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Lancet, The, ISSN 0140-6736, 2009, Volume 374, Issue 9684, pp. 145 - 158
Summary Hyperparathyroidism is due to increased activity of the parathyroid glands, either from an intrinsic abnormal change altering excretion of parathyroid... 
Internal Medicine | VITAMIN-D SUPPLEMENTATION | GLOMERULAR-FILTRATION-RATE | MEDICINE, GENERAL & INTERNAL | BONE-MINERAL DENSITY | CALCIUM-SENSING RECEPTOR | ASYMPTOMATIC PRIMARY HYPERPARATHYROIDISM | CALCIMIMETIC CINACALCET HCL | PARATHYROID-HORMONE ASSAY | QUALITY-OF-LIFE | FAMILIAL HYPOCALCIURIC HYPERCALCEMIA | LEFT-VENTRICULAR HYPERTROPHY | Hyperparathyroidism, Primary - diagnosis | Hyperparathyroidism, Secondary - therapy | Prevalence | Vitamin D Deficiency - complications | Humans | Parathyroidectomy | Incidence | Patient Selection | Calcium - physiology | Hyperparathyroidism, Secondary - epidemiology | Hyperparathyroidism, Primary - therapy | Hyperparathyroidism, Primary - epidemiology | Parathyroid Hormone - physiology | Acute Disease | Diagnosis, Differential | Algorithms | Hyperparathyroidism, Secondary - diagnosis | Mass Screening | Homeostasis - physiology | Kidney Failure, Chronic - complications | Hyperparathyroidism, Primary - etiology | Decision Trees | Causality | Hyperparathyroidism, Secondary - etiology | Practice Guidelines as Topic | Complications and side effects | Care and treatment | Hormone therapy | Diphosphonates | Postmenopausal women | Research | Health aspects | Hyperparathyroidism | Risk factors | Comparative studies | Thyroid gland | Radiation therapy | Vitamin D | Hemodialysis | Cells
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2012, Volume 122, Issue 7, pp. 2543 - 2553
Chronic kidney disease-mineral and bone disorder (CKD-MBD) is associated with secondary hyperparathyroidism (HPT) and serum elevations in the phosphaturic... 
MEDICINE, RESEARCH & EXPERIMENTAL | FAMILIAL TUMORAL CALCINOSIS | VITAMIN-D METABOLISM | CARDIOVASCULAR EVENTS | VASCULAR CALCIFICATION | HEMODIALYSIS-PATIENTS | PARATHYROID-HORMONE | PHYSIOLOGICAL-ROLE | FIBROBLAST GROWTH FACTOR-23 | LEFT-VENTRICULAR HYPERTROPHY | STAGE RENAL-DISEASE | Chronic Kidney Disease-Mineral and Bone Disorder - blood | Kidney - pathology | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Hyperparathyroidism, Secondary - physiopathology | Male | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Parathyroid Hormone - blood | Tibia - pathology | Fibroblast Growth Factors - immunology | Luciferases - genetics | Renal Insufficiency, Chronic - metabolism | Calcium - blood | Fibroblast Growth Factors - metabolism | Phosphates - blood | Tibia - metabolism | Genes, Reporter | Kidney - physiopathology | CHO Cells | Biomarkers - metabolism | Cricetinae | Hyperparathyroidism, Secondary - metabolism | Glomerular Filtration Rate | Rats | Myocardium - pathology | Vascular Calcification - pathology | Calcitriol - blood | Rats, Sprague-Dawley | Renal Insufficiency, Chronic - physiopathology | Aorta - pathology | Renal Insufficiency, Chronic - blood | Hyperparathyroidism, Secondary - blood | Animals | Mice | Mice, Inbred BALB C | Fibroblast Growth Factors - antagonists & inhibitors | Hemodynamics | Antibodies, Monoclonal, Murine-Derived - pharmacology | Luciferases - biosynthesis | Complications and side effects | United States | Mortality | Research | Kidney diseases | Hyperparathyroidism | Risk factors
Journal Article
Journal Article
Familial isolated hyperparathyroidism caused by single adenoma, 12/2005
Hyper parathyroidism is associated with increased secretion of parathyroid hormone. Primary hyper parathyroidism is due to adenoma or hyperplasia and very... 
Familial primary hyperparathyroidism, parathyroid adenoma, parathyroidectomy
Journal
Nature Genetics, ISSN 1061-4036, 12/2002, Volume 32, Issue 4, pp. 676 - 680
Journal Article
Head & Neck, ISSN 1043-3074, 02/2015, Volume 37, Issue 2, pp. 299 - 307
Journal Article
Journal Article