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1. Ocular immunology and inflammation
International Ocular Inflammation Society
Ocular immunology and inflammation, ISSN 0927-3948, 1993
Eye | Immunological aspects | Inflammation | Diseases
Journal
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2. Full Text A rush to judgment on Th17
by Steinman, Lawrence
Journal of Experimental Medicine, ISSN 0022-1007, 07/2008, Volume 205, Issue 7, pp. 1517 - 1522
Some immunologists have characterized T helper (Th)17 T cells as the master mediators of tissue damage in a variety of pathological conditions. New data now... 
MEDICINE, RESEARCH & EXPERIMENTAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | GROWTH-FACTOR-BETA | ROR-GAMMA-T | MULTIPLE-SCLEROSIS | IFN-GAMMA | EFFECTOR RESPONSE | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | BIOLOGICAL-ACTIVITIES | IMMUNOLOGY | T-H-17 CELLS | T-HELPER-CELLS | Dendritic Cells - immunology | Humans | Central Nervous System - pathology | Encephalomyelitis, Autoimmune, Experimental - immunology | Interleukin-23 - genetics | Encephalomyelitis, Autoimmune, Experimental - genetics | Disease Susceptibility | Macrophages - pathology | Interleukin-17 - genetics | Interleukin-12 Subunit p35 - genetics | Interleukin-23 - immunology | Mice, Knockout | Models, Immunological | Up-Regulation - immunology | Uveitis - immunology | Mice | Uveitis - genetics | Proteins - antagonists & inhibitors | Eye - pathology | Chemokines, CXC - immunology | Interleukin-17 - immunology | Autoimmunity - genetics | Neutralization Tests | Adoptive Transfer | Paralysis - pathology | Th1 Cells - immunology | Central Nervous System - immunology | Eye - immunology | Nitric Oxide Synthase Type II - immunology | Female | Interferon-gamma - genetics | Macrophages - immunology | Neutrophils - pathology | Inflammation Mediators - immunology | Cell Line | Encephalomyelitis, Autoimmune, Experimental - pathology | Myelin Sheath - immunology | Myelin Sheath - pathology | Gene Expression Regulation - immunology | Uveitis - prevention & control | Mice, Inbred C57BL | Neutrophils - immunology | Paralysis - immunology | Up-Regulation - genetics | Proteins - immunology | Antibodies - pharmacology | Gene Expression Regulation - drug effects | Animals | Interleukin-12 Subunit p35 - immunology | Interleukin-17 - antagonists & inhibitors | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Antigens - immunology | Autoimmunity - drug effects | Uveitis - pathology | Commentaries
Journal Article
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3. Ocular immunology
by Smolin, Gilbert and O'Connor, G. Richard 1928- (George Richard)
1981, ISBN 9780812106886, x, 322
Eye | Immunological aspects | Eye diseases | Immunology | Diseases
Book
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4. Full Text Mannan-binding lectin-associated serine protease (MASP)-1 is crucial for lectin pathway activation in human serum whereas neither MASP-1 nor MASP-3 is required for alternative pathway function
by Degn, Søren E and Jensen, Lisbeth and Hansen, Annette G and Duman, Duygu and Tekin, Mustafa and Jensenius, Jens C and Thiel, Steffen
Journal of Immunology, ISSN 0022-1767, 10/2012, Volume 189, Issue 8, pp. 3957 - 3969
The lectin pathway of complement is an important component of innate immunity. Its activation has been thought to occur via recognition of pathogens by... 
