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2012, Türk Tarih Kurumu Yayınları.IV/B-3 Dizi, ISBN 9789751625175, Volume sa. 1., xxii, 1034 sayfa
Book
Nature, ISSN 0028-0836, 05/2010, Volume 465, Issue 7297, pp. 492 - 496
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 501, Issue 7466, pp. 247 - 251
Successful infection by enteric bacterial pathogens depends on the ability of the bacteria to colonize the gut, replicate in host tissues and disseminate to... 
SYSTEM | INDUCED APOPTOSIS | ROLES | MULTIDISCIPLINARY SCIENCES | DISEASE | CITROBACTER-RODENTIUM | VIRULENCE | PROTEINS | NLEB | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Fas Ligand Protein - metabolism | Humans | fas Receptor - deficiency | Caspase 8 - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - chemistry | Male | Citrobacter rodentium - pathogenicity | fas Receptor - metabolism | Enteropathogenic Escherichia coli - metabolism | Enteropathogenic Escherichia coli - pathogenicity | Cell Death | HEK293 Cells | Female | TNF Receptor-Associated Death Domain Protein - metabolism | Protein Structure, Tertiary | Citrobacter rodentium - physiology | Signal Transduction | Fas-Associated Death Domain Protein - metabolism | Escherichia coli Infections - microbiology | Gastrointestinal Tract - microbiology | Escherichia coli Proteins - metabolism | Escherichia coli Infections - metabolism | Fas-Associated Death Domain Protein - chemistry | N-Acetylglucosaminyltransferases - metabolism | Fas Ligand Protein - antagonists & inhibitors | Animals | TNF Receptor-Associated Death Domain Protein - chemistry | Escherichia coli Infections - pathology | Virulence Factors - metabolism | Mice | Enzyme Activation | HeLa Cells | Cell receptors | Bacterial infections | Pathogenic microorganisms | Microbiology | Physiological aspects | Research | Stomach diseases | Proteins | Yeast | Microscopy | Rodents | Infections | Kinases | Apoptosis | Index Medicus | EPEC | apoptosis | caspase-8 | death domain
Journal Article
2006, Medical intelligence unit, ISBN 0387321721, 160
"Fas Signaling" is focused on the signaling mechanisms and biology of the prototypic death receptor Fas, also called CD95 or APO-1. The chapters of this book... 
CD antigens | Adaptor Proteins, Signal Transducing | Cellular signal transduction | Membrane Glycoproteins | Antigens, CD95 | Clinical & internal medicine | Oncology | Molecular Medicine | Biomedicine | Immunology
Book
International Journal of Biological Sciences, ISSN 1449-2288, 2018, Volume 14, Issue 14, pp. 2103 - 2113
Journal Article
Journal Article
Bioscience, Biotechnology, and Biochemistry, ISSN 0916-8451, 02/2016, Volume 80, Issue 2, pp. 250 - 256
Advanced glycation end-products (AGEs) are extremely accumulated in the retinal vascular and epithelial cells of diabetes mellitus (DM) patients, particularly... 
apoptosis | human retinal cells | mitochondrial dysfunction | advanced glycation end-product (AGE) | Fas-FasL | Mitochondrial dysfunction | Advanced glycation end-product (AGE) | Human retinal cells | Apoptosis | SYSTEM | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOOD SCIENCE & TECHNOLOGY | INVOLVEMENT | RATIO | PROLIFERATION | BCL-2 | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | LIVER | NITRIC-OXIDE | CHEMISTRY, APPLIED | EXPRESSION | PERICYTES | Retina - drug effects | RNA, Small Interfering - genetics | Fas Ligand Protein - metabolism | Retina - metabolism | Epithelial Cells - metabolism | Recombinant Fusion Proteins - pharmacology | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Caspase 3 - metabolism | Caspase 8 - metabolism | Membrane Potential, Mitochondrial - drug effects | fas Receptor - metabolism | Caspase 8 - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Caspase 3 - genetics | Fas Ligand Protein - agonists | fas Receptor - genetics | Cell Line | Serum Albumin, Bovine - pharmacology | Signal Transduction | Gene Expression Regulation | fas Receptor - antagonists & inhibitors | Mitochondria - metabolism | Mitochondria - drug effects | fas Receptor - agonists | Fas Ligand Protein - antagonists & inhibitors | Glycation End Products, Advanced - pharmacology | Fas Ligand Protein - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2010, Volume 53, Issue 6, pp. 1069 - 1077
Journal Article
European Journal of Cancer, ISSN 0959-8049, 2013, Volume 49, Issue 12, pp. 2734 - 2746
Abstract Fas signalling has been shown to induce the epithelial–mesenchymal transition (EMT) to promote gastrointestinal (GI) cancer metastasis, but its... 
