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Journal Article
Nature, ISSN 0028-0836, 02/2005, Volume 433, Issue 7025, pp. 523 - 527
Toll-like receptor 2 (TLR2) is required for the recognition of numerous molecular components of bacteria, fungi and protozoa. The breadth of the ligand... 
TOLL-LIKE RECEPTOR-2 | IMMUNE-RESPONSE | PATTERN-RECOGNITION | LIPOTEICHOIC ACID | GENE | METABOLISM | MULTIDISCIPLINARY SCIENCES | FATTY-ACID | LIPOPOLYSACCHARIDE | IDENTIFICATION | LIPOPROTEINS | Peptidoglycan - metabolism | Toll-Like Receptor 2 | CD36 Antigens - genetics | Membrane Glycoproteins - metabolism | Staphylococcus aureus - physiology | Humans | Membrane Glycoproteins - chemistry | Peptidoglycan - chemistry | Zymosan - pharmacology | Mutagenesis - drug effects | Toll-Like Receptors | CD36 Antigens - metabolism | Gene Deletion | Receptors, Cell Surface - chemistry | Staphylococcal Infections - microbiology | Dimerization | Oligopeptides - chemistry | Staphylococcal Infections - metabolism | Cell Line | Oligopeptides - chemical synthesis | Glycerides - chemistry | Staphylococcal Infections - genetics | Signal Transduction | Staphylococcal Infections - immunology | Mice, Inbred C57BL | Membrane Glycoproteins - agonists | Receptors, Cell Surface - agonists | Receptors, Cell Surface - metabolism | Glycerides - metabolism | Mutation - genetics | Immunologic Deficiency Syndromes - chemically induced | Ethylnitrosourea - pharmacology | Mice, Knockout | Staphylococcus aureus - chemistry | Homozygote | Glycerides - pharmacology | Phenotype | Animals | Aging - physiology | Receptors, Cell Surface - deficiency | Mice | Oligopeptides - pharmacology | Peptidoglycan - pharmacology | Lipopeptides | Tumor Necrosis Factor-alpha - biosynthesis | Membrane Glycoproteins - deficiency | Rodents | Immunology | Sensors
Journal Article
Environmental and Molecular Mutagenesis, ISSN 0893-6692, 05/2013, Volume 54, Issue 4, pp. 268 - 280
The events or factors that lead from normal cell function to conditions and diseases such as aging or cancer reflect complex interactions between cells and... 
HPRT mutation | cell fate decisions | damage tolerance | gene environment interaction | cellular stress response | damage avoidance | Damage tolerance | Damage avoidance | Cell fate decisions | Cellular stress response | Gene environment interaction | 6-THIOGUANINE-RESISTANT LYMPHOCYTES | LESCH-NYHAN SYNDROME | GENE-EXPRESSION PROFILES | INDUCED PREMATURE SENESCENCE | DNA MISMATCH REPAIR | ENVIRONMENTAL SCIENCES | REVERSE-TRANSCRIPTASE INHIBITORS | HUMAN LYMPHOBLASTOID-CELLS | GENETICS & HEREDITY | EXPOSED IN-VIVO | ETHYL-N-NITROSOUREA | TOXICOLOGY | HPRT GENE | Mutagens - toxicity | Epithelial Cells - drug effects | Humans | Ethylnitrosourea - toxicity | Amphotericin B - toxicity | Dose-Response Relationship, Drug | Epithelial Cells - physiology | Lamivudine - toxicity | Guanine Nucleotides - pharmacology | Stress, Physiological - drug effects | Environmental Pollutants - toxicity | Cell Line | Cell Survival - drug effects | Epithelium - drug effects | Epithelium - metabolism | Amphotericin B - pharmacology | Mutagens - pharmacology | Environmental Pollutants - pharmacology | Zidovudine - pharmacology | Calibration | Environmental Exposure | Thionucleotides - pharmacology | Ethylnitrosourea - pharmacology | Animals | Guanine Nucleotides - toxicity | Zidovudine - toxicity | Lamivudine - pharmacology | Mice | Thionucleotides - toxicity | Toxicity Tests | Purines | Analysis | Environmental aspects | DNA polymerases | Lamivudine | Epithelium | Thioguanine | Zidovudine | Index Medicus
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 08/1997, Volume 17, Issue 8, pp. 4792 - 4800
Journal Article
Environmental and Molecular Mutagenesis, ISSN 0893-6692, 05/2013, Volume 54, Issue 4, pp. 281 - 293
Cellular stress responses consist of a complex network of pathways and linked processes that, when perturbed, are postulated to have roles in the pathogenesis... 
