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Journal Article
Journal Article
Journal Article
Neuron, ISSN 0896-6273, 02/2012, Volume 73, Issue 3, pp. 445 - 452
Loss of retinal ganglion cells (RGCs) accounts for visual function deficits after optic nerve injury, but how axonal insults lead to neuronal death remains... 
OPEN-ANGLE GLAUCOMA | OXIDATIVE STRESS | OPTIC-NERVE | MESSENGER-RNA | ER STRESS | TRANSCRIPTION FACTOR XBP-1 | OCULAR HYPERTENSION | ENDOPLASMIC-RETICULUM | RETINITIS-PIGMENTOSA | NEUROSCIENCES | MOUSE MODELS | Glaucoma - etiology | Axotomy - methods | Dependovirus - genetics | Caspase 3 - metabolism | Cell Survival - genetics | Green Fluorescent Proteins - genetics | RNA, Messenger - metabolism | X-Box Binding Protein 1 | Retinal Ganglion Cells - pathology | DNA-Binding Proteins - metabolism | Tubulin - metabolism | Flow Cytometry | Cell Death - genetics | Optic Nerve Injuries - pathology | Glaucoma - physiopathology | Gene Expression Regulation - genetics | Optic Nerve Injuries - etiology | Mice, Inbred C57BL | Optic Nerve Injuries - physiopathology | Gene Expression Regulation - physiology | Rats | Transcription Factor CHOP - deficiency | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Rats, Sprague-Dawley | Regulatory Factor X Transcription Factors | Amino Acids | Protein Folding | Mice, Knockout | Transcription Factors - metabolism | Animals | Mice | Glaucoma - genetics | Medical colleges | Neurons | Cell death | Analysis | Ophthalmology | Ganglion | Public health | Protein binding | Phosphorylation | Transcription factors | Optic nerve | Protein folding | Rodents | Hypoxia | Hybridization | Kinases | Apoptosis | Index Medicus
Journal Article