxide Synthase - deficiency | Sulfhydryl Reagents - pharmacology | Mice | Mutagenesis - physiology | Dizocilpine Maleate - pharmacology | Nitric Oxide - metabolism | Methyl aspartate | Neurosciences | Neurons | Nitric oxide | Proteins | Microscopy | Experiments | Rodents
Journal Article
Journal of Dental Research, ISSN 0022-0345, 3/2004, Volume 83, Issue 3, pp. 216 - 221
Incompletely infiltrated collagen fibrils in acid-etched dentin are susceptible to degradation. We hypothesize that degradation can occur in the absence of...
Matrix metalloproteinases (MMPs) | Dentin matrix | Gelatinase | ADHESIVE/DENTIN INTERFACE | HUMAN TEETH | ROOT DENTIN | RESIN-DENTIN BONDS | matrix metallo-proteinases (MMPs) | PROTEINASES | IDENTIFICATION | dentin matrix | MATRIX METALLOPROTEINASES | NANOLEAKAGE | DENTISTRY, ORAL SURGERY & MEDICINE | WATER STORAGE | IN-VIVO | gelatinase | Anti-Infective Agents, Local - pharmacology | Dentin - metabolism | Humans | Chlorhexidine - pharmacology | Benzamidines - pharmacology | Microscopy, Electron | Aminocaproic Acid - pharmacology | Collagen - metabolism | Dentin - drug effects | Time Factors | Cysteine Proteinase Inhibitors - pharmacology | Matrix Metalloproteinase Inhibitors | Phenylmethylsulfonyl Fluoride - pharmacology | Ethylmaleimide - pharmacology | Matrix Metalloproteinases - metabolism | Acid Etching, Dental | Saliva, Artificial - chemistry | Serine Proteinase Inhibitors - pharmacology | Aging - metabolism
Matrix metalloproteinases (MMPs) | Dentin matrix | Gelatinase | ADHESIVE/DENTIN INTERFACE | HUMAN TEETH | ROOT DENTIN | RESIN-DENTIN BONDS | matrix metallo-proteinases (MMPs) | PROTEINASES | IDENTIFICATION | dentin matrix | MATRIX METALLOPROTEINASES | NANOLEAKAGE | DENTISTRY, ORAL SURGERY & MEDICINE | WATER STORAGE | IN-VIVO | gelatinase | Anti-Infective Agents, Local - pharmacology | Dentin - metabolism | Humans | Chlorhexidine - pharmacology | Benzamidines - pharmacology | Microscopy, Electron | Aminocaproic Acid - pharmacology | Collagen - metabolism | Dentin - drug effects | Time Factors | Cysteine Proteinase Inhibitors - pharmacology | Matrix Metalloproteinase Inhibitors | Phenylmethylsulfonyl Fluoride - pharmacology | Ethylmaleimide - pharmacology | Matrix Metalloproteinases - metabolism | Acid Etching, Dental | Saliva, Artificial - chemistry | Serine Proteinase Inhibitors - pharmacology | Aging - metabolism
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 12/2001, Volume 89, Issue 12, pp. 1177 - 1183
Mitochondrial ATP-sensitive potassium (mitoKATP) channels have been suggested as triggers and end effectors in myocardial ischemic preconditioning. However,...
Heart | channel | Mitochondria | Superoxide | ATP-sensitive K | Channel reconstitution | CARDIAC & CARDIOVASCULAR SYSTEMS | OXYGEN RADICALS | MECHANISM | mitochondria | MEMBRANE | TRIGGERS | CARDIOMYOCYTES | heart | channel reconstitution | INHIBITION | PERIPHERAL VASCULAR DISEASE | AGENTS | superoxide | CARDIOPROTECTION | HEMATOLOGY | ATP-sensitive K+ channel | K+ CHANNELS | HEART-MITOCHONDRIA | Potassium - metabolism | Hydroxy Acids - pharmacology | Diazoxide - pharmacology | Guanosine Triphosphate - pharmacology | Mitochondria, Heart - metabolism | Myocardium - chemistry | Mitochondria, Heart - drug effects | Adenosine Triphosphate - pharmacology | Dose-Response Relationship, Drug | Potassium Channels - metabolism | Glyburide - pharmacology | Cattle | Myocardium - metabolism | Superoxides - metabolism | Xanthine - pharmacology | Ethylmaleimide - pharmacology | Benzamides - pharmacology | Membrane Potentials - drug effects | Xanthine Oxidase - metabolism | Mitochondria, Heart - chemistry | Decanoic Acids - pharmacology | Superoxides - pharmacology | Xanthine Oxidase - pharmacology | Potassium Channels - drug effects | Xanthine - metabolism | Animals | Potassium Channels - chemistry | Sulfhydryl Reagents - pharmacology | Lipid Bilayers - chemistry | Lipid Bilayers - metabolism | Subcellular Fractions - chemistry | Ion Channel Gating - drug effects
Heart | channel | Mitochondria | Superoxide | ATP-sensitive K | Channel reconstitution | CARDIAC & CARDIOVASCULAR SYSTEMS | OXYGEN RADICALS | MECHANISM | mitochondria | MEMBRANE | TRIGGERS | CARDIOMYOCYTES | heart | channel reconstitution | INHIBITION | PERIPHERAL VASCULAR DISEASE | AGENTS | superoxide | CARDIOPROTECTION | HEMATOLOGY | ATP-sensitive K+ channel | K+ CHANNELS | HEART-MITOCHONDRIA | Potassium - metabolism | Hydroxy Acids - pharmacology | Diazoxide - pharmacology | Guanosine Triphosphate - pharmacology | Mitochondria, Heart - metabolism | Myocardium - chemistry | Mitochondria, Heart - drug effects | Adenosine Triphosphate - pharmacology | Dose-Response Relationship, Drug | Potassium Channels - metabolism | Glyburide - pharmacology | Cattle | Myocardium - metabolism | Superoxides - metabolism | Xanthine - pharmacology | Ethylmaleimide - pharmacology | Benzamides - pharmacology | Membrane Potentials - drug effects | Xanthine Oxidase - metabolism | Mitochondria, Heart - chemistry | Decanoic Acids - pharmacology | Superoxides - pharmacology | Xanthine Oxidase - pharmacology | Potassium Channels - drug effects | Xanthine - metabolism | Animals | Potassium Channels - chemistry | Sulfhydryl Reagents - pharmacology | Lipid Bilayers - chemistry | Lipid Bilayers - metabolism | Subcellular Fractions - chemistry | Ion Channel Gating - drug effects
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 03/2012, Volume 302, Issue 5, pp. 821 - 833
Pattnaik BR, Hughes BA. Effects of KCNQ channel modulators on the M-type potassium current in primate retinal pigment epithelium. Am J Physiol Cell Physiol...
Patch clamp | Ion channels | CYSTEINE-MODIFYING REAGENT | LOCALIZATION | patch clamp | PHYSIOLOGY | ANTICONVULSANT RETIGABINE | VOLUME | ION | CULTURED-CELLS | ion channels | CELL BIOLOGY | PHARMACOLOGY | NEURONS | RECTIFYING K+ CURRENT | EXPRESSION | Potassium - metabolism | Retinal Pigment Epithelium - metabolism | Tetraethylammonium - pharmacology | Cells, Cultured | Macaca fascicularis | Macaca mulatta | Sulfonamides - pharmacology | Protein Subunits - metabolism | KCNQ Potassium Channels - antagonists & inhibitors | Patch-Clamp Techniques | Animals | Chromans - pharmacology | KCNQ Potassium Channels - metabolism | Ethylmaleimide - pharmacology | Indoles - pharmacology | Phenylenediamines - pharmacology | Neurons - metabolism | Protein Subunits - antagonists & inhibitors | Pyridines - pharmacology | Organometallic Compounds - pharmacology | Potassium Channel Blockers - pharmacology | Retinal Pigment Epithelium - cytology | Carbamates - pharmacology | Retinal Pigment Epithelium - drug effects | Physiological aspects | Primates | Potassium in the body | Retina | Potassium channels | Epithelium
Patch clamp | Ion channels | CYSTEINE-MODIFYING REAGENT | LOCALIZATION | patch clamp | PHYSIOLOGY | ANTICONVULSANT RETIGABINE | VOLUME | ION | CULTURED-CELLS | ion channels | CELL BIOLOGY | PHARMACOLOGY | NEURONS | RECTIFYING K+ CURRENT | EXPRESSION | Potassium - metabolism | Retinal Pigment Epithelium - metabolism | Tetraethylammonium - pharmacology | Cells, Cultured | Macaca fascicularis | Macaca mulatta | Sulfonamides - pharmacology | Protein Subunits - metabolism | KCNQ Potassium Channels - antagonists & inhibitors | Patch-Clamp Techniques | Animals | Chromans - pharmacology | KCNQ Potassium Channels - metabolism | Ethylmaleimide - pharmacology | Indoles - pharmacology | Phenylenediamines - pharmacology | Neurons - metabolism | Protein Subunits - antagonists & inhibitors | Pyridines - pharmacology | Organometallic Compounds - pharmacology | Potassium Channel Blockers - pharmacology | Retinal Pigment Epithelium - cytology | Carbamates - pharmacology | Retinal Pigment Epithelium - drug effects | Physiological aspects | Primates | Potassium in the body | Retina | Potassium channels | Epithelium
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2008, Volume 283, Issue 39, pp. 26312 - 26323
The mitochondrial permeability transition pore (MPTP) plays a key role in cell death, yet its molecular identity remains uncertain. Although knock-out studies...
DEPENDENT ANION CHANNEL | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | UBIQUINONE ANALOGS | REPERFUSION INJURY | INORGANIC-PHOSPHATE | RAT-HEART MITOCHONDRIA | ADP/ATP CARRIER | MATRIX VOLUME | ADENINE-NUCLEOTIDE TRANSLOCASE | CELL-DEATH | Mitochondria, Heart - metabolism | Cyclosporine - pharmacology | Mitochondria, Liver - metabolism | Protein Conformation - drug effects | Phosphate Transport Proteins - metabolism | Cyclophilins - metabolism | Mitochondrial ADP, ATP Translocases - antagonists & inhibitors | Mitochondrial ADP, ATP Translocases - metabolism | Cyclophilins - antagonists & inhibitors | Mitochondrial Proteins - metabolism | Protein Binding - drug effects | Dibenzocycloheptenes - pharmacology | Ethylmaleimide - pharmacology | Ion Transport - drug effects | Arsenicals - pharmacology | Mitochondrial Proteins - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Mitochondrial Membranes - metabolism | Benzoquinones - pharmacology | Animals | Cell Membrane Permeability - physiology | Cell Membrane Permeability - drug effects | Spiro Compounds - pharmacology | Protein Binding - physiology
DEPENDENT ANION CHANNEL | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | UBIQUINONE ANALOGS | REPERFUSION INJURY | INORGANIC-PHOSPHATE | RAT-HEART MITOCHONDRIA | ADP/ATP CARRIER | MATRIX VOLUME | ADENINE-NUCLEOTIDE TRANSLOCASE | CELL-DEATH | Mitochondria, Heart - metabolism | Cyclosporine - pharmacology | Mitochondria, Liver - metabolism | Protein Conformation - drug effects | Phosphate Transport Proteins - metabolism | Cyclophilins - metabolism | Mitochondrial ADP, ATP Translocases - antagonists & inhibitors | Mitochondrial ADP, ATP Translocases - metabolism | Cyclophilins - antagonists & inhibitors | Mitochondrial Proteins - metabolism | Protein Binding - drug effects | Dibenzocycloheptenes - pharmacology | Ethylmaleimide - pharmacology | Ion Transport - drug effects | Arsenicals - pharmacology | Mitochondrial Proteins - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Mitochondrial Membranes - metabolism | Benzoquinones - pharmacology | Animals | Cell Membrane Permeability - physiology | Cell Membrane Permeability - drug effects | Spiro Compounds - pharmacology | Protein Binding - physiology
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/1994, Volume 14, Issue 5, pp. 2924 - 2932
Formation of reactive oxygen species and disfunction of the excitatory amino acid (EAA) system are thought to be key events in the development of neuronal...
