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Journal of Immunology, ISSN 0022-1767, 01/2011, Volume 186, Issue 2, pp. 1022 - 1031
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, pp. e0164102 - e0164102
Objective Therapeutic agents that are transformable via introducing cleavable linkage by locally enriched MMP-2 within inflamed synovium would enhance... 
RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | CELLS | ACTIVATION | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | ALPHA-V-BETA-3 INTEGRIN | DISEASE | INDUCIBLE GENE H3 | ARRIVE GUIDELINES | DELIVERY | Recombinant Proteins - therapeutic use | NIH 3T3 Cells | Arthritis, Experimental - drug therapy | RANK Ligand - metabolism | Recombinant Fusion Proteins - pharmacology | Recombinant Fusion Proteins - therapeutic use | Molecular Sequence Data | Male | Recombinant Proteins - biosynthesis | RNA, Messenger - metabolism | Arthritis, Experimental - metabolism | Arthritis, Experimental - pathology | Transforming Growth Factor beta - therapeutic use | Mice, Inbred DBA | Vascular Cell Adhesion Molecule-1 - genetics | Chemokine CCL2 - metabolism | Extracellular Matrix Proteins - therapeutic use | Severity of Illness Index | Amino Acid Sequence | Matrix Metalloproteinase 2 - metabolism | Extracellular Matrix Proteins - genetics | Extracellular Matrix Proteins - pharmacology | Chemokine CCL2 - genetics | Recombinant Proteins - pharmacology | Cell Adhesion - drug effects | Down-Regulation - drug effects | RANK Ligand - genetics | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Animals | Transforming Growth Factor beta - genetics | Recombinant Fusion Proteins - genetics | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Chronic Disease | Microscopy, Fluorescence | Rheumatoid factor | Peptides | Proteases | Analysis | Collagen | Bone morphogenetic proteins | Arthritis | Glycine | Health aspects | Matrix metalloproteinases | Pathogenesis | Transforming growth factor | Matrix metalloproteinase | Synovium | Cell adhesion & migration | Proteins | Angiogenesis | Metalloproteinase | Localization | Growth factors | Enzymes | Sequences | Immunoglobulins | Effectiveness | Internal medicine | Cloning | Rheumatology | Pharmacology | Inflammation | Chemical compounds | Adhesion | Gelatinase A | Medicine | Rheumatoid arthritis | In vivo methods and tests | Cell migration | Index Medicus
Journal Article
Dental Materials, ISSN 0109-5641, 2011, Volume 27, Issue 5, pp. 465 - 477
Abstract Objectives This study examined the use of sodium trimetaphosphate (STMP) as a biomimetic analog of matrix phosphoproteins for remineralization of... 
Advanced Basic Science | Dentistry | Degradation | Biomimetic | Artificial carious lesion | Collagen | Remineralization | Sodium trimetaphosphate | CARIOUS DENTIN | MATERIALS SCIENCE, BIOMATERIALS | CONFOCAL RAMAN MICROSPECTROSCOPY | IN-VITRO REMINERALIZATION | LESIONS | CALCIUM-PHOSPHATE | CRYSTAL-GROWTH | DENTISTRY, ORAL SURGERY & MEDICINE | ADHESIVE SYSTEMS | BOND STRENGTH | 2 LAYERS | Cariostatic Agents - therapeutic use | Humans | Composite Resins - chemistry | Dental Cements - chemistry | Methacrylates - chemistry | Calcium - chemistry | X-Ray Microtomography | Tooth Remineralization - methods | Dental Caries - pathology | Collagen - drug effects | Time Factors | Phosphoproteins - therapeutic use | Extracellular Matrix Proteins - therapeutic use | Dentin-Bonding Agents - chemistry | Biomimetic Materials - therapeutic use | Microscopy, Electron, Transmission | Hydroxides - chemistry | Collagen - ultrastructure | Silicon Dioxide - chemistry | Calcium Phosphates - chemistry | Dentin - ultrastructure | Materials Testing | Minerals - analysis | Acrylic Resins - chemistry | Dentin - drug effects | Tooth Demineralization - drug therapy | Dental Caries - drug therapy | Tooth Demineralization - pathology | Saliva, Artificial - chemistry | Polyphosphates - therapeutic use | Phosphates | Dental caries | Usage | Biomimetics | Phosphoproteins | Dentin | Adhesives | Dental materials | Transmission electron microscopy | Sodium | Collagens | Nanostructure | Lesions | remineralization | collagen | sodium trimetaphosphate | degradation | biomimetic | artificial carious lesion
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2006, Volume 103, Issue 13, pp. 4946 - 4951
Microvascular dysfunction is a major cause of impaired wound healing seen in diabetic patients. Therefore, reestablishment of structural and functional... 