SYSTEM | INDIVIDUALS | PATTERN-RECOGNITION MOLECULES | COMPLEMENT FACTOR-D | MBL | 3MC SYNDROME | STOICHIOMETRY | MUTATIONS | IMMUNOLOGY | IDENTIFICATION | DEFICIENCY | Craniofacial Abnormalities - immunology | Transcriptional Activation - genetics | Complement Pathway, Mannose-Binding Lectin - immunology | Blepharoptosis - genetics | Eye Abnormalities - enzymology | Mannose-Binding Protein-Associated Serine Proteases - physiology | Eye Abnormalities - immunology | Humans | Heart Defects, Congenital - immunology | Craniosynostoses - enzymology | Blepharoptosis - immunology | Developmental Disabilities - genetics | Transcriptional Activation - immunology | Strabismus - genetics | Abdominal Muscles - abnormalities | Abnormalities, Multiple - immunology | Heart Defects, Congenital - genetics | Developmental Disabilities - enzymology | Blepharoptosis - enzymology | Heart Defects, Congenital - enzymology | Mannose-Binding Protein-Associated Serine Proteases - genetics | Complement Pathway, Alternative - genetics | Abnormalities, Multiple - genetics | Craniofacial Abnormalities - genetics | Strabismus - enzymology | Abdominal Muscles - immunology | Craniosynostoses - genetics | Complement Pathway, Mannose-Binding Lectin - genetics | Cryptorchidism - enzymology | Abdominal Muscles - enzymology | Hip Dislocation, Congenital - genetics | Codon, Nonsense | Eye Abnormalities - genetics | Animals | Craniofacial Abnormalities - enzymology | Craniosynostoses - immunology | Cryptorchidism - immunology | Hip Dislocation, Congenital - immunology | Complement Pathway, Alternative - immunology | Strabismus - immunology | Abnormalities, Multiple - enzymology | Developmental Disabilities - immunology | Hip Dislocation, Congenital - enzymology | Cryptorchidism - genetics
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5. Ocular immunology
by Smolin, Gilbert and O'Connor, G. Richard 1928- (George Richard)
1986, 2nd ed. --, ISBN 9780316801881, x, 360
Eye | Immunological aspects | Eye diseases | Immunology | Diseases
Book
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6. Full Text Neutrophil IL-1β processing induced by pneumolysin is mediated by the NLRP3/ASC inflammasome and caspase-1 activation and is dependent on K+ efflux
by Karmakar, Mausita and Katsnelson, Michael and Malak, Hesham A and Greene, Neil G and Howell, Scott J and Hise, Amy G and Camilli, Andrew and Kadioglu, Aras and Dubyak, George R and Pearlman, Eric
Journal of Immunology, ISSN 0022-1767, 02/2015, Volume 194, Issue 4, pp. 1763 - 1775
Although neutrophils are the most abundant cells in acute infection and inflammation, relatively little attention has been paid to their role in inflammasome... 
STREPTOCOCCUS-PNEUMONIAE | INTERLEUKIN-1-BETA | DEFICIENT | INNATE | KERATITIS | DEATH | MECHANISMS | NLRC4 | PYROPTOSIS | IMMUNOLOGY | CONTRIBUTES | Pneumococcal Infections | Potassium - metabolism | Eye Infections, Bacterial - immunology | Keratitis - immunology | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Caspase 1 - metabolism | Caspase 1 - immunology | Enzyme Activation - immunology | Signal Transduction - immunology | Flow Cytometry | Interleukin-1beta - metabolism | Bacterial Proteins - immunology | Eye Infections, Bacterial - metabolism | Neutrophils - metabolism | Carrier Proteins - immunology | Disease Models, Animal | Apoptosis Regulatory Proteins - immunology | Enzyme-Linked Immunosorbent Assay | Mice, Inbred C57BL | Neutrophils - immunology | Interleukin-1beta - immunology | Blotting, Western | Mice, Knockout | Spectrophotometry, Atomic | Animals | CARD Signaling Adaptor Proteins | Inflammasomes - immunology | Fluorescent Antibody Technique | Keratitis - metabolism | Mice | Streptolysins - immunology
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7. Full Text Mesenchymal stem/stromal cells precondition lung monocytes/macrophages to produce tolerance against allo- and autoimmunity in the eye
by Jung Hwa Ko and Hyun Ju Lee and Hyun Jeong Jeong and Mee Kum Kim and Won Ryang Wee and Sun-ok Yoon and Hosoon Choi and Darwin J. Prockop and Joo Youn Oh
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2016, Volume 113, Issue 1, pp. 158 - 163
Intravenously administered mesenchymal stem/stromal cells (MSCs) engraft only transiently in recipients, but confer long-term therapeutic benefits in patients... 