antioxidants | disulfide-reducing agents | glutamate uptake | protein thiol groups | astrocytes | excitotoxicity | redox modulation | oxygen free radicals | TISSUE-INJURY | NMDA RECEPTOR | ANTIOXIDANTS | PROTEIN THIOL GROUPS | LIPID-PEROXIDATION | INVITRO ANOXIA | NEURONAL INJURY | BRAIN SYNAPTOSOMES | GLUTAMATE UPTAKE | ASTROCYTES | NEUROSCIENCES | EXCITOTOXICITY | AMINO-ACIDS | CENTRAL-NERVOUS-SYSTEM | OXYGEN FREE RADICALS | RETINAL GLIAL-CELLS | DISULFIDE-REDUCING AGENTS | REDOX MODULATION | METHYL-D-ASPARTATE | Catalase - pharmacology | 2-Amino-5-phosphonovalerate - pharmacology | Free Radical Scavengers | Glutamates - metabolism | Xanthines - pharmacology | Cerebral Cortex - metabolism | Quinoxalines - pharmacology | Glutathione - pharmacology | Time Factors | Dithionitrobenzoic Acid - pharmacology | L-Lactate Dehydrogenase - analysis | Ethylmaleimide - pharmacology | Biological Transport - drug effects | Dithiothreitol - pharmacology | Animals, Newborn | 6-Cyano-7-nitroquinoxaline-2,3-dione | Astrocytes - drug effects | Superoxide Dismutase - pharmacology | Xanthine Oxidase - pharmacology | Cells, Cultured | Hydrogen Peroxide - pharmacology | Rats | Antioxidants - pharmacology | Xanthine | Glutamic Acid | Animals | p-Chloromercuribenzoic Acid | Free Radicals - pharmacology | Kinetics | Chloromercuribenzoates - pharmacology | Astrocytes - metabolism
antioxidants | disulfide-reducing agents | glutamate uptake | protein thiol groups | astrocytes | excitotoxicity | redox modulation | oxygen free radicals | TISSUE-INJURY | NMDA RECEPTOR | ANTIOXIDANTS | PROTEIN THIOL GROUPS | LIPID-PEROXIDATION | INVITRO ANOXIA | NEURONAL INJURY | BRAIN SYNAPTOSOMES | GLUTAMATE UPTAKE | ASTROCYTES | NEUROSCIENCES | EXCITOTOXICITY | AMINO-ACIDS | CENTRAL-NERVOUS-SYSTEM | OXYGEN FREE RADICALS | RETINAL GLIAL-CELLS | DISULFIDE-REDUCING AGENTS | REDOX MODULATION | METHYL-D-ASPARTATE | Catalase - pharmacology | 2-Amino-5-phosphonovalerate - pharmacology | Free Radical Scavengers | Glutamates - metabolism | Xanthines - pharmacology | Cerebral Cortex - metabolism | Quinoxalines - pharmacology | Glutathione - pharmacology | Time Factors | Dithionitrobenzoic Acid - pharmacology | L-Lactate Dehydrogenase - analysis | Ethylmaleimide - pharmacology | Biological Transport - drug effects | Dithiothreitol - pharmacology | Animals, Newborn | 6-Cyano-7-nitroquinoxaline-2,3-dione | Astrocytes - drug effects | Superoxide Dismutase - pharmacology | Xanthine Oxidase - pharmacology | Cells, Cultured | Hydrogen Peroxide - pharmacology | Rats | Antioxidants - pharmacology | Xanthine | Glutamic Acid | Animals | p-Chloromercuribenzoic Acid | Free Radicals - pharmacology | Kinetics | Chloromercuribenzoates - pharmacology | Astrocytes - metabolism
Journal Article
Neuropharmacology, ISSN 0028-3908, 10/2017, Volume 125, pp. 156 - 165
The role of nitric oxide (NO) in nociceptive transmission at the spinal cord level remains uncertain. Increased activity of spinal -methyl- -aspartate (NMDA)...
