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Nature Genetics, ISSN 1061-4036, 07/2012, Volume 44, Issue 7, pp. 751 - 759
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2015, Volume 21, Issue 11, pp. 1344 - 1349
Journal Article
Journal Article
Journal Article
Cell, ISSN 0092-8674, 2002, Volume 109, Issue 1, pp. 113 - 124
Runx3/Pebp2αC null mouse gastric mucosa exhibits hyperplasias due to stimulated proliferation and suppressed apoptosis in epithelial cells, and the cells are... 
CLEIDOCRANIAL DYSPLASIA | CONFORMATION POLYMORPHISM ANALYSIS | DOMAIN | AMPLIFICATION | EPITHELIAL-CELLS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUTATIONS | DIFFERENTIATION | FREQUENT LOSS | HETEROZYGOSITY | CELL BIOLOGY | Apoptosis - drug effects | Humans | Transcription Factors - deficiency | Epithelium - physiopathology | Stomach Neoplasms - metabolism | Apoptosis - genetics | Male | Gastric Mucosa - metabolism | Stomach Neoplasms - physiopathology | DNA-Binding Proteins - deficiency | DNA Methylation | Hyperplasia - genetics | Cell Transformation, Neoplastic - genetics | Tumor Suppressor Proteins - deficiency | Gene Deletion | Tumor Suppressor Proteins - genetics | Female | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Gene Expression Regulation, Neoplastic - genetics | Cell Division - genetics | Stomach - physiopathology | Stomach Neoplasms - genetics | Gene Targeting | Epithelium - pathology | Stomach - pathology | Epithelium - metabolism | Exons - genetics | Protein Structure, Tertiary - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Core Binding Factor Alpha 3 Subunit | Cell Division - drug effects | Mice, Knockout | Hyperplasia - pathology | Transforming Growth Factor beta - pharmacology | Animals | Hyperplasia - physiopathology | Mice | Cell Transformation, Neoplastic - drug effects | Transforming Growth Factor beta - metabolism | Cell research | Cell death | Analysis | Hyperplasia | Physiological aspects | Causes of | Genetic aspects | Gene expression | Stomach cancer | Mucous membrane
Journal Article
Oncology Letters, ISSN 1792-1074, 07/2015, Volume 10, Issue 1, pp. 392 - 398
High-risk human papillomavirus (HPV)16/18 infection in the development of lung cancer has previously been identified, and fragile histidine triad (FHIT) loss... 
FHIT loss | Human papillomavirus | p53 mutation | Lung cancer | 3P14.2 | FREQUENT | ADENOCARCINOMA | CERVICAL-CANCER | lung cancer | GENE | ONCOLOGY | human papillomavirus | FRAGILE SITES | HUMAN PAPILLOMA-VIRUS | EXPRESSION
Journal Article
Journal Article