epigenetic cell memory | gene‐environment interactions | checkpoint dysfunction | cellular stress response | genomic instability | senescence bypass | Gene-environment interactions | Epigenetic cell memory | Senescence bypass | Checkpoint dysfunction | Cellular stress response | Genomic instability | CELLS | OXIDATIVE STRESS | DNA METHYLATION | INDUCED PREMATURE SENESCENCE | CANCER | CHECKPOINT ADAPTATION | ENVIRONMENTAL SCIENCES | REACTIVE OXYGEN | gene-environment interactions | GENETICS & HEREDITY | EPIGENETIC EFFECTORS | S-PHASE | TOXICOLOGY | HPRT GENE | Mutagens - toxicity | Epithelial Cells - drug effects | Humans | Ethylnitrosourea - toxicity | Amphotericin B - toxicity | Dose-Response Relationship, Drug | Epithelial Cells - physiology | Lamivudine - toxicity | Guanine Nucleotides - pharmacology | Stress, Physiological - drug effects | Cell Death - drug effects | Cell Line | Cell Survival - drug effects | Epithelium - drug effects | Epithelium - metabolism | Amphotericin B - pharmacology | Mutagens - pharmacology | Zidovudine - pharmacology | Thionucleotides - pharmacology | Ethylnitrosourea - pharmacology | Cell Shape - drug effects | Phenotype | Animals | Mutation - drug effects | Guanine Nucleotides - toxicity | Hypoxanthine Phosphoribosyltransferase - genetics | Cell Cycle Checkpoints - drug effects | Zidovudine - toxicity | Cell Proliferation - drug effects | Lamivudine - pharmacology | Mice | DNA Damage | Thionucleotides - toxicity | Toxicity Tests | Environmental aspects | Purines | Lamivudine | Zidovudine | Analysis | Stem cells | Index Medicus
Journal Article
Cancer Discovery, ISSN 2159-8274, 06/2018, Volume 8, Issue 6, pp. 714 - 729
Journal Article
Psychopharmacology, ISSN 0033-3158, 1/2013, Volume 225, Issue 2, pp. 407 - 419
Journal Article
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 04/2010, Volume 31, Issue 7, pp. 1281 - 1291
Journal Article
Journal Article
Journal Article
Molecular Cancer, ISSN 1476-4598, 06/2009, Volume 8, Issue 1, pp. 38 - 38
Background: Tuberous sclerosis (TSC) is a hamartoma syndrome in which renal and lung tumors cause the greatest morbidity. Loss of either TSC1 or TSC2 in TSC... 
RAPAMYCIN | COMPLEX | LYMPHANGIOLEIOMYOMATOSIS | INHIBITION | AKT/PKB | ONCOLOGY | PHOSPHORYLATION | ANGIOMYOLIPOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | AKT | TUMORS | SIROLIMUS | Multiprotein Complexes | Antineoplastic Agents - therapeutic use | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Quinolines - pharmacology | Mechanistic Target of Rapamycin Complex 1 | Proteins | Tuberous Sclerosis - pathology | Ethylnitrosourea | Antineoplastic Agents - pharmacology | Imidazoles - therapeutic use | Everolimus | Disease Models, Animal | Sirolimus - analogs & derivatives | Sirolimus - therapeutic use | Mice, Inbred C57BL | Carrier Proteins - antagonists & inhibitors | Neoplasm Recurrence, Local | Mice, Transgenic | Transcription Factors - antagonists & inhibitors | Imidazoles - pharmacology | Sirolimus - pharmacology | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Transcription Factors - metabolism | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Animals | Carrier Proteins - metabolism | Tuberous Sclerosis - drug therapy | Signal Transduction - drug effects | Kidney Neoplasms - pathology | Mice | Quinolines - therapeutic use | TOR Serine-Threonine Kinases | Kidney Neoplasms - drug therapy | Usage | Tuberous sclerosis | Genetic aspects | Rapamycin | Models | Diagnosis | Research | Drug therapy | Health aspects
Journal Article