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1. Full Text Rare Structural Variants Disrupt Multiple Genes in Neurodevelopmental Pathways in Schizophrenia
by Tom Walsh and Jon M. McClellan and Shane E. McCarthy and Anjené M. Addington and Sarah B. Pierce and Greg M. Cooper and Alex S. Nord and Mary Kusenda and Dheeraj Malhotra and Abhishek Bhandari and Sunday M. Stray and Caitlin F. Rippey and Patricia Roccanova and Vlad Makarov and B. Lakshmi and Robert L. Findling and Linmarie Sikich and Thomas Stromberg and Barry Merriman and Nitin Gogtay and Philip Butler and Kristen Eckstrand and Laila Noory and Peter Gochman and Robert Long and Zugen Chen and Sean Davis and Carl Baker and Evan E. Eichler and Paul S. Meltzer and Stanley F. Nelson and Andrew B. Singleton and Ming K. Lee and Judith L. Rapoport and Mary-Claire King and Jonathan Sebat
Science, ISSN 0036-8075, 4/2008, Volume 320, Issue 5875, pp. 539 - 543
Schizophrenia is a devastating neurodevelopmental disorder whose genetic influences remain elusive. We hypothesize that individually rare structural variants... 
Autistic disorder | Genes | Genomics | DNA | Medical genetics | Schizophrenia | Amino acids | Reports | Genetic mutation | Chromosomes | Diseases | NEURONAL MIGRATION | NEUREGULIN | NMDA RECEPTOR | GLUTAMATE | NEUROTRANSMITTERS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | DIFFERENTIATION | CHILDHOOD-ONSET SCHIZOPHRENIA | EXPRESSION | COMMON DISEASE | Gene Duplication | Receptor, Epidermal Growth Factor - genetics | Oligonucleotide Array Sequence Analysis | Excitatory Amino Acid Transporter 1 - chemistry | Humans | Excitatory Amino Acid Transporter 1 - physiology | Molecular Sequence Data | Male | Neurons - cytology | Brain - growth & development | Excitatory Amino Acid Transporter 1 - genetics | Case-Control Studies | Brain - metabolism | Schizophrenia - genetics | Schizophrenia - physiopathology | Gene Deletion | Neurons - physiology | Adult | Female | Receptor, Epidermal Growth Factor - physiology | Child | Amino Acid Sequence | Brain - cytology | Genetic Predisposition to Disease | Signal Transduction | Receptor, Epidermal Growth Factor - chemistry | Adolescent | Age of Onset | Polymorphism, Single Nucleotide | Mutation | Genome, Human | Receptor, ErbB-4 | Chromosome replication | Gene mutations | Neurobiology | Physiological aspects | Influence | Genetic aspects | Chromosome deletion | Genetics | Psychiatry
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2. Full Text Increases in circulating amino acids with in-feed antibiotics correlated with gene expression of intestinal amino acid transporters in piglets
by Yu, Miao and Mu, Chunlong and Yang, Yuxiang and Zhang, Chuanjian and Su, Yong and Huang, Zan and Yu, Kaifan and Zhu, Weiyun
Amino Acids, ISSN 0939-4451, 9/2017, Volume 49, Issue 9, pp. 1587 - 1599
In-feed antibiotics have been commonly used to promote the growth performance of piglets. The antibiotics can increase protein utilization, but the underlying... 