Monocyte/macrophage | Mesenchymal stem/stromal cell | Corneal allotransplantation | Experimental autoimmune uveitis | Immune tolerance | STEM-CELLS | ACTIVATION | experimental autoimmune uveitis | HMSCS | corneal allotransplantation | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | PROSTAGLANDIN-E2 | immune tolerance | MSCS | INFLAMMATION | IN-VIVO | REGULATORY T-CELLS | ANTIINFLAMMATORY PROTEIN TSG-6 | monocyte/macrophage | mesenchymal stem/stromal cell | Corneal Transplantation | Cell Adhesion Molecules - genetics | Graft Survival - immunology | CD11b Antigen - immunology | Cornea - immunology | Genes, MHC Class II - immunology | Monocytes - immunology | Gene Knockdown Techniques | Mesenchymal Stromal Cells - immunology | T-Lymphocytes, Regulatory - immunology | Leukocyte Common Antigens - immunology | Administration, Intravenous | Animals | Autoimmunity - immunology | Immune Tolerance - immunology | Uveitis - immunology | Immune Tolerance - genetics | Mice | Interleukin-10 - immunology | Lung - immunology | Macrophages - immunology | Mesenchymal Stem Cell Transplantation | Disease Models, Animal | Eye | Macrophages | Health aspects | Stem cells | stromal cell | Biological Sciences | mesenchymal stem | monocyte | macrophage
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8. Full Text STING regulates intracellular DNA-mediated, type i interferon-dependent innate immunity
by Ishikawa, Hiroki and Ma, Zhe and Barber, Glen N
Nature, ISSN 0028-0836, 10/2009, Volume 461, Issue 7265, pp. 788 - 792
The innate immune system is critical for the early detection of invading pathogens and for initiating cellular host defence countermeasures, which include the... 
ACTIVATION | HEPATITIS-C VIRUS | RECOGNITION | RNA | MULTIDISCIPLINARY SCIENCES | INFLAMMASOME | RIG-I | CYTOSOLIC DNA | KAPPA-B | ADAPTER PROTEIN | CYTOPLASMIC DNA | DNA, Bacterial - metabolism | Vesicular Transport Proteins - metabolism | Vaccines, DNA - immunology | Endoplasmic Reticulum - metabolism | DNA, Bacterial - immunology | Antigen-Presenting Cells - microbiology | Listeria monocytogenes - immunology | Interferon Type I - immunology | Interferon Type I - biosynthesis | Membrane Proteins - deficiency | Cytoplasmic Vesicles - metabolism | Plasmids - genetics | Herpesvirus 1, Human - immunology | Membrane Proteins - metabolism | Fibroblasts - microbiology | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Fibroblasts - virology | T-Lymphocytes, Cytotoxic - immunology | Antigen-Presenting Cells - metabolism | Intracellular Space - immunology | Membrane Proteins - genetics | Plasmids - immunology | DNA, Viral - metabolism | Antigen-Presenting Cells - immunology | Antigen-Presenting Cells - virology | Mice, Knockout | Protein Transport | DNA, Viral - immunology | Immunity, Innate - immunology | Animals | Intracellular Space - metabolism | Listeria monocytogenes - genetics | Fibroblasts - immunology | Herpesvirus 1, Human - genetics | Mice | Herpesvirus 1, Human - pathogenicity | Physiological aspects | Genetic aspects | Interferon | Immunity | Proteins | Viruses | Immunization | E coli | Research & development--R&D | Cells
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9. Full Text An Ocular Commensal Protects against Corneal Infection by Driving an Interleukin-17 Response from Mucosal γδ T Cells
by St. Leger, Anthony J and Desai, Jigar V and Drummond, Rebecca A and Kugadas, Abirami and Almaghrabi, Fatimah and Silver, Phyllis and Raychaudhuri, Kumarkrishna and Gadjeva, Mihaela and Iwakura, Yoichiro and Lionakis, Michail S and Caspi, Rachel R
Immunity, ISSN 1074-7613, 07/2017, Volume 47, Issue 1, pp. 148 - 158.e5
Mucosal sites such as the intestine, oral cavity, nasopharynx, and vagina all have associated commensal flora. The surface of the eye is also a mucosal site,... 
