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1. Full Text Fibroblast Growth Factor 23 and Risks of Mortality and End-Stage Renal Disease in Patients With Chronic Kidney Disease
by Isakova, Tamara and Xie, Huiliang and Yang, Wei and Xie, Dawei and Anderson, Amanda Hyre and Scialla, Julia and Wahl, Patricia and Gutiérrez, Orlando M and Steigerwalt, Susan and He, Jiang and Schwartz, Stanley and Lo, Joan and Ojo, Akinlolu and Sondheimer, James and Hsu, Chi-yuan and Lash, James and Leonard, Mary and Kusek, John W and Feldman, Harold I and Wolf, Myles and Chronic Renal Insufficiency Cohort (CRIC) Study Group, for the and CRIC Study Grp and Chronic Renal Insufficiency Cohort (CRIC) Study Group
JAMA, ISSN 0098-7484, 06/2011, Volume 305, Issue 23, pp. 2432 - 2439
CONTEXT A high level of the phosphate-regulating hormone fibroblast growth factor 23 (FGF-23) is associated with mortality in patients with end-stage renal... 
PHOSPHORUS | MEDICINE, GENERAL & INTERNAL | FIBROBLAST-GROWTH-FACTOR-23 | VASCULAR DYSFUNCTION | PARATHYROID-HORMONE | HEMODIALYSIS | DEATH | PROGRESSION | FGF RECEPTOR | CALCITRIOL | PREDICTION | Kidney Failure, Chronic - mortality | Glomerular Filtration Rate | Prognosis | Prospective Studies | Outcome Assessment (Health Care) | Humans | Middle Aged | Risk Factors | Kidney Diseases - mortality | Male | Biomarkers - blood | Disease Progression | Kidney Failure, Chronic - blood | Fibroblast Growth Factors - blood | Female | Aged | Kidney Diseases - blood | Chronic Disease | Care and treatment | Patient outcomes | Fibroblast growth factors | Kidney diseases | Diagnosis | Health aspects | Risk factors | Hormones | Mortality | Chronic illnesses
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2. Full Text Fibroblast growth factor 23 is elevated before parathyroid hormone and phosphate in chronic kidney disease
by Isakova, Tamara and Wahl, Patricia and Vargas, Gabriela S and Gutiérrez, Orlando M and Scialla, Julia and Xie, Huiliang and Appleby, Dina and Nessel, Lisa and Bellovich, Keith and Chen, Jing and Hamm, Lee and Gadegbeku, Crystal and Horwitz, Edward and Townsend, Raymond R and Anderson, Cheryl A.M and Lash, James P and Hsu, Chi-yuan and Leonard, Mary B and Wolf, Myles and on behalf of the Chronic Renal Insufficiency Cohort (CRIC) Study Group and CRIC Study Grp and on behalf of the Chronic Renal Insufficiency Cohort (CRIC) Study Group
Kidney International, ISSN 0085-2538, 06/2011, Volume 79, Issue 12, pp. 1370 - 1378
Fibroblast growth factor 23 (FGF23) regulates phosphorus metabolism and is a strong predictor of mortality in dialysis patients. FGF23 is thought to be an... 
phosphate | FGF23 | chronic kidney disease | MORTALITY | VITAMIN-D | KLOTHO | FIBROBLAST-GROWTH-FACTOR-23 | SECONDARY HYPERPARATHYROIDISM | RENAL-DISEASE | FGF RECEPTOR | PATHOGENESIS | CALCIUM | UROLOGY & NEPHROLOGY | PHOSPHORUS-METABOLISM | Predictive Value of Tests | Up-Regulation | Biomarkers - urine | Prospective Studies | United States | Humans | Middle Aged | Hyperparathyroidism, Secondary - physiopathology | Male | Parathyroid Hormone - blood | Renal Insufficiency, Chronic - complications | Hyperparathyroidism, Secondary - urine | Hyperphosphatemia - blood | Time Factors | Phosphates - urine | Phosphates - blood | Female | Hyperphosphatemia - urine | Renal Insufficiency, Chronic - diagnosis | Severity of Illness Index | Glomerular Filtration Rate | Phosphorus, Dietary - metabolism | Biomarkers - blood | Renal Insufficiency, Chronic - physiopathology | Renal Insufficiency, Chronic - blood | Hyperparathyroidism, Secondary - blood | Hyperparathyroidism, Secondary - diagnosis | Fibroblast Growth Factors - blood | Hyperphosphatemia - etiology | Aged | Hyperparathyroidism, Secondary - etiology | Hyperphosphatemia - physiopathology | Hyperphosphatemia - diagnosis | Renal Insufficiency, Chronic - urine
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3. Full Text Cardiac actions of fibroblast growth factor 23
by Faul, Christian, PhD
Bone, ISSN 8756-3282, 2016, Volume 100, pp. 69 - 79
Abstract Fibroblast growth factors (FGF ) are mitogenic signal mediators that induce cell proliferation and survival. Although cardiac myocytes are... 
