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Psychopharmacology, ISSN 0033-3158, 6/2014, Volume 231, Issue 11, pp. 2291 - 2298
α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid (AMPA) receptor stimulation has been proposed to be a common neural mechanism of metabotropic glutamate... 
Neurosciences | AMPA receptor | Biomedicine | Ketamine | Serotonin | Metabotropic glutamate 2/3 receptor antagonist | Depression | Pharmacology/Toxicology | Psychiatry | 5-HT1A receptor | Novelty-suppressed feeding test | 5-HT1Areceptor | ANTIDEPRESSANT | Novelty-suppressedfeedingtest | RESISTANT MAJOR DEPRESSION | 5-HT1A RECEPTORS | PSYCHIATRY | INVOLVEMENT | RELEASE | NEUROSCIENCES | PREFRONTAL CORTEX | MGS0039 | ANIMAL-MODELS | PHARMACOLOGY & PHARMACY | MICE | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Serotonin 5-HT1 Receptor Antagonists - pharmacology | Receptors, N-Methyl-D-Aspartate - metabolism | Male | Receptors, Metabotropic Glutamate - metabolism | Amino Acids - pharmacology | Quinoxalines - pharmacology | Piperidines - pharmacology | Dioxoles - pharmacology | Antidepressive Agents - pharmacology | Ritanserin - pharmacology | Fenclonine - pharmacology | Receptors, AMPA - metabolism | Tryptophan Hydroxylase - metabolism | Xanthenes - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Serotonin 5-HT2 Receptor Antagonists - pharmacology | Piperazines - pharmacology | Feeding Behavior - physiology | Feeding Behavior - drug effects | Neuropsychological Tests | Animals | Pyridines - pharmacology | Ketamine - pharmacology | Tryptophan Hydroxylase - antagonists & inhibitors | Receptors, Metabotropic Glutamate - antagonists & inhibitors | Receptors, AMPA - antagonists & inhibitors | Neurons | Amino acids | Index Medicus
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2011, Volume 69, Issue 8, pp. 754 - 761
Journal Article
Psychological Medicine, ISSN 0033-2917, 05/2016, Volume 46, Issue 7, pp. 1459 - 1472
Background. Ketamine and non-ketamine N-methyl-D-aspartate receptor antagonists (NMDAR antagonists) recently demonstrated antidepressant efficacy for the... 
depression | meta-analyses | ketamine | Bipolar depression | N-methyl-d-aspartate receptor antagonists | trajectories | TREATMENT-RESISTANT DEPRESSION | PSYCHIATRY | ADD-ON TRIAL | RANDOMIZED CONTROLLED-TRIAL | MAJOR DEPRESSION | IV KETAMINE | PSYCHOLOGY, CLINICAL | PSYCHOLOGY | PROOF-OF-CONCEPT | INTRAVENOUS KETAMINE | N-methyl-D-aspartate receptor antagonists | ANTIDEPRESSANT EFFICACY | CHANNEL BLOCKER | RATING-SCALE | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Excitatory Amino Acid Antagonists - pharmacokinetics | Humans | Excitatory Amino Acid Antagonists - administration & dosage | Excitatory Amino Acid Antagonists - pharmacology | Bipolar Disorder - drug therapy | Ketamine - administration & dosage | Ketamine - adverse effects | Outcome Assessment (Health Care) - statistics & numerical data | Ketamine - pharmacokinetics | Antidepressive Agents - administration & dosage | Antidepressive Agents - pharmacology | Antidepressive Agents - adverse effects | Antidepressive Agents - pharmacokinetics | Ketamine - pharmacology | Excitatory Amino Acid Antagonists - adverse effects | Depressive Disorder, Major - drug therapy | Discontinued | Depressive personality disorders | Intravenous administration | Toxicity | Clinical trials | Glutamic acid receptors | Mental depression | Dosage | Clinical outcomes | Randomized controlled trials | Confidence intervals | Psychotropic drugs | Hostility | Remission | Safety | Antidepressant drugs | Bipolar affective disorder | Efficacy | Depression | N-Methyl-D-aspartic acid receptors | Meta-analysis | Refractory depression | Side effects | Ketamine | Psychopharmacology | Superiority | Index Medicus
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 08/2013, Volume 38, Issue 9, pp. 1609 - 1616
We have previously demonstrated that lipopolysaccharide (LPS) induces depressive-like behavior by activating indoleamine 2,3 dioxygenase (IDO; O'Connor et al,... 
