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Journal Article
Journal Article
Experimental Cell Research, ISSN 0014-4827, 08/2013, Volume 319, Issue 14, pp. 2282 - 2295
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2014, Volume 289, Issue 36, pp. 25079 - 25087
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 04/2012, Volume 302, Issue 8, pp. G794 - G804
Exogenous GLP-2 and IGF-I induce a differential intestinal response in IGF binding protein-3 and -5 double knockout mice. Am J Physiol Gastrointest Liver... 
Intestinal adaptation | Epiregulin | ErbB ligands | Glucagon-like peptide-2 receptor | Proglucagon | SYSTEM | SMALL-BOWEL RESECTION | PHYSIOLOGY | glucagon-like peptide-2 receptor | ENTERAL NUTRIENTS | intestinal adaptation | OVEREXPRESSION | proglucagon | epiregulin | GLUCAGON-LIKE PEPTIDE-2 | ADAPTATION | PARENTERALLY FED RATS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | ASSOCIATION | GROWTH-FACTOR-I | Insulin-Like Growth Factor Binding Protein 3 - genetics | Intestines - drug effects | Genes, erbB - physiology | Insulin-Like Growth Factor I - pharmacology | Colon - drug effects | Male | Colon - anatomy & histology | Body Weight - genetics | Intestinal Mucosa - drug effects | Intestinal Mucosa - growth & development | Intestines - anatomy & histology | RNA - isolation & purification | Glucagon-Like Peptide 2 - pharmacology | Intestine, Small - growth & development | Organ Size - genetics | Colon - growth & development | Insulin-Like Growth Factor Binding Protein 5 - physiology | Real-Time Polymerase Chain Reaction | Insulin-Like Growth Factor Binding Protein 3 - physiology | Spleen - growth & development | Mice, Inbred C57BL | Intestines - growth & development | Kidney - growth & development | Organ Size - physiology | Mice, Knockout | RNA - biosynthesis | Animals | Analysis of Variance | Intestine, Small - anatomy & histology | Intestine, Small - drug effects | Mice | Body Weight - physiology | Insulin-Like Growth Factor Binding Protein 5 - genetics | Intestines | Peptides | Physiological aspects | Genetic aspects | Research | Binding proteins | Gene expression | Health aspects | Growth factors | Proteins | Digestive system | Rodents | Binding sites | Index Medicus | Mucosal Biology
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 07/2002, Volume 94, Issue 14, pp. 1099 - 1106
Journal Article
Circulation Research, ISSN 0009-7330, 10/2009, Volume 105, Issue 9, pp. 897 - 905
RATIONALE:Insulin-like growth factor binding protein (IGFBP)-3 modulates vascular development by regulating endothelial progenitor cell (EPC) behavior,... 
Insulin-like growth factor binding protein-3 | Vascular repair | Nitric oxide | Hematopoietic stem cells | nitric oxide | STEM-CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | RAT RETINA | LOW-DENSITY-LIPOPROTEIN | RECEPTOR | ENDOTHELIAL-CELL APOPTOSIS | FACTOR-I | CHOROIDAL NEOVASCULARIZATION | vascular repair | SPHINGOSINE 1-PHOSPHATE | RETINOPATHY | PERIPHERAL VASCULAR DISEASE | hematopoietic stem cells | HEMATOLOGY | insulin-like growth factor binding protein-3 | Cell Proliferation | Mesenteric Arteries - physiopathology | Humans | Male | Green Fluorescent Proteins - genetics | Retinal Neovascularization - metabolism | Retinopathy of Prematurity - physiopathology | Vasodilation | Retinal Vessels - pathology | Insulin-Like Growth Factor Binding Protein 3 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Infant, Newborn | Cytoprotection | Disease Models, Animal | Animals, Newborn | Gene Transfer Techniques | Sphingomyelins - metabolism | Retinal Vessels - metabolism | Signal Transduction | Endothelial Cells - metabolism | Rats | Mice, Transgenic | Mesenteric Arteries - metabolism | Cell Adhesion Molecules - metabolism | Rats, Sprague-Dawley | Cerebral Arteries - metabolism | Stem Cells - pathology | Mice | Retinopathy of Prematurity - metabolism | Endothelial Cells - pathology | Retinal Neovascularization - physiopathology | Retinal Neovascularization - pathology | Astrocytes - metabolism | Cell Movement | Insulin-Like Growth Factor Binding Protein 3 - genetics | Phosphatidylinositol 3-Kinases - metabolism | Phosphoproteins - metabolism | Stem Cells - metabolism | Cell Death | Female | Microfilament Proteins - metabolism | Nitric Oxide Synthase Type III - metabolism | Ceramides - metabolism | Mice, Inbred C57BL | Cells, Cultured | Cerebral Arteries - physiopathology | Pregnancy | Animals | Retinal Vessels - physiopathology | Scavenger Receptors, Class B - metabolism | Nitric Oxide - metabolism | Retinopathy of Prematurity - pathology | Index Medicus | Vascular Repair | Hematopoietic Stem Cells
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e98641 - e98641
Insulin-like-factor-binding-protein 3 (IGFBP-3) is known to modulate the activity of insulin-like growth factors (IGFs) besides having a number of... 
CELLS | PLASMA-LEVELS | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | IGF-BINDING PROTEIN-3 | CANCER-RISK | GROWTH-FACTOR (IGF)-I | TUMOR-GROWTH | Melanoma - blood | Humans | Middle Aged | Insulin-Like Growth Factor Binding Protein 3 - blood | Glycogen Synthase Kinase 3 beta | Male | Insulin-Like Growth Factor Binding Protein 3 - pharmacology | Neoplasm Metastasis | Insulin-Like Growth Factor Binding Protein 3 - metabolism | Melanocytes - drug effects | Melanocytes - pathology | Adult | Female | Proto-Oncogene Proteins c-akt - metabolism | Melanoma - metabolism | Recombinant Proteins - metabolism | Tumor Microenvironment - drug effects | Neoplasm Invasiveness | Melanoma - pathology | Recombinant Proteins - pharmacology | Glycogen Synthase Kinase 3 - metabolism | Disease Progression | Recombinant Proteins - blood | Up-Regulation - drug effects | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Survival Analysis | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Biological products | Chemical properties | Growth | Protein binding | Drugs | Colorectal cancer | AKT protein | Insulin-like growth factors | Metastasis | Cancer therapies | Blood | Metastases | Proteins | Animal tissues | Cell cycle | Insulin-like growth factor-binding protein 3 | Inhibition | Growth factors | Cell survival | Internal medicine | Medical treatment | Melanoma | Plastic surgery | Pharmacology | Insulin | Chemical compounds | Patients | Survival | College campuses | Medicine | Medical prognosis | Cell lines | Prostate cancer | Tyrosinase | Cell migration | Melanin | Index Medicus
Journal Article