X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (950) 950
Publication (168) 168
Book Review (15) 15
Book Chapter (8) 8
Book / eBook (7) 7
Conference Proceeding (2) 2
Dissertation (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (803) 803
neurosciences (641) 641
index medicus (555) 555
male (361) 361
mice (356) 356
glutamate (335) 335
rats (321) 321
humans (299) 299
glutamic acid - metabolism (217) 217
excitatory amino acid antagonists - pharmacology (202) 202
brain (196) 196
neurons (195) 195
female (192) 192
mice, knockout (169) 169
disease models, animal (148) 148
neurology (148) 148
biochemistry & molecular biology (146) 146
rats, sprague-dawley (139) 139
neurons - metabolism (136) 136
excitatory amino acid transporter 2 - metabolism (132) 132
mice, inbred c57bl (130) 130
pharmacology & pharmacy (129) 129
rodents (127) 127
article (125) 125
excitatory amino-acids (125) 125
neurons - drug effects (115) 115
cells, cultured (113) 113
amino acids (111) 111
rat-brain (111) 111
expression (103) 103
in vitro techniques (102) 102
oxidative stress (102) 102
astrocytes (100) 100
nervous system (99) 99
central-nervous-system (98) 98
excitotoxicity (96) 96
rats, wistar (96) 96
astrocytes - metabolism (94) 94
animals, newborn (92) 92
psychiatry (91) 91
hippocampus (90) 90
analysis (88) 88
receptors, n-methyl-d-aspartate - metabolism (86) 86
brain - metabolism (85) 85
dose-response relationship, drug (83) 83
methyl aspartate (83) 83
schizophrenia (81) 81
hippocampus - metabolism (80) 80
time factors (80) 80
long-term potentiation (79) 79
physiological aspects (77) 77
excitatory amino acid transporter 1 - metabolism (75) 75
neurons - physiology (75) 75
clinical neurology (74) 74
biomedicine (73) 73
cell biology (73) 73
physiology (69) 69
proteins (68) 68
synaptic transmission - physiology (67) 67
epilepsy (65) 65
in-vivo (65) 65
gaba (64) 64
neuroscience (64) 64
excitatory amino acid agonists - pharmacology (63) 63
mice, transgenic (63) 63
neurodegeneration (63) 63
dizocilpine maleate - pharmacology (61) 61
immunohistochemistry (61) 61
methyl-d-aspartate (61) 61
excitatory postsynaptic potentials - drug effects (60) 60
patch-clamp techniques (59) 59
amyotrophic-lateral-sclerosis (58) 58
research (58) 58
gene expression (57) 57
glutamate transporter (56) 56
calcium - metabolism (55) 55
excitatory amino acid transporter 2 - genetics (55) 55
nmda receptors (55) 55
neurons - pathology (53) 53
excitatory postsynaptic potentials - physiology (52) 52
metabolism (52) 52
prefrontal cortex (52) 52
synaptic transmission (50) 50
analysis of variance (49) 49
hippocampus - cytology (49) 49
hippocampus - drug effects (49) 49
nmda receptor (48) 48
synaptic transmission - drug effects (48) 48
amino acid transport system x-ag - metabolism (46) 46
dopamine (46) 46
neuroprotection (46) 46
rat (46) 46
synaptic plasticity (46) 46
research article (45) 45
alzheimers-disease (44) 44
apoptosis (44) 44
cerebral cortex - metabolism (44) 44
aspartate (43) 43
motor activity - drug effects (43) 43
receptors, n-methyl-d-aspartate - antagonists & inhibitors (43) 43
more...
Library Location Library Location
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Neuropsychopharmacology, ISSN 0893-133X, 02/2015, Volume 40, Issue 7, pp. 1569 - 1579
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 04/2012, Volume 32, Issue 17, pp. 6000 - 6013
The extracellular levels of excitatory amino acids are kept low by the action of the glutamate transporters. Glutamate/aspartate transporter ( GLAST) and... 
MOLECULAR-MECHANISM | RAT CEREBRAL-CORTEX | DIFFERENTIAL SYNAPTIC LOCALIZATION | D-ASPARTATE | IN-SITU HYBRIDIZATION | MONOCLONAL-ANTIBODY | CHLORIDE CHANNEL | GROWTH-FACTOR | HIPPOCAMPAL-NEURONS | GABA SYNTHESIS | NEUROSCIENCES | Membrane Potentials - genetics | Age Factors | Rats, Wistar | Gene Expression Regulation, Developmental - genetics | Parvalbumins - metabolism | Male | Dendrites - ultrastructure | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Microscopy, Immunoelectron | Vesicular Glutamate Transport Protein 1 | Proteolipids | Kidney - metabolism | Central Nervous System - anatomy & histology | Excitatory Amino Acid Agents - pharmacology | Neurons - metabolism | Neurons - drug effects | Gene Expression Regulation, Developmental - physiology | Synaptophysin - metabolism | Animals, Newborn | Membrane Potentials - drug effects | Dendrites - metabolism | Myelin Basic Protein - metabolism | Excitatory Amino Acid Transporter 3 - deficiency | Excitatory Amino Acid Transporter 2 - deficiency | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Rats | D-Aspartic Acid - metabolism | Excitatory Amino Acid Transporter 3 - genetics | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | Excitatory Amino Acid Transporter 2 - metabolism | Subcellular Fractions - metabolism | Mice, Knockout | Patch-Clamp Techniques | Animals | Excitatory Amino Acid Transporter 3 - metabolism | Central Nervous System - cytology | Glutamate Decarboxylase - metabolism | Mice | In Vitro Techniques | Aspartic Acid - pharmacology
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2002, Volume 99, Issue 3, pp. 1604 - 1609
Journal Article
Journal Article
Glia, ISSN 0894-1491, 09/2018, Volume 66, Issue 9, pp. 1988 - 1998
Glutamate is a major excitatory neurotransmitter and plays an important role in neuropathic pain, which is frequently caused by nerve damage. According to... 
ceftriaxone | neuropathic pain | GLT1 | midbrain | spinal cord | glutamate transporter | ASTROCYTE ACTIVATION | COCAINE-SEEKING | MODEL | NEUROSCIENCES | SPINAL DORSAL-HORN | PROTEIN EXPRESSION | UP-REGULATION | CORD-INJURY | BRAIN | ESTROUS-CYCLE | Sciatic Nerve - injuries | Neural Pathways - drug effects | Spinal Cord - drug effects | Spinal Cord - metabolism | Analgesics, Non-Narcotic - pharmacology | Neuralgia - metabolism | Periaqueductal Gray - drug effects | Ceftriaxone - pharmacology | Male | Neural Pathways - pathology | Neuralgia - pathology | Touch | Hyperalgesia - pathology | Spinal Cord - pathology | Neuralgia - etiology | Excitatory Amino Acid Transporter 2 - genetics | Disease Models, Animal | Hyperalgesia - metabolism | Excitatory Amino Acid Transporter 2 - deficiency | Mice, Inbred C57BL | Mice, Transgenic | Hot Temperature | Neuralgia - drug therapy | Periaqueductal Gray - metabolism | Animals | Hyperalgesia - drug therapy | Neural Pathways - metabolism | Periaqueductal Gray - pathology | Amino acids | Care and treatment | Pain | Glutamate | Brain | Spinal cord | Hypersensitivity | Periaqueductal gray area | Neuropathy | Ceftriaxone | Spinal cord injury | Neuronal-glial interactions | Rodents | Sensory neurons | Mice | Glutamatergic transmission | Glutamic acid transporter | Transporter | Neurotransmission | Injuries | Dorsal horn
Journal Article
Glia, ISSN 0894-1491, 04/2018, Volume 66, Issue 4, pp. 777 - 788
Glial glutamate transporter GLT1 plays a key role in the maintenance of extracellular glutamate homeostasis. Recent human genetic studies have suggested that... 
