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The New England Journal of Medicine, ISSN 0028-4793, 07/2017, Volume 377, Issue 1, pp. 52 - 61
CD55 prevents convertase enzyme formation in the complement cascade, acting as a brake on complement activation. Inactivating mutations in CD55 result in... 
MEDICINE, GENERAL & INTERNAL | ACTIVATION | INFLAMMATORY-BOWEL-DISEASE | HEMOLYTIC-UREMIC SYNDROME | CELL RESPONSES | COMPLEMENT REGULATORY PROTEIN | INAB PHENOTYPE | DECAY-ACCELERATING FACTOR | PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | T-CELLS | Intestine, Small - pathology | Humans | CD55 Antigens - genetics | Child, Preschool | Infant | Male | Protein-Losing Enteropathies - genetics | Syndrome | Complement System Proteins - metabolism | CD55 Antigens - blood | Complement Activation - genetics | Homozygote | T-Lymphocytes - metabolism | Complement Inactivating Agents - pharmacology | Pedigree | Protein-Losing Enteropathies - complications | Statistics, Nonparametric | Immunoglobulin A - blood | Female | Thrombosis - genetics | Mutation | Child | Complement Activation - drug effects | Proteins | Molecular targeted therapy | Gene mutations | Gastrointestinal diseases | Analysis | Homeostasis | Research | Thrombosis | Blood clot | Gastrointestinal tract diseases | Complement component C5a | Lymphocytes T | Cell activation | Immunology | Pain | Intestine | Hepatology | Gastroenterology | Malabsorption | Thromboembolism | Digestive tract | Age | Edema | Medical research | Lymphatic system | Hypersensitivity | Diarrhea | Inflammation | Heredity | Protein deficiency | Patients | Hereditary diseases | Complement activation | Infectious diseases | Decay-accelerating factor
Journal Article
The American Journal of Pathology, ISSN 0002-9440, 07/2019, Volume 189, Issue 7, pp. 1386 - 1401
Recent studies traced inflammatory bowel disease in some patients to deficiency of CD55 [decay-accelerating factor (DAF)], but the mechanism underlying the... 
ANTERIOR-CHAMBER | C5A RECEPTOR | COMPLEMENT | ACTIVATION | SOLUBLE-ANTIGEN | IMMUNE PRIVILEGE | REGULATORY T-CELLS | DECAY-ACCELERATING FACTOR | ORAL TOLERANCE | PATHOLOGY | EXPRESSION
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 10/2017, Volume 377, Issue 15, pp. 1499 - 1500
To the Editor: Ozen et al. (July 6 issue) 1 describe 11 study participants with loss-of-function variants in CD55 and complement hyperactivation, angiopathic... 
MEDICINE, GENERAL & INTERNAL | DECAY-ACCELERATING FACTOR | COMPLEMENT | Protein-Losing Enteropathies | Fontan Procedure | Humans
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2017, Volume 214, Issue 9, pp. 2715 - 2732
Journal Article
European Journal of Immunology, ISSN 0014-2980, 06/2013, Volume 43, Issue 6, pp. 1617 - 1629
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 44, pp. 17188 - 17199
Enterohemorrhagic Escherichia coli (EHEC) is one of several E. coli pathotypes that infect the intestinal tract and cause disease. Formation of the... 
host-pathogen interaction | EHEC | CELLS | infection | COMPLEMENT | proteomics | StcE | TRANSPORTERS | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR DAF | neutrophil | COMPUTATIONAL PLATFORM | INHIBITION | O157-H7 | cell surface | DECAY-ACCELERATING FACTOR | metalloprotease | EXPRESSION | CD55 | PROTEASE | EPHA2 | Microbiology
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2017, Volume 377, Issue 1, pp. 87 - 89
CD55 (complement decay-accelerating factor) inhibits the alternative and classical arms of the complement pathway. Three patients with protein-losing... 
Proteins | Genotype & phenotype | Decay-accelerating factor | Gastrointestinal diseases | Monoclonal antibodies | Mutation | Patients
Journal Article
Transplantation, ISSN 0041-1337, 04/2012, Volume 93, Issue 7, pp. 686 - 692
Journal Article
The FEBS Journal, ISSN 1742-464X, 02/2013, Volume 280, Issue 3, pp. 840 - 854
We previously reported that nitric oxide (NO) reduces the rate of bacteremia and maternal mortality in pregnant rats with uterine infection by Escherichia coli... 
S‐nitrosylation | nitric oxide | Sp1 | decay accelerating factor | mRNA stability | S-nitrosylation | BIOCHEMISTRY & MOLECULAR BIOLOGY | NECROSIS-FACTOR-ALPHA | RICH ELEMENTS | TRANSLATION | INTESTINAL EPITHELIAL-CELLS | MESSENGER-RNA | GESTATIONAL PYELONEPHRITIS | INTRAUTERINE INFECTION | PROMOTER | EXPRESSION | FACTOR GENE | Immunoprecipitation | Fimbriae Proteins - metabolism | Humans | 3' Untranslated Regions - genetics | CD55 Antigens - genetics | Sp1 Transcription Factor - metabolism | RNA, Messenger - metabolism | Promoter Regions, Genetic - genetics | Cysteine - genetics | Nitroso Compounds - pharmacology | Nitroso Compounds - metabolism | Escherichia coli - metabolism | Cysteine - metabolism | Nitric Oxide Donors - pharmacology | Sp1 Transcription Factor - physiology | Nitric Oxide Donors - metabolism | RNA, Messenger - genetics | ELAV Proteins - metabolism | Binding Sites - genetics | Down-Regulation - drug effects | ELAV Proteins - physiology | Blotting, Western | Regulatory Sequences, Nucleic Acid - genetics | Down-Regulation - physiology | Fimbriae Proteins - genetics | Sp1 Transcription Factor - genetics | Escherichia coli - genetics | Cell Line, Tumor | Protein Binding | CD55 Antigens - metabolism | Mutation | Nitric Oxide - metabolism | ELAV Proteins - genetics | Messenger RNA | Promoters (Genetics) | Nitric oxide | Escherichia coli | Protein binding | Proteins | E coli | Ribonucleic acid--RNA | Binding sites
Journal Article