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Journal Article
Annals of Oncology, ISSN 0923-7534, 03/2010, Volume 21, Issue 10, pp. 1999 - 2004
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 05/2008, Volume 26, Issue 15, pp. 2512 - 2518
Journal Article
Ophthalmology, ISSN 0161-6420, 2014, Volume 121, Issue 5, pp. 994 - 1000
Purpose Treatment for ocular surface squamous neoplasia (OSSN) has historically been surgery, but nonsurgical interventions are increasingly used. Treatment... 
AMERICAN-JOINT-COMMITTEE | CORNEAL | SOLID TUMORS | MITOMYCIN-C | OPHTHALMOLOGY | TERM-FOLLOW-UP | CANCER-CLASSIFICATION | CONJUNCTIVAL INTRAEPITHELIAL NEOPLASIA | HUMAN-PAPILLOMAVIRUS | TOPICAL INTERFERON-ALPHA-2B | CELL CARCINOMA | Recombinant Proteins - therapeutic use | Corneal Diseases - therapy | Conjunctival Neoplasms - drug therapy | Conjunctival Neoplasms - surgery | Carcinoma in Situ - drug therapy | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Carcinoma, Squamous Cell - surgery | Male | Antineoplastic Agents - therapeutic use | Eye Neoplasms - therapy | Eye Neoplasms - surgery | Case-Control Studies | Ophthalmologic Surgical Procedures | Eye Neoplasms - drug therapy | Carcinoma in Situ - surgery | Aged, 80 and over | Ophthalmic Solutions | Adult | Female | Corneal Diseases - pathology | Postoperative Complications | Visual Acuity - physiology | Carcinoma in Situ - pathology | Risk Factors | Eye Neoplasms - pathology | Interferon-alpha - therapeutic use | Carcinoma, Squamous Cell - therapy | Corneal Diseases - drug therapy | Treatment Outcome | Neoplasm Recurrence, Local - diagnosis | Carcinoma in Situ - therapy | Conjunctival Neoplasms - therapy | Conjunctival Neoplasms - pathology | Corneal Diseases - surgery | Carcinoma, Squamous Cell - drug therapy | Aged | Neoplasm Staging | Cryotherapy | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 04/2012, Volume 18, Issue 4, pp. 521 - 528
Tyrosine kinase inhibitors (TKIs) elicit high response rates among individuals with kinase-driven malignancies, including chronic myeloid leukemia (CML) and... 
CHRONIC MYELOGENOUS LEUKEMIA | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | GEFITINIB | BIOCHEMISTRY & MOLECULAR BIOLOGY | EYE DISEASES | CHRONIC MYELOID-LEUKEMIA | CELL BIOLOGY | CELL LUNG-CANCER | FACTOR RECEPTOR EGFR | GROWTH | ACTIVATING MUTATIONS | MUTANT EGFR | Enzyme-Linked Immunosorbent Assay - methods | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Follow-Up Studies | Apoptosis - drug effects | Humans | Middle Aged | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Male | BH3 Interacting Domain Death Agonist Protein - genetics | Dose-Response Relationship, Drug | Protein Isoforms - metabolism | Transfection | Bcl-2-Like Protein 11 | Statistics, Nonparametric | Aged, 80 and over | Apoptosis Regulatory Proteins - genetics | Adult | Female | Gene Expression Regulation, Neoplastic - drug effects | Lung Neoplasms - genetics | Annexins - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Membrane Proteins - genetics | Gene Frequency | Exons - genetics | Genotype | Proto-Oncogene Proteins - genetics | International Cooperation | Drug Resistance, Neoplasm - genetics | Cell Line, Tumor | Polymorphism, Genetic - genetics | Aged | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Cohort Studies | Drug Resistance, Neoplasm - drug effects | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | Sequence Deletion - genetics | Antimitotic agents | Physiological aspects | Protein tyrosine kinase | Dosage and administration | Genetic aspects | Research | Antineoplastic agents | Tumor proteins | Genetic polymorphisms | Inhibitor drugs | Mutation | Kinases | Gene expression | Apoptosis | Polymorphism | Index Medicus
Journal Article
Cancer Immunology, Immunotherapy, ISSN 0340-7004, 7/2014, Volume 63, Issue 7, pp. 675 - 683
Journal Article
Science Signaling, ISSN 1945-0877, 10/2015, Volume 8, Issue 397, pp. ra98 - ra98
Yes-associated protein 1 (YAP1) is a transcriptional coactivator in the Hippo signaling pathway. Increased YAP1 activity promotes the growth of tumors,... 
PHOTODYNAMIC THERAPY | PATHWAY | COLORECTAL-CANCER | BIOCHEMISTRY & MOLECULAR BIOLOGY | HIPPO | GROWTH | MACULAR DEGENERATION | CELL-PROLIFERATION | CHOROIDAL NEOVASCULARIZATION | TEAD/TEF FAMILY | TUMORIGENESIS | CELL BIOLOGY | Transcription, Genetic - drug effects | Phosphorylation | Adenocarcinoma - pathology | Molecular Weight | Apoptosis - drug effects | Colonic Neoplasms - drug therapy | Humans | Neoplasm Proteins - physiology | Colonic Neoplasms - chemically induced | Neoplasm Proteins - antagonists & inhibitors | Phosphoproteins - antagonists & inhibitors | Adenomatous Polyposis Coli - pathology | Autophagy - drug effects | Genes, APC | Neoplasm Proteins - drug effects | Proteasome Endopeptidase Complex - drug effects | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Adenoma - drug therapy | HEK293 Cells | Antineoplastic Agents - pharmacology | Phosphoproteins - physiology | Adenomatous Polyposis Coli - genetics | Mice, Inbred C57BL | Transcription Factors - antagonists & inhibitors | Porphyrins - pharmacology | Adenocarcinoma - drug therapy | Cell Division - drug effects | Mice, Knockout | Xenograft Model Antitumor Assays | Adaptor Proteins, Signal Transducing - physiology | Animals | Adenomatous Polyposis Coli - drug therapy | Colonic Neoplasms - pathology | Adenoma - pathology | Cell Line, Tumor | Mice | Protein Processing, Post-Translational | STAT3 Transcription Factor - antagonists & inhibitors | Protein Multimerization - drug effects | Index Medicus
Journal Article
Journal Article