X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (201) 201
index medicus (199) 199
dna repair (137) 137
animals (133) 133
dna damage (119) 119
fanconi anemia - genetics (107) 107
fanconi anemia complementation group d2 protein - metabolism (101) 101
fanconi's anemia (96) 96
cancer (90) 90
mice (87) 87
fanconi anemia complementation group d2 protein - genetics (81) 81
fanconi anemia (77) 77
proteins (74) 74
cell biology (72) 72
homologous recombination (69) 69
genetic aspects (66) 66
mutation (66) 66
fanconi anemia - metabolism (65) 65
research (64) 64
biochemistry & molecular biology (63) 63
hemic and lymphatic diseases (61) 61
deoxyribonucleic acid--dna (57) 57
anemia (56) 56
pathway (55) 55
article (54) 54
female (52) 52
dna (51) 51
genetics & heredity (50) 50
oncology (46) 46
congenital, hereditary, and neonatal diseases and abnormalities (44) 44
cross-link repair (44) 44
fanconi-anemia (43) 43
genomic instability (43) 43
fancd2 (42) 42
fanconi syndrome (41) 41
repair (41) 41
nutritional and metabolic diseases (40) 40
multidisciplinary sciences (39) 39
mice, knockout (38) 38
biology (37) 37
cell line (36) 36
cell line, tumor (36) 36
fanconi anemia - pathology (36) 36
fanconi anemia complementation group proteins - metabolism (36) 36
dna-damage response (34) 34
male (34) 34
protein (34) 34
ubiquitination (34) 34
cell cycle (33) 33
double-strand breaks (33) 33
fanconi anemia complementation group d2 protein - deficiency (33) 33
health aspects (33) 33
medicine (33) 33
research article (33) 33
dna repair - genetics (31) 31
dna replication (31) 31
bone marrow (30) 30
dna-repair (30) 30
fanconi-anemia pathway (30) 30
physiological aspects (30) 30
signal transduction (29) 29
genetics (28) 28
genomes (28) 28
cells, cultured (27) 27
deoxyribonucleic acid (27) 27
fanconi anemia complementation group proteins - genetics (27) 27
genes (27) 27
dna-binding proteins - genetics (25) 25
dna-binding proteins - metabolism (25) 25
analysis (24) 24
apoptosis (24) 24
dna-damage (24) 24
monoubiquitinated fancd2 (24) 24
science (24) 24
breast cancer (23) 23
cells (23) 23
defects (23) 23
hematology (23) 23
risk factors (23) 23
mice, inbred c57bl (22) 22
hela cells (21) 21
chromatin (20) 20
gene expression (20) 20
genetic predisposition to disease (20) 20
histones - metabolism (19) 19
mammalian-cells (19) 19
replication (19) 19
stem cells (19) 19
targeted disruption (19) 19
tumors (19) 19
damage (18) 18
gene (18) 18
hematopoietic stem cells - metabolism (18) 18
molecular biology (18) 18
oxidative stress (18) 18
phosphorylation (18) 18
protein binding (18) 18
mutations (17) 17
nuclear proteins - genetics (17) 17
brca1 (16) 16
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 2280 - 14
Defects in DNA repair can cause various genetic diseases with severe pathological phenotypes. Fanconi anemia ( FA) is a rare disease characterized by bone... 
