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2000, Methods in molecular biology, ISBN 0896036243, Volume 139, xiv, 370
It is now widely accepted that much of the dynamic function of cells and tissues is regulated from outside the cell by the extracellular matrix (ECM). In... 
Extracellular matrix | Extracellular matrix proteins | Laboratory manuals | Cytology | Cell Biology | Life Sciences
Book
2017, Methods in molecular biology, ISBN 9781493972531, Volume 1660.
This volume examines established methods and protocols to isolate and characterize extracellular vesicles (EVs) and their composition, among other techniques... 
Extracellular Vesicles
Web Resource
2017, Methods in molecular biology, ISBN 9781493972531, Volume 1660.
This volume examines established methods and protocols to isolate and characterize extracellular vesicles (EVs) and their composition, among other techniques... 
Extracellular Vesicles
Web Resource
Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 36, pp. 24035 - 24048
The role of endogenous inducers of inflammation is poorly understood. To produce the proinflammatory master cytokine interleukin (IL)-1β, macrophages need... 
GENE | LEUCINE-RICH PROTEOGLYCANS | CASPASE-1 | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE IMMUNE-RESPONSE | HYALURONAN | IL-1-BETA | SECRETION | ATP | NALP3 INFLAMMASOME | Toll-Like Receptor 2 - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Immunity, Innate - genetics | Extracellular Matrix - metabolism | Caspase 1 - metabolism | Male | Ureteral Obstruction - metabolism | Interleukin-1beta - genetics | Inflammation - metabolism | Kidney - metabolism | Carrier Proteins - immunology | Signal Transduction | Proteoglycans - immunology | Extracellular Matrix Proteins - genetics | Proteoglycans - metabolism | Toll-Like Receptor 4 - genetics | Biglycan | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Ureteral Obstruction - immunology | Macrophages - metabolism | Receptors, Purinergic P2 - metabolism | Toll-Like Receptor 2 - immunology | Mice | Proteoglycans - genetics | Extracellular Matrix - immunology | Kidney - immunology | Caspase 1 - immunology | Extracellular Matrix - genetics | Receptors, Purinergic P2 - genetics | Ureteral Obstruction - genetics | Interleukin-1beta - biosynthesis | Lung - metabolism | Receptors, Purinergic P2 - immunology | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Carrier Proteins - biosynthesis | Interleukin-1beta - immunology | Inflammation - immunology | Protein Structure, Tertiary - genetics | Receptors, Purinergic P2X7 | Carrier Proteins - genetics | Immunity, Innate - immunology | Animals | Caspase 1 - genetics | Extracellular Matrix Proteins - immunology | Receptors, Purinergic P2X4 | Inflammation - genetics | Lung - immunology | Animal models | Reactive oxygen species | Oligomerization | Proteoglycans | Transcription | Lung | Inflammation | Macrophages | Kidney | Stress | Caspase-1 | Purine P2X receptors | Signal transduction | Cell activation | TLR2 protein | Interleukin 1 | Toll-like receptors | Extracellular matrix | Sepsis | Cooperativity | Injuries | Index Medicus | Glycobiology and Extracellular Matrices
Journal Article
2009, 2nd ed., Methods in molecular biology, ISBN 9781597454131, Volume 522., xi, 375
Book
Journal of Clinical Investigation, ISSN 0021-9738, 04/2014, Volume 124, Issue 4, pp. 1622 - 1635
Journal Article
Brain, ISSN 0006-8950, 2010, Volume 133, Issue 8, pp. 2331 - 2347
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 12/2009, Volume 28, Issue 49, pp. 4326 - 4343
Mammographically dense breast tissue is one of the greatest risk factors for developing breast carcinoma, yet the associated molecular mechanisms remain... 
3D matrix/focal adhesion | Breast cancer | Mechanotransduction | Epithelial morphogenesis | Cell-extracellular matrix interaction | Breast tissue density | CANCER RISK | ACTIVATED PROTEIN-KINASE | mechanotransduction | BIOCHEMISTRY & MOLECULAR BIOLOGY | breast cancer | MALIGNANT PHENOTYPE | CARCINOMA IN-SITU | CELL BIOLOGY | MAMMOGRAPHIC DENSITY | STRESS FIBERS | ONCOLOGY | epithelial morphogenesis | EPITHELIAL-SPECIFIC DISRUPTION | GROWTH-FACTORS | GENETICS & HEREDITY | EXTRACELLULAR-MATRIX | FOCAL ADHESION KINASE | breast tissue density | cell-extracellular matrix interaction | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Cell Count | Humans | Breast Neoplasms - etiology | Gene Expression Profiling | Extracellular Signal-Regulated MAP Kinases - metabolism | Mammary Glands, Human - metabolism | Mechanotransduction, Cellular - physiology | Carcinoma - etiology | Extracellular Matrix - chemistry | Breast Neoplasms - metabolism | Cell Transformation, Neoplastic - genetics | Extracellular Matrix - physiology | Gene Expression - physiology | Female | Mammary Glands, Human - physiology | Extracellular Signal-Regulated MAP Kinases - physiology | Focal Adhesion Kinase 1 - metabolism | Mechanotransduction, Cellular - genetics | Mammary Glands, Human - cytology | Risk Factors | Cells, Cultured | Focal Adhesion Kinase 1 - physiology | Cell Transformation, Neoplastic - metabolism | Phenotype | Breast Neoplasms - genetics | Carcinoma - genetics | Carcinoma - metabolism | Physiological aspects | Development and progression | Genetic aspects | Research | Gene expression | Protein kinases | Risk factors | Genotype & phenotype | Cellular biology | Index Medicus | 3D-Matrix | Focal Adhesion | Cell-Extracellular Matrix Interaction | Epithelial Morphogenesis | Breast Cancer | Breast Tissue Density | Tumor-Stroma Interaction
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 08/2011, Volume 108, Issue 32, pp. E440 - E449
Astrocytes regulate synaptic connectivity in the CNS through secreted signals. Here we identified two astrocyte-secreted proteins, hevin and SPARC, as... 
Extracellular matrix | Synaptic cleft 1 | Matricellular protein | Osteonectin | SPARC-Like | osteonectin | SC1 | synaptic cleft 1 | RAT | EXTRACELLULAR-MATRIX GLYCOPROTEIN | INTEGRIN | MULTIDISCIPLINARY SCIENCES | RECEPTOR | matricellular protein | CELL-SURVIVAL | SUPERIOR COLLICULUS | SPARC-Like 1 | EXPRESSION | extracellular matrix | SUPERFICIAL LAYERS | BRAIN | Central Nervous System - ultrastructure | Central Nervous System - metabolism | Extracellular Matrix Proteins - deficiency | Humans | Superior Colliculi - ultrastructure | Calcium-Binding Proteins - antagonists & inhibitors | Culture Media, Conditioned - pharmacology | Retinal Ganglion Cells - metabolism | Retinal Ganglion Cells - cytology | Osteonectin - deficiency | Synapses - metabolism | HEK293 Cells | Neurogenesis - drug effects | Extracellular Matrix Proteins - metabolism | Astrocytes - cytology | Calcium-Binding Proteins - chemistry | Calcium-Binding Proteins - metabolism | Protein Structure, Tertiary | Astrocytes - drug effects | Retinal Ganglion Cells - ultrastructure | Synapses - drug effects | Extracellular Matrix Proteins - chemistry | Superior Colliculi - metabolism | Extracellular Matrix Proteins - antagonists & inhibitors | Superior Colliculi - drug effects | Osteonectin - metabolism | Rats | Calcium-Binding Proteins - deficiency | Synapses - ultrastructure | Osteonectin - chemistry | Rats, Sprague-Dawley | Astrocytes - ultrastructure | Animals | Central Nervous System - cytology | Superior Colliculi - cytology | Mice | Retinal Ganglion Cells - drug effects | Astrocytes - secretion | Proteins | Brain | Rodents | Cells | Retinal ganglion cells | Synaptogenesis | Astrocytes | Neural networks | Central nervous system | Superior colliculus | Synapses | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, pp. e90565 - e90565
Airway remodelling describes the histopathological changes leading to fixed airway obstruction in patients with asthma and includes extra-cellular matrix (ECM)... 
SURVIVAL | GROWTH-FACTOR-BETA | COLLAGEN | ELASTASE | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | GENE-EXPRESSION | INTERLEUKIN-13 | FIBRONECTIN | CELL PROLIFERATION | TENASCIN-C | Tenascin - genetics | Gene Expression - drug effects | Humans | Extracellular Matrix - metabolism | Immunoblotting | Tenascin - metabolism | Tenascin - pharmacology | Dose-Response Relationship, Drug | Muscle, Smooth - drug effects | RNA Interference | Indoles - pharmacology | Myocytes, Smooth Muscle - drug effects | Bradykinin - pharmacology | Extracellular Matrix Proteins - metabolism | Matrix Metalloproteinase 1 - genetics | Vasodilator Agents - pharmacology | Asthma - physiopathology | Extracellular Matrix Proteins - genetics | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Extracellular Matrix Proteins - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Asthma - genetics | Hydroxamic Acids | Integrin beta1 - metabolism | Integrin beta3 - metabolism | Muscle Contraction - drug effects | Muscle, Smooth - physiopathology | Respiratory System - drug effects | Respiratory System - physiopathology | Collagen Type I - pharmacology | Matrix Metalloproteinase 1 - metabolism | Asthma - pathology | Mitogen-Activated Protein Kinases - metabolism | Viral antibodies | Antibodies | Smooth muscle | Collagen | Integrins | Asthma | Tenascin | Cell culture | Blocking antibodies | Matrix metalloproteinase | Remodeling | Experiments | Respiratory tract | Proteins | Genotype & phenotype | Airway management | Bradykinin | Extracellular matrix | Metalloproteinase | Growth factors | Interstitial collagenase | Gels | Cytokines | Tenascin C | Muscles | Extracellular signal-regulated kinase | MAP kinase | JNK protein | siRNA | Gene expression | Patients | Muscle contraction | Bundles | Medicine | Signaling | Biopsy | Index Medicus
Journal Article