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Nature Reviews Rheumatology, ISSN 1759-4790, 04/2017, Volume 13, Issue 4, pp. 217 - 233
TNF blockers are highly efficacious at dampening inflammation and reducing symptoms in rheumatic diseases such as rheumatoid arthritis, psoriatic arthritis and... 
LIGAND 1A TL1A | COLLAGEN-INDUCED ARTHRITIS | FIBROBLAST-LIKE SYNOVIOCYTES | TUMOR-NECROSIS-FACTOR | SYSTEMIC-LUPUS-ERYTHEMATOSUS | LYMPHOTOXIN-BETA-RECEPTOR | SYNOVIAL FIBROBLASTS | CD4(+) T-CELLS | CD40 LIGAND | MONOCLONAL-ANTIBODY | RHEUMATOLOGY | Fas Ligand Protein - metabolism | Rheumatic Diseases - drug therapy | OX40 Ligand - metabolism | CD27 Ligand - antagonists & inhibitors | Dendritic Cells - immunology | Humans | Lymphotoxin-alpha - metabolism | Molecular Targeted Therapy | 4-1BB Ligand - antagonists & inhibitors | TNF-Related Apoptosis-Inducing Ligand - antagonists & inhibitors | CD40 Ligand - metabolism | CD27 Ligand - metabolism | Cell Death | Rheumatic Diseases - immunology | Signal Transduction | Lymphocyte Activation | Tumor Necrosis Factors - metabolism | 4-1BB Ligand - metabolism | Immune Tolerance | Tumor Necrosis Factor Ligand Superfamily Member 15 - metabolism | TNF-Related Apoptosis-Inducing Ligand - metabolism | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | Tumor Necrosis Factors - immunology | OX40 Ligand - antagonists & inhibitors | Cytokine TWEAK | Fas Ligand Protein - antagonists & inhibitors | Animals | Tumor Necrosis Factor Ligand Superfamily Member 15 - antagonists & inhibitors | T-Lymphocytes - immunology | Tumor Necrosis Factor Ligand Superfamily Member 14 - antagonists & inhibitors | CD40 Ligand - antagonists & inhibitors | Lymphotoxin-alpha - antagonists & inhibitors | Tumor Necrosis Factors - antagonists & inhibitors | Care and treatment | Cytokines | Tumor necrosis factor | Rheumatic diseases | Development and progression | Genetic aspects | Health aspects
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e78355
Approximately 30% of patients with Epstein-Barr virus (EBV)-positive advanced nasopharyngeal carcinoma (NPC) display chemoresistance to cisplatin-based... 
CANCER-CELLS | B-CELLS | SURVIVAL | MEMBRANE-PROTEIN 1 | MULTIDISCIPLINARY SCIENCES | EBV | MIR-21 | LATENCY | LIGAND | EXPRESSION | LINES | RNA-Binding Proteins - genetics | Carcinoma | Apoptosis - drug effects | Humans | Nasopharyngeal Neoplasms - virology | MicroRNAs - metabolism | Viral Matrix Proteins - pharmacology | Tetrazolium Salts | Forkhead Transcription Factors - metabolism | Luciferases | Thiazoles | Apoptosis Regulatory Proteins - genetics | Inhibitory Concentration 50 | Viral Matrix Proteins - metabolism | Colony-Forming Units Assay | Gene Expression Regulation, Neoplastic - drug effects | Nasopharyngeal Neoplasms - drug therapy | Real-Time Polymerase Chain Reaction | RNA-Binding Proteins - antagonists & inhibitors | Cell Line | Nasopharyngeal Carcinoma | Cisplatin - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Drug Resistance, Neoplasm - genetics | Fas Ligand Protein - antagonists & inhibitors | Cisplatin - therapeutic use | Signal Transduction - drug effects | Apoptosis Regulatory Proteins - antagonists & inhibitors | Fluorescent Antibody Technique | MicroRNAs - genetics | Fas Ligand Protein - genetics | Forkhead Box Protein O3 | Infection | Prognosis | MicroRNA | Tumor necrosis factor | Epstein-Barr virus | B cells | Health aspects | Cisplatin | Apoptosis | Cancer | Cell proliferation | Laboratories | Genes | Crosstalk | Chemoresistance | Viruses | Oncology | Homology | AKT protein | Infections | Metastasis | Proteins | Nasopharyngeal carcinoma | Disease resistance | Signal transduction | Coding | Tumor necrosis factor-TNF | Tumorigenesis | Bioindicators | FOXO3 protein | MiRNA | Ribonucleic acid--RNA | 1-Phosphatidylinositol 3-kinase | Membrane proteins | Medicine | Signaling | Chemotherapy | FasL protein | Cell death | MicroRNAs | Medical prognosis | Cell lines | Biomarkers | Ligands | Latent membrane protein 1 | RNA | miRNA | Ribonucleic acid
Journal Article
Nature, ISSN 0028-0836, 05/2010, Volume 465, Issue 7297, pp. 492 - 496
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 1404 - 15
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 501, Issue 7466, pp. 247 - 251
Successful infection by enteric bacterial pathogens depends on the ability of the bacteria to colonize the gut, replicate in host tissues and disseminate to... 
SYSTEM | INDUCED APOPTOSIS | ROLES | MULTIDISCIPLINARY SCIENCES | DISEASE | CITROBACTER-RODENTIUM | VIRULENCE | PROTEINS | NLEB | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Fas Ligand Protein - metabolism | Humans | fas Receptor - deficiency | Caspase 8 - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - chemistry | Male | Citrobacter rodentium - pathogenicity | fas Receptor - metabolism | Enteropathogenic Escherichia coli - metabolism | Enteropathogenic Escherichia coli - pathogenicity | Cell Death | HEK293 Cells | Female | TNF Receptor-Associated Death Domain Protein - metabolism | Protein Structure, Tertiary | Citrobacter rodentium - physiology | Signal Transduction | Fas-Associated Death Domain Protein - metabolism | Escherichia coli Infections - microbiology | Gastