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Journal of Biological Chemistry, ISSN 0021-9258, 05/2013, Volume 288, Issue 22, pp. 15699 - 15711
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 412 - 427
Endothelial progenitor cells are known to reverse acute kidney injury by paracrine mechanisms. We previously found that microvesicles released from these... 
exosome | ischemia–reperfusion | acute kidney injury | ischemia-reperfusion | HORIZONTAL TRANSFER | STEM-CELLS | CONTRIBUTE | REGENERATION | FAILURE | EXPERIMENTAL GLOMERULONEPHRITIS | REPAIR | EPITHELIAL-CELLS | MESSENGER-RNA | UROLOGY & NEPHROLOGY | PLATELET-ACTIVATING-FACTOR | Chemotaxis, Leukocyte | Cell Proliferation | Epithelial Cells - metabolism | Kidney - blood supply | Capillaries - pathology | Kidney - pathology | Rats, Wistar | Endothelial Cells - transplantation | Cell-Derived Microparticles - transplantation | Male | MicroRNAs - metabolism | Acute Kidney Injury - genetics | Stem Cells - metabolism | Cell Hypoxia | Stem Cell Transplantation | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Cell-Derived Microparticles - pathology | Cell-Derived Microparticles - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Capillaries - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Ribonuclease III - genetics | Ribonuclease III - metabolism | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Endothelial Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Rats | Epithelial Cells - pathology | Acute Kidney Injury - prevention & control | Regeneration | Oligonucleotides - metabolism | Animals | Reperfusion Injury - prevention & control | Fibrosis | Stem Cells - pathology | Acute Kidney Injury - metabolism | Endothelial Cells - pathology | Apoptosis | Index Medicus | Cell proliferation | Intravenous administration | Paracrine signalling | mRNA | Leukocytes | Ribonuclease | Endothelial cells | Kidney | Angiogenesis | Ischemia | Stem cells | Hypoxia | miRNA | Capillaries | Injuries
Journal Article
Science Translational Medicine, ISSN 1946-6234, 12/2015, Volume 7, Issue 318, pp. 318ra200 - 318ra200
Autoantibodies to components of apoptotic cells, such as anti-perlecan antibodies, contribute to rejection in organ transplant recipients. However, mechanisms... 
SOLID-ORGAN TRANSPLANTATION | MEDICINE, RESEARCH & EXPERIMENTAL | ACUTE KIDNEY INJURY | SYSTEMIC-LUPUS-ERYTHEMATOSUS | CORONARY-ARTERY DISEASE | EXTRACELLULAR VESICLES | ANTIVIMENTIN ANTIBODIES | PERLECAN FRAGMENT LG3 | NON-HLA ANTIBODIES | MESENCHYMAL STEM-CELLS | CHRONIC ALLOGRAFT NEPHROPATHY | CELL BIOLOGY | Cell-Derived Microparticles - enzymology | Humans | Muscle, Smooth, Vascular - immunology | Immunity, Humoral | Ischemia - immunology | Time Factors | Peptide Fragments - immunology | Exosomes - immunology | Acute Kidney Injury - immunology | Proteomics - methods | Graft Rejection - pathology | Proteasome Endopeptidase Complex - immunology | Ischemia - pathology | Aorta - transplantation | Disease Models, Animal | Biomarkers - metabolism | Acute Kidney Injury - pathology | Myocytes, Smooth Muscle - enzymology | Rats | Aorta - immunology | Acute Kidney Injury - enzymology | Aorta - pathology | Human Umbilical Vein Endothelial Cells - enzymology | Exosomes - enzymology | Human Umbilical Vein Endothelial Cells - pathology | Mice, Inbred BALB C | Graft Rejection - immunology | Proteasome Endopeptidase Complex - metabolism | Muscle, Smooth, Vascular - enzymology | Ischemia - enzymology | Myocytes, Smooth Muscle - pathology | Human Umbilical Vein Endothelial Cells - immunology | Exosomes - pathology | Autoantibodies - biosynthesis | Allografts | Cell-Derived Microparticles - pathology | Kidney Tubules, Proximal - enzymology | Aorta - enzymology | Graft Rejection - enzymology | Kidney Tubules, Proximal - immunology | Kidney Tubules, Proximal - pathology | Peptide Fragments - metabolism | Mice, Inbred C57BL | Cells, Cultured | Heparan Sulfate Proteoglycans - immunology | Autoantibodies - immunology | Muscle, Smooth, Vascular - pathology | Animals | Apoptosis - immunology | Cell-Derived Microparticles - immunology | Heparan Sulfate Proteoglycans - metabolism | Myocytes, Smooth Muscle - immunology | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2013, Volume 110, Issue 18, pp. 7312 - 7317
Hypoxia, or low oxygen tension, is a major regulator of tumor development and aggressiveness. However, how cancer cells adapt to hypoxia and communicate with... 
