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The Journal of Pathology, ISSN 0022-3417, 09/2007, Volume 213, Issue 1, pp. 82 - 90
Fibroblast growth factor receptors (FGFRs) mediate the tumourigenic effects of FGFs in prostate cancer. These receptors are therefore potential therapeutic... 
prostate cancer | tissue microarray | laser capture microdissection | fibroblast growth factor receptors | Laser capture microdissection | Tissue microarray | Prostate cancer | Fibroblast growth factor receptors | FACTOR FAMILY | ISOFORMS | FGF | SPECIFICITY | RESTORATION | PATHOLOGY | TUMORIGENICITY | ARG ALLELE | EPITHELIAL-CELLS | GENES | PROGRESSION | Immunohistochemistry | Prostatic Neoplasms - metabolism | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Cell Proliferation | Microdissection | Oligonucleotide Array Sequence Analysis | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Humans | Gene Expression Regulation, Neoplastic | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Male | Gene Expression Profiling | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Case-Control Studies | Receptors, Fibroblast Growth Factor - analysis | Prostatic Neoplasms - genetics | RNA Interference | Receptor, Fibroblast Growth Factor, Type 2 - analysis | Receptors, Fibroblast Growth Factor - genetics | Transcription, Genetic | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Receptor, Fibroblast Growth Factor, Type 1 - analysis | Receptor, Fibroblast Growth Factor, Type 3 - genetics | RNA, Small Interfering - pharmacology | Receptor, Fibroblast Growth Factor, Type 4 - analysis | Microscopy, Confocal | Receptors, Fibroblast Growth Factor - metabolism | Cell Line, Tumor | Polymorphism, Single Nucleotide | Receptor, Fibroblast Growth Factor, Type 3 - analysis | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Protein Isoforms - genetics | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, pp. e0172407 - e0172407
Introduction Calciphylaxis/calcific uremic arteriolopathy affects mainly end-stage kidney disease patients but is also associated with malignant disorders such... 
MORTALITY | PREDICTORS | MULTIDISCIPLINARY SCIENCES | VASCULAR CALCIFICATION | DISEASE | MALIGNANT-MELANOMA | MODEL | DEFICIENCY | 5'-Nucleotidase - genetics | Calciphylaxis - etiology | Genetic Predisposition to Disease | Humans | Middle Aged | Fibroblast Growth Factors - genetics | Male | Receptors, Calcitriol - genetics | Calciphylaxis - genetics | Case-Control Studies | Uremia - genetics | Renal Dialysis - adverse effects | Aged, 80 and over | Female | Registries | Aged | Polymorphism, Single Nucleotide | Uremia - etiology | Germany | GPI-Linked Proteins - genetics | Vitamin D | Analysis | Calcification | Calcifediol | Genetic aspects | Alfacalcidol | Risk factors | Genetic polymorphisms | Nucleotidase | End-stage renal disease | Nephrology | Calcium | Fibroblast growth factor 23 | Hemodialysis | Genes | Cardiovascular disease | Single-nucleotide polymorphism | Minerals | Bone diseases | Myeloma | Epidemiology | CD73 antigen | Stanniocalcin | Gold | Osteocalcin | Statistical analysis | 5'-Nucleotidase | Mortality | Melanoma | Health risks | Breast cancer | Regression analysis | Risk analysis | Patients | Statistics | Studies | Hypotheses | Calciphylaxis | Genotyping | Klotho protein | Tagging | Dialysis | Kidney diseases | Mutation | Vitamin D receptors | Health risk assessment | Kidney transplantation | Cancer | Index Medicus
Journal Article
The EMBO Journal, ISSN 0261-4189, 02/2011, Volume 30, Issue 4, pp. 783 - 795
The epithelial–mesenchymal transition (EMT) is a crucial event in wound healing, tissue repair, and cancer progression in adult tissues. Here, we demonstrate... 
