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Nature, ISSN 0028-0836, 02/2011, Volume 470, Issue 7332, pp. 115 - 121
The activated B-cell-like (ABC) subtype of diffuse large B-cell lymphoma (DLBCL) remains the least curable form of this malignancy despite recent advances in... 
TNFAIP3 A20 | PATHWAYS | SURVIVAL | B-CELL LYMPHOMA | GENE | SIGNATURES | SUBTYPES | MULTIDISCIPLINARY SCIENCES | KINASE | INHIBITORS | NF-KAPPA-B | Interleukin-1 Receptor-Associated Kinases - metabolism | Phosphorylation | Interleukin-1 Receptor-Associated Kinases - genetics | Interleukin-1 Receptor-Associated Kinases - biosynthesis | Humans | Molecular Sequence Data | NF-kappa B - metabolism | Janus Kinases - metabolism | Myeloid Differentiation Factor 88 - chemistry | RNA Interference | Burkitt Lymphoma - genetics | Toll-Like Receptors - metabolism | STAT3 Transcription Factor - metabolism | Protein Structure, Tertiary | Amino Acid Sequence | Oncogenes - genetics | Cytokines - metabolism | Lymphoma, Large B-Cell, Diffuse - pathology | Signal Transduction | Cell Survival | Cytokines - secretion | Mutant Proteins - genetics | Myeloid Differentiation Factor 88 - genetics | Mutant Proteins - metabolism | Mutation - genetics | Lymphoma, B-Cell, Marginal Zone - genetics | Receptors, Interleukin-1 - metabolism | Sequence Analysis, RNA | Lymphoma, Large B-Cell, Diffuse - classification | Mutant Proteins - chemistry | Cell Line, Tumor | Hydrophobic and Hydrophilic Interactions | High-Throughput Nucleotide Sequencing | Lymphoma, Large B-Cell, Diffuse - genetics | Myeloid Differentiation Factor 88 - metabolism | Amino Acid Substitution | Lymphomas | Genetic aspects | Models | Gene mutations | Observations | Abnormalities | Mutation | Kinases | Toxicity | Apoptosis | Tumors
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 11/2013, Volume 170, Issue 5, pp. 1078 - 1091
Background and Purpose The gallnut of Rhus chinensis MILL and its main constituent penta‐O‐galloyl‐β‐D‐glucose (PGG) inhibited NF‐κB activation in... 
macrophage | NF‐κB | inflammation | MyD88 | penta‐O‐galloyl‐β‐D‐glucose | NF-κB | penta-O-galloyl-β-D-glucose | ULCERATIVE-COLITIS | NF-B | ARRIVE GUIDELINES | RHUS-CHINENSIS | INTESTINAL INFLAMMATION | IRAK-1 ACTIVATION | EXPERIMENTAL COLITIS | IN-VIVO | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | penta-O-galloyl--D-glucose | SCHLECHTENDALIA-CHINENSIS | NF-KAPPA-B | Interleukin-1 Receptor-Associated Kinases - metabolism | Colon - enzymology | Phosphorylation | Colon - drug effects | Gastrointestinal Agents - administration & dosage | Molecular Sequence Data | Colon - immunology | Male | NF-kappa B - metabolism | Trinitrobenzenesulfonic Acid | Dose-Response Relationship, Drug | Transfection | RNA Interference | Colitis - chemically induced | Myeloid Differentiation Factor 88 - antagonists & inhibitors | Inflammation Mediators - metabolism | Hydrolyzable Tannins - pharmacology | Anti-Inflammatory Agents - administration & dosage | Colitis - immunology | Macrophages, Peritoneal - drug effects | Disease Models, Animal | NF-kappa B - antagonists & inhibitors | Administration, Oral | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Macrophages, Peritoneal - enzymology | Myeloid Differentiation Factor 88 - genetics | Macrophages, Peritoneal - immunology | Toll-Like Receptor 4 - metabolism | Hydrolyzable Tannins - administration & dosage | Gastrointestinal Agents - pharmacology | Animals | Colitis - enzymology | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mice | Colitis - prevention & control | Myeloid Differentiation Factor 88 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Research Papers
Journal Article
Science, ISSN 0036-8075, 8/2008, Volume 321, Issue 5889, pp. 691 - 696
MyD88 is a key downstream adapter for most Toll-like receptors (TLRs) and interleukin-1 receptors (IL-1Rs). MyD88 deficiency in mice leads to susceptibility to... 
