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Journal Article
Oncogene, ISSN 1476-5594, 2014, Volume 34, Issue 15, pp. 1979 - 1990
.... Since VAV2 serves as a GEF for the small Rho GTPase Rac1, a key player in cell motility and adhesion, we explored the vimentin-VAV2 pathway as a potential novel regulator of lung cancer cell motility... 
BIOCHEMISTRY & MOLECULAR BIOLOGY | MATRIX ADHESION | EPITHELIAL-MESENCHYMAL TRANSITIONS | CELL BIOLOGY | ACTIN STRESS FIBERS | ONCOLOGY | ENDOTHELIAL-CELLS | PROSTATE-CANCER | RAC1 ACTIVATION | GENETICS & HEREDITY | NUCLEOTIDE EXCHANGE FACTOR | GROWTH-FACTOR | INTERMEDIATE-FILAMENTS | CARCINOMA METASTASIS | Lung Neoplasms - genetics | Phosphorylation | Proto-Oncogene Proteins c-vav - genetics | Signal Transduction | Vimentin - metabolism | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Neoplasm Metastasis | Animals | Heterografts | Transfection | Cell Adhesion - physiology | HEK293 Cells | Cell Line, Tumor | Mice | Focal Adhesion Kinase 1 - metabolism | Proto-Oncogene Proteins c-vav - metabolism | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics | Care and treatment | Lung cancer | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Intermediate filament proteins | Proteins | Metastasis | Kinases | Cell adhesion & migration | Vimentin | Motility | Invasiveness | Rac1 protein | Tumor cell lines | Guanine | Metastases | Medical prognosis | Cell adhesion | Focal adhesion kinase | Guanine nucleotide exchange factor | Guanosinetriphosphatase | Cell motility | FAK | Focal Adhesion | Cell Adhesion
Journal Article
Journal Article
Journal Article
The Journal of pathology, ISSN 0022-3417, 2017, Volume 242, Issue 3, pp. 358 - 370
Focal adhesion kinase (FAK) inhibitors have been developed as potential anticancer agents and are undergoing clinical trials... 
tumour angiogenesis | focal adhesion kinase | LOCALIZATION | ACTIVATION | PERMEABILITY | PHOSPHORYLATION | BARRIER FUNCTION | TUMOR ANGIOGENESIS | PATHOLOGY | SRC-FAMILY KINASES | CANCER | ONCOLOGY | SMOOTH-MUSCLE-CELLS | AUTOPHOSPHORYLATION | Cell Division - genetics | Focal Adhesion Kinase 1 - genetics | Neoplasm Transplantation | Antineoplastic Agents, Hormonal - pharmacology | Melanoma - blood supply | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Capillary Permeability - drug effects | Melanoma - enzymology | Endothelium, Vascular - enzymology | Focal Adhesion Protein-Tyrosine Kinases - deficiency | Mutation - genetics | Capillary Permeability - genetics | Focal Adhesion Protein-Tyrosine Kinases - genetics | Homozygote | Neovascularization, Pathologic - enzymology | Animals | Melanoma - genetics | Tamoxifen - pharmacology | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Mice | Cell Hypoxia - genetics | Focal Adhesion Kinase 1 - metabolism | Tyrosine | Fibronectins | Platelet-derived growth factor | Endothelial growth factors | Analysis | Melanoma | Phenols | Genetic aspects | Neovascularization | Permeability | Endothelium | Residues | Phosphorylation | Clinical trials | Activation | Kinases | Blood | Fibronectin | Anticancer properties | Angiogenesis | Antitumor agents | Leakage | Medical research | Vessels | Blood vessels | Tamoxifen | Cadherin | Src protein | Adhesion | Pathology | Signaling | Inhibitors | Hypoxia | In vivo methods and tests | Mutation | Focal adhesion kinase | Paxillin | In vitro methods and tests | Tumors | Original Paper | Original Papers
Journal Article
Nature cell biology, ISSN 1476-4679, 2004, Volume 6, Issue 2, pp. 154 - 161
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 03/2009, Volume 24, Issue 3, pp. 411 - 424
.... Focal adhesions are prime candidates for transducing external stimuli. Focal adhesion kinase (FAK... 
mechanical loading | integrins | osteoblasts | stress/strain | osteopontin | Osteopontin | Stress/strain | Mechanical loading | Osteoblasts | Integrins
Journal Article