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Immunity, ISSN 1074-7613, 03/2013, Volume 38, Issue 3, pp. 475 - 488
Follicular B cell survival requires signaling from BAFFR, a receptor for BAFF and the B cell antigen receptor (BCR). This “tonic” BCR survival signal is... 
TACI | AKT | BCMA | IMMUNOLOGY | MATURATION | ACTIVATING FACTOR | AUTOIMMUNE-DISEASE | SYK TYROSINE KINASE | MICE LACKING | FAMILY | ANTIGEN | Oligonucleotide Array Sequence Analysis | Protein-Tyrosine Kinases - metabolism | Cell Survival - genetics | Immunoblotting | Gene Expression Profiling | Intracellular Signaling Peptides and Proteins - metabolism | Receptors, Antigen, B-Cell - metabolism | Extracellular Signal-Regulated MAP Kinases - immunology | Phosphatidylinositol 3-Kinases - immunology | Phosphorylation - drug effects | B-Cell Activation Factor Receptor - genetics | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | 3-Phosphoinositide-Dependent Protein Kinases | B-Lymphocytes - metabolism | Protein-Serine-Threonine Kinases - metabolism | B-Cell Activation Factor Receptor - immunology | Cell Survival - drug effects | RNA, Untranslated | Mice, Transgenic | Mice, Knockout | B-Cell Activation Factor Receptor - metabolism | Models, Immunological | B-Lymphocytes - immunology | Signal Transduction - drug effects | Mice | CD79 Antigens - immunology | Intracellular Signaling Peptides and Proteins - immunology | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Protein-Tyrosine Kinases - immunology | Signal Transduction - immunology | Protein-Tyrosine Kinases - genetics | Flow Cytometry | B-Cell Activating Factor - immunology | Protein-Serine-Threonine Kinases - genetics | Cell Survival - immunology | Proteins - immunology | Receptors, Antigen, B-Cell - immunology | CD79 Antigens - metabolism | Proteins - genetics | Receptor Cross-Talk - immunology | B-Lymphocytes - drug effects | Animals | Proteins - metabolism | Tamoxifen - pharmacology | Protein-Serine-Threonine Kinases - immunology | B-Cell Activating Factor - pharmacology | Proteins | Genetic research | Cells | Bone marrow | Antigens | Kinases | Rodents | Immune system | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 11/2008, Volume 205, Issue 12, pp. 2763 - 2779
Progressive loss of T cell functionality is a hallmark of chronic infection with human immunodeficiency virus 1 (HIV-1). We have identified a novel population... 
MEDICINE, RESEARCH & EXPERIMENTAL | SKEWED MATURATION | ACTIVATION | MULTIPLE-SCLEROSIS | IMMUNODEFICIENCY-VIRUS-INFECTION | IMPAIRED FUNCTION | CD4(+) | IMMUNOLOGY | CHRONIC VIRAL-INFECTION | DISEASE PROGRESSION | LYMPHOCYTES | ANTIGEN | GATA3 Transcription Factor - genetics | T-Lymphocyte Subsets - immunology | Antigens, CD - immunology | HIV-1 - pathogenicity | T-Lymphocyte Subsets - cytology | GATA3 Transcription Factor - immunology | Humans | STAT5 Transcription Factor - immunology | T-Box Domain Proteins - immunology | Antigens, CD - genetics | Extracellular Signal-Regulated MAP Kinases - genetics | Extracellular Signal-Regulated MAP Kinases - immunology | HIV Infections - immunology | STAT5 Transcription Factor - genetics | Signal Transduction - immunology | HIV Infections - pathology | Apoptosis Regulatory Proteins - genetics | Apoptosis Regulatory Proteins - immunology | Membrane Proteins - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Membrane Proteins - immunology | p38 Mitogen-Activated Protein Kinases - immunology | Hepatitis A Virus Cellular Receptor 2 | T-Box Domain Proteins - genetics | Disease Progression | Antiretroviral Therapy, Highly Active | HIV-1 - immunology | Phenotype | Animals | T-Lymphocytes - cytology | HIV Infections - drug therapy | Programmed Cell Death 1 Receptor | T-Lymphocytes - immunology | HIV Infections - physiopathology | HLA Antigens | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 699 - 714
Journal Article
Nature Immunology, ISSN 1529-2908, 06/2011, Volume 12, Issue 6, pp. 544 - 550
The molecular mechanisms that underlie T cell quiescence are poorly understood. Here we report that mature naive CD8(+) T cells lacking the transcription... 