UBIQUITINATION | DAMAGE RESPONSE | CHROMATIN | CROSS-LINK REPAIR | HUMAN-CELLS | MULTIDISCIPLINARY SCIENCES | SENSITIVITY | HOMOLOGOUS RECOMBINATION | BRCA1 | ENRICHMENT ANALYSIS | ASSOCIATION | Genetic Therapy | Ubiquitin-Specific Proteases - genetics | Fanconi Anemia Complementation Group D2 Protein - genetics | Fanconi Anemia - metabolism | Humans | DNA Repair - physiology | DNA Repair - genetics | Fanconi Anemia Complementation Group C Protein - genetics | Fanconi Anemia Complementation Group A Protein - genetics | Fanconi Anemia Complementation Group D2 Protein - deficiency | Fanconi Anemia Complementation Group A Protein - metabolism | Fanconi Anemia Complementation Group Proteins - metabolism | Ubiquitination | Fanconi Anemia Complementation Group G Protein - genetics | Ubiquitin-Specific Proteases - deficiency | Fanconi Anemia Complementation Group G Protein - deficiency | BRCA1 Protein - metabolism | Fanconi Anemia - genetics | Ubiquitin-Specific Proteases - metabolism | Chromosomal Instability | Rad51 Recombinase - metabolism | Cell Line | Fanconi Anemia Complementation Group Proteins - deficiency | Fanconi Anemia Complementation Group Proteins - genetics | Fanconi Anemia Complementation Group G Protein - metabolism | Gene Knockout Techniques | Fanconi Anemia Complementation Group C Protein - metabolism | Fanconi Anemia Complementation Group D2 Protein - metabolism | Fanconi Anemia Complementation Group A Protein - deficiency | CRISPR-Cas Systems | Fanconi Anemia Complementation Group C Protein - deficiency | Fanconi Anemia - therapy | DNA Damage | Histones - metabolism | Mutation
Journal Article
Nature, ISSN 0028-0836, 07/2011, Volume 475, Issue 7354, pp. 53 - 59
Reactive aldehydes are common carcinogens. They are also by-products of several metabolic pathways and, without enzymatic catabolism, may accumulate and cause... 
DNA-ADDUCTS | METABOLISM | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | FETAL ALCOHOL SYNDROME | ETHANOL | CHILDHOOD LEUKEMIA | KNOCKOUT MICE | ACETALDEHYDE | FANCONI-ANEMIA | CONSUMPTION | Embryo, Mammalian - drug effects | Acetaldehyde - metabolism | Clone Cells - drug effects | Teratogens - toxicity | Ethanol - toxicity | Fanconi Anemia Complementation Group D2 Protein - genetics | Weaning | Aldehyde Dehydrogenase, Mitochondrial | Male | DNA Repair - genetics | Aldehydes - metabolism | Aldehydes - antagonists & inhibitors | Fanconi Anemia Complementation Group D2 Protein - deficiency | Precursor Cell Lymphoblastic Leukemia-Lymphoma - chemically induced | Hematopoiesis - drug effects | Fetal Alcohol Spectrum Disorders - etiology | Gene Deletion | Aldehyde Dehydrogenase - deficiency | Embryo Loss - etiology | DNA Damage - genetics | Fanconi Anemia - pathology | Female | Fanconi Anemia - genetics | Aldehyde Dehydrogenase - metabolism | B-Lymphocytes - metabolism | Bone Marrow - drug effects | Cell Line | Cell Survival - drug effects | Aldehydes - toxicity | Bone Marrow - physiopathology | Ethanol - metabolism | Acetaldehyde - toxicity | Embryo Loss - chemically induced | Mice, Inbred C57BL | Aldehyde Dehydrogenase - genetics | Genes, Essential | Embryo, Mammalian - abnormalities | Pregnancy | B-Lymphocytes - drug effects | Animals | Embryo, Mammalian - embryology | Fanconi Anemia Complementation Group D2 Protein - metabolism | Bone Marrow - pathology | Chickens | Alleles | Mice | Precursor Cell Lymphoblastic Leukemia-Lymphoma - etiology | Teratogens - metabolism | Causes of | Research | Aldehydes | Health aspects | Fanconi's anemia | Mutation | DNA repair | Stem cells | Cancer
Journal Article
Nature, ISSN 0028-0836, 02/2015, Volume 518, Issue 7538, pp. 258 - 262
Journal Article
Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 502, Issue 7471, pp. 381 - 384
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 3915 - 14
Journal Article
Journal Article
STEM CELLS, ISSN 1066-5099, 11/2015, Volume 33, Issue 11, pp. 3382 - 3396
Journal Article
Journal Article