Life Sciences | Biochemistry, general | Amino acid transporter | Analytical Chemistry | Life Sciences, general | Antibiotics | Biochemical Engineering | Proteomics | Neurobiology | Piglets | Amino acid receptors | Amino acids profiles | PIGS | NUTRIENT ABSORPTION | BACTERIA | BIOCHEMISTRY & MOLECULAR BIOLOGY | DIETARY SUPPLEMENTATION | GASTROINTESTINAL-TRACT | WEANED PIGLETS | METABOLIC PROFILES | GUT MICROBIOTA | MOLECULAR-CLONING | HEALTH | Amino Acids - blood | Sodium-Potassium-Exchanging ATPase - genetics | Receptors, G-Protein-Coupled - metabolism | Large Neutral Amino Acid-Transporter 1 - metabolism | Weaning | Amino Acid Transport System ASC - agonists | Receptors, G-Protein-Coupled - agonists | RNA, Messenger - metabolism | Receptors, Calcium-Sensing - metabolism | Quinoxalines - pharmacology | TRPV Cation Channels - metabolism | Animal Feed - analysis | Swine | Weight Gain - drug effects | Biological Transport - drug effects | Amino Acid Transport System ASC - genetics | Excitatory Amino Acid Transporter 3 - agonists | Oxytetracycline - pharmacology | Animals, Newborn | Kitasamycin - pharmacology | RNA, Messenger - genetics | TRPV Cation Channels - agonists | Amino Acid Transport System ASC - metabolism | Large Neutral Amino Acid-Transporter 1 - genetics | Excitatory Amino Acid Transporter 3 - genetics | Peptide Transporter 1 - genetics | RNA, Messenger - agonists | Receptors, Calcium-Sensing - agonists | TRPV Cation Channels - genetics | Gene Expression Regulation - drug effects | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Excitatory Amino Acid Transporter 3 - metabolism | Receptors, Calcium-Sensing - genetics | Anti-Bacterial Agents - pharmacology | Peptide Transporter 1 - agonists | Peptide Transporter 1 - metabolism | Receptors, G-Protein-Coupled - genetics | RNA | Analysis | Genetic research | Amino acids | Gene expression | Adenosine triphosphatase | Na+/K+-exchanging ATPase | Birth | Small intestine | Animal feed | Hogs | Receptors | Diet | Correlation analysis | Sodium-glutamic acid cotransporter | Calcium-sensing receptors
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3. Full Text Excitatory amino acid transporter expression by astrocytes is neuroprotective against microglial excitotoxicity
by Liang, Jianfeng and Takeuchi, Hideyuki and Doi, Yukiko and Kawanokuchi, Jun and Sonobe, Yoshifumi and Jin, Shijie and Yawata, Izumi and Li, Hua and Yasuoka, Satoko and Mizuno, Tetsuya and Suzumura, Akio
Brain Research, ISSN 0006-8993, 2008, Volume 1210, pp. 11 - 19
Abstract Glutamate-induced excitotoxicity is considered as a major cause of neurodegenerative disease. Excitatory amino acid transporters (EAATs) on glial... 
Neurology | Astrocyte | Glutamate | Excitotoxicity | EAAT | Microglia | NERVOUS-SYSTEM | CELLS | RELEASE | AFFINITY GLUTAMATE TRANSPORTER | NEUROSCIENCES | GLAST | CLONING | NEURONS | RAT-BRAIN | excitotoxicity | ACTIVATED MICROGLIA | astrocyte | microglia | glutamate | GLT-1 | Microglia - metabolism | Excitatory Amino Acid Transporter 5 - genetics | Coculture Techniques | Amino Acid Transport Systems, Acidic - metabolism | Nerve Degeneration - physiopathology | Nerve Degeneration - genetics | Gliosis - physiopathology | Excitatory Amino Acid Transporter 1 - genetics | RNA, Messenger - metabolism | Nerve Degeneration - metabolism | Cytoprotection - drug effects | Excitatory Amino Acid Transporter 2 - genetics | Astrocytes - cytology | Microglia - cytology | Astrocytes - drug effects | Gliosis - genetics | Glutamic Acid - pharmacology | Microglia - drug effects | Gliosis - metabolism | Mice, Inbred C57BL | Cells, Cultured | Excitatory Amino Acid Transporter 1 - metabolism | Cell Communication - genetics | Excitatory Amino Acid Transporter 3 - genetics | Excitatory Amino Acid Transporter 2 - metabolism | Excitatory Amino Acid Transporter 5 - metabolism | Animals | Excitatory Amino Acid Transporter 3 - metabolism | Excitatory Amino Acid Transporter 4 - genetics | Excitatory Amino Acid Transporter 4 - metabolism | Cell Communication - drug effects | Amino Acid Transport Systems, Acidic - genetics | Cytoprotection - genetics | Neurotoxins - metabolism | Glutamic Acid - metabolism | Mice | Astrocytes - metabolism | Index Medicus
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4. Full Text Transcriptomic and quantitative proteomic analysis of transporters and drug metabolizing enzymes in freshly isolated human brain microvessels
by Shawahna, Ramzi and Uchida, Yasuo and Declèves, Xavier and Ohtsuki, Sumio and Yousif, Salah and Dauchy, Sandrine and Jacob, Aude and Chassoux, Francine and Daumas-Duport, Catherine and Couraud, Pierre-Olivier and Terasaki, Tetsuya and Scherrmann, Jean-Michel
Molecular Pharmaceutics, ISSN 1543-8384, 08/2011, Volume 8, Issue 4, pp. 1332 - 1341
We have investigated the transcriptomic and/or proteomic patterns of 71 solute carrier (SLC) and organic solute (OST) transporters, 34 ATP-binding cassette... 