ocular surface disease | γδ T cells | IL-17 | host defense | ocular commensal bacteria | microbiome | mucosal immunity | Pseudomonas aeruginosa - immunology | Humans | Cornea - immunology | Immunity, Mucosal | Corynebacterium - immunology | Receptors, Antigen, T-Cell, gamma-delta - genetics | Candidiasis - microbiology | Corynebacterium Infections - microbiology | Eye Infections - microbiology | Neutrophil Infiltration | Corynebacterium Infections - immunology | Eye Infections - immunology | Neutrophils - microbiology | Candidiasis - immunology | Disease Models, Animal | Cornea - microbiology | Microbiota - immunology | Receptors, Antigen, T-Cell, gamma-delta - metabolism | Mice, Inbred C57BL | Neutrophils - immunology | Interleukin-17 - genetics | Tears - immunology | Candida albicans - immunology | Pseudomonas Infections - microbiology | Mice, Knockout | Pseudomonas Infections - immunology | Host-Pathogen Interactions | Interleukin-17 - metabolism | Animals | T-Lymphocytes - immunology | Mice | Cornea | Commensals | Disease | Mucosa | Interleukin | Homeostasis | Infections | Lymphocytes T | Nasopharynx | Immunity | Recruitment | Eye | Immunology | Lymphocytes | Intestine | Rodents | Bacteria | Immune system | Cytokines | Neutrophils | Vagina | Antimicrobial agents | Interleukin 17 | Studies | Oral cavity | Antibiotics | Flora | Tears
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10. Full Text Breakdown of immune privilege and spontaneous autoimmunity in mice expressing a transgenic T cell receptor specific for a retinal autoantigen
by Horai, Reiko and Silver, Phyllis B and Chen, Jun and Agarwal, Rajeev K and Chong, Wai Po and Jittayasothorn, Yingyos and Mattapallil, Mary J and Nguyen, Sonia and Natarajan, Kannan and Villasmil, Rafael and Wang, Peng and Karabekian, Zaruhi and Lytton, Simon D and Chan, Chi-Chao and Caspi, Rachel R
Journal of Autoimmunity, ISSN 0896-8411, 2013, Volume 44, pp. 21 - 33
Abstract Despite presence of circulating retina-specific T cells in healthy individuals, ocular immune privilege usually averts development of autoimmune... 
Allergy and Immunology | Spontaneous disease | Immune privilege | TCR transgenic mouse | Autoimmune uveitis | TOLERANCE | NEO-SELF ANTIGEN | IMMUNOLOGY | BINDING PROTEIN | EYE | REGULATORY CELLS | UVEORETINITIS | NONOBESE DIABETIC MICE | INFLAMMATION | DISEASE | UVEITIS | Eye Proteins - immunology | Humans | Autoimmune Diseases - immunology | Mice, Transgenic | Retina - immunology | Th1 Cells - immunology | Receptors, Antigen, T-Cell - biosynthesis | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Animals | Autoimmunity - immunology | Uveitis - immunology | Autoantigens - immunology | Th17 Cells - immunology | Receptors, Antigen, T-Cell - immunology | Mice | Immunologic Memory - immunology | Cytokines - immunology | Retinol-Binding Proteins - immunology | Autoimmunity | Genetic engineering | T cells | Binding proteins | Analysis | Protein binding
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11. Mucosal immunology and virology
by Tyring, Stephen
2006, ISBN 1846282012, ix, 212 p. ; ill.
An understanding of virus infection and the underlying role of the immune system in protection against these diseases is vital in today's medical climate.... 
Immunological aspects | Immunology | Mucous membrane | Immunity, Mucosal | Diseases | Clinical & internal medicine | Virus diseases | Infectious Diseases | Otorhinolaryngology | Pneumology/Respiratory System | Medicine & Public Health | Dermatology | Gastroenterology
Book
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12. Full Text Identification of autoantigens recognized by the 2F5 and 4E10 broadly neutralizing HIV-1 antibodies
by Yang, Guang and Holl, T. Matt and Liu, Yang and Li, Yi and Lu, Xiaozhi and Nicely, Nathan I and Kepler, Thomas B and Alam Munir, S and Liao, Hua-Xin and Cain, Derek W and Spicer, Leonard and VandeBerg, John L and Haynes, Barton F and Kelsoe, Garnett
Journal of Experimental Medicine, ISSN 0022-1007, 02/2013, Volume 210, Issue 2, pp. 241 - 256
Many human monoclonal antibodies that neutralize multiple clades of HIV-1 are polyreactive and bind avidly to mammalian autoantigens. Indeed, the generation of... 