Angiogenesis | Messenger RNA | Glioma | Cell lines | Hypoxia | Exosomes | Brain hypoxia | Endothelial cells | Tumors | Cancer | Blood vessels | CNS | Biomarker | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | blood vessels | MICROVESICLES | biomarker | CANCER | GLIOBLASTOMA | AUTOCRINE VEGF | MICROENVIRONMENT | ENDOTHELIAL-CELLS | GROWTH | GENE-EXPRESSION | CAVEOLIN-1 | Exosomes - metabolism | Blood Vessels - pathology | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Brain Neoplasms - blood supply | Neoplasm Proteins - metabolism | Neovascularization, Pathologic - pathology | Glioma - genetics | Cell Transformation, Neoplastic - genetics | Pericytes - pathology | Glioma - pathology | Female | Autocrine Communication | Paracrine Communication | Endothelial Cells - metabolism | Pericytes - metabolism | Brain Neoplasms - genetics | Signal Transduction - genetics | Transcriptome - genetics | Mice, SCID | Glioma - blood supply | Xenograft Model Antitumor Assays | Animals | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Mice | Tissue Donors | Cell Hypoxia - genetics | Cell Transformation, Neoplastic - pathology | Endothelial Cells - pathology | Proteome - metabolism | Physiological aspects | Metastasis | Research | Gliomas | Health aspects | Cancer cells | Signal transduction | Genotype & phenotype | RNA-protein interactions | Cellular biology | Index Medicus | Biological Sciences | Clinical Medicine | Basic Medicine | Neurologi | Neurology | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Cell and Molecular Biology | Klinisk medicin | Cancer and Oncology | Cell- och molekylärbiologi | Cancer och onkologi
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 131, Issue 4, pp. 1194 - 1203.e14
Background Exosomes are nanovesicles involved in intercellular communication. Their roles in various diseases are often contextual, depending on the cell type... 
Allergy and Immunology | monocytes | allergy | epithelial cells | IL-13 | exosomes | Asthma | ACTIVATION | DENDRITIC CELLS | MACROPHAGES | IMMUNOLOGY | MATURATION | MEMBRANE-VESICLES | IMMUNE-RESPONSES | IN-VIVO | MICE | SUPPRESSOR-CELLS | T-CELLS | Exosomes - metabolism | Inflammation - pathology | Asthma - metabolism | Epithelial Cells - metabolism | Interleukin-13 - immunology | Exosomes - pathology | Male | Monocytes - metabolism | Monocytes - immunology | Respiratory Mucosa - pathology | Inflammation - metabolism | Respiratory Mucosa - immunology | Asthma - immunology | Exosomes - immunology | Monocytes - pathology | Lung - metabolism | Chemotaxis - immunology | Macrophages - immunology | Lung - pathology | Aniline Compounds - pharmacology | Macrophages - pathology | Cells, Cultured | Epithelial Cells - pathology | Inflammation - immunology | Chemotaxis - drug effects | Interleukin-13 - pharmacology | Cell Differentiation - immunology | Macrophages - metabolism | Animals | Benzylidene Compounds - pharmacology | Cell Differentiation - drug effects | Epithelial Cells - immunology | Mice | Mice, Inbred BALB C | Bronchoalveolar Lavage Fluid - chemistry | Respiratory Mucosa - metabolism | Asthma - pathology | Lung - immunology | Immunogenetics | Proteins | Enzymes | Flow cytometry | Tuberculosis | Lymphocytes | Experiments | Immune system | Communication | Index Medicus | Abridged Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 06/2015, Volume 17, Issue 6, pp. 816 - 826
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2018, Volume 20, Issue 3, pp. 307 - 319
Reactive oxygen species (ROS) contribute to tissue damage and remodelling mediated by the inflammatory response after injury. Here we show that ROS, which... 
STEM-CELLS | PHAGOCYTOSIS | ACTIVATION | SUPEROXIDE-PRODUCTION | ROS | KINASE | PTEN | NEURONS | MECHANISMS | OUTGROWTH | CELL BIOLOGY | Ganglia, Spinal - injuries | Sciatic Nerve - injuries | Axons - enzymology | Reactive Oxygen Species - metabolism | Exosomes - pathology | Sciatic Nerve - physiopathology | Spinal Cord Injuries - genetics | Spinal Cord Injuries - pathology | Endocytosis | CX3C Chemokine Receptor 1 - metabolism | Ganglia, Spinal - enzymology | Peripheral Nerve Injuries - physiopathology | Spinal Cord Injuries - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Dyneins - metabolism | Cell Line | NADPH Oxidase 2 - deficiency | Macrophages - pathology | Signal Transduction | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Endosomes - pathology | Nerve Regeneration | Macrophages - enzymology | Mice, Knockout | Animals | Peripheral Nerve Injuries - enzymology | NADPH Oxidase 2 - metabolism | Axons - pathology | Ganglia, Spinal - pathology | Exosomes - enzymology | Endosomes - enzymology | Spinal Cord Injuries - physiopathology | NADPH Oxidase 2 - genetics | Nerve Degeneration | Peripheral Nerve Injuries - genetics | Sciatic Nerve - enzymology | Axons | Regeneration | Reactive oxygen species | NADP (Coenzyme) | Nervous system | Cellular signal transduction | Research | Gene expression | Oxidase | Recovery of function | Spinal injuries | CX3CR1 protein | Inflammatory response | AKT protein | Exosomes | Macrophages | Inactivation | NAD(P)H oxidase | Signal transduction | Dorsal root ganglia | Neurodegeneration | Dieback | CYBB protein | Degeneration | Cell body | Oxygen | Deactivation | Inflammation | Ganglia | 1-Phosphatidylinositol 3-kinase | Signaling | Dynein | PTEN protein | Sciatic nerve | Endosomes | Index Medicus
Journal Article
Journal Article