blood-brain barrier | quantitative LC-MS/MS | transporters | drug-metabolizing enzymes | human | PREGNANE-X RECEPTOR | MEDICINE, RESEARCH & EXPERIMENTAL | BARRIER | P-GLYCOPROTEIN | UDP-GLUCURONOSYLTRANSFERASE | CONSTITUTIVE ANDROSTANE RECEPTOR | PHARMACOLOGY & PHARMACY | GLUTATHIONE-S-TRANSFERASE | RAT-BRAIN | PROTEINS | UP-REGULATION | EXPRESSION | ATP Binding Cassette Transporter, Sub-Family G, Member 2 | Glutamate Plasma Membrane Transport Proteins - metabolism | Humans | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Microvessels - metabolism | Glucose Transporter Type 1 - metabolism | Glutamate Plasma Membrane Transport Proteins - genetics | Excitatory Amino Acid Transporter 1 - genetics | Neoplasm Proteins - metabolism | Brain - metabolism | Brain - blood supply | Tandem Mass Spectrometry | Glutathione Transferase - genetics | ATP-Binding Cassette Transporters - genetics | ATP-Binding Cassette Transporters - metabolism | Chromatography, Liquid | Proteomics - methods | Neoplasm Proteins - genetics | Transcriptome - physiology | Glutathione S-Transferase pi - genetics | Glutathione S-Transferase pi - metabolism | Glutathione Transferase - metabolism | Excitatory Amino Acid Transporter 1 - metabolism | Transcriptome - genetics | Glucose Transporter Type 1 - genetics | ATP-Binding Cassette, Sub-Family B, Member 1 - genetics | ATP Binding Cassette Transporter, Sub-Family B | In Vitro Techniques
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5. Full Text δ‐Opioid receptors up‐regulate excitatory amino acid transporters in mouse astrocytes
by Liang, Jianfeng and Chao, Dongman and Sandhu, Harleen K and Yu, Yanbing and Zhang, Li and Balboni, Gianfranco and Kim, Dong H and Xia, Ying
British Journal of Pharmacology, ISSN 0007-1188, 12/2014, Volume 171, Issue 23, pp. 5417 - 5430
Background and PurposeAstrocytic excitatory amino acid transporters (EAATs) regulate extracellular glutamate concentrations and play a role in preventing... 
ACTIVATION | MOTOR-NEURON DEGENERATION | CONCISE GUIDE | PHARMACOLOGY | GLUTAMATE TRANSPORTER | MU | PHARMACOLOGY & PHARMACY | AMYOTROPHIC-LATERAL-SCLEROSIS | RAT-BRAIN | EXPRESSION | GLT-1 | Naltrexone - analogs & derivatives | Receptors, Opioid, delta - metabolism | Astrocytes - drug effects | RNA, Small Interfering - genetics | Up-Regulation | Mice, Inbred C57BL | Cells, Cultured | Excitatory Amino Acid Transporter 3 - genetics | Excitatory Amino Acid Transporter 1 - genetics | Excitatory Amino Acid Transporter 2 - biosynthesis | Naltrexone - pharmacology | Animals | Excitatory Amino Acid Transporter 3 - metabolism | Receptors, Opioid, delta - genetics | Narcotic Antagonists - pharmacology | Benzimidazoles - pharmacology | Receptors, Opioid, delta - antagonists & inhibitors | Excitatory Amino Acid Transporter 1 - biosynthesis | Excitatory Amino Acid Transporter 2 - genetics | Oligopeptides - pharmacology | Receptors, Opioid, delta - agonists | Astrocytes - metabolism | Research Papers
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6. Full Text SLC1 glutamate transporters
by Grewer, Christof and Gameiro, Armanda and Rauen, Thomas
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 1/2014, Volume 466, Issue 1, pp. 3 - 24
The plasma membrane transporters for the neurotransmitter glutamate belong to the solute carrier 1 family. They are secondary active transporters, taking up... 