Signal transduction | Dorsal horn neurons | Ion channel | Synaptic transmission | Synaptic plasticity | Neuropathic pain | DORSAL-HORN NEURONS | CHLORIDE HOMEOSTASIS | NEUROSCIENCES | MECHANICAL TRANSMISSION | SUBSTANTIA-GELATINOSA NEURONS | S-NITROSYLATION | REDOX MODULATORY SITE | L-ARGININE | PHARMACOLOGY & PHARMACY | MOLECULAR-BASIS | TONIC CHOLINERGIC INHIBITION | Cyclic GMP - pharmacology | Posterior Horn Cells - drug effects | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Posterior Horn Cells - metabolism | Soluble Guanylyl Cyclase - metabolism | Nitric Oxide - pharmacology | Analgesics, Non-Narcotic - pharmacology | Neuralgia - metabolism | Receptors, N-Methyl-D-Aspartate - metabolism | Male | Touch | Cyclic GMP - analogs & derivatives | Nitric Oxide Synthase Type I - antagonists & inhibitors | Spinal Nerves - drug effects | Ethylmaleimide - pharmacology | Spinal Nerves - injuries | Disease Models, Animal | Hyperalgesia - metabolism | Soluble Guanylyl Cyclase - antagonists & inhibitors | Tissue Culture Techniques | Spinal Nerves - metabolism | Excitatory Amino Acid Antagonists - pharmacology | Hot Temperature | Rats, Sprague-Dawley | Neuralgia - drug therapy | Animals | Hyperalgesia - drug therapy | Arginine - pharmacology | Nitric Oxide Synthase Type I - metabolism | Central Nervous System Agents - pharmacology | S-Nitroso-N-Acetylpenicillamine - pharmacology | Allergy | Amino acids | Allergic reaction | Nitric oxide | Methyl aspartate | Care and treatment | Neurosciences | Pain | Arginine | Neurons | Cellular signal transduction | Chronic pain | neuropathic pain | synaptic transmission | synaptic plasticity | signal transduction | ion channel | dorsal horn neurons
Signal transduction | Dorsal horn neurons | Ion channel | Synaptic transmission | Synaptic plasticity | Neuropathic pain | DORSAL-HORN NEURONS | CHLORIDE HOMEOSTASIS | NEUROSCIENCES | MECHANICAL TRANSMISSION | SUBSTANTIA-GELATINOSA NEURONS | S-NITROSYLATION | REDOX MODULATORY SITE | L-ARGININE | PHARMACOLOGY & PHARMACY | MOLECULAR-BASIS | TONIC CHOLINERGIC INHIBITION | Cyclic GMP - pharmacology | Posterior Horn Cells - drug effects | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Posterior Horn Cells - metabolism | Soluble Guanylyl Cyclase - metabolism | Nitric Oxide - pharmacology | Analgesics, Non-Narcotic - pharmacology | Neuralgia - metabolism | Receptors, N-Methyl-D-Aspartate - metabolism | Male | Touch | Cyclic GMP - analogs & derivatives | Nitric Oxide Synthase Type I - antagonists & inhibitors | Spinal Nerves - drug effects | Ethylmaleimide - pharmacology | Spinal Nerves - injuries | Disease Models, Animal | Hyperalgesia - metabolism | Soluble Guanylyl Cyclase - antagonists & inhibitors | Tissue Culture Techniques | Spinal Nerves - metabolism | Excitatory Amino Acid Antagonists - pharmacology | Hot Temperature | Rats, Sprague-Dawley | Neuralgia - drug therapy | Animals | Hyperalgesia - drug therapy | Arginine - pharmacology | Nitric Oxide Synthase Type I - metabolism | Central Nervous System Agents - pharmacology | S-Nitroso-N-Acetylpenicillamine - pharmacology | Allergy | Amino acids | Allergic reaction | Nitric oxide | Methyl aspartate | Care and treatment | Neurosciences | Pain | Arginine | Neurons | Cellular signal transduction | Chronic pain | neuropathic pain | synaptic transmission | synaptic plasticity | signal transduction | ion channel | dorsal horn neurons
Journal Article
8.
Full Text
Reducing and Oxidizing Agents Sensitize Heat-Activated Vanilloid Receptor (TRPV1) Current
Molecular Pharmacology, ISSN 0026-895X, 07/2006, Volume 70, Issue 1, pp. 383 - 394
We have previously reported that the reducing agent dithiothreitol (DTT) strongly increases thermally induced activity of the transient receptor potential...
CAPSAICIN RECEPTOR | DISULFIDE BONDS | THERMAL HYPERALGESIA | RESINIFERATOXIN BINDING | RAT PERIPHERAL NOCICEPTORS | DEPENDENT PROTEIN-KINASE | PHARMACOLOGY & PHARMACY | CHANNELS | REDOX MODULATION | CYSTEINE RESIDUES | DIRECT PHOSPHORYLATION | Membrane Potentials - drug effects | Cell Line | Oxidants - pharmacology | TRPV Cation Channels - physiology | Capsaicin - pharmacology | Humans | Hydrogen Peroxide - pharmacology | Mutant Proteins - genetics | Rats | Diamide - pharmacology | Hot Temperature | Mutant Proteins - physiology | Mutation - genetics | Mutation, Missense - genetics | Reducing Agents - pharmacology | TRPV Cation Channels - genetics | Dose-Response Relationship, Drug | Patch-Clamp Techniques | Animals | Transfection | Dithionitrobenzoic Acid - pharmacology | Ethylmaleimide - pharmacology | Sulfhydryl Reagents - pharmacology | Dithiothreitol - pharmacology
CAPSAICIN RECEPTOR | DISULFIDE BONDS | THERMAL HYPERALGESIA | RESINIFERATOXIN BINDING | RAT PERIPHERAL NOCICEPTORS | DEPENDENT PROTEIN-KINASE | PHARMACOLOGY & PHARMACY | CHANNELS | REDOX MODULATION | CYSTEINE RESIDUES | DIRECT PHOSPHORYLATION | Membrane Potentials - drug effects | Cell Line | Oxidants - pharmacology | TRPV Cation Channels - physiology | Capsaicin - pharmacology | Humans | Hydrogen Peroxide - pharmacology | Mutant Proteins - genetics | Rats | Diamide - pharmacology | Hot Temperature | Mutant Proteins - physiology | Mutation - genetics | Mutation, Missense - genetics | Reducing Agents - pharmacology | TRPV Cation Channels - genetics | Dose-Response Relationship, Drug | Patch-Clamp Techniques | Animals | Transfection | Dithionitrobenzoic Acid - pharmacology | Ethylmaleimide - pharmacology | Sulfhydryl Reagents - pharmacology | Dithiothreitol - pharmacology
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 03/2012, Volume 679, Issue 1-3, pp. 40 - 50
We previously reported that both nitric oxide (NO) generated from NO synthase by bombesin and NO generated from SIN-1 (NO donor) activate the brain...