Angiogenesis | Wound healing | Diabetes complications | Blood vessels | Mice | Skin | Diabetes | Lymphatic vessels | Blood | Blood flow | Therapeutic protein | Nitric oxide | Cutaneous wound | Growth factor | LONG-TERM | therapeutic protein | nitric oxide | MULTIDISCIPLINARY SCIENCES | cutaneous wound | ANGIOPOIETIN-1 | growth factor | STIMULATES ANGIOGENESIS | REPAIR | COMP-ANG1 | NITRIC-OXIDE | MICE | COMPLICATIONS | diabetes | EXPRESSION | TIE2 RECEPTOR | Diabetes Mellitus - pathology | Recombinant Proteins - therapeutic use | Tail - pathology | Matrilin Proteins | Regional Blood Flow - drug effects | Tail - blood supply | Extracellular Matrix Proteins - therapeutic use | Ear - blood supply | Wound Healing - drug effects | Skin - pathology | Disease Models, Animal | Tail - drug effects | Glycoproteins - genetics | Wounds and Injuries - pathology | Wounds and Injuries - blood | Extracellular Matrix Proteins - genetics | Glycoproteins - therapeutic use | Mice, Inbred C57BL | Nitric Oxide Synthase Type II - deficiency | Diabetes Mellitus - drug therapy | Nitric Oxide Synthase Type III | Recombinant Proteins - genetics | Mice, Knockout | Lymphangiogenesis - drug effects | Skin - blood supply | Angiopoietin-1 - genetics | Animals | Nitric Oxide Synthase Type II - genetics | Wounds and Injuries - drug therapy | Angiopoietin-1 - therapeutic use | Ear - pathology | Skin - drug effects | Nitric Oxide Synthase Type II - metabolism | Blood circulation disorders | Research | Proteins | Cellular biology | Rodents | Injuries | Index Medicus | Biological Sciences
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 01/2017, Volume 18, Issue 1, pp. 65 - 65
After peripheral nerve injury, motor and sensory axons are able to regenerate but inaccuracy of target reinnervation leads to poor functional recovery.... 
BDNF | Motor axons | Extracellular matrix | Reinnervation | Neurotrophic factors | NGF | NT3 | Nerve regeneration | Sensory axons | TUBE REPAIR | FUNCTIONAL RECOVERY | RAT | NEURONS IN-VITRO | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | RELEASE | CHEMISTRY, MULTIDISCIPLINARY | NERVE GROWTH-FACTOR | PERIPHERAL-NERVE | NEURITE OUTGROWTH | neurotrophic factors | nerve regeneration | reinnervation | SCHWANN-CELLS | extracellular matrix | motor axons | sensory axons | Motor Neurons - physiology | Cells, Cultured | Extracellular Matrix Proteins - pharmacology | Rats | Nerve Growth Factors - administration & dosage | Axons - physiology | Rats, Sprague-Dawley | Nerve Growth Factors - therapeutic use | Nerve Growth Factors - pharmacology | Microspheres | Animals | Sensory Receptor Cells - physiology | Nerve Regeneration - drug effects | Female | Extracellular Matrix Proteins - therapeutic use | Peripheral Nerve Injuries - drug therapy | Extracellular Matrix Proteins - administration & dosage | Motor neurons | Spinal cord | Axonogenesis | Brain slice preparation | Recovery of function | Nerve growth factor | Neurotrophin 3 | Motor task performance | Fibronectin | Polylactide-co-glycolide | Regeneration | Laminin | Brain-derived neurotrophic factor | Rodents | Glycolic acid | Collagen | Sensory neurons | Axon guidance | Silicones | Sciatic nerve | Index Medicus
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 09/2013, Volume 28, Issue 9, pp. 1912 - 1924
(PRELP)-P-hbd is a peptide corresponding to the N-terminal heparin binding domain of the matrix protein proline/arginine-rich end leucine-rich repeat protein... 