Structure function analysis | SLC1 | Biomedicine | Human Physiology | Glutamate transporter pharmacology | Regulation | Glutamate transporters | Neurotransmission | 3' UNTRANSLATED REGION | PROTEIN-KINASE-C | PHYSIOLOGY | GLIAL GLUTAMATE | AMYOTROPHIC-LATERAL-SCLEROSIS | POLYMERASE-II TRANSCRIPTION | GATED CHLORIDE CHANNEL | AMINO-ACID TRANSPORTER | THREO-BETA-BENZYLOXYASPARTATE | FLUORENE-ASPARTIC-ACID | NF-KAPPA-B | Excitatory Amino Acid Transporter 1 - antagonists & inhibitors | Amino Acid Sequence | Animals | Excitatory Amino Acid Transporter 1 - chemistry | Humans | Excitatory Amino Acid Agents - pharmacology | Molecular Sequence Data | Excitatory Amino Acid Transporter 1 - metabolism | Glutamic Acid - metabolism | Excitatory Amino Acid Transporter 1 - genetics | Glutamate | Amino acids | Analysis
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7. Full Text Astroglial glutamate transporter deficiency increases synaptic excitability and leads to pathological repetitive behaviors in mice
by Aida, Tomomi and Yoshida, Junichi and Nomura, Masatoshi and Tanimura, Asami and Iino, Yusuke and Soma, Miho and Bai, Ning and Ito, Yukiko and Cui, Wanpeng and Aizawa, Hidenori and Yanagisawa, Michiko and Nagai, Terumi and Takata, Norio and Tanaka, Kenji F and Takayanagi, Ryoichi and Kano, Masanobu and Götz, Magdalena and Hirase, Hajime and Tanaka, Kohichi
Neuropsychopharmacology, ISSN 0893-133X, 02/2015, Volume 40, Issue 7, pp. 1569 - 1579
An increase in the ratio of cellular excitation to inhibition (E/I ratio) has been proposed to underlie the pathogenesis of neuropsychiatric disorders, such as... 
AUTISM | PSYCHIATRY | SCHIZOPHRENIA | NEUROSCIENCES | TOURETTE-SYNDROME | OBSESSIVE-COMPULSIVE DISORDER | PATHOPHYSIOLOGY | NMDA RECEPTOR ANTAGONISTS | GLAST | MUTANT MICE | CIRCUITRY | ANIMAL-MODELS | PHARMACOLOGY & PHARMACY | Seizures - genetics | Cerebral Cortex - pathology | Nerve Degeneration - genetics | Male | Synapses - genetics | Excitatory Amino Acid Transporter 1 - genetics | Cumulative Trauma Disorders - genetics | Female | Excitatory Amino Acid Transporter 2 - genetics | Excitatory Postsynaptic Potentials - genetics | Disease Models, Animal | Animals, Newborn | Excitatory Amino Acid Transporter 1 - deficiency | Gene Expression Regulation - genetics | Excitatory Amino Acid Transporter 2 - deficiency | Mice, Transgenic | Enzyme Inhibitors - therapeutic use | Cumulative Trauma Disorders - drug therapy | Nerve Tissue Proteins - metabolism | Proteins - genetics | Anxiety - genetics | Animals | Cumulative Trauma Disorders - pathology | Hyperalgesia - genetics | Cumulative Trauma Disorders - complications | Mice | Nerve Degeneration - etiology | Original
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8. Full Text Allosteric modulation of an excitatory amino acid transporter: The subtype-selective inhibitor UCPH-101 exerts sustained inhibition of EAAT1 through an intramonomeric site in the trimerization domain
by Abrahamsen, Bjarke and Schneider, Nicole and Erichsen, Mette N and Huynh, Tri H. V and Fahlke, Christoph and Bunch, Lennart and Jensen, Anders A
Journal of Neuroscience, ISSN 0270-6474, 01/2013, Volume 33, Issue 3, pp. 1068 - 1087
In the present study, the mechanism of action and molecular basis for the activity of the first class of selective inhibitors of the human excitatory amino... 
