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Oncogene, ISSN 0950-9232, 03/2008, Volume 27, Issue 12, pp. 1677 - 1686
INK4 | FOXO | Akt
Journal Article
BLOOD, ISSN 0006-4971, 12/2018, Volume 132, Issue 25, pp. 2616 - 2618
Journal Article
Molecular Systems Biology, ISSN 1744-4292, 08/2010, Volume 6, Issue 1, pp. 399 - n/a
Journal Article
by Choi, D and Oh, KJ and Koo, SH
HEPATOLOGY, ISSN 0270-9139, 05/2013, Volume 57, Issue 5, pp. 2091 - 2091
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 01/2015, Volume 747, pp. 71 - 87
Proliferation of hepatic stellate cells (HSCs) is vital for the development of fibrosis during liver injury. In this study, we describe that arctigenin (ATG),... 
Arctigenin | p27Kip1 | Hepatic stellate cell | Proliferation | Liver fibrosis | Forkhead box O (FOXO) | p27 | Forkhead box O(FOXO)
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, p. e0167491
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 10/2019
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 10/2019, Volume 139, Issue 10, pp. 2098 - 2107
Psoriasis is a T lymphocyte–driven systemic inflammatory disease. Regulatory T cells (Tregs) are essential for establishing and maintaining immune tolerance.... 
BET | FOXO TRANSCRIPTION FACTORS | PROTEINS | EXPRESSION | DIFFERENTIATE | DERMATOLOGY
Journal Article
BLOOD, ISSN 0006-4971, 04/2012, Volume 119, Issue 15, pp. 3371 - 3372
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 10/2011, Volume 51, Issue 8, pp. 1575 - 1582
The superoxide free radical (O ) has been viewed as a likely major contributor to aging. If this is correct, then superoxide dismutase (SOD), which removes O ,... 
Caenorhabditis elegans | Free radicals | Oxidative damage | daf-16/FoxO | Aging | ER stress | Superoxide dismutase | BIOCHEMISTRY & MOLECULAR BIOLOGY | TISSUE | EXTENSION | STRESS RESISTANCE | LONGEVITY | HORMESIS | MUTANTS | ROS-GENERATOR | DAF-16 | ENDOCRINOLOGY & METABOLISM | EXPRESSION | C. ELEGANS | Superoxide Dismutase - genetics | Oxidation-Reduction | Animals, Genetically Modified | Heat-Shock Proteins - metabolism | Transcriptional Activation | Transgenes - genetics | Cells, Cultured | Oxidative Stress - genetics | Protein-Serine-Threonine Kinases - genetics | Caenorhabditis elegans Proteins - metabolism | Hydrogen Peroxide - metabolism | Catalase - metabolism | Transcription Factors - metabolism | Caenorhabditis elegans - physiology | Carrier Proteins - genetics | Heat-Shock Proteins - genetics | Animals | Carrier Proteins - metabolism | Superoxide Dismutase-1 | Caenorhabditis elegans Proteins - genetics | Forkhead Transcription Factors | Protein-Serine-Threonine Kinases - metabolism | Superoxide Dismutase - metabolism | Heat shock proteins | Acetylcysteine | Peroxides | Superoxide | CML, carboxymethyllysine | FoxO | daf-16 | IIS, insulin | Original Contribution | HNE, 4-hydroxynonenal | NAC, N-acetylcysteine | RNAi, RNA-mediated interference | co-OE, co-overexpression | HSF-1, heat shock factor-1 | SOD, superoxide dismutase | OE, overexpression | IGF-1 signaling | O2•−, superoxide anion | AMPK, AMP-dependent kinase | ROS, reactive oxygen species
Journal Article