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1. Full Text Platelet-Rich Plasma in a Murine Model
by Denapoli, Priscila Martins Andrade and Stilhano, Roberta Sessa and Ingham, Sheila Jean McNeill and Han, Sang Won and Abdalla, Rene Jorge
American Journal of Sports Medicine, ISSN 0363-5465, 08/2016, Volume 44, Issue 8, pp. 1962 - 1971
leukocytes | muscle contusion | platelet-rich plasma | VEGF | Flt-1
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2. Full Text The vascular endothelial growth factor receptor (VEGFR-1) supports growth and survival of human breast carcinoma
by Wu, Yan and Hooper, Andrea T and Zhong, Zhaojing and Witte, Larry and Bohlen, Peter and Rafii, Shahin and Hicklin, Daniel J
International Journal of Cancer, ISSN 0020-7136, 10/2006, Volume 119, Issue 7, pp. 1519 - 1529
Vascular endothelial growth factor receptor 1 (VEGFR-1) is present on endothelial cells and subsets of human tumor cells, raising the hypothesis that... 
Angiogenesis | Breast cancer | VEGFR-1 (Flt-1) | Endothelium | Placental growth factor (PlGF) | MIGRATION | endothelium | SIGNALING PATHWAYS | angiogenesis | breast cancer | TUMOR ANGIOGENESIS | FLT-1 | CANCER | VEGFR-I (Flt-1) | INHIBITION | ONCOLOGY | placental growth factor (PIGF) | AUTOCRINE | SMOOTH-MUSCLE-CELLS | SEMAPHORIN SEMA3F | EXPRESSION | Cell Proliferation | Breast Neoplasms - immunology | Humans | Pregnancy Proteins - genetics | Gene Expression Regulation, Neoplastic | Pregnancy Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Vascular Endothelial Growth Factor A - genetics | Breast Neoplasms - metabolism | Sensitivity and Specificity | Vascular Endothelial Growth Factor Receptor-1 - immunology | Antibodies - immunology | Proto-Oncogene Proteins c-akt - metabolism | Vascular Endothelial Growth Factor Receptor-1 - genetics | Signal Transduction | Cell Survival | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Cell Line, Tumor | Placenta Growth Factor | Mice | Index Medicus
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3. Full Text Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfimction, hypertension, and proteinuria in preeclampsia
by Maynard, SE and Min, JY and Merchan, J and Lim, KH and Li, JY and Mondal, S and Libermann, TA and Morgan, LP and Sellke, FW and Stillman, IE and Epstein, FH and Sukhatme, VP and Karumanchi, SA
JOURNAL OF CLINICAL INVESTIGATION, ISSN 0021-9738, 03/2003, Volume 111, Issue 5, pp. 649 - 658
Preeclampsia, a syndrome affecting 5% of pregnancies, causes substantial maternal and fetal morbidity and mortality. The pathophysiology of preeclampsia... 
MEDICINE, RESEARCH & EXPERIMENTAL | PREGNANCIES | ANGIOGENESIS | VEGF | SERUM-LEVELS | NITRIC-OXIDE | FLT-1 | FACTOR RECEPTOR | BINDING | GROWTH-FACTOR CONCENTRATIONS
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4. Full Text PIGF/VEGFR-1 Signaling Promotes Macrophage Polarization and Accelerated Tumor Progression in Obesity
by Incio, J and Tam, J and Rahbari, NN and Suboj, P and McManus, DT and Chin, SM and Vardam, TD and Batista, A and Babykutty, S and Jung, K and Khachatryan, A and Hato, T and Ligibel, JA and Krop, IE and Puchner, SB and Schlett, CL and Hoffmman, U and Ancukiewicz, M and Shibuya, M and Carmeliet, P and Soares, R and Duda, DG and Jain, RK and Fukumura, D
CLINICAL CANCER RESEARCH, ISSN 1078-0432, 06/2016, Volume 22, Issue 12, pp. 2993 - 3004
Purpose: Obesity promotes pancreatic and breast cancer progression via mechanisms that are poorly understood. Although obesity is associated with increased... 