AMPA receptor | depression | lipopolysaccharide | NMDA receptor | inflammation | ketamine | BACILLUS-CALMETTE-GUERIN | PSYCHIATRY | MACROPHAGES | INDOLEAMINE 2,3-DIOXYGENASE | INDUCTION | NEUROSCIENCES | HIPPOCAMPUS | QUINOLINIC ACID | SICKNESS | IMMUNE ACTIVATION | PHARMACOLOGY & PHARMACY | BRAIN | Indoleamine-Pyrrole 2,3,-Dioxygenase - metabolism | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Body Weight - drug effects | Motor Activity - drug effects | Male | Ketamine - antagonists & inhibitors | Lipopolysaccharides - antagonists & inhibitors | Brain - metabolism | Quinoxalines - pharmacology | Depression - drug therapy | Drug Interactions | Liver - drug effects | Depression - chemically induced | Immobility Response, Tonic - drug effects | Antidepressive Agents - pharmacology | Brain-Derived Neurotrophic Factor - biosynthesis | Ketamine - therapeutic use | Cytokines - metabolism | Drug Administration Schedule | Food Preferences - drug effects | Liver - metabolism | Mice, Inbred C57BL | Excitatory Amino Acid Antagonists - pharmacology | Antidepressive Agents - therapeutic use | Brain - drug effects | Eating - drug effects | Animals | Signal Transduction - drug effects | Mice | Ketamine - pharmacology | Receptors, AMPA - antagonists & inhibitors | Index Medicus | Antidepressants | biological psychiatry | animal models | behavioral science | bipolar | unipolar | Original
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2006, Volume 26, Issue 11, pp. 2852 - 2861
Glutamate receptor (GluR) ion channels mediate fast synaptic transmission in the mammalian CNS. Numerous crystallographic studies, the majority on the... 
Glutamate receptor | Kainate | Antagonist | Structure | Crystallography | Gating | ACTIVATION | NMDA RECEPTORS | MECHANISM | MODEL | kainate | antagonist | NEUROSCIENCES | structure | GLUTAMATE-RECEPTOR | crystallography | AMPA RECEPTORS | glutamate receptor | gating | DYNAMICS | PARTIAL AGONIST ACTION | ION CHANNELS | SUBUNIT | Excitatory Amino Acid Antagonists - metabolism | Receptors, Kainic Acid - chemistry | Protein Conformation - drug effects | Thymine - analogs & derivatives | Receptors, AMPA - chemistry | Alanine - chemistry | Crystallography, X-Ray | Receptors, Kainic Acid - antagonists & inhibitors | Oocytes | Quinoxalines - pharmacology | Alanine - analogs & derivatives | Recombinant Fusion Proteins - antagonists & inhibitors | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Excitatory Amino Acid Antagonists - chemistry | Thymine - pharmacology | Binding Sites | Alanine - pharmacology | Thymine - metabolism | Thymine - chemistry | Glutamic Acid - pharmacology | Xenopus laevis | Models, Molecular | Rats | Alanine - metabolism | Excitatory Amino Acid Antagonists - pharmacology | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - metabolism | Recombinant Fusion Proteins - chemistry | Patch-Clamp Techniques | Animals | Hydrogen Bonding | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - chemistry | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - pharmacology | Protein Binding | Glutamic Acid - metabolism | Glutamic Acid - chemistry | Receptors, AMPA - antagonists & inhibitors | Index Medicus | BIOLOGY | GENES | CRYSTAL STRUCTURE | MATERIALS SCIENCE | CHAINS | DIMERS | CONFORMATIONAL CHANGES
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2010, Volume 67, Issue 9, pp. 831 - 839
Background Corticotropin-releasing factor (CRF) and gamma-aminobutyric acid (GABA)ergic systems in the central amygdala (CeA) are implicated in the... 