conditional knockout mice | sudden unexpected death in epilepsy (SUDEP) | seizure‐induced cell death | infantile epilepsy | NMDA receptor | astrocyte | seizure-induced cell death | ENCEPHALOPATHIES | GRIN2A MUTATIONS | PURKINJE CELL SYNAPSES | SPINAL-CORD | MODEL | NEUROSCIENCES | DE-NOVO MUTATIONS | EPILEPSY | DYSFUNCTION | EXPRESSION | BRAIN | Neurons - pathology | Homeodomain Proteins - metabolism | Receptors, N-Methyl-D-Aspartate - metabolism | Seizures - drug therapy | Male | Seizures - metabolism | Brain - growth & development | Anticonvulsants - pharmacology | Brain - metabolism | Seizures - pathology | Forkhead Transcription Factors - metabolism | Excitatory Amino Acid Transporter 2 - genetics | Neurons - metabolism | Disease Models, Animal | Neurodegenerative Diseases - pathology | Excitatory Amino Acid Transporter 2 - deficiency | Mice, Inbred C57BL | Nuclear Proteins - metabolism | Neurodegenerative Diseases - metabolism | Transcription Factors - genetics | Forkhead Transcription Factors - genetics | Homeodomain Proteins - genetics | Mice, Knockout | Brain - drug effects | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Animals | Cell Death - physiology | Brain - pathology | Pentylenetetrazole | Pyrazines - pharmacology | Seizures (Medicine) | Glutamate | Neurons | Cell death | Epilepsy | Brain | Spinal cord | Glutamic acid receptors (ionotropic) | Pathogenesis | Brain stem | Body weight | Forebrain | Homeostasis | Recombinase | Neuronal-glial interactions | Receptors | Clonal deletion | Neurodegeneration | Rodents | Deletion | Ventricular zone | Age | Seizures | Seizing | Cortex | N-Methyl-D-aspartic acid receptors | Cre recombinase | Mice | Glutamic acid transporter | Ventricle | Mutation | Diencephalon | Transporter
Journal Article
Psychopharmacology, ISSN 0033-3158, 5/2018, Volume 235, Issue 5, pp. 1371 - 1387
GLT-1 is the major glutamate transporter in the brain and is expressed predominantly in astrocytes but is also present in excitatory axon terminals. To... 
Neurosciences | Biomedicine | Amphetamine | Dopamine | Glutamate transporter | Glutamate homeostasis | Locomotor response | Behavior | Pharmacology/Toxicology | Glutamate uptake | Psychiatry | EXTRACELLULAR DOPAMINE | PREPULSE INHIBITION | PSYCHIATRY | ALZHEIMERS-DISEASE | IN-SITU HYBRIDIZATION | COCAINE | PSYCHOMOTOR STIMULANTS | SYNAPTIC PLASTICITY | NEUROSCIENCES | VENTRAL TEGMENTAL AREA | RAT NUCLEUS-ACCUMBENS | PHARMACOLOGY & PHARMACY | GROUP-II | Nucleus Accumbens - metabolism | Fear - drug effects | Hippocampus - drug effects | Mice, 129 Strain | Fear - physiology | Gene Deletion | Female | Excitatory Amino Acid Transporter 2 - genetics | Neurons - metabolism | Neurons - drug effects | Interpersonal Relations | Dopamine - metabolism | Locomotion - drug effects | Astrocytes - drug effects | Excitatory Amino Acid Transporter 2 - deficiency | Mice, Inbred C57BL | Amphetamine - pharmacology | Excitatory Amino Acid Transporter 2 - metabolism | Mice, Knockout | Hippocampus - metabolism | Phenotype | Animals | Mice | Locomotion - physiology | Nucleus Accumbens - drug effects | Astrocytes - metabolism | Neurons | Analysis | Phenols | Amino acids | Social aspects | Glutamate | Central nervous system agents | Neurophysiology | Presynapse | Drugs | Brain | Drug abuse | Memory | Cognitive ability | Homeostasis | Recombinase | Neurogenesis | Learning | Clonal deletion | Rodents | Deletion | Gating | Locomotor activity | Phenotypes | Astrocytes | Abnormalities | Nucleus accumbens | Sensorimotor gating | Synapsin | Psychopharmacology | Phenotyping | Amphetamines | Glutamic acid transporter | Transporter | Hippocampus | glutamate transporter | locomotor response | glutamate homeostasis | glutamate uptake | amphetamine | behavior | dopamine
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e36853
Background: Brains of patients with schizophrenia show both neurodevelopmental and functional deficits that suggest aberrant glutamate neurotransmission.... 
