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PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 11, p. e47842
Birt-Hogg-Dube (BHD) is a tumor suppressor gene syndrome associated with fibrofolliculomas, cystic lung disease, and chromophobe renal cell carcinoma. In... 
EPITHELIAL-MESENCHYMAL TRANSITION | SPONTANEOUS PNEUMOTHORAX | FLCN | TUBEROUS SCLEROSIS COMPLEX | MULTIDISCIPLINARY SCIENCES | RHO | MTOR ACTIVATION | MUTATIONS | DESMOPLAKIN | BHD | FAMILY | Hair - abnormalities | Humans | Hair - metabolism | Wound Healing | Adherens Junctions - metabolism | Catenins - metabolism | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Epidermis - abnormalities | Integrases - metabolism | Zonula Occludens-1 Protein - metabolism | Proto-Oncogene Proteins - metabolism | Cell Line | Desmosomes - metabolism | Epidermis - metabolism | Epidermis - pathology | Hair - pathology | Tumor Suppressor Proteins - metabolism | gamma Catenin - metabolism | Proto-Oncogene Proteins - genetics | Keratin-14 - metabolism | Proto-Oncogene Proteins - deficiency | Cell Adhesion | Animals | Models, Biological | rho GTP-Binding Proteins - metabolism | Dogs | Protein Binding | Mice | Plakophilins - metabolism | Cell Movement | Physiological aspects | Keratin | Genetic aspects | Genes | Tumors | TOR protein | Eyelid | Pathogenesis | Hyperplasia | Cell junctions | Critical care | Cardiovascular disease | Biology | Recombinase | Kinases | Autophagy | Medical schools | Cell adhesion & migration | Cell adhesion molecules | Proteins | Animal tissues | Cell adhesion | Polarity | Tumorigenesis | p0071 Protein | Biomedical engineering | Medical research | Lung diseases | RhoA protein | Epidermis | Adherens junctions | Rapamycin | Adhesion | Rho-associated kinase | Flox | Medicine | Signaling | Hospitals | Tumor suppressor genes | Mutation | Emphysema | Prostate | Cytoplasm | Clear cell-type renal cell carcinoma | Kidney transplantation | Cancer
Journal Article
Developmental Biology, ISSN 0012-1606, 11/2013, Volume 383, Issue 2, pp. 227 - 238
Sproutys (Sprys) are downstream targets and negative feedback regulators of the FGF–Ras–ERK signaling pathway. Our previous studies have shown that Spry1 and... 
Foxc2 | Foxc1 | FGF | Eyelids | C-Jun | Wnt | Sprouty | ERK | MIGRATION | SIGNALS | RECEPTOR | SPROUTY GENES | DEVELOPMENTAL BIOLOGY | CELL FATE | REGULATOR | EPITHELIUM | LEADING-EDGE | RETINA | Phosphorylation | Cell Proliferation | Hedgehog Proteins - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Phosphoproteins - metabolism | Bone Morphogenetic Protein 4 - metabolism | Embryo, Mammalian - metabolism | Fibroblast Growth Factors - metabolism | Forkhead Transcription Factors - metabolism | Gene Expression Regulation, Developmental | Membrane Proteins - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Wnt Signaling Pathway | Eyelids - embryology | Eyelids - physiology | Epithelium - metabolism | Membrane Proteins - genetics | Phosphoproteins - genetics | Forkhead Transcription Factors - genetics | Mutation - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Embryo, Mammalian - cytology | Proto-Oncogene Proteins c-jun - metabolism | Adaptor Proteins, Signal Transducing - genetics | Eyelids - enzymology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Eyelids - cytology | Cell Movement | Epidermis - cytology | Polymerization | Bone morphogenetic proteins | Muscle proteins | Stem cells | c-Jun | eyelids
Journal Article
Journal Article
Journal Article
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 2012, Volume 4, Issue 3, pp. 192 - 205
Ankyloblepharon‐ectodermal defects‐cleft lip/palate (AEC) syndrome, which is characterized by cleft palate and severe defects of the skin, is an autosomal... 
AEC syndrome | p63 knock‐in | p63 target genes | epidermis | FGF signalling | Epidermis | P63 target genes | P63 knock-in | MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | HAIR | ANKYLOBLEPHARON | TRANSCRIPTION | P53 HOMOLOG | p63 knock-in | CRUCIAL ROLE | GROWTH-FACTOR RECEPTOR | LIMB | TARGETS | MUTATIONS | Cell Proliferation | Humans | Eyelids - abnormalities | Male | Cleft Palate - genetics | Stem Cells - cytology | Stem Cells - metabolism | Ectodermal Dysplasia - physiopathology | Eyelids - metabolism | Fibroblast Growth Factors - metabolism | Ectoderm - metabolism | Ectodermal Dysplasia - metabolism | Eyelids - physiopathology | Tumor Suppressor Proteins - genetics | Female | Eye Abnormalities - metabolism | Tumor Suppressor Proteins - metabolism | Signal Transduction | Mice, Inbred C57BL | Mice, Transgenic | Cleft Palate - physiopathology | Transcription Factors - genetics | Cleft Lip - metabolism | Eye Abnormalities - genetics | Mice, Knockout | Transcription Factors - metabolism | Ectoderm - cytology | Cleft Lip - genetics | Animals | Cleft Lip - physiopathology | Eye Abnormalities - physiopathology | Cleft Palate - metabolism | Ectodermal Dysplasia - genetics | Mice | Mutation | Growth | Analysis | Stem cells | Fibroblast growth factors | Skin | Universities and colleges | Genetic transcription | Mitogens | Cell proliferation | Fibroblast growth factor | Medical research | Phenotypes | Transcription factors | Epithelial cells | Pathogenesis | Gene expression | Progenitor cells | Defects | Hereditary diseases | Cleft lip/palate | Cell adhesion & migration | Proteins | Genotype & phenotype | Cell growth | Mutagenesis | Protein kinase | Fibroblast growth factor receptor 7 | Fibroblast growth factor receptor 2 | Fibroblast growth factor receptors
Journal Article