B-CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNE-RESPONSE | PROXIMAL EXTERNAL REGION | PASSIVE TRANSFER | IMMUNODEFICIENCY-VIRUS TYPE-1 | CENTRAL TOLERANCE | GERMINAL-CENTERS | IMMUNOLOGY | HUMAN MONOCLONAL-ANTIBODIES | GP41 ANTIBODIES | TRANSGENIC MICE | RNA-Binding Proteins - genetics | Hydrolases - genetics | env Gene Products, Human Immunodeficiency Virus - immunology | HIV Envelope Protein gp41 - immunology | Humans | Phylogeny | HIV Envelope Protein gp41 - chemistry | Autoantigens - genetics | Epitopes - immunology | Antibodies, Neutralizing - immunology | HIV Antibodies - immunology | Conserved Sequence | Antibodies, Monoclonal - immunology | env Gene Products, Human Immunodeficiency Virus - chemistry | Amino Acid Sequence | Cell Line | Immunization | RNA-Binding Proteins - chemistry | RNA-Binding Proteins - immunology | RNA Splicing Factors | Immune Tolerance | Opossums - immunology | Autoantigens - chemistry | Epitopes - genetics | HIV-1 - immunology | Animals | B-Lymphocytes - immunology | Hydrolases - immunology | Autoantigens - immunology | Hydrophobic and Hydrophilic Interactions | Epitopes - chemistry | Hydrolases - chemistry | Mice
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13. Full Text Induced Autoimmunity to Heat Shock Proteins Elicits Glaucomatous Loss of Retinal Ganglion Cell Neurons via Activated T-Cell-Derived Fas-Ligand
by Wax, Martin B and Tezel, Gulgun and Yang, Junjie and Peng, Guanghua and Patil, Rajkumar V and Agarwal, Neeraj and Sappington, Rebecca M and Calkins, David J
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 46, pp. 12085 - 12096
Glaucomatous optic neuropathy causes blindness through the degeneration of retinal ganglion cells (RGCs) and their axons, which comprise the optic nerve.... 
Autoimmunity | Glaucoma | T-cells | Heat shock proteins | FasL | Microglia | glaucoma | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | NORMAL-PRESSURE GLAUCOMA | ENDOTOXIN-INDUCED UVEITIS | NEUROSCIENCES | autoimmunity | MULTIPLE-SCLEROSIS | IMMUNE SURVEILLANCE | OCULAR HYPERTENSION | CLASS-I | CENTRAL-NERVOUS-SYSTEM | microglia | heat shock proteins | SERUM AUTOANTIBODIES | Nerve Degeneration - physiopathology | Rats, Inbred Lew | Retinal Degeneration - immunology | Male | Retinal Ganglion Cells - metabolism | Autoimmune Diseases of the Nervous System - pathology | Retinal Degeneration - metabolism | Fas Ligand Protein - immunology | Nerve Degeneration - metabolism | Retinal Ganglion Cells - pathology | Autoimmune Diseases of the Nervous System - physiopathology | fas Receptor - immunology | Lymphocyte Activation - immunology | Microglia - immunology | Autoimmunity - immunology | Glaucoma - metabolism | Glaucoma - physiopathology | Animals, Newborn | Cell Line | Rats | Retinal Ganglion Cells - immunology | Nerve Degeneration - immunology | Retinal Degeneration - physiopathology | Rats, Sprague-Dawley | Heat-Shock Proteins - immunology | Autoimmune Diseases of the Nervous System - immunology | Autoantibodies - immunology | Intraocular Pressure - immunology | Animals | Apoptosis - immunology | Glaucoma - immunology | T-Lymphocytes - immunology
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14. Full Text Interleukin-13 Promotes Susceptibility to Chlamydial Infection of the Respiratory and Genital Tracts
by Asquith, Kelly L and Horvat, Jay C and Kaiko, Gerard E and Carey, Alison J and Beagley, Kenneth W and Hansbro, Philip M and Foster, Paul S
PLoS Pathogens, ISSN 1553-7366, 05/2011, Volume 7, Issue 5, p. e1001339
Chlamydiae are intracellular bacteria that commonly cause infections of the respiratory and genital tracts, which are major clinical problems. Infections are... 
ALLERGIC INFLAMMATION | TRACHOMATIS INFECTION | MICROBIOLOGY | KNOCKOUT MICE | AIRWAYS HYPERREACTIVITY | PNEUMONIAE INFECTION | VIROLOGY | INTERFERON-GAMMA | TRANSCUTANEOUS IMMUNIZATION | NITRIC-OXIDE | ALVEOLAR MACROPHAGES | PARASITOLOGY | MURIDARUM INFECTION | Lung - microbiology | Interleukin-13 - immunology | Interferon-gamma - metabolism | CD4-Positive T-Lymphocytes - immunology | Chlamydia muridarum - immunology | Respiratory Tract Infections - microbiology | Chlamydia Infections - immunology | Disease Susceptibility - immunology | Interleukin-10 - metabolism | Female | Macrophages - immunology | Macrophages - microbiology | Vaginal Diseases - immunology | Bacterial Load | Immunity, Innate | Mice, Knockout | Animals | Chlamydia Infections - microbiology | Chlamydia muridarum - pathogenicity | Epithelial Cells - immunology | Epithelial Cells - microbiology | Respiratory Tract Infections - immunology | Vaginal Diseases - microbiology | Mice | Mice, Inbred BALB C | Lung - immunology | Chlamydia infections | Prevention | Care and treatment | Interleukins | Causes of | Development and progression | Disease susceptibility | Genetic aspects | Research | Health aspects | Flow cytometry | Sexually transmitted diseases--STD | Bacterial infections | Lungs | Nitric oxide | Eye diseases | Infections | Chronic obstructive pulmonary disease | Alzheimers disease | Asthma | Sexually transmitted diseases | STD
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15. Full Text STING-Dependent Cytosolic DNA Sensing Promotes Radiation-Induced Type I Interferon-Dependent Antitumor Immunity in Immunogenic Tumors
by Deng, Liufu and Liang, Hua and Xu, Meng and Yang, Xuanming and Burnette, Byron and Arina, Ainhoa and Li, Xiao-Dong and Mauceri, Helena and Beckett, Michael and Darga, Thomas and Huang, Xiaona and Gajewski, Thomas F and Chen, Zhijian J and Fu, Yang-Xin and Weichselbaum, Ralph R
Immunity, ISSN 1074-7613, 11/2014, Volume 41, Issue 5, pp. 843 - 852
Ionizing radiation-mediated tumor regression depends on type I interferon (IFN) and the adaptive immune response, but several pathways control I IFN induction.... 
ADAPTIVE IMMUNITY | 2ND-MESSENGER | CYCLIC GMP-AMP | DENDRITIC CELLS | RECOGNITION | INFLAMMATION | ANTIMICROBIAL PEPTIDE | RADIOTHERAPY | IMMUNOLOGY | INNATE IMMUNITY | NUCLEIC-ACIDS | Interferon-beta - immunology | Dendritic Cells - immunology | Adaptor Proteins, Vesicular Transport - genetics | Xanthones - pharmacology | Signal Transduction - immunology | RNA Interference | Neoplasms - radiotherapy | Antineoplastic Agents - pharmacology | Receptor, Interferon alpha-beta - genetics | Radiation, Ionizing | Adaptive Immunity | Nucleotides, Cyclic - pharmacology | DNA - immunology | Membrane Proteins - genetics | Mice, Inbred C57BL | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Interferon-beta - biosynthesis | Immunity, Innate | Mice, Knockout | Animals | Cross-Priming - immunology | Interferon-beta - pharmacology | Receptor, Interferon alpha-beta - immunology | Neoplasms - immunology | Mice | RNA, Small Interfering | Nucleotidyltransferases - immunology | Ionizing radiation | Immune response | Dendritic cells | DNA | Interferon | Biological response modifiers | Molecular biology | Tumors | Proteins | Transcription factors | Lymphocytes | Rodents | Radiation therapy | Kinases | Deoxyribonucleic acid--DNA | Immune system
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