S-Nitrosylation | Brain | Adrenomedullary outflow | Bombesin | Cyclooxygenase-1 | Nitric oxide | POTASSIUM CHANNELS | NITRIC-OXIDE SYNTHASE | SYMPATHETIC PREGANGLIONIC NEURONS | THORACIC SPINAL-CORD | DORSAL VAGAL COMPLEX | PARAVENTRICULAR NUCLEUS | PLASMA NORADRENALINE | PHARMACOLOGY & PHARMACY | GASTRIC-ACID SECRETION | CENTRAL-NERVOUS-SYSTEM | THROMBOXANE A | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - administration & dosage | Cysteine - analogs & derivatives | Oxadiazoles - administration & dosage | Rats, Wistar | Injections, Intraventricular | Imidazoles - administration & dosage | Male | Molsidomine - analogs & derivatives | Molsidomine - administration & dosage | S-Nitrosothiols - metabolism | Bombesin - administration & dosage | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Bombesin - pharmacology | Oxadiazoles - pharmacology | Dithiothreitol - administration & dosage | Sulfhydryl Reagents - administration & dosage | Ethylmaleimide - pharmacology | Adrenal Medulla - drug effects | Cysteine - metabolism | Nitric Oxide Donors - pharmacology | Dithiothreitol - pharmacology | Quinoxalines - administration & dosage | Rats | Imidazoles - pharmacology | Catecholamines - blood | Benzoates - administration & dosage | Brain - drug effects | Nitric Oxide Donors - administration & dosage | Paraventricular Hypothalamic Nucleus - drug effects | Adrenal Medulla - secretion | Animals | Bombesin - antagonists & inhibitors | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Molsidomine - pharmacology | Paraventricular Hypothalamic Nucleus - metabolism | Benzoates - pharmacology | Molsidomine - antagonists & inhibitors | Sulfhydryl Reagents - pharmacology | Ethylmaleimide - administration & dosage | Nitrogen oxide | Cysteine | Catecholamines | Neuropeptides | Cells | Thromboxanes | Thiols | Neurons | Adrenal medulla | Thromboxane A2 | Hypothalamus | Paraventricular nucleus | Guanylate cyclase | Nitric-oxide synthase | Dithiothreitol | N-Ethylmaleimide
S-Nitrosylation | Brain | Adrenomedullary outflow | Bombesin | Cyclooxygenase-1 | Nitric oxide | POTASSIUM CHANNELS | NITRIC-OXIDE SYNTHASE | SYMPATHETIC PREGANGLIONIC NEURONS | THORACIC SPINAL-CORD | DORSAL VAGAL COMPLEX | PARAVENTRICULAR NUCLEUS | PLASMA NORADRENALINE | PHARMACOLOGY & PHARMACY | GASTRIC-ACID SECRETION | CENTRAL-NERVOUS-SYSTEM | THROMBOXANE A | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - administration & dosage | Cysteine - analogs & derivatives | Oxadiazoles - administration & dosage | Rats, Wistar | Injections, Intraventricular | Imidazoles - administration & dosage | Male | Molsidomine - analogs & derivatives | Molsidomine - administration & dosage | S-Nitrosothiols - metabolism | Bombesin - administration & dosage | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Bombesin - pharmacology | Oxadiazoles - pharmacology | Dithiothreitol - administration & dosage | Sulfhydryl Reagents - administration & dosage | Ethylmaleimide - pharmacology | Adrenal Medulla - drug effects | Cysteine - metabolism | Nitric Oxide Donors - pharmacology | Dithiothreitol - pharmacology | Quinoxalines - administration & dosage | Rats | Imidazoles - pharmacology | Catecholamines - blood | Benzoates - administration & dosage | Brain - drug effects | Nitric Oxide Donors - administration & dosage | Paraventricular Hypothalamic Nucleus - drug effects | Adrenal Medulla - secretion | Animals | Bombesin - antagonists & inhibitors | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Molsidomine - pharmacology | Paraventricular Hypothalamic Nucleus - metabolism | Benzoates - pharmacology | Molsidomine - antagonists & inhibitors | Sulfhydryl Reagents - pharmacology | Ethylmaleimide - administration & dosage | Nitrogen oxide | Cysteine | Catecholamines | Neuropeptides | Cells | Thromboxanes | Thiols | Neurons | Adrenal medulla | Thromboxane A2 | Hypothalamus | Paraventricular nucleus | Guanylate cyclase | Nitric-oxide synthase | Dithiothreitol | N-Ethylmaleimide
Journal Article
Archives of Oral Biology, ISSN 0003-9969, 2010, Volume 55, Issue 8, pp. 545 - 549
Abstract Objective The purpose of the present study was to characterize the collagenolytic activity in a sonicated extract of Tannerella forsythia and to...
Advanced Basic Science | Dentistry | Tannerella forsythia | Extracellular matrix | Cysteine | proMMP-9 | Type I collagen | proMMP-2 | CELLS | PORPHYROMONAS-GINGIVALIS | ARG-GINGIPAIN | BACTERIAL | GENE | DENTISTRY, ORAL SURGERY & MEDICINE | INFECTED ROOT CANALS | COLLAGENASE ACTIVITY | PROTEASE | Leucine - pharmacology | Humans | Protease Inhibitors - pharmacology | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 2 - drug effects | Cysteine - pharmacology | Edetic Acid - pharmacology | Gelatinases - metabolism | Phenylmethylsulfonyl Fluoride - pharmacology | Ethylmaleimide - pharmacology | Leucine - analogs & derivatives | Tosylphenylalanyl Chloromethyl Ketone - pharmacology | Iodoacetic Acid - pharmacology | Serine Proteinase Inhibitors - pharmacology | Cathepsins - antagonists & inhibitors | Collagen Type I - metabolism | Electrophoresis, Polyacrylamide Gel | Enzyme Inhibitors - pharmacology | Iodoacetamide - pharmacology | Chelating Agents - pharmacology | Tosyllysine Chloromethyl Ketone - pharmacology | Bacteroides - metabolism | Calcium Chloride - pharmacology | Leupeptins - pharmacology | Enzyme Precursors - metabolism | Cysteine Proteinase Inhibitors - pharmacology | Cell Line, Tumor | Matrix Metalloproteinase 9 - drug effects | Enzyme Activation
Advanced Basic Science | Dentistry | Tannerella forsythia | Extracellular matrix | Cysteine | proMMP-9 | Type I collagen | proMMP-2 | CELLS | PORPHYROMONAS-GINGIVALIS | ARG-GINGIPAIN | BACTERIAL | GENE | DENTISTRY, ORAL SURGERY & MEDICINE | INFECTED ROOT CANALS | COLLAGENASE ACTIVITY | PROTEASE | Leucine - pharmacology | Humans | Protease Inhibitors - pharmacology | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 2 - drug effects | Cysteine - pharmacology | Edetic Acid - pharmacology | Gelatinases - metabolism | Phenylmethylsulfonyl Fluoride - pharmacology | Ethylmaleimide - pharmacology | Leucine - analogs & derivatives | Tosylphenylalanyl Chloromethyl Ketone - pharmacology | Iodoacetic Acid - pharmacology | Serine Proteinase Inhibitors - pharmacology | Cathepsins - antagonists & inhibitors | Collagen Type I - metabolism | Electrophoresis, Polyacrylamide Gel | Enzyme Inhibitors - pharmacology | Iodoacetamide - pharmacology | Chelating Agents - pharmacology | Tosyllysine Chloromethyl Ketone - pharmacology | Bacteroides - metabolism | Calcium Chloride - pharmacology | Leupeptins - pharmacology | Enzyme Precursors - metabolism | Cysteine Proteinase Inhibitors - pharmacology | Cell Line, Tumor | Matrix Metalloproteinase 9 - drug effects | Enzyme Activation
Journal Article
Journal of General Physiology, ISSN 0022-1295, 09/2005, Volume 126, Issue 3, pp. 243 - 262
We have further tested the hypothesis that receptor-mediated modulation of KCNQ channels involves depletion of phosphatidylinositol 4,5-bisphosphate (PIP2) by...
