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Journal of Clinical Investigation, ISSN 0021-9738, 04/2012, Volume 122, Issue 4, pp. 1469 - 1486
Breast cancers are highly heterogeneous but can be grouped into subtypes based on several criteria, including level of expression of certain markers.... 
EPITHELIAL-MESENCHYMAL TRANSITION | MEDICINE, RESEARCH & EXPERIMENTAL | BASAL-LIKE | STEM-CELLS | DNA METHYLATION | CLAUDIN-LOW | TRANSCRIPTION FACTOR SNAIL | GENE-EXPRESSION | PHENOTYPE | TUMOR-CELLS | HISTONE METHYLATION | Histocompatibility Antigens - genetics | Neoplasm Transplantation | Adenocarcinoma - pathology | Humans | Middle Aged | Neoplasm Proteins - physiology | Recombinant Fusion Proteins - physiology | Neoplasm Proteins - antagonists & inhibitors | Cell Line, Tumor - transplantation | Gene Expression Profiling | Cadherins - biosynthesis | Breast Neoplasms - metabolism | Gene Knockdown Techniques | DNA (Cytosine-5-)-Methyltransferases - metabolism | DNA Methylation | Histone-Lysine N-Methyltransferase - antagonists & inhibitors | Adenocarcinoma - metabolism | Adult | Female | Cadherins - genetics | Neoplasm Proteins - genetics | Cell Line, Tumor - metabolism | Snail Family Transcription Factors | Gene Expression Regulation, Neoplastic - genetics | Histone-Lysine N-Methyltransferase - genetics | Transcription Factors - physiology | DNA (Cytosine-5-)-Methyltransferase 1 | Neoplasm Invasiveness | Histocompatibility Antigens - physiology | Transcription Factors - genetics | Mice, Inbred ICR | Animals | Histone-Lysine N-Methyltransferase - metabolism | Breast Neoplasms - pathology | Aged | Mice | Protein Processing, Post-Translational | Histones - metabolism | Methylation | Histone-Lysine N-Methyltransferase - physiology | Physiological aspects | Cadherins | Breast cancer | Genetic aspects | Research | Transforming growth factors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
by Wu, Y and Zhou, B.P
British Journal of Cancer, ISSN 0007-0920, 02/2010, Volume 102, Issue 4, pp. 639 - 644
Tumour necrosis factor-alpha (TNF-alpha) is an important inflammatory factor that acts as a master switch in establishing an intricate link between... 
NF-κB | TNF-α | Snail | EMT | TRANSCRIPTION FACTORS | B ACTIVATION | METASTASIS | TNF-alpha | SNAIL TRANSCRIPTION | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | TUMOR-NECROSIS-FACTOR | NF-kappa B | ONCOLOGY | INFLAMMATION | GENE-EXPRESSION | PROGRESSION | Minireviews
Journal Article
Cancer research, ISSN 0008-5472, 06/2019, Volume 79, Issue 16, pp. 4135 - 4148
Snail is a key regulator of epithelial-mesenchymal transition (EMT) which is a major step in tumor metastasis. While the induction of Snail transcription... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | LOCALIZATION | PROTEIN | METASTASIS | ONCOLOGY | KINASE | TRANSCRIPTION FACTOR SNAIL | GENE-EXPRESSION | REPRESSOR | EMT | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e56664 - e56664
Journal Article
The EMBO Journal, ISSN 0261-4189, 11/2013, Volume 32, Issue 23, pp. 3079 - 3095
Here, we show that expression of ZNF281/ZBP‐99 is controlled by SNAIL and miR‐34a/b/c in a coherent feed‐forward loop: the epithelial–mesenchymal transition... 