Orthopedics | Cardiac hypertrophy | Chronic kidney disease | Fibroblast growth factors | Uremic cardiomyopathy | Receptor tyrosine kinases | CARDIOVASCULAR EVENTS | ANGIOTENSIN-II | FACTOR RECEPTOR | LEFT-VENTRICULAR HYPERTROPHY | PHOSPHATE HOMEOSTASIS | FACTOR 21 PROTECTS | FGF-RECEPTOR | SERUM-LEVELS | MYOCARDIAL HYPERTROPHY | ENDOCRINOLOGY & METABOLISM | CHRONIC KIDNEY-DISEASE | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Humans | Fibroblast Growth Factors - genetics | Signal Transduction - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Renal Insufficiency, Chronic - metabolism | Cardiomyopathies - genetics | Fibroblast Growth Factors - metabolism | Animals | Receptor Protein-Tyrosine Kinases - genetics | Cardiomyopathies - metabolism | Renal Insufficiency, Chronic - genetics | Signal Transduction - physiology | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Heart failure | Heart | Chronic kidney failure | Cell death | Analysis | Stress (Physiology) | Tyrosine | Phosphates | Medical colleges | T cells | Sulfates | Cells | Epidermal growth factor | Parathyroid hormone | Mitogens | Protein kinases
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4. Full Text FGF receptors control vitamin D and phosphate homeostasis by mediating renal FGF‐23 signaling and regulating FGF‐23 expression in bone
by Wöhrle, Simon and Bonny, Olivier and Beluch, Noemie and Gaulis, Swann and Stamm, Christelle and Scheibler, Marcel and Müller, Matthias and Kinzel, Bernd and Thuery, Anne and Brueggen, Joseph and Hynes, Nancy E and Sellers, William R and Hofmann, Francesco and Graus‐Porta, Diana
Journal of Bone and Mineral Research, ISSN 0884-0431, 10/2011, Volume 26, Issue 10, pp. 2486 - 2497
The functional interaction between fibroblast growth factor 23 (FGF‐23) and Klotho in the control of vitamin D and phosphate homeostasis is manifested by the... 
KLOTHO | FGF RECEPTOR INHIBITOR | PHOSPHATE HOMEOSTASIS | FGF‐23 | VITAMIN D METABOLISM | phosphate homeostasis | vitamin D metabolism | FGF receptor inhibitor | Klotho | FGF-23 | FAMILIAL TUMORAL CALCINOSIS | D METABOLISM | O-GLYCOSYLATION | DOMINANT HYPOPHOSPHATEMIC RICKETS | IN-VIVO | ENDOCRINOLOGY & METABOLISM | MISSENSE MUTATION | PARATHYROID-HORMONE | FIBROBLAST GROWTH FACTOR-23 | OSTEOGLOPHONIC DYSPLASIA | TRANSGENIC MICE | Cell Line | Gene Expression Regulation | Blotting, Western | Fibroblast Growth Factors - metabolism | Kidney - metabolism | Animals | Fibroblast Growth Factors - blood | Bone and Bones - metabolism | Homeostasis - physiology | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Vitamin D - physiology | Receptors, Fibroblast Growth Factor - physiology | Real-Time Polymerase Chain Reaction
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5. Full Text Vascular klotho deficiency potentiates the development of human artery calcification and mediates resistance to fibroblast growth factor 23
by Lim, Kenneth and Lu, Tzong-Shi and Molostvov, Guerman and Lee, Christina and Lam, F.T and Zehnder, Daniel and Hsiao, Li-Li
Circulation, ISSN 0009-7322, 05/2012, Volume 125, Issue 18, pp. 2243 - 2255
Background-Klotho is known to function as a cofactor for the phosphatonin, fibroblast growth factor (FGF)-23 at the kidney. FGF-23 levels rise in chronic... 