CYSTEINE-MODIFYING REAGENT | PROTEIN-KINASE-C | PHYSIOLOGY | INOSITOL 1,4,5-TRISPHOSPHATE | PHOSPHATIDYLINOSITOL 4,5-BISPHOSPHATE | MUSCARINIC MODULATION | SYMPATHETIC NEURONS | RECEPTOR-MEDIATED INHIBITION | ION CHANNELS | NEUROBLASTOMA-CELLS | K+ CHANNELS | Oxotremorine - pharmacology | Protein Kinase C - genetics | Muscarinic Agonists - pharmacology | Humans | Alkylation | KCNQ Potassium Channels | Phosphatidylinositol 4,5-Diphosphate - metabolism | Phosphatidylinositol Diacylglycerol-Lyase - metabolism | Transfection | Potassium Channels, Voltage-Gated - metabolism | Receptor, Muscarinic M1 - drug effects | Time Factors | Oxotremorine - analogs & derivatives | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Potassium Channels, Voltage-Gated - drug effects | Ethylmaleimide - pharmacology | Inositol Phosphates - metabolism | CHO Cells | KCNQ1 Potassium Channel | Membrane Potentials - drug effects | Phosphatidylinositol Diacylglycerol-Lyase - antagonists & inhibitors | Cricetinae | Calcium - pharmacology | Potassium Channels, Voltage-Gated - antagonists & inhibitors | Enzyme Activation - drug effects | Sulfonamides - pharmacology | Phospholipid Ethers - pharmacology | Receptor, Muscarinic M1 - genetics | Animals | Receptor, Muscarinic M1 - metabolism
CYSTEINE-MODIFYING REAGENT | PROTEIN-KINASE-C | PHYSIOLOGY | INOSITOL 1,4,5-TRISPHOSPHATE | PHOSPHATIDYLINOSITOL 4,5-BISPHOSPHATE | MUSCARINIC MODULATION | SYMPATHETIC NEURONS | RECEPTOR-MEDIATED INHIBITION | ION CHANNELS | NEUROBLASTOMA-CELLS | K+ CHANNELS | Oxotremorine - pharmacology | Protein Kinase C - genetics | Muscarinic Agonists - pharmacology | Humans | Alkylation | KCNQ Potassium Channels | Phosphatidylinositol 4,5-Diphosphate - metabolism | Phosphatidylinositol Diacylglycerol-Lyase - metabolism | Transfection | Potassium Channels, Voltage-Gated - metabolism | Receptor, Muscarinic M1 - drug effects | Time Factors | Oxotremorine - analogs & derivatives | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Potassium Channels, Voltage-Gated - drug effects | Ethylmaleimide - pharmacology | Inositol Phosphates - metabolism | CHO Cells | KCNQ1 Potassium Channel | Membrane Potentials - drug effects | Phosphatidylinositol Diacylglycerol-Lyase - antagonists & inhibitors | Cricetinae | Calcium - pharmacology | Potassium Channels, Voltage-Gated - antagonists & inhibitors | Enzyme Activation - drug effects | Sulfonamides - pharmacology | Phospholipid Ethers - pharmacology | Receptor, Muscarinic M1 - genetics | Animals | Receptor, Muscarinic M1 - metabolism
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2000, Volume 20, Issue 5, pp. 1710 - 1721
Channels from KCNQ2 and KCNQ3 genes have been suggested to underlie the neuronal M-type K(+) current. The M current is modulated by muscarinic agonists via...