BREAST-CANCER | ACTIVATION | ADIPOSE-TISSUE DEVELOPMENT | METASTASIS | ANGIOGENESIS | ONCOLOGY | BONE-MARROW | BODY-MASS INDEX | FACTOR RECEPTOR-1 | FLT-1 | ENDOTHELIAL GROWTH-FACTOR
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5. Full Text Vascular Permeability Factor (VPF)/Vascular Endothelial Growth Factor (VEGF) Receptor-1 Down-modulates VPF/VEGF Receptor-2-mediated Endothelial Cell Proliferation, but Not Migration, through Phosphatidylinositol 3-Kinase-dependent Pathways
by Huiyan Zeng and Harold F. Dvorak and Debabrata Mukhopadhyay
Journal of Biological Chemistry, ISSN 0021-9258, 07/2001, Volume 276, Issue 29, pp. 26969 - 26979
Vascular permeability factor (VPF)/vascular endothelial growth factor (VEGF) achieves its multiple functions by activating two receptor tyrosine kinases, Flt-1... 
SIGNAL-TRANSDUCTION | ANGIOGENESIS | 3-KINASE | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | KDR | EXTRACELLULAR-MATRIX | TUMOR-CELLS | FLT-1 | IDENTIFICATION | EXPRESSION
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6. Full Text Mechanical forces induce changes in VEGF and VEGFR-1/sFlt-1 expression in human chondrocytes
by Beckmann, Rainer and Houben, Astrid and Tohidnezhad, Mersedeh and Kweider, Nisreen and Fragoulis, Athanassios and Wruck, Christoph J and Brandenburg, Lars O and Hermanns-Sachweh, Benita and Goldring, Mary B and Pufe, Thomas and Jahr, Holger
International Journal of Molecular Sciences, ISSN 1661-6596, 09/2014, Volume 15, Issue 9, pp. 15456 - 15474
Expression of the pro-angiogenic vascular endothelial growth factor (VEGF) stimulates angiogenesis and correlates with the progression of osteoarthritis.... 
sVEGFR-1/FLT-1 | Cyclic stretch | VEGFR-1/FLT-1 | VEGF-A | C-28/I2 | Human chondrocyte | Strain | strain | MATRIX | ANGIOGENESIS | STRETCH | BIOCHEMISTRY & MOLECULAR BIOLOGY | human chondrocyte | cyclic stretch | CHEMISTRY, MULTIDISCIPLINARY | ARTICULAR-CARTILAGE | OSTEOARTHRITIS | TRANSCRIPTIONAL REGULATION | CELL | FACTOR GENE | ENDOTHELIAL GROWTH-FACTOR | Cell Line | Promoter Regions, Genetic | Vascular Endothelial Growth Factor A - biosynthesis | Microscopy, Electron, Scanning | Enzyme-Linked Immunosorbent Assay | Cartilage, Articular - cytology | Humans | Cells, Cultured | Gene Expression Regulation | Stress, Mechanical | Vascular Endothelial Growth Factor Receptor-1 - biosynthesis | Vascular Endothelial Growth Factor A - genetics | Time Factors | Chondrocytes - ultrastructure | Cell Shape | Primary Cell Culture | In Vitro Techniques | Chondrocytes - metabolism | Genes, Reporter | Vascular Endothelial Growth Factor Receptor-1 - genetics | Gene expression | Mechanical engineering | Vascular endothelial growth factor | Index Medicus
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7. Full Text Endothelial cells dynamically compete for the tip cell position during angiogenic sprouting
by Jakobsson, L and Franco, C.A and Bentley, K and Collins, R.T and Ponsioen, B and Aspalter, I.M and Rosewell, I and Busse, M and Thurston, G and Medvinsky, A and Schulte-Merker, S and Gerhardt, H
Nature Cell Biology, ISSN 1465-7392, 2010, Volume 12, Issue 10, pp. 943 - 953
Sprouting angiogenesis requires the coordinated behaviour of endothelial cells, regulated by Notch and vascular endothelial growth factor receptor (VEGFR)... 