colorectal cancer | metastasis | miR‐34a | ZNF281 | stemness | Stemness | Metastasis | MiR-34a | Colorectal cancer | TUMOR PROGRESSION | miR-34a | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION FACTOR SNAIL | BETA-CATENIN | MIR-200 FAMILY | NEGATIVE FEEDBACK LOOP | CELL BIOLOGY | WNT/BETA-CATENIN | COLORECTAL-CANCER | EMBRYONIC STEM-CELLS | GENE-EXPRESSION | Colorectal Neoplasms - genetics | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | Molecular Sequence Data | Male | Wound Healing | Case-Control Studies | Breast Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Lung Neoplasms - secondary | Base Sequence | Epithelial-Mesenchymal Transition | Female | Snail Family Transcription Factors | Colorectal Neoplasms - metabolism | Fibroblasts - metabolism | Lung Neoplasms - genetics | Neoplasm Invasiveness | Spheroids, Cellular - metabolism | Cells, Cultured | Mice, SCID | Sequence Homology, Nucleic Acid | Transcription Factors - metabolism | Animals | Breast Neoplasms - genetics | Tumor Suppressor Protein p53 | Breast Neoplasms - pathology | Mice, Inbred NOD | Trans-Activators - metabolism | Fibroblasts - cytology | Mice | MicroRNAs - genetics | Colorectal Neoplasms - pathology | Cell Movement | Proteins | Gene expression | Cell adhesion & migration | Index Medicus
Journal Article
Cancer Letters, ISSN 0304-3835, 03/2015, Volume 358, Issue 2, pp. 124 - 135
Upregulation of CXCR2 in tumor cells has been documented in several types of cancer. As one of its ligands, CXCL5 is associated with neutrophil infiltration... 
Hepatocellular carcinoma | Epithelial–mesenchymal transition | Metastasis | CXCL5 | CXCR2 | Epithelial-mesenchymal transition | TUMOR PROGRESSION | CURATIVE RESECTION | CANCER | HEPATOCELLULAR-CARCINOMA | INVASION | ONCOLOGY | POOR-PROGNOSIS | IN-VIVO | SNAIL | Carcinoma, Hepatocellular - mortality | Prognosis | Humans | Middle Aged | Glycogen Synthase Kinase 3 beta | Male | Phosphatidylinositol 3-Kinases - metabolism | Chemokine CXCL5 - metabolism | Epithelial-Mesenchymal Transition - genetics | Liver Neoplasms - mortality | Neoplasm Metastasis | Heterografts | Neoplasm Grading | Carcinoma, Hepatocellular - genetics | Adult | Female | Liver Neoplasms - pathology | Proto-Oncogene Proteins c-akt - metabolism | Snail Family Transcription Factors | Disease Models, Animal | Gene Expression | Liver Neoplasms - genetics | Signal Transduction | Tumor Burden | Glycogen Synthase Kinase 3 - metabolism | Disease Progression | Chemokine CXCL5 - genetics | Transcription Factors - metabolism | Animals | Receptors, Interleukin-8B - metabolism | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Receptors, Interleukin-8B - genetics | Cell Line, Tumor | Aged | Neoplasm Staging | Carcinoma, Hepatocellular - metabolism | Animal models | Senescence | Mesenchyme | Xenotransplantation | AKT protein | Activation | Kinases | Multivariate analysis | Cell spreading | Metastases | Proteins | Liver cancer | Signal transduction | Rodents | Xenografts | Tumorigenesis | Phenotypes | Tumor cells | CXCR2 protein | Therapeutic applications | Cells | 1-Phosphatidylinositol 3-kinase | Signaling | Plasmids | Medical prognosis | Cell lines | Infiltration | Regulation | Laboratory animals | Immunofluorescence | Liver cirrhosis | Cell migration | Cancer | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 02/2016, Volume 35, Issue 8, pp. 1049 - 1057
Journal Article
Nature Cell Biology, ISSN 1465-7392, 10/2004, Volume 6, Issue 10, pp. 931 - 940
The phenotypic changes of increased motility and invasiveness of cancer cells are reminiscent of the epithelial-mesenchymal transition (EMT) that occurs during... 
BREAST-CANCER | REPRESSION | PATHWAY | E-CADHERIN EXPRESSION | GLYCOGEN-SYNTHASE KINASE-3 | TRANSCRIPTION FACTOR SNAIL | TUMOR-CELLS | NF-KAPPA-B | CARCINOMA | PROGRESSION | CELL BIOLOGY
Journal Article