M current | channel | Patch clamp | G-protein | Muscarinic receptor | Calcium | INTRACELLULAR CA2 | RECEPTOR SUBTYPES | CA2+ CHANNELS | CALCIUM CHANNELS | RAT SYMPATHETIC NEURONS | BETA-GAMMA-SUBUNITS | IDIOPATHIC EPILEPSY | N-ETHYLMALEIMIDE | NEUROSCIENCES | PROTEIN-KINASES | POTASSIUM CHANNEL | Oxotremorine - pharmacology | Potassium Channels, Voltage-Gated | Tetraethylammonium - pharmacology | Calcium - metabolism | Muscarinic Agonists - pharmacology | Calcium Signaling - physiology | Humans | Cytoplasm - metabolism | Male | Neurons - cytology | Receptors, Muscarinic - metabolism | Potassium Channels - metabolism | KCNQ3 Potassium Channel | Muscarinic Antagonists - pharmacology | Gene Expression - physiology | KCNQ2 Potassium Channel | Cloning, Molecular | Egtazic Acid - analogs & derivatives | Neurons - physiology | Ethylmaleimide - pharmacology | Receptors, Muscarinic - genetics | Neurons - chemistry | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Atropine - pharmacology | Chelating Agents - pharmacology | Superior Cervical Ganglion - cytology | Rats, Sprague-Dawley | Anthracenes - pharmacology | Potassium Channels - genetics | Patch-Clamp Techniques | Animals | Egtazic Acid - pharmacology | Calcium Signaling - drug effects | Thapsigargin - pharmacology | Second Messenger Systems - physiology | Staurosporine - pharmacology | GTP-Binding Proteins - metabolism | K+ channel | ARTICLE | patch clamp | calcium | muscarinic receptor
M current | channel | Patch clamp | G-protein | Muscarinic receptor | Calcium | INTRACELLULAR CA2 | RECEPTOR SUBTYPES | CA2+ CHANNELS | CALCIUM CHANNELS | RAT SYMPATHETIC NEURONS | BETA-GAMMA-SUBUNITS | IDIOPATHIC EPILEPSY | N-ETHYLMALEIMIDE | NEUROSCIENCES | PROTEIN-KINASES | POTASSIUM CHANNEL | Oxotremorine - pharmacology | Potassium Channels, Voltage-Gated | Tetraethylammonium - pharmacology | Calcium - metabolism | Muscarinic Agonists - pharmacology | Calcium Signaling - physiology | Humans | Cytoplasm - metabolism | Male | Neurons - cytology | Receptors, Muscarinic - metabolism | Potassium Channels - metabolism | KCNQ3 Potassium Channel | Muscarinic Antagonists - pharmacology | Gene Expression - physiology | KCNQ2 Potassium Channel | Cloning, Molecular | Egtazic Acid - analogs & derivatives | Neurons - physiology | Ethylmaleimide - pharmacology | Receptors, Muscarinic - genetics | Neurons - chemistry | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Atropine - pharmacology | Chelating Agents - pharmacology | Superior Cervical Ganglion - cytology | Rats, Sprague-Dawley | Anthracenes - pharmacology | Potassium Channels - genetics | Patch-Clamp Techniques | Animals | Egtazic Acid - pharmacology | Calcium Signaling - drug effects | Thapsigargin - pharmacology | Second Messenger Systems - physiology | Staurosporine - pharmacology | GTP-Binding Proteins - metabolism | K+ channel | ARTICLE | patch clamp | calcium | muscarinic receptor
Journal Article
BMC Neuroscience, ISSN 1471-2202, 05/2012, Volume 13, Issue 1, pp. 53 - 53
Background: ATP is an extracellular signaling molecule with many ascribed functions in sensory systems, including the olfactory epithelium. The mechanism(s) by...
SYSTEM | CELLS | IN-VITRO | NEUROTRANSMISSION | PURINERGIC RECEPTOR ACTIVATION | ACETYLCHOLINE-RELEASE | NEURONS | NEUROMUSCULAR-JUNCTION | CONNEXIN HEMICHANNELS | EXPRESSION | NEUROSCIENCES | Gadolinium - pharmacology | Humans | Qb-SNARE Proteins - metabolism | Molecular Sequence Data | Phosphopyruvate Hydratase - metabolism | Exocytosis - drug effects | Adenosine Triphosphate - pharmacology | Drug Interactions | Transfection | Qc-SNARE Proteins - metabolism | Time Factors | Adenosine Triphosphate - metabolism | Quinacrine - metabolism | Ethylmaleimide - pharmacology | Sensory Receptor Cells - metabolism | Bromodeoxyuridine - metabolism | Pyridoxal Phosphate - analogs & derivatives | Organ Culture Techniques | Animals, Newborn | Receptors, Purinergic P2X2 - genetics | Calcium - pharmacology | Cytidine Triphosphate - pharmacology | Pyridoxal Phosphate - pharmacology | Uridine Triphosphate - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Adenosine Triphosphate - analogs & derivatives | Carbenoxolone - pharmacology | Microscopy, Confocal | Animals | Sensory Receptor Cells - drug effects | Analysis of Variance | Bacterial Toxins - pharmacology | Receptors, Purinergic P2X2 - metabolism | Mice | Olfactory Mucosa - cytology | Enterotoxins - pharmacology | Purinergic Agents - pharmacology | Infants (Newborn) | Luciferase | Central nervous system | Hostages | G proteins | Product introduction | Epithelium | Cell differentiation | Adenosine triphosphate | Probenecid | Colleges & universities | Homeostasis | ABC transporters | Life sciences | Cells | Adenosine triphosphatase | Manuscripts
SYSTEM | CELLS | IN-VITRO | NEUROTRANSMISSION | PURINERGIC RECEPTOR ACTIVATION | ACETYLCHOLINE-RELEASE | NEURONS | NEUROMUSCULAR-JUNCTION | CONNEXIN HEMICHANNELS | EXPRESSION | NEUROSCIENCES | Gadolinium - pharmacology | Humans | Qb-SNARE Proteins - metabolism | Molecular Sequence Data | Phosphopyruvate Hydratase - metabolism | Exocytosis - drug effects | Adenosine Triphosphate - pharmacology | Drug Interactions | Transfection | Qc-SNARE Proteins - metabolism | Time Factors | Adenosine Triphosphate - metabolism | Quinacrine - metabolism | Ethylmaleimide - pharmacology | Sensory Receptor Cells - metabolism | Bromodeoxyuridine - metabolism | Pyridoxal Phosphate - analogs & derivatives | Organ Culture Techniques | Animals, Newborn | Receptors, Purinergic P2X2 - genetics | Calcium - pharmacology | Cytidine Triphosphate - pharmacology | Pyridoxal Phosphate - pharmacology | Uridine Triphosphate - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Adenosine Triphosphate - analogs & derivatives | Carbenoxolone - pharmacology | Microscopy, Confocal | Animals | Sensory Receptor Cells - drug effects | Analysis of Variance | Bacterial Toxins - pharmacology | Receptors, Purinergic P2X2 - metabolism | Mice | Olfactory Mucosa - cytology | Enterotoxins - pharmacology | Purinergic Agents - pharmacology | Infants (Newborn) | Luciferase | Central nervous system | Hostages | G proteins | Product introduction | Epithelium | Cell differentiation | Adenosine triphosphate | Probenecid | Colleges & universities | Homeostasis | ABC transporters | Life sciences | Cells | Adenosine triphosphatase | Manuscripts
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2003, Volume 278, Issue 33, pp. 31192 - 31201
A great deal of data has been amassed suggesting that cationic peptides are able to translocate into eucaryotic cells in a temperature-independent manner....