TRACHEAL BRANCHING MORPHOGENESIS | TYROSINE KINASE | VEGF | IN-VIVO | NETWORK FORMATION | NOTCH | MICE | GROWTH-FACTOR | FLT-1 | RED FLUORESCENT PROTEIN | CELL BIOLOGY | Retinal Vessels - metabolism | Signal Transduction | Endothelial Cells - metabolism | Receptors, Notch - metabolism | Mice, Inbred C57BL | Computational Biology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Zebrafish - embryology | Neovascularization, Physiologic - physiology | Animals | Endothelial Cells - cytology | Computer Simulation | Membrane Proteins - metabolism | Mice | Drosophila - metabolism | Physiological aspects | Vertebrae | Genetic aspects | Drosophila | Index Medicus
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8. Full Text Loss of HIF-1α in endothelial cells disrupts a hypoxia-driven VEGF autocrine loop necessary for tumorigenesis
by Tang, Nan and Wang, Lianchun and Esko, Jeffrey and Giordano, Frank J and Huang, Yan and Gerber, Hans-Peter and Ferrara, Napoleone and Johnson, Randall S
Cancer Cell, ISSN 1535-6108, 2004, Volume 6, Issue 5, pp. 485 - 495
We deleted the hypoxia-responsive transcription factor HIF-1α in endothelial cells (EC) to determine its role during neovascularization. We found that loss of... 
INDUCIBLE FACTOR-1-ALPHA | STEM-CELLS | ONCOLOGY | IN-VIVO | THERAPEUTIC IMPLICATIONS | MICE | PATHOLOGICAL ANGIOGENESIS | TUMOR ANGIOGENESIS | FLT-1 | TRANSCRIPTION FACTOR | GROWTH-FACTOR RECEPTOR-1 | CELL BIOLOGY
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9. Full Text Tumor refractoriness to anti- VEGF treatment is mediated by CD11b + Gr1 + myeloid cells
by Malik, Ajay K and Schanz, Stefanie and Gerber, Hans-Peter and Zhong, Cuiling and Fuh, Germaine and Wu, Xiumin and Baldwin, Megan E and Ferrara, Napoleone and Shojaei, Farbod
Nature Biotechnology, ISSN 1087-0156, 08/2007, Volume 25, Issue 8, pp. 911 - 920
Vascular endothelial growth factor ( VEGF) is an essential regulator of normal and abnormal blood vessel growth. A monoclonal antibody ( mAb) that targets VEGF... 
SIGNAL-TRANSDUCTION | PROGENITOR CELLS | STEM-CELLS | DENDRITIC CELLS | RECEPTOR FLT-1 | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | COMBINATION THERAPY | TYROSINE KINASE | DRUG-RESISTANCE | HUMAN MONOCYTES | ENDOTHELIAL GROWTH-FACTOR | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Drug Resistance, Neoplasm | Treatment Outcome | Antineoplastic Agents - administration & dosage | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Leukemia, Myeloid - drug therapy | Animals | Leukemia, Myeloid - metabolism | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Mice | Neovascularization, Pathologic - metabolism | CD11b Antigen - metabolism | Biotechnology | Cellular biology | Gene expression | Rodents | Tumors | Cancer | Index Medicus
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10. Full Text Hypoxia down-regulates sFlt-1 (sVEGFR-1) expression in human microvascular endothelial cells by a mechanism involving mRNA alternative processing
by Ikeda, Takayuki and Sun, Li and Tsuruoka, Naoki and Ishigaki, Yasuhito and Yoshitomi, Yasuo and Yoshitake, Yoshino and Yonekura, Hideto
Biochemical Journal, ISSN 0264-6021, 06/2011, Volume 436, Issue 2, pp. 399 - 407
sFlt-1 (soluble Flt-1) potently inhibits angiogenesis by binding extracellularly to VEGF (vascular endothelial growth factor). In the present paper, we report... 