Vascular calcification | Chronic kidney disease | Inflammation | Vitamin D | Klotho | VITAMIN-D | chronic kidney disease | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | ENDOTHELIAL DYSFUNCTION | HORMONE KLOTHO | vitamin D | vascular calcification | IN-VITRO | inflammation | FGF-RECEPTOR | HEMODIALYSIS-PATIENTS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | EXPRESSION | Cell Line | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Serum Response Factor - metabolism | Humans | Nuclear Proteins - metabolism | Aorta - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Glucuronidase - deficiency | MAP Kinase Signaling System | Renal Insufficiency, Chronic - metabolism | Fibroblast Growth Factors - metabolism | Vascular Calcification - metabolism | Glucuronidase - biosynthesis | Receptors, Calcitriol - agonists | Trans-Activators - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Myocytes, Smooth Muscle - metabolism
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6. Full Text Fibroblast growth factor 23 accelerates phosphate-induced vascular calcification in the absence of Klotho deficiency
by Jimbo, Rika and Kawakami-Mori, Fumiko and Mu, Shengyu and Hirohama, Daigoro and Majtan, Bohumil and Shimizu, Yuichiro and Yatomi, Yutaka and Fukumoto, Seiji and Fujita, Toshiro and Shimosawa, Tatsuo
Kidney International, ISSN 0085-2538, 05/2014, Volume 85, Issue 5, pp. 1103 - 1111
Fibroblast growth factor 23 (FGF23) is a phosphate-regulating hormone that acts primarily on the kidney and parathyroid. With declining kidney function there... 
phosphate | chronic kidney disease | Klotho | fibroblast growth factor 23 | vascular calcification | Vascular calcification | Chronic kidney disease | Fibroblast growth factor 23 | Phosphate | VITAMIN-D | AORTIC CALCIFICATION | OSTEOBLAST DIFFERENTIATION | CARDIOVASCULAR EVENTS | FGF RECEPTOR 1 | ENDOTHELIAL-CELLS | ARTERIAL CALCIFICATION | HEMODIALYSIS-PATIENTS | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | Phosphorylation | Muscle, Smooth, Vascular - metabolism | Glucuronidase - metabolism | Myocytes, Smooth Muscle - pathology | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Aorta - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Renal Insufficiency, Chronic - complications | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Aortic Diseases - chemically induced | Aortic Diseases - metabolism | Glucuronidase - deficiency | Dose-Response Relationship, Drug | Renal Insufficiency, Chronic - metabolism | Fibroblast Growth Factors - metabolism | Vascular Calcification - metabolism | Transfection | Phosphates - toxicity | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Aorta - drug effects | Osteoblasts - drug effects | Cells, Cultured | Vascular Calcification - pathology | Rats, Sprague-Dawley | Aorta - pathology | Osteoblasts - pathology | Muscle, Smooth, Vascular - pathology | Animals | Glucuronidase - genetics | Signal Transduction - drug effects | Cell Differentiation - drug effects | Vascular Calcification - chemically induced | Recombinant Proteins - toxicity | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Osteoblasts - metabolism | Fibroblast Growth Factors - toxicity
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7. Full Text Physiological Regulation and Disorders of Phosphate Metabolism -Pivotal Role of Fibroblast Growth Factor 23
by Fukumoto, Seiji
Internal Medicine, ISSN 0918-2918, 2008, Volume 47, Issue 5, pp. 337 - 343
Fibroblast growth factor (FGF) 23 has been identified as the last member of FGF family. FGF23 reduces serum phosphate level by suppressing proximal tubular... 
fibroblast growth factor | Klotho | hyperphosphatemia | hypophosphatemia | hormone | Hormone | Hyperphosphatemia | Fibroblast growth factor | Hypophosphatemia | BETA-KLOTHO | OSTEOMALACIA | FAMILIAL TUMORAL CALCINOSIS | PPAR-ALPHA | HEREDITARY HYPOPHOSPHATEMIC RICKETS | FIBROBLAST-GROWTH-FACTOR | FGF RECEPTOR | HYPERCALCIURIA | MEDICINE, GENERAL & INTERNAL | GENE | MISSENSE MUTATION | Phosphates - metabolism | Hypophosphatemia - physiopathology | Familial Hypophosphatemic Rickets - physiopathology | Humans | Osteomalacia - physiopathology | Calcification, Physiologic - physiology | Glucuronidase | Hyperphosphatemia - physiopathology | Fibroblast Growth Factors - physiology
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8. Full Text Serum fibroblast growth factor‐23 (FGF‐23) and fracture risk in elderly men
by Mirza, Majd AI and Karlsson, Magnus K and Mellström, Dan and Orwoll, Eric and Ohlsson, Claes and Ljunggren, Östen and Larsson, Tobias E and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Uppsala universitet and Institutionen för medicinska vetenskaper
Journal of Bone and Mineral Research, ISSN 0884-0431, 04/2011, Volume 26, Issue 4, pp. 857 - 864
A normal mineral metabolism is integral for skeletal development and preservation of bone integrity. Fibroblast growth factor 23 (FGF‐23) is a bone‐derived... 