SURVIVAL | ACTIVATION | PATHWAY | IN-VIVO | GENE-EXPRESSION | IL-7 RECEPTOR | MICE | IMMUNOLOGY | HOMEOSTATIC PROLIFERATION | INTERLEUKIN-7 RECEPTOR | THYMOCYTE | Forkhead Transcription Factors - immunology | Cell Proliferation | Nitriles - pharmacology | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Immunoblotting | Interleukin-7 - pharmacology | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - immunology | Receptors, Interleukin-7 - genetics | Flow Cytometry | T-Lymphocytes - metabolism | Forkhead Transcription Factors - metabolism | T-Lymphocytes - drug effects | CD8-Positive T-Lymphocytes - metabolism | Female | MAP Kinase Kinase Kinases - antagonists & inhibitors | Receptors, Interleukin-7 - immunology | Repressor Proteins - metabolism | Butadienes - pharmacology | Receptors, Interleukin-7 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Repressor Proteins - genetics | Mice, Transgenic | Imidazoles - pharmacology | MAP Kinase Kinase Kinases - metabolism | Forkhead Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | MAP Kinase Kinase Kinases - immunology | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | CD8-Positive T-Lymphocytes - drug effects | Repressor Proteins - immunology | Protein Binding | T-Lymphocytes - immunology | Forkhead Box Protein O1 | Mice | Pyridines - pharmacology | CD8-Positive T-Lymphocytes - immunology | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2013, Volume 288, Issue 38, pp. 27519 - 27532
The C-type lectin macrophage galactose-type lectin (MGL) exerts an immunosuppressive role reflected by its interaction with terminal GalNAc moieties, such as... 
PROTEIN | GENE | RECOGNITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | O-GLYCOSYLATION | GLYCANS | GNT-V | CHAPERONE | TRANSCRIPTIONAL REGULATION | CD45 | EXPRESSION | Protein Kinase C - genetics | Glucosyltransferases - immunology | Leukocyte Common Antigens - metabolism | Humans | Lectins, C-Type - immunology | Antigens, Tumor-Associated, Carbohydrate - immunology | Galactosyltransferases - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Lectins, C-Type - genetics | Extracellular Signal-Regulated MAP Kinases - genetics | MAP Kinase Signaling System - immunology | Extracellular Signal-Regulated MAP Kinases - immunology | Galactosyltransferases - immunology | CD4-Positive T-Lymphocytes - immunology | Leukocyte Common Antigens - immunology | Lectins, C-Type - metabolism | Calcineurin - genetics | MAP Kinase Signaling System - genetics | Calcineurin - immunology | Protein Kinase C - metabolism | Molecular Chaperones - immunology | Glucosyltransferases - genetics | Molecular Chaperones - biosynthesis | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Molecular Chaperones - genetics | Galactosyltransferases - biosynthesis | Protein Kinase C - immunology | Antigens, Tumor-Associated, Carbohydrate - metabolism | Antigens, Tumor-Associated, Carbohydrate - genetics | Leukocyte Common Antigens - genetics | Glucosyltransferases - biosynthesis | Lymphocyte Activation - physiology | Calcineurin - metabolism | Index Medicus | Lectin | Signaling | C-type Lectin | T Cell | Glycobiology and Extracellular Matrices | Glycosylation | ERK
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 10, pp. 6269 - 6281
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 12/2013, Volume 94, Issue 6, pp. 1113 - 1121
AMPK attenuates CD40‐mediated inflammatory activity in myeloid APC; its activity in both APC and T cells contributes to T cell functional polarization. AMPK is... 
antigen processing/presentation | Th1/Th2 cells | monocytes/macrophages | costimulation | inflammation | Inflammation | Monocytes/macrophages | Costimulation | Antigen processing/presentation | DC | Myeloid Cells - cytology | Myeloid Cells - enzymology | AMP-Activated Protein Kinases - metabolism | Antigen-Presenting Cells - cytology | Interleukin-17 - immunology | Extracellular Signal-Regulated MAP Kinases - metabolism | Th1 Cells - immunology | Extracellular Signal-Regulated MAP Kinases - genetics | MAP Kinase Signaling System - immunology | Transcription Factor RelA - genetics | Extracellular Signal-Regulated MAP Kinases - immunology | CD40 Antigens - metabolism | MAP Kinase Signaling System - genetics | Antigen Presentation | AMP-Activated Protein Kinases - immunology | Myeloid Cells - immunology | Transcription Factor RelA - immunology | Th17 Cells - cytology | CD40 Antigens - genetics | Th17 Cells - enzymology | Interleukin-17 - genetics | Antigen-Presenting Cells - immunology | Mice, Knockout | Interleukin-17 - metabolism | Animals | MAP Kinase Signaling System - drug effects | Transcription Factor RelA - metabolism | Lipopolysaccharides - pharmacology | Th17 Cells - immunology | Th1 Cells - enzymology | Mice | Th1 Cells - cytology | AMP-Activated Protein Kinases - genetics | Antigen-Presenting Cells - enzymology | CD40 Antigens - immunology | Index Medicus | presentation | monocytes | macrophages | Spotlight on Leading Edge Research | antigen processing | Th1 | Th2 cells
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2016, Volume 291, Issue 38, pp. 20030 - 20041
Journal Article