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Journal Article
Journal of biomedical science, ISSN 1021-7770, 09/2015, Volume 22, Issue 1, pp. 77 - 77
...) targeting Fanconi anemia (FA)/BRCA pathway upstream genes on the sensitivity of cisplatin-sensitive (A549 and SK-MES-1) and -resistant (A549/DDP... 
RNA interference | Fanconi anemia/BRCA pathway | Lung cancer | Cisplatin | Chemoresistance | Core complex | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Lung Neoplasms - genetics | Lung Neoplasms - drug therapy | Fanconi Anemia Complementation Group D2 Protein - genetics | Humans | Lung Neoplasms - metabolism | Fanconi Anemia Complementation Group F Protein - metabolism | Fanconi Anemia Complementation Group L Protein - genetics | Fanconi Anemia Complementation Group D2 Protein - antagonists & inhibitors | Lung Neoplasms - pathology | Signal Transduction - genetics | Cisplatin - pharmacology | Fanconi Anemia Complementation Group F Protein - genetics | Fanconi Anemia Complementation Group L Protein - antagonists & inhibitors | BRCA1 Protein - genetics | Drug Resistance, Neoplasm - genetics | BRCA1 Protein - antagonists & inhibitors | Fanconi Anemia Complementation Group D2 Protein - metabolism | RNA Interference | Signal Transduction - drug effects | BRCA1 Protein - metabolism | Fanconi Anemia Complementation Group F Protein - antagonists & inhibitors | Cell Line, Tumor | Fanconi Anemia Complementation Group L Protein - metabolism | Drug Resistance, Neoplasm - drug effects | RNA | DNA damage | Genes | Respiratory agents | Drug resistance | Antineoplastic agents | Fanconi's anemia | Antimitotic agents | Prevention | Chemotherapy | Cancer cells | Genetic research | Genetic aspects | Drug therapy | Health aspects | Cancer | Ribonucleic acid--RNA | Anemia | Index Medicus
Journal Article
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 05/2007, Volume 117, Issue 5, pp. 1440 - 1449
Journal Article
Molecular cell, ISSN 1097-2765, 08/2018, Volume 71, Issue 4, pp. 621 - 628.e4
FANCA is a component of the Fanconi anemia (FA) core complex that activates DNA interstrand crosslink repair by monoubiquitination of FANCD2... 
FANCA | strand exchange | DNA double-strand break repair | Fanconi anemia | single-strand annealing | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Epithelial Cells - metabolism | Baculoviridae - metabolism | Humans | Baculoviridae - genetics | Moths | DNA Breaks, Double-Stranded | Fanconi Anemia Complementation Group A Protein - genetics | Fanconi Anemia Complementation Group A Protein - metabolism | Fanconi Anemia Complementation Group Proteins - metabolism | Fanconi Anemia Complementation Group G Protein - genetics | Rad52 DNA Repair and Recombination Protein - metabolism | DNA End-Joining Repair | DNA Mismatch Repair | Cloning, Molecular | Osteoblasts - cytology | Epithelial Cells - cytology | Recombinant Proteins - metabolism | Gene Expression | Recombinational DNA Repair | Genetic Vectors - chemistry | Fanconi Anemia Complementation Group Proteins - genetics | Genetic Vectors - metabolism | Fanconi Anemia Complementation Group G Protein - metabolism | Recombinant Proteins - genetics | DNA - metabolism | Rad52 DNA Repair and Recombination Protein - genetics | DNA - genetics | Animals | Cell Line, Tumor | Osteoblasts - metabolism | Yuan (China) | Medical colleges | Annealing | DNA | Genetic research | Molecular biology | Fanconi's anemia | Index Medicus | DNA double strand break repair
Journal Article
Molecular cell, ISSN 1097-2765, 07/2012, Volume 47, Issue 1, pp. 61 - 75
Journal Article
Nature (London), ISSN 1476-4687, 01/2018, Volume 553, Issue 7687, pp. 171 - 177
Haematopoietic stem cells renew blood. Accumulation of DNA damage in these cells promotes their decline, while misrepair of this damage initiates malignancies.... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Acetaldehyde - metabolism | Fanconi Anemia Complementation Group D2 Protein - genetics | Fanconi Anemia - metabolism | Ethanol - administration & dosage | Hematopoietic Stem Cells - pathology | Male | Fanconi Anemia Complementation Group D2 Protein - deficiency | Tumor Suppressor Protein p53 - genetics | DNA Breaks, Double-Stranded - drug effects | DNA End-Joining Repair | Gene Deletion | Genomic Instability - drug effects | Fanconi Anemia - pathology | Female | Alcohol Dehydrogenase - deficiency | Fanconi Anemia - genetics | Recombinational DNA Repair - drug effects | Alcohol Dehydrogenase - metabolism | Hematopoietic Stem Cells - drug effects | Cell Survival - drug effects | Ethanol - metabolism | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Hematopoietic Stem Cell Transplantation | Hematopoietic Stem Cells - metabolism | Ku Autoantigen - metabolism | Genes, p53 - genetics | Tumor Suppressor Protein p53 - deficiency | Whole Genome Sequencing | Ethanol - pharmacology | Animals | Fanconi Anemia Complementation Group D2 Protein - metabolism | Alcohol Dehydrogenase - genetics | Mice | Mutation | Aldehydes | Health aspects | Chromosomes | Hematopoietic stem cells | Alcohols | Recombination | Dehydrogenases | Cell survival | Ethanol | p53 Protein | DNA damage | Alcohol | Transplantation | Genomes | Chromosome rearrangements | DNA repair | Gene sequencing | Acetaldehyde | Genomic instability | Mutagenesis | Clonal deletion | Stem cells | Cell cycle | Alcoholic beverages | Damage accumulation | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article
Nature structural & molecular biology, ISSN 1545-9985, 10/2008, Volume 15, Issue 11, pp. 1138 - 1146
Journal Article