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Journal of the National Cancer Institute, ISSN 0027-8874, 07/2012, Volume 104, Issue 13, pp. 975 - 981
The success of targeted therapies for cancer is undisputed; strong preclinical evidence has resulted in the approval of several new agents for cancer... 
LUNG-CANCER | KINASE INHIBITION | SOLID TUMORS | ONCOLOGY | PHASE-II | IGF RECEPTOR | MONOCLONAL-ANTIBODY | BREAST-CANCER-CELLS | ANTITUMOR-ACTIVITY | FACTOR-I RECEPTOR | SARCOMA FAMILY | Neoplasms - metabolism | Breast Neoplasms - surgery | Lung Neoplasms - drug therapy | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Carcinoma, Squamous Cell - metabolism | Humans | Lung Neoplasms - metabolism | Antibodies, Monoclonal - therapeutic use | Antineoplastic Agents - therapeutic use | Endocrine Disruptors | Breast Neoplasms - metabolism | Early Termination of Clinical Trials | Human Growth Hormone - metabolism | Biomarkers, Tumor - metabolism | Female | Antineoplastic Agents - pharmacology | Human Growth Hormone - administration & dosage | Hyperinsulinism - complications | Ovariectomy | Signal Transduction | Risk Factors | Treatment Outcome | Clinical Trials as Topic | Evidence-Based Medicine | Disease Progression | Breast Neoplasms - prevention & control | Neoplasms - drug therapy | Carcinoma, Squamous Cell - drug therapy | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Body Height | Hypophysectomy | Immunoglobulins, Intravenous | Insulin-Like Growth Factor I - metabolism | Physiological aspects | Care and treatment | Enzyme inhibitors | Research | Insulin-like growth factor 1 | Cancer
Journal Article
JACC: Cardiovascular Interventions, ISSN 1936-8798, 2013, Volume 6, Issue 11, pp. 1111 - 1128
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2018, Volume 13, Issue 6, p. e0200070
Journal Article
British Journal of Cancer, ISSN 0007-0920, 01/2011, Volume 104, Issue 1, pp. 75 - 82
BACKGROUND: Activating mutations of FGFR3 are frequently identified in superficial urothelial carcinoma (UC) and increased expression of FGFR1 and FGFR3 are... 
TKI-258 | urothelial carcinoma | tyrosine kinase inhibitor | FGFR3 | PD173074 | FGFR1 | MULTIPLE-MYELOMA | GENE-MUTATIONS | BLADDER-CANCER | TRANSLOCATION | PROLIFERATION | KINASE INHIBITOR | CHIR-258 | ONCOLOGY | THERAPEUTIC TARGET | EXPRESSION | ACTIVATING MUTATIONS | Urothelium - metabolism | Apoptosis - drug effects | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Male | Receptor, Fibroblast Growth Factor, Type 3 - antagonists & inhibitors | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Immunoenzyme Techniques | Urinary Bladder Neoplasms - prevention & control | Carcinoma, Transitional Cell - prevention & control | Quinolones - therapeutic use | Urinary Bladder Neoplasms - pathology | Phosphorylation - drug effects | Pyrroles - therapeutic use | Urinary Bladder Neoplasms - metabolism | Benzimidazoles - therapeutic use | Cells, Cultured | Carcinoma, Transitional Cell - metabolism | Mutation - genetics | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Blotting, Western | Xenograft Model Antitumor Assays | Animals | Carcinoma, Transitional Cell - pathology | Pyrimidines - therapeutic use | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Cell Cycle - drug effects | In Vitro Techniques | Urothelium - drug effects | Protein-Tyrosine Kinases - antagonists & inhibitors | Translational Therapeutics
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Journal Article