Psychiatry | Alcohol | dependence | mRNA | electrophysiology | microdialysis | CENTRAL EXTENDED AMYGDALA | GABAERGIC TRANSMISSION | CENTRAL NUCLEUS | DRINKING | PSYCHIATRY | RATS | ANXIETY-LIKE BEHAVIOR | NEUROSCIENCES | GABA RELEASE | ANIMAL-MODELS | ETHANOL WITHDRAWAL | CRF1 ANTAGONIST | Amygdala - drug effects | Microdialysis - methods | Rats, Wistar | gamma-Aminobutyric Acid - metabolism | Body Weight - drug effects | Ethanol - administration & dosage | Male | RNA, Messenger - metabolism | Cyclophilins - pharmacology | Time Factors | Neurons - physiology | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Corticotropin-Releasing Hormone - genetics | Neurons - drug effects | Alcoholism - pathology | Alcoholism - blood | Valine - analogs & derivatives | Amygdala - metabolism | Rats | Excitatory Amino Acid Antagonists - pharmacology | Pyrimidines - pharmacology | Rats, Sprague-Dawley | Amygdala - pathology | Corticotropin-Releasing Hormone - pharmacology | GABA Antagonists - pharmacology | Hormone Antagonists - pharmacology | Patch-Clamp Techniques | Animals | Phosphinic Acids - pharmacology | Alcoholism - metabolism | Central Nervous System Depressants - administration & dosage | Valine - pharmacology | In Vitro Techniques | Propanolamines - pharmacology | Inhibitory Postsynaptic Potentials - drug effects | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 04/2013, Volume 78, Issue 1, pp. 81 - 93
Journal Article
Neuron, ISSN 0896-6273, 2010, Volume 68, Issue 1, pp. 113 - 126
Endocannabinoids and their receptor CB1 play key roles in brain function. Astrocytes express CB1Rs that are activated by endocannabinoids released by neurons.... 
ENDOGENOUS CANNABINOIDS | ACTIVATION | CA1 PYRAMIDAL CELLS | EXCITATORY SYNAPSES | NEURONS | LONG-TERM POTENTIATION | GLIAL-CELLS | NEUROSCIENCES | MODULATION | HIPPOCAMPAL SYNAPSES | PLASTICITY | Cannabinoid Receptor Modulators - metabolism | Cannabinoid Receptor Modulators - pharmacology | Glycine - analogs & derivatives | Synaptic Transmission - physiology | Synaptic Transmission - genetics | Calcium - metabolism | Excitatory Postsynaptic Potentials - drug effects | Patch-Clamp Techniques - methods | Resorcinols - pharmacology | Drug Interactions | Photolysis | Piperidines - pharmacology | Pyramidal Cells - physiology | Egtazic Acid - analogs & derivatives | Synaptic Transmission - drug effects | Pyramidal Cells - drug effects | Electric Stimulation - methods | Endocannabinoids | Excitatory Postsynaptic Potentials - genetics | Pyrazoles - pharmacology | Animals, Newborn | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Biophysics | Chelating Agents - pharmacology | Hippocampus - cytology | Mice, Knockout | Astrocytes - physiology | Animals | Egtazic Acid - pharmacology | Glycine - pharmacology | Cannabinoid Receptor Modulators - antagonists & inhibitors | Benzoates - pharmacology | Thapsigargin - pharmacology | Mice | Pyridines - pharmacology | Receptor, Cannabinoid, CB1 - deficiency | In Vitro Techniques | Glutamate | Neurons | Studies | Retina | Transmitters | Index Medicus | Potentiation | Brain | Neurotransmitter release | Astrocytes | Glutamic acid receptors | Cannabinoid CB1 receptors | synaptic depression | Calcium signalling | Pyramidal cells | Synaptic transmission | Endogenous stimuli | Receptor mechanisms | Glutamic acid receptors (metabotropic) | Hippocampus | Neurotransmission
Journal Article