Angiogenesis | Vascular endothelial cell | Hypoxia | mRNA 3′-end processing | Soluble Flt-1 | Vascular endothelial growth factor (VEGF) | hypoxia | angiogenesis | BIOCHEMISTRY & MOLECULAR BIOLOGY | mRNA 3 '-end processing | POLYADENYLATION | vascular endothelial growth factor (VEGF) | FACTOR CSTF-64 | RECEPTOR | PREECLAMPSIA | CLEAVAGE | POSSIBLE PARTICIPATION | soluble Flt-1 | ELEVATED LEVELS | GROWTH-FACTOR | PERICYTES | vascular endothelial cell | Vascular Endothelial Growth Factor Receptor-1 - antagonists & inhibitors | Microcirculation - genetics | Alternative Splicing - genetics | Humans | RNA, Messenger - genetics | Cells, Cultured | Molecular Sequence Data | Vascular Endothelial Growth Factor Receptor-1 - biosynthesis | Down-Regulation - genetics | RNA, Messenger - biosynthesis | Base Sequence | RNA, Messenger - antagonists & inhibitors | RNA 3' End Processing - genetics | Cell Hypoxia - genetics | Endothelial Cells - physiology | Vascular Endothelial Growth Factor Receptor-1 - genetics | Index Medicus | hnRNP, heterogeneous nuclear ribonucleoprotein | RT, reverse transcription | sVEGFR, soluble VEGFR | PHD, prolyl hydroxylase | HEK-293TN cell, HEK (human embryonic kidney)-293 cell expressing the large T-antigen of SV40 (simian virus 40) and the neomycin-resistance gene | mFlt-1, membrane-bound Flt-1 | CstF, cleavage stimulation factor | FBS, fetal bovine serum | KDR, kinase insert domain-containing receptor | HUVEC, human umbilical vein EC | VEGF, vascular endothelial growth factor | MVEC, microvascular EC | EC, endothelial cell | HIF, hypoxia-inducible factor | sFlt-1, soluble Flt-1 | VEGFR, VEGF receptor | bFGF, basic fibroblast growth factor | DMOG, dimethyloxaloylglycine | Cst F64, 64-kDa subunit of CstF | CFIM25, 25-kDa subunit of the cleavage factor Im | ARE, A+U-rich element | HMVEC, human MVEC
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11. Full Text Dynamic alterations in decoy VEGF receptor-1 stability regulate angiogenesis
by Boucher, Joshua M and Clark, Ryan P and Chong, Diana C and Citrin, Kathryn M and Wylie, Lyndsay A and Bautch, Victoria L
Nature Communications, ISSN 2041-1723, 06/2017, Volume 8, Issue 1, pp. 15699 - 15699
Blood vessel expansion is driven by sprouting angiogenesis of endothelial cells, and is essential for development, wound healing and disease.... 
SIGNAL-TRANSDUCTION | PROTEIN STABILITY | PALMITOYLATION | TYROSINE KINASE | ACYL TRANSFERASES | CELL-MIGRATION | MULTIDISCIPLINARY SCIENCES | MOUSE | LYMPHANGIOGENESIS | FLT-1 VEGFR-1 | ENDOTHELIAL GROWTH-FACTOR | Index Medicus
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12. Full Text Placental growth factor reconstitutes hematopoiesis by recruiting VEGFR1(+) stem cells from bone-marrow microenvironment
by Hattori, K and Heissig, B and Wu, Y and Dias, S and Tejada, R and Ferris, B and Hicklin, DJ and Zhu, ZP and Bohlen, P and Witte, L and Hendrikx, J and Hackett, NR and Crystal, RG and Moore, MAS and Werb, Z and Lyden, D and Rafii, S
NATURE MEDICINE, ISSN 1078-8956, 08/2002, Volume 8, Issue 8, pp. 841 - 849
The mechanism by which angiogenic factors recruit bone marrow ( BM)-derived quiescent endothelial and hematopoietic stem cells ( HSCs) is not known. Here, we... 