Hematology, Oncology and Palliative Medicine | Fas signalling | GSK-3β | Epithelial–mesenchymal transition | Metastasis | Gastrointestinal cancer | Epithelial-mesenchymal transition | Fas signaling | GLIOMA-CELLS | PROTEIN-KINASE | TRANSCRIPTION | TUMOR-CELLS | COLON-CANCER | ONCOLOGY | GASTRIC-CANCER | GSK3-BETA ACTIVITY | COLORECTAL-CANCER | GENE-EXPRESSION | GSK-3 beta | PROGRESSION | Immunohistochemistry | Fas Ligand Protein - metabolism | Gastrointestinal Neoplasms - genetics | Humans | Middle Aged | Glycogen Synthase Kinase 3 beta | Immunoblotting | Male | fas Receptor - metabolism | Gastrointestinal Neoplasms - pathology | Fas Ligand Protein - pharmacology | RNA Interference | Epithelial-Mesenchymal Transition | fas Receptor - genetics | Female | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Snail Family Transcription Factors | Gastrointestinal Neoplasms - metabolism | Kaplan-Meier Estimate | Signal Transduction - genetics | Transcription Factors - genetics | Glycogen Synthase Kinase 3 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | beta Catenin - genetics | Transcription Factors - metabolism | Glycogen Synthase Kinase 3 - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Models, Genetic | Fas Ligand Protein - genetics | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Journal Article
Inflammation Research, ISSN 1023-3830, 8/2017, Volume 66, Issue 8, pp. 679 - 690
The aim of this study was to elucidate the role of apoptosis mediated through Fas/FasL pathway using the mouse model of atopic dermatitis (AD). AD was induced... 
Allergology | Ovalbumin | Neurology | Immunology | Biomedicine | Dermatology | Atopic dermatitis | Rheumatology | Fas/FasL | Inflammation | Pharmacology/Toxicology | Apoptosis | FAS LIGAND | TISSUE-INJURY | KERATINOCYTES | IMMUNOLOGY | CELL BIOLOGY | ANTIVIRAL RESPONSE | REGULATORY T-CELLS | MICE | SKIN | HSV-2 INFECTION | EXPRESSION | Allergens | Dermatitis, Atopic - genetics | Skin - metabolism | Male | Fas Ligand Protein - immunology | Immunoglobulin E - blood | CD4-Positive T-Lymphocytes - immunology | fas Receptor - immunology | Dermatitis, Atopic - immunology | fas Receptor - genetics | Female | Cytokines - genetics | Macrophages - immunology | Skin - pathology | Disease Models, Animal | Skin - immunology | Mice, Inbred C57BL | Neutrophils - immunology | Mice, Transgenic | Dermatitis, Atopic - pathology | Collagen - metabolism | Animals | Dermatitis, Atopic - blood | Fas Ligand Protein - genetics | Immunohistochemistry | Albumin | Mice | T cells | Analysis | Animal models | Fas antigen | Pathogenesis | Staining | Immunoglobulin E | Inflammatory response | Lymphocytes T | mRNA | Macrophages | Dermatitis | Interleukin 4 | Interleukin 5 | Lymphocytes | Rodents | Interleukin 1 | Deposition | Thickening | Immunoregulation | Leukocytes (neutrophilic) | Interleukin 13 | Gene expression | Real time | Interleukin 17 | CD4 antigen | Polymerase chain reaction | FasL protein | Collagen | CXCL10 protein | Sampling methods | Skin | Index Medicus | FasL | Fas | Original Research Paper
Journal Article
The FEBS Journal, ISSN 1742-464X, 08/2018, Volume 285, Issue 15, pp. 2813 - 2827
Journal Article