MANNOSE-6-PHOSPHATE RECEPTORS | RECEPTOR-MEDIATED ENDOCYTOSIS | LIPID-BILAYERS | ANTENNAPEDIA HOMEODOMAIN | TRANSFERRIN | PANTP PEPTIDE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ANTIMICROBIAL PEPTIDES | PLASMA-MEMBRANE | PROTEIN TRANSDUCTION | 3RD HELIX | Temperature | Cations - metabolism | Homeodomain Proteins - metabolism | Humans | Nocodazole - pharmacology | Molecular Sequence Data | Antennapedia Homeodomain Protein | Fixatives - pharmacology | Oligopeptides - physiology | Ethylmaleimide - pharmacology | Antineoplastic Agents - pharmacology | Cell Membrane - metabolism | Amino Acid Sequence | Peptide Fragments - metabolism | Endocytosis - drug effects | Enzyme Inhibitors - pharmacology | Homeodomain Proteins - chemistry | Endocytosis - physiology | Nuclear Proteins | Peptide Fragments - chemistry | K562 Cells | Transcription Factors | Vinblastine - pharmacology | Lipid Bilayers - metabolism | Antineoplastic Agents, Phytogenic - pharmacology | Hydrogen-Ion Concentration
MANNOSE-6-PHOSPHATE RECEPTORS | RECEPTOR-MEDIATED ENDOCYTOSIS | LIPID-BILAYERS | ANTENNAPEDIA HOMEODOMAIN | TRANSFERRIN | PANTP PEPTIDE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ANTIMICROBIAL PEPTIDES | PLASMA-MEMBRANE | PROTEIN TRANSDUCTION | 3RD HELIX | Temperature | Cations - metabolism | Homeodomain Proteins - metabolism | Humans | Nocodazole - pharmacology | Molecular Sequence Data | Antennapedia Homeodomain Protein | Fixatives - pharmacology | Oligopeptides - physiology | Ethylmaleimide - pharmacology | Antineoplastic Agents - pharmacology | Cell Membrane - metabolism | Amino Acid Sequence | Peptide Fragments - metabolism | Endocytosis - drug effects | Enzyme Inhibitors - pharmacology | Homeodomain Proteins - chemistry | Endocytosis - physiology | Nuclear Proteins | Peptide Fragments - chemistry | K562 Cells | Transcription Factors | Vinblastine - pharmacology | Lipid Bilayers - metabolism | Antineoplastic Agents, Phytogenic - pharmacology | Hydrogen-Ion Concentration
Journal Article
Journal of Cell Science, ISSN 0021-9533, 2001, Volume 114, Issue 20, pp. 3619 - 3629
Autophagy is a normal degradative pathway that involves the sequestration of cytoplasmic portions and intracellular organelles in a membrane vacuole called the...
NEM-sensitive protein | Starvation | Vinblastine | PI3-kinase | Autophagy | LC3 | autophagy | HT-29 CELLS | ISOLATED RAT HEPATOCYTES | SACCHAROMYCES-CEREVISIAE | CELL BIOLOGY | starvation | MEMBRANE-FUSION | YEAST | SEQUESTRATION | PROTEIN CONJUGATION SYSTEM | vinblastine | PATHWAY | VESICULAR TRANSPORT | DEGRADATION | Cricetinae | Vacuoles - ultrastructure | Adenine - analogs & derivatives | Cadaverine - analogs & derivatives | Phagosomes - metabolism | Enzyme Inhibitors - pharmacology | Adenine - pharmacology | Autophagy - physiology | Recombinant Fusion Proteins - metabolism | Cycloheximide - pharmacology | Amino Acids - metabolism | Cadaverine - metabolism | Animals | Androstadienes - pharmacology | Vacuoles - metabolism | Ethylmaleimide - pharmacology | Phagosomes - ultrastructure | Vinblastine - pharmacology | Protein Synthesis Inhibitors - pharmacology | Fluorescent Dyes | CHO Cells
NEM-sensitive protein | Starvation | Vinblastine | PI3-kinase | Autophagy | LC3 | autophagy | HT-29 CELLS | ISOLATED RAT HEPATOCYTES | SACCHAROMYCES-CEREVISIAE | CELL BIOLOGY | starvation | MEMBRANE-FUSION | YEAST | SEQUESTRATION | PROTEIN CONJUGATION SYSTEM | vinblastine | PATHWAY | VESICULAR TRANSPORT | DEGRADATION | Cricetinae | Vacuoles - ultrastructure | Adenine - analogs & derivatives | Cadaverine - analogs & derivatives | Phagosomes - metabolism | Enzyme Inhibitors - pharmacology | Adenine - pharmacology | Autophagy - physiology | Recombinant Fusion Proteins - metabolism | Cycloheximide - pharmacology | Amino Acids - metabolism | Cadaverine - metabolism | Animals | Androstadienes - pharmacology | Vacuoles - metabolism | Ethylmaleimide - pharmacology | Phagosomes - ultrastructure | Vinblastine - pharmacology | Protein Synthesis Inhibitors - pharmacology | Fluorescent Dyes | CHO Cells
Journal Article