PROGENITOR CELLS | FACTOR VEGF | MEDICINE, RESEARCH & EXPERIMENTAL | MOBILIZATION | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR RECEPTOR-1 | TUMOR ANGIOGENESIS | FLT-1 | CELL BIOLOGY | INHIBITION | ENDOTHELIAL-CELLS | MICE
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13. Full Text MicroRNA-15b contributes to ginsenoside-Rg(1)-induced angiogenesis through increased expression. of VEGFR-2
by Chan, LS and Yue, PYK and Wong, YY and Wong, RNS
BIOCHEMICAL PHARMACOLOGY, ISSN 0006-2952, 08/2013, Volume 86, Issue 3, pp. 392 - 400
Ginsenoside-Rg(1) (Rg(1)) has been identified as potent proangiogenic agent, which plays an important role in wound healing promotion or treatment of ischemic... 
PANAX-GINSENG | MECHANISM | HYPOXIA | FLT-1 | SIGNAL-TRANSDUCTION | Angiogenesis | Ginsenoside-Rg | GENOMICS | Microrna-15b | PATHWAY | VEGFR-2 | BIOLOGY | PHARMACOLOGY & PHARMACY | GINSENOSIDE RG1 | RECEPTORS | HUVECs
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14. Full Text Novel anti-VEGF chimeric molecules delivered by AAV vectors for inhibition of retinal neovascularization
by Pechan, P and Rubin, H and Lukason, M and Ardinger, J and DuFresne, E and Hauswirth, W.W and Wadsworth, S.C and Scaria, A
Gene Therapy, ISSN 0969-7128, 01/2009, Volume 16, Issue 1, pp. 10 - 16
Vascular endothelial growth factor (VEGF) is important in pathological neovascularization, which is a key component of diseases such as the wet form of... 
MEDICINE, RESEARCH & EXPERIMENTAL | VEGFR1 | ADENOASSOCIATED VIRAL VECTOR | IMMUNOGLOBULIN-LIKE DOMAIN | LIGAND-BINDING | VEGF | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACULAR DEGENERATION | THERAPEUTIC LEVELS | OCULAR NEOVASCULARIZATION | Fc fragment | FACTOR RECEPTOR FLT-1 | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | retinal neovascularization | adeno-associated virus 2 | GENETICS & HEREDITY | GENE-TRANSFER | Flt-1 | ENDOTHELIAL GROWTH-FACTOR | Protein Structure, Tertiary | Dependovirus - genetics | Gene Expression | Retina - metabolism | Genetic Vectors - administration & dosage | Humans | Mice, Inbred C57BL | RNA, Messenger - analysis | Retinal Neovascularization - metabolism | Retinal Neovascularization - therapy | Vascular Endothelial Growth Factor A - metabolism | Recombinant Fusion Proteins - metabolism | Genetic Vectors - genetics | Animals | Genetic Engineering | Fluorescent Antibody Technique | Protein Binding | Recombinant Fusion Proteins - genetics | Mice | Transgenes | Immunoglobulin Fc Fragments - genetics | Transduction, Genetic - methods | Genetic Therapy - methods | Vascular Endothelial Growth Factor Receptor-1 - genetics | Usage | Care and treatment | Genetic vectors | Genetic aspects | Research | Retinal diseases | Vascular endothelial growth factor | Health aspects | Retina | Ophthalmology | Gene therapy | Molecular biology | Binding sites | Index Medicus
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15. Full Text VEGF(164)-mediated inflammation is required for pathological, but not physiological, ischemia-induced retinal neovascularization
by Ishida, S and Usui, T and Yamashiro, K and Kaji, Y and Amano, S and Ogura, Y and Hida, T and Oguchi, Y and Ambati, J and Miller, JW and Gragoudas, ES and Ng, YS and D'Amore, PA and Shima, DT and Adamis, AP
JOURNAL OF EXPERIMENTAL MEDICINE, ISSN 0022-1007, 08/2003, Volume 198, Issue 3, pp. 483 - 489
Hypoxia-induced VEGF governs both physiological retinal vascular development and pathological retinal neovascularization. In the current paper, the mechanisms... 