PRELP | BONE LOSS | BREAST CANCER METASTASIS | hbd | OSTEOPOROSIS | (PRELP)-P-hbd | PRELP BINDS | DENOSUMAB | ENDOCRINOLOGY & METABOLISM | CANCER | Bone and Bones - pathology | Humans | Bone Neoplasms - secondary | Osteoporosis - drug therapy | Structure-Activity Relationship | Glycoproteins - pharmacology | Bone and Bones - drug effects | Bone and Bones - metabolism | Protein Binding - drug effects | Mammary Neoplasms, Experimental - pathology | Extracellular Matrix Proteins - adverse effects | Female | Bone Neoplasms - drug therapy | Extracellular Matrix Proteins - therapeutic use | Glycoproteins - chemistry | Mammary Neoplasms, Experimental - complications | Extracellular Matrix Proteins - chemistry | Osteoclasts - pathology | Glycoproteins - therapeutic use | Bone Resorption - drug therapy | Extracellular Matrix Proteins - pharmacology | Glycoproteins - adverse effects | Bone Resorption - complications | Osteoclasts - metabolism | Animals | Osteoporosis - pathology | Mice, Nude | Osteoporosis - complications | Bone Resorption - pathology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Osteoclasts - drug effects | Glycosaminoglycans | Peptides | Analysis | Estrogen | Amino acids | Development and progression | Anticoagulants (Medicine) | Sulfates | Protein binding | Tretinoin | Tumors | Proteins | Osteoporosis | Breast cancer | Index Medicus | Rheumatology and Autoimmunity | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Reumatologi och inflammation | Klinisk medicin
Journal Article
Journal Article
Journal Article
Experimental Neurology, ISSN 0014-4886, 2009, Volume 216, Issue 2, pp. 390 - 397
Temporal lobe epilepsy (TLE) is often accompanied by granule cell dispersion (GCD), a migration defect of granule cells in the dentate gyrus. We have... 
Neuronal migration | Dentate gyrus | dab1 | apoER2 | Temporal lobe epilepsy | Extracellular matrix | VLDLR | Neurogenesis | Ammon's horn sclerosis | LIPOPROTEIN RECEPTORS | PROTEIN | TEMPORAL-LOBE EPILEPSY | NEUROSCIENCES | DISRUPTION | NEURONAL MIGRATION DISORDERS | HIPPOCAMPAL SCLEROSIS | MOUSE MODEL | MICE | RADIAL GLIAL SCAFFOLD | Neuroprotective Agents - therapeutic use | Nerve Tissue Proteins - therapeutic use | LDL-Receptor Related Proteins | Male | RNA, Messenger - metabolism | Drug Delivery Systems | Receptors, Lipoprotein - metabolism | Adaptor Protein Complex 1 - metabolism | Adaptor Protein Complex 1 - genetics | Dentate Gyrus - drug effects | Time Factors | Cell Adhesion Molecules, Neuronal - therapeutic use | Cell Adhesion Molecules, Neuronal - metabolism | Extracellular Matrix Proteins - therapeutic use | Kainic Acid - toxicity | Neurons - drug effects | Extracellular Matrix Proteins - metabolism | Disease Models, Animal | Receptors, LDL - genetics | Mice, Inbred C57BL | Receptors, Lipoprotein - genetics | Receptors, LDL - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Cell Movement - drug effects | Animals | Cell Count - methods | Excitatory Amino Acid Agonists - toxicity | Analysis of Variance | Epilepsy - chemically induced | Serine Endopeptidases - therapeutic use | Epilepsy - drug therapy | Mice | Serine Endopeptidases - metabolism | Dentate Gyrus - pathology | Epilepsy - pathology | Neural Inhibition - drug effects | Prevention | Neurons | Epilepsy | Analysis | Index Medicus
Journal Article
Journal Article