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Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0165497
Zebrafish heart regeneration depends on cardiac cell proliferation, epicardium activation and transient reparative tissue deposition. The contribution and the... 
CRYOINJURY | FIN REGENERATION | MULTIDISCIPLINARY SCIENCES | SUPERFAMILY | EXTRACELLULAR-MATRIX | MECHANISMS | DIVERSITY | MUTATIONS | ADULT ZEBRAFISH | EXPRESSION | CARDIAC REGENERATION | Vimentin - metabolism | Heart - physiology | Pericardium - metabolism | Collagen Type I - genetics | In Situ Hybridization | Pericardium - pathology | Myocardium - metabolism | Dioxoles - pharmacology | Benzamides - pharmacology | Heart Ventricles - pathology | Collagen Type I - metabolism | Zebrafish Proteins - metabolism | Myocardium - pathology | Regeneration - physiology | Animals, Genetically Modified - metabolism | Connective Tissue - metabolism | Up-Regulation - drug effects | Animals | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Zebrafish - metabolism | Collagen Type XII - genetics | Collagen Type XII - metabolism | Heart Ventricles - metabolism | Transforming Growth Factor beta - metabolism | Microscopy, Fluorescence | Heart | Bone morphogenetic proteins | Glycoproteins | Transforming growth factors | Collagen | Analysis | Cell proliferation | Collagen (type I) | Collagens | Biology | Tissue analysis | Connective tissues | Proteins | Signal transduction | Cell activation | Rodents | Extracellular matrix | Heart diseases | Deposition | Immunoglobulins | Epicardium | Zebrafish | Cardiomyocytes | Mammals | Gene expression | Studies | Regeneration | Signaling | Inhibitors | Fibrosis | Ventricle | Cryoinjury
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 2014, Volume 34, Issue 8, pp. 1674 - 1680
Atherothrombosis is the main cause of myocardial infarction and ischemic stroke. Although the extrinsic (tissue factor-factor VIIa [FVIIa]) pathway is... 
Blood coagulation | Fibrin | Plaque rupture | Blood platelets | Thrombus | Tissue factor | Factor XII | blood platelets | ACTIVATION | tissue factor | blood coagulation | plaque rupture | thrombus | COAGULATION-FACTOR-XII | INHIBITION | PLASMA | HUMAN ATHEROSCLEROTIC PLAQUES | IN-VIVO | PERIPHERAL VASCULAR DISEASE | PLATELET GLYCOPROTEIN-VI | GENERATION | PROVIDES PROTECTION | fibrin | HEMATOLOGY | factor XII | Carotid Arteries - metabolism | Factor XIIa - metabolism | Apolipoproteins E - deficiency | Factor XII Deficiency - blood | Atherosclerosis - genetics | Humans | Aortic Diseases - blood | Carotid Artery Diseases - complications | Time Factors | Carotid Artery Diseases - pathology | Blood Coagulation | Factor XII Deficiency - genetics | Thrombosis - blood | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Factor XII - genetics | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Atherosclerosis - complications | Plaque, Atherosclerotic | Factor VIIa - metabolism | Mice, Knockout | Atherosclerosis - blood | Thrombosis - pathology | Aortic Diseases - genetics | Animals | Blood Platelets - metabolism | Thrombosis - etiology | Apolipoproteins E - genetics | Carotid Artery Diseases - blood | Factor XII - metabolism | Carotid Artery Diseases - genetics | Aortic Diseases - complications | Thrombosis - genetics | Carotid Arteries - pathology | Mice | Factor XI - metabolism | Thromboplastin - metabolism | Cholesterol, Dietary | Rupture, Spontaneous
Journal Article
11.