Receptors | Phenotypes | Transfection | Cytokines | HEK293 cells | Genes | Cell lines | Fibroblasts | Agonists | Reports | Children | MYD88-DEFICIENT MICE | HERPES-SIMPLEX-VIRUS | PROTECTIVE IMMUNITY | MYELOID DIFFERENTIATION FACTOR-88 | PSEUDOMONAS-AERUGINOSA | MULTIDISCIPLINARY SCIENCES | HUMORAL IMMUNITY | STAPHYLOCOCCUS-AUREUS | AERUGINOSA LUNG INFECTION | HOST RESPONSE | CUTTING EDGE | Humans | Child, Preschool | Male | Mutation, Missense | Bacterial Infections - genetics | Pneumococcal Infections - immunology | Bacterial Infections - immunology | Gene Deletion | Pneumococcal Infections - genetics | Female | Toll-Like Receptors - metabolism | Child | Staphylococcal Infections - genetics | Disease Susceptibility | Cytokines - metabolism | Signal Transduction | Staphylococcal Infections - immunology | Receptors, Interleukin-1 - immunology | Toll-Like Receptors - immunology | Myeloid Differentiation Factor 88 - genetics | Immunity, Innate | Pseudomonas Infections - genetics | Pseudomonas Infections - immunology | Receptors, Interleukin-1 - metabolism | Animals | Myeloid Differentiation Factor 88 - deficiency | Adolescent | Mice | Myeloid Differentiation Factor 88 - metabolism | Cell Line, Transformed | Genetic aspects | Bacterial infections | Pediatrics | Bacteria | Biomedical research | Genetic disorders | Immunology | Pneumococcal Infections | Life Sciences | Toll-Like Receptors | Bacterial Infections | Myeloid Differentiation Factor 88 | Bacteriology | Staphylococcal Infections | Microbiology and Parasitology | Pseudomonas Infections | Receptors, Interleukin-1
Journal Article
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 08/2015, Volume 373, Issue 6, pp. 584 - 586
Journal Article
Blood, ISSN 0006-4971, 08/2013, Volume 122, Issue 7, pp. 1222 - 1232
Journal Article
The Journal of Immunology, ISSN 0022-1767, 01/2008, Volume 180, Issue 2, pp. 1169 - 1178
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 179 - 188
TLR activation on CD11c(+) DCs triggers DC maturation, which is critical for T cell activation. Given the expansion of CD11c(+) DCs during the progression of... 
ADAPTIVE IMMUNITY | MEDICINE, RESEARCH & EXPERIMENTAL | REDUCES ATHEROSCLEROSIS | DENDRITIC CELLS | REGULATORY T-CELLS | PLAQUE PHENOTYPE | APOE(-/-) MICE | CHRONIC INFLAMMATION | ARTERIAL INTIMA | AUTOIMMUNE-DISEASE | APOLIPOPROTEIN-E | T-Lymphocytes, Regulatory - metabolism | Chemokine CCL2 - immunology | Atherosclerosis - genetics | Dendritic Cells - immunology | Dendritic Cells - pathology | Monocytes - metabolism | Monocytes - immunology | T-Lymphocytes, Regulatory - pathology | Transplantation, Homologous | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Bone Marrow Transplantation | Monocytes - pathology | Myeloid Differentiation Factor 88 - immunology | Chemokine CCL2 - metabolism | Toll-Like Receptors - metabolism | Dendritic Cells - metabolism | Receptors, LDL - genetics | Atherosclerosis - pathology | Atherosclerosis - immunology | Lymphocyte Activation | Toll-Like Receptors - immunology | Myeloid Differentiation Factor 88 - genetics | Receptors, LDL - metabolism | Chemokine CCL2 - genetics | Signal Transduction - genetics | Receptors, LDL - immunology | Atherosclerosis - metabolism | Mice, Knockout | Animals | Toll-Like Receptors - genetics | Signal Transduction - physiology | Mice | Myeloid Differentiation Factor 88 - metabolism | Causes of | Physiological aspects | Research | T cells | Risk factors | Atherosclerosis
Journal Article
Journal Article