OSTEOMALACIA | RICKETS | BONE MINERAL DENSITY | BMD | FRACTURES | VITAMIN D | FGF‐23 | CALCITRIOL | bone mineral density | osteomalacia | calcitriol | FGF-23 | vitamin D | rickets | fractures | FIBROBLAST-GROWTH-FACTOR-23 | VASCULAR DYSFUNCTION | TUMOR-INDUCED OSTEOMALACIA | HYPOPHOSPHATEMIC RICKETS | PHOSPHATE HOMEOSTASIS | ENDOCRINOLOGY & METABOLISM | OSTEOPOROTIC FRACTURES | OLDER MEN | BONE | CHRONIC KIDNEY-DISEASE | FGF23 | Bone Density | Prospective Studies | Age Factors | Humans | Risk Factors | Kaplan-Meier Estimate | Proportional Hazards Models | Spinal Fractures - epidemiology | Fractures, Bone - epidemiology | Male | Hip Fractures - blood | Hip Fractures - epidemiology | Sweden - epidemiology | Glomerular Filtration Rate - physiology | Fractures, Bone - blood | Fractures, Bone - etiology | Spinal Fractures - blood | Fibroblast Growth Factors - blood | Osteoporosis - complications | Aged, 80 and over | Aged | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
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9. Full Text Transgenic mice expressing fibroblast growth factor 23 under the control of the alpha 1(I) collagen promoter exhibit growth retardation, osteomalacia, and disturbed phosphate homeostasis
by Larsson, T and Marsell, R and Schipani, E and Ohlsson, C and Ljunggren, O and Tenenhouse, HS and Juppner, H and Jonsson, KB
ENDOCRINOLOGY, ISSN 0013-7227, 07/2004, Volume 145, Issue 7, pp. 3087 - 3094
Mutations in the fibroblast growth factor 23 gene, FGF23, cause autosomal dominant hypophosphatemic rickets (ADHR). The gene product, FGF-23, is produced by... 
PROTEOLYTIC CLEAVAGE | RICKETS | COTRANSPORTER GENES | FGF-23 | FACTOR (FGF)-23 | ENDOCRINOLOGY & METABOLISM | SKELETAL PHENOTYPE | TARGETED ABLATION | LINKED HYPOPHOSPHATEMIC HYP | ONCOGENIC OSTEOMALACIA | PHEX
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10. Full Text Fibroblast growth factor 23 and the heart
by Faul, Christian
Current Opinion in Nephrology and Hypertension, ISSN 1062-4821, 07/2012, Volume 21, Issue 4, pp. 369 - 375
PURPOSE OF REVIEWElevated serum levels of the phosphaturic hormone fibroblast growth factor 23 (FGF23) are strongly associated with chronic kidney disease... 
fibroblast growth factor receptor signaling | left ventricular hypertrophy | calcineurin | cardiorenal syndrome | CRYSTAL-STRUCTURE | PERITONEAL-DIALYSIS | ISCHEMIC-HEART | LEFT-VENTRICULAR HYPERTROPHY | FGF RECEPTOR | PHOSPHATE | STRUCTURAL BASIS | CARDIAC-HYPERTROPHY | UROLOGY & NEPHROLOGY | PERIPHERAL VASCULAR DISEASE | STAGE RENAL-DISEASE | Biomarkers - metabolism | Cardiovascular Diseases - etiology | Up-Regulation | Prognosis | Cardiovascular Diseases - metabolism | Signal Transduction | Humans | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - metabolism | Glucuronidase - metabolism | Myocardium - pathology | Cardiovascular Diseases - pathology | Fibroblast Growth Factors - metabolism | Ventricular Remodeling | Animals | Fibroblast Growth Factors - blood | Myocardium - metabolism | Cardiovascular Diseases - blood | Kidney Diseases - blood | Kidney Diseases - complications | Chronic Disease | Kidney Diseases - metabolism | Index Medicus
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