FGF‐2 | TGF‐β | δEF1 | FGF receptor | EMT | FGF-2 | TGF-β | dEF1 | DELTA-EF1 | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | IDENTIFICATION | SUPPRESSION | FIBROBLASTS | CELL BIOLOGY | PROSTATE ADENOCARCINOMA | COREPRESSORS | TGF-beta | SQUAMOUS-CELL CARCINOMA | CANCER PROGRESSION | TUMOR-GROWTH | Neoplasms - metabolism | Protein Binding - genetics | Myofibroblasts - physiology | Epithelial-Mesenchymal Transition - physiology | Homeodomain Proteins - metabolism | Humans | Actins - metabolism | Fibroblast Growth Factor 2 - pharmacology | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Actins - genetics | DNA-Binding Proteins - metabolism | Myofibroblasts - metabolism | Cell Differentiation - genetics | Protein Isoforms - metabolism | Neoplasms - genetics | Protein Binding - drug effects | Receptors, Fibroblast Growth Factor - genetics | Fibroblast Growth Factor 2 - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Cell Differentiation - physiology | Neoplasm Invasiveness | Alternative Splicing - genetics | Cells, Cultured | Transforming Growth Factor beta - physiology | Alcohol Oxidoreductases - metabolism | Signal Transduction - genetics | Myofibroblasts - drug effects | Protein Isoforms - physiology | Transcription Factors - metabolism | Transforming Growth Factor beta - pharmacology | Alternative Splicing - drug effects | Cell Differentiation - drug effects | Models, Biological | Receptors, Fibroblast Growth Factor - metabolism | Neoplasms - pathology | Protein Isoforms - genetics | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2016, Volume 291, Issue 36, pp. 18632 - 18642
Parathyroid hormone (PTH) and FGF23 are the primary hormones regulating acute phosphate homeostasis. Human renal proximal tubule cells (RPTECs) were used to... 
RENAL PROXIMAL TUBULE | PROTEIN-KINASE-A | MDCK CELLS | ACTIVATION | PTH | FGF-RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | I COTRANSPORTERS | FACTOR RECEPTOR GENES | CALCIUM-TRANSPORT | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Receptor, Parathyroid Hormone, Type 1 - metabolism | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Phosphoproteins - genetics | Phosphoproteins - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Fibroblast Growth Factor, Type 4 - biosynthesis | Parathyroid Hormone - genetics | Sodium-Hydrogen Exchangers - metabolism | Sodium-Phosphate Cotransporter Proteins, Type IIa - metabolism | Sodium-Phosphate Cotransporter Proteins, Type IIa - genetics | Fibroblast Growth Factors - metabolism | Phosphates - metabolism | Glucuronidase - biosynthesis | Glucuronidase - genetics | Parathyroid Hormone - metabolism | Receptor, Parathyroid Hormone, Type 1 - genetics | Signal Transduction - physiology | Sodium-Hydrogen Exchangers - genetics | Receptor, Fibroblast Growth Factor, Type 3 - biosynthesis | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Cell Line, Transformed | Index Medicus | parathyroid hormone | NPT2A | klotho | PDZ Protein | fibroblast growth factor receptor (FGFR) | NHERF1 | alternative splicing | G protein-coupled receptor (GPCR) | transport | Cell Biology
Journal Article
Cell Metabolism, ISSN 1550-4131, 12/2015, Volume 22, Issue 6, pp. 1020 - 1032
Chronic kidney disease (CKD) is a worldwide public health threat that increases risk of death due to cardiovascular complications, including left ventricular... 