FACTOR VEGF | MEDICINE, RESEARCH & EXPERIMENTAL | angiogenesis | VEGF | CELL-ADHESION | immunity | IMMUNOLOGY | FLT-1 | retina | POTENCY | leukocyte | RETINOPATHY | ADHESION MOLECULE-1 ICAM-1 | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR
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16. Full Text The dynamics of revascularization after white matter infarction monitored in Flt1-tdsRed and Flk1-GFP mice
by Shimauchi-Ohtaki, Hiroya and Kurachi, Masashi and Naruse, Masae and Shibasaki, Koji and Sugio, Shouta and Matsumoto, Ken and Ema, Masatsugu and Yoshimoto, Yuhei and Ishizaki, Yasuki
Neuroscience Letters, ISSN 0304-3940, 01/2019, Volume 692, pp. 70 - 76
Subcortical white matter infarction causes ischemic demyelination and loss of brain functions, as the result of disturbances of the blood flow. Although... 
Angiogenesis | Pericyte | Flt1 | Endothelial cell | VEGF | PDGF | White matter infarction | Flk1 | CELLS | BLOOD-VESSEL FORMATION | MODEL | FLT-1 | NEUROSCIENCES | STROKE | GROWTH | PERICYTES | Index Medicus
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17. Full Text Corneal avascularity is due to soluble VEGF receptor-1
by Collinson, J. Martin and Shibuya, Masabumi and Baffi, Judit Z and Albuquerque, Romulo J. C and Newcomb, Michael T and Samuelson, Don A and Jani, Pooja D and Suthar, Tushar and Sakurai, Eiji and Ambati, Balamurali K and Raisler, Brian J and Bogdanovich, Sasha and Witta, Jassir and Agnew, Dalen W and Khurana, Tejvir S and Singh, Nirbhai and Curiel, David T and Ogura, Yuichiro and Richter, Elizabeth and Marsh, Helene and Baldwin, Megan E and Kwan, Donna and Nozaki, Miho and Green, W. Richard and Caldwell, Ruth B and Lin, Qing and Mahasreshti, Parameshwar J and Ikeda, Sakae and Ambati, Jayakrishna and St. Leger, Judy and Leiper, Lucy J and De Falco, Sandro and Takeda, Atsunobu and Kleinman, Mark E and Gerber, Hans-Peter and Ferrara, Napoleone and Orecchia, Angela
Nature, ISSN 0028-0836, 10/2006, Volume 443, Issue 7114, pp. 993 - 997
Corneal avascularity-the absence of blood vessels in the cornea-is required for optical clarity and optimal vision, and has led to the cornea being widely used... 
ANIRIDIA | INHIBITION | ANGIOGENESIS | ABNORMALITIES | GENETIC ABLATION | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | GROWTH | MICE | NEOVASCULARIZATION | FLT-1 | Vascular Endothelial Growth Factor Receptor-1 - deficiency | RNA, Messenger - genetics | Solubility | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Cornea - metabolism | Animals | Cornea - blood supply | Trichechus | Gene Deletion | Mice | Neovascularization, Physiologic | Vascular Endothelial Growth Factor Receptor-1 - genetics | Genotype & phenotype | Cornea | Gene expression | Genetic recombination | Veins & arteries | Index Medicus
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