GLAST | AUTISM | DEVELOPING RAT | GLUTAMATE | BIOLOGY | GENE-EXPRESSION | INJURY | SCHIZOPHRENIA | MICE | NEUROPATHOLOGY | PREFRONTAL CORTEX | Brain - embryology | Receptors, N-Methyl-D-Aspartate - metabolism | DNA Primers - genetics | Olfactory Bulb - metabolism | Brain - abnormalities | Excitatory Amino Acid Transporter 1 - genetics | Olfactory Bulb - embryology | Cerebral Cortex - metabolism | Receptors, N-Methyl-D-Aspartate - genetics | Brain - metabolism | Base Sequence | Female | Excitatory Amino Acid Transporter 2 - genetics | Cerebral Cortex - abnormalities | Excitatory Amino Acid Transporter 1 - deficiency | Hippocampus - embryology | Excitatory Amino Acid Transporter 2 - deficiency | Excitatory Amino Acid Transporter 1 - metabolism | Excitatory Amino Acid Transporter 2 - metabolism | Mice, Knockout | Pregnancy | Hippocampus - metabolism | Animals | Cerebral Cortex - embryology | Receptors, N-Methyl-D-Aspartate - deficiency | Mice | Hippocampus - abnormalities | Olfactory Bulb - abnormalities | Viral antibodies | Brain | Embryonic development | Neurons | Pregnant women | Antibodies | Schizophrenia | Amino acids | Glutamate | Neurophysiology | Neurosciences | Traumatic brain injury | Mental disorders | Laboratories | Glutamic acid receptors (ionotropic) | Intellectual disabilities | Genes | Science | Genomes | Glutamic acid receptors | Paraffin | Defects | Consortia | Psychotropic drugs | Receptors | Developmental stages | Clonal deletion | Rodents | Microtubule-associated protein 2 | Deletion | Thalamus | Medical research | Cortex | N-Methyl-D-aspartic acid receptors | Pharmacology | Embryos | Autism | Signaling | Brain research | Hypoxia | Olfactory bulb | Glutamic acid transporter | Glutamatergic transmission | Aberration | Transporter | Neurotransmission | Hippocampus
Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 11/2008, Volume 11, Issue 11, pp. 1294 - 1301
Cellular abnormalities in amyotrophic lateral sclerosis (ALS) are not limited to motor neurons. Astrocyte dysfunction also occurs in human ALS and transgenic... 
GLIAL-RESTRICTED PRECURSORS | TRANSGENIC RAT MODEL | GLUTAMATE TRANSPORTERS | ADULT RATS | MOUSE MODEL | EXTEND SURVIVAL | ALS MODEL | AMYOTROPHIC-LATERAL-SCLEROSIS | HNT NEURONS | NEUROSCIENCES | STEM-CELL GRAFTS | Superoxide Dismutase - genetics | Phrenic Nerve - physiopathology | Humans | Glial Fibrillary Acidic Protein - metabolism | Cell Movement - physiology | Hindlimb - physiopathology | Motor Neuron Disease - therapy | Spinal Cord - pathology | Motor Neuron Disease - physiopathology | Cell Differentiation - physiology | Cell Survival - physiology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Neural Conduction - genetics | Animals, Genetically Modified | Excitatory Amino Acid Transporter 2 - deficiency | Rats | Cell Transplantation | Spinal Cord - surgery | Motor Neuron Disease - pathology | Action Potentials - physiology | Astrocytes - physiology | Animals | Green Fluorescent Proteins - biosynthesis | Microfilament Proteins | Muscle, Skeletal - physiopathology | Stem Cells - physiology | Spinal Cord - physiopathology | Physiological aspects | Motor neurons | Nervous system | Care and treatment | Degeneration | Astrocytes | astroglia | grafting | neurodegeneration | ALS | neural precursor cell | progenitor | amyotrophic lateral sclerosis | SOD1 | non-cell autonomous | stem cell | motor neuron | neuroprotection | transplantation | lineage-restricted precursor | glial precursor | replacement | astrocyte
Journal Article