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Journal Article
Nature Medicine, ISSN 1078-8956, 03/2013, Volume 19, Issue 3, pp. 351 - 357
Previous attempts to identify neuroprotective targets by studying the ischemic cascade and devising ways to suppress it have failed to translate to efficacious... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | BRAIN-INJURY | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | HEAT-SHOCK PROTEIN | RAT MODEL | DELAYED NEURONAL DEATH | CELL BIOLOGY | HIPPOCAMPUS | RAPAMYCIN PROTECTS | CA1 | TRANSIENT FOREBRAIN ISCHEMIA | Rats, Wistar | Multiprotein Complexes | Male | Hypoxia-Ischemia, Brain - prevention & control | Autophagy - drug effects | Neuroprotective Agents - metabolism | Mechanistic Target of Rapamycin Complex 1 | RNA Interference | Tumor Suppressor Proteins - genetics | Prosencephalon - blood supply | CA3 Region, Hippocampal - metabolism | CA1 Region, Hippocampal - metabolism | Hypoxia-Ischemia, Brain - metabolism | Tumor Suppressor Proteins - metabolism | Adenine - analogs & derivatives | CA1 Region, Hippocampal - physiology | CA3 Region, Hippocampal - physiology | Cells, Cultured | Ischemic Preconditioning | Prosencephalon - injuries | Rats | Adenine - pharmacology | Sirolimus - pharmacology | Animals | Proteins - metabolism | Hypoxia | RNA, Small Interfering | TOR Serine-Threonine Kinases | Proteins - antagonists & inhibitors | Stroke (Disease) | Complications and side effects | Autophagy (Cytology) | Care and treatment | Physiological aspects | Genetic aspects | Research | Pharmacology | Ischemia | Gene expression | Neurons | Autophagy | global cerebral ischemia | endogenous neuroprotection | autophagy | CA3 | proteomics | hippocampus | hamartin | preconditioning
Journal Article