SUBSTRATE INTERACTIONS | NEUROTRANSMITTER TRANSPORTERS | SEROTONIN TRANSPORTER | INTERNALIZATION | NEURONAL GLUTAMATE TRANSPORTER | METABOTROPIC GLUTAMATE-RECEPTOR-SUBTYPE-5 | PHARMACOLOGICAL CHARACTERIZATION | TARGETS | RECEPTORS | EXPRESSION | NEUROSCIENCES | Allosteric Regulation - drug effects | Humans | Cells, Cultured | Rats | Excitatory Amino Acid Transporter 1 - metabolism | Excitatory Amino Acid Transporter 3 - genetics | Excitatory Amino Acid Transporter 1 - genetics | Excitatory Amino Acid Transporter 2 - metabolism | Excitatory Amino Acid Transporter 1 - antagonists & inhibitors | Animals | Excitatory Amino Acid Transporter 3 - metabolism | Excitatory Amino Acid Transporter 4 - genetics | Excitatory Amino Acid Transporter 4 - metabolism | Biological Transport - physiology | Excitatory Amino Acid Transporter 2 - genetics | Benzopyrans - pharmacology | Index Medicus
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9. Full Text Abnormal expression of glutamate transporters in temporal lobe areas in elderly patients with schizophrenia
by Shan, Dan and Lucas, Elizabeth K and Drummond, Jana B and Haroutunian, Vahram and Meador-Woodruff, James H and McCullumsmith, Robert E
Schizophrenia Research, ISSN 0920-9964, 2013, Volume 144, Issue 1, pp. 1 - 8
Abstract Glutamate transporters facilitate the buffering, clearance and cycling of glutamate and play an important role in maintaining synaptic and... 
Psychiatry | Schizophrenia | Superior temporal gyrus | Excitatory amino acid transporter (EAAT) | Glutamate | Vesicular glutamate transporter (VGLUT) | Hippocampus | DORSOLATERAL PREFRONTAL CORTEX | ANTERIOR CINGULATE CORTEX | REDUCED NEURONAL SIZE | MESSENGER-RNA EXPRESSION | PSYCHIATRY | BIPOLAR DISORDER | VENTRAL HIPPOCAMPAL LESION | SYNAPTIC VESICLE | AMINO-ACID TRANSPORTER-2 | GLIAL-CELL DENSITY | MAJOR DEPRESSIVE DISORDER | Excitatory Amino Acid Transporter 3 - biosynthesis | Schizophrenia - metabolism | Glutamic Acid - genetics | Humans | Haloperidol - administration & dosage | Glutamate Plasma Membrane Transport Proteins - genetics | Male | Hippocampus - drug effects | Excitatory Amino Acid Transporter 1 - genetics | Excitatory Amino Acid Transporter 2 - biosynthesis | Vesicular Glutamate Transport Protein 1 - genetics | Vesicular Glutamate Transport Protein 2 - biosynthesis | Schizophrenia - genetics | Aged, 80 and over | Female | Excitatory Amino Acid Transporter 1 - biosynthesis | Excitatory Amino Acid Transporter 2 - genetics | Temporal Lobe - pathology | Disease Models, Animal | Vesicular Glutamate Transport Protein 2 - genetics | Rats | Glutamate Plasma Membrane Transport Proteins - biosynthesis | Hippocampus - pathology | Excitatory Amino Acid Transporter 3 - genetics | Temporal Lobe - metabolism | Rats, Sprague-Dawley | Vesicular Glutamate Transport Proteins - genetics | Antipsychotic Agents - administration & dosage | Gene Expression Regulation - drug effects | Hippocampus - metabolism | Animals | Aged | Glutamic Acid - metabolism | Schizophrenia - drug therapy | Temporal Lobe - drug effects | Vesicular Glutamate Transport Proteins - biosynthesis | Vesicular Glutamate Transport Protein 1 - biosynthesis | Proteins | Antipsychotic drugs | Analysis | Aged patients | Amino acids | excitatory amino acid transporter (EAAT) | vesicular glutamate transporter (VGLUT) | schizophrenia | hippocampus | glutamate | superior temporal gyrus
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10. Full Text Variations in Promoter Activity Reveal a Differential Expression and Physiology of Glutamate Transporters by Glia in the Developing and Mature CNS
by Regan, Melissa R and Huang, Yanhua H and Kim, Yu Shin and Dykes-Hoberg, Margaret I and Jin, Lin and Watkins, Andrew M and Bergles, Dwight E and Rothstein, Jeffrey D
Journal of Neuroscience, ISSN 0270-6474, 06/2007, Volume 27, Issue 25, pp. 6607 - 6619
Glutamate transporters regulate excitatory neurotransmission and prevent glutamate- mediated excitotoxicity in the CNS. To better study the cellular and... 
GLAST | Oligodendroglial | Astroglia | BAC | Transgenic | Membrane | Glutamate | GLT-1 | astroglia | NERVOUS-SYSTEM | membrane | AXONAL DAMAGE | oligodendroglial | IN-SITU HYBRIDIZATION | AMYOTROPHIC-LATERAL-SCLEROSIS | MOTOR-NEURON DISEASE | NEUROSCIENCES | HUMAN ASTROCYTES | transgenic | RAT-BRAIN | OLIGODENDROCYTE EXCITOTOXICITY | glutamate | COMPARATIVE IMMUNOCYTOCHEMISTRY | TRANSGENIC MICE | Cell Line | Excitatory Amino Acid Transporter 2 - physiology | Central Nervous System - metabolism | Vesicular Glutamate Transport Proteins - physiology | Humans | Excitatory Amino Acid Transporter 1 - physiology | Mice, Transgenic | Excitatory Amino Acid Transporter 1 - genetics | Excitatory Amino Acid Transporter 2 - biosynthesis | Vesicular Glutamate Transport Proteins - genetics | Central Nervous System - growth & development | Central Nervous System - physiology | Animals | Promoter Regions, Genetic - physiology | Mice, Mutant Strains | Neuroglia - metabolism | Excitatory Amino Acid Transporter 1 - biosynthesis | Excitatory Amino Acid Transporter 2 - genetics | Mice | Vesicular Glutamate Transport Proteins - biosynthesis | Gene Expression Regulation, Developmental - physiology
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11. Full Text Glutamatergic alterations and mitochondrial impairment in a murine model of Alzheimer disease
by Cassano, Tommaso and Serviddio, Gaetano and Gaetani, Silvana and Romano, Adele and Dipasquale, Pasqua and Cianci, Silvia and Bellanti, Francesco and Laconca, Leonardo and Romano, Antonino Davide and Padalino, Iolanda and LaFerla, Frank M and Nicoletti, Ferdinando and Cuomo, Vincenzo and Vendemiale, Gianluigi
Neurobiology of Aging, ISSN 0197-4580, 2012, Volume 33, Issue 6, pp. 1121.e1 - 1121.e12
Abstract Deficits in glutamate neurotransmission and mitochondrial functions were detected in the frontal cortex (FC) and hippopcampus (HIPP) of aged... 
Neurology | Internal Medicine | Mitochondria | Microdialysis | 3×Tg-AD mice | Frontal cortex | Glutamate | Alzheimer's disease | Hippocampus | H+ LEAK | TRIPLE-TRANSGENIC MODEL | TAU PATHOLOGY | NEUROSCIENCES | EXCITATORY AMINO-ACIDS | GERIATRICS & GERONTOLOGY | 3 X Tg-AD mice | PROTON LEAK | IN-VIVO | MOUSE MODEL | A-BETA | MICE | DYSFUNCTION | Synaptosomal-Associated Protein 25 - genetics | Vesicular Glutamate Transport Protein 1 - metabolism | Glutamic Acid - genetics | Glutamic Acid - physiology | Male | Mice, Transgenic | Excitatory Amino Acid Transporter 1 - metabolism | Mitochondria - metabolism | Mitochondria - pathology | Excitatory Amino Acid Transporter 1 - genetics | Excitatory Amino Acid Transporter 2 - metabolism | Vesicular Glutamate Transport Protein 1 - genetics | Alzheimer Disease - pathology | Animals | Alzheimer Disease - metabolism | Excitatory Amino Acid Transporter 2 - genetics | Mice | Synaptosomal-Associated Protein 25 - metabolism | Alzheimer Disease - genetics | Disease Models, Animal | Hydrogen peroxide | Amino acids | Aspartate | Peroxides | Mitochondrial DNA | Muscle proteins | Intermediate filament proteins | Adenosine triphosphate | Protons | Animal models | Neurodegenerative diseases | Glial fibrillary acidic protein | excitatory amino acids | Data processing | F0F1-ATPase | Bioenergetics | Cortex (frontal) | Aging | Glutamic acid transporter | Membrane potential | Electron transport | ATP | Neurotransmission
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