SIGNAL-TRANSDUCTION | MORTALITY | FACTOR FAMILY | KLOTHO | POINT MUTATION | TYROSINE KINASE | ENDOCRINOLOGY & METABOLISM | CHRONIC KIDNEY-DISEASE | CANCER PROGRESSION | EXPRESSION | FGF RECEPTOR | CELL BIOLOGY | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Humans | Glucuronidase - metabolism | Fibroblast Growth Factors - genetics | Male | Renal Insufficiency, Chronic - metabolism | Fibroblast Growth Factors - metabolism | Hypertrophy, Left Ventricular - pathology | HEK293 Cells | Receptor, Fibroblast Growth Factor, Type 4 - deficiency | Female | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Disease Models, Animal | Phospholipase C gamma - metabolism | Mutagenesis, Site-Directed | Myocytes, Cardiac - cytology | Signal Transduction | Hypertrophy, Left Ventricular - metabolism | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Cells, Cultured | Rats | Rats, Sprague-Dawley | Gene Knock-In Techniques | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Animals | Glucuronidase - genetics | Myocytes, Cardiac - metabolism | Mice | Calcineurin - metabolism | Hypertension | Medical colleges | Chronic kidney failure | Heart enlargement | Stem cells | Physiological aspects | Fibroblast growth factors | Phospholipases | Drug discovery | T cells | Health aspects | Heart | Resveratrol | Development and progression | Kidney diseases | Cells | Index Medicus
Journal Article
Cancer Cell, ISSN 1535-6108, 10/2017, Volume 32, Issue 4, pp. 474 - 489.e6
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2005, Volume 102, Issue 51, pp. 18455 - 18460
Journal Article
Journal Article
Cellular Signalling, ISSN 0898-6568, 01/2018, Volume 42, pp. 144 - 154
Receptor tyrosine kinases (RTKs) form multiprotein complexes that initiate and propagate intracellular signals and determine the RTK-specific signalling... 
Fibroblast growth factor | receptor tyrosine kinase | interactome | signal transduction | FGFR3 | ACTIVATION | PROTEIN | FGF | PHOSPHORYLATION | CELL BIOLOGY | ARREST | CELL-MIGRATION | MOUSE MODEL | SKELETAL DYSPLASIA | S6 KINASE | C-SRC | Tyrosine | Phosphatases | Analysis | Phenols | Cellular signal transduction | Fibroblast growth factors | Cells | Index Medicus
Journal Article
Science, ISSN 0036-8075, 5/1997, Volume 276, Issue 5314, pp. 955 - 960
A new class of protein tyrosine kinase inhibitors was identified that is based on an oxindole core (indolinones). Two compounds from this class inhibited the... 
Molecules | Receptors | Oxygen | Phosphorylation | Phenyls | NIH 3T3 cells | Hydrogen bonds | Atoms | Antibodies | Reports | Insulin | HUMAN ASTROCYTOMAS | INSULIN-RECEPTOR | POINT MUTATION | MULTIDISCIPLINARY SCIENCES | GENES | K RO MULTIDISCIPLINARY SCIENCES | BREAST-CARCINOMA CELLS | CROUZON-SYNDROME | FGF FAMILY | SIGNAL TRANSDUCTION | EXPRESSION | CANCER | Receptor, Fibroblast Growth Factor, Type 1 | Protein-Tyrosine Kinases - metabolism | Crystallography, X-Ray | Piperazines - metabolism | Piperazines - chemistry | Phosphotyrosine - metabolism | Adenosine Triphosphate - metabolism | Enzyme Inhibitors - chemistry | Protein-Tyrosine Kinases - chemistry | Amino Acid Sequence | Pyrroles - metabolism | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Enzyme Inhibitors - metabolism | Enzyme Inhibitors - pharmacology | Receptors, Fibroblast Growth Factor - antagonists & inhibitors | Models, Molecular | Receptors, Fibroblast Growth Factor - chemistry | Receptor, Insulin - antagonists & inhibitors | Piperazines - pharmacology | Receptor Protein-Tyrosine Kinases | Pyrroles - pharmacology | Animals | Hydrogen Bonding | Receptors, Platelet-Derived Growth Factor - metabolism | Receptors, Fibroblast Growth Factor - metabolism | Pyrroles - chemistry | Receptor, Insulin - metabolism | Mice | 3T3 Cells | Protein-Tyrosine Kinases - antagonists & inhibitors | Cell proliferation | Protein tyrosine kinase | Fibroblast growth factors | Research | Cellular control mechanisms | Proteins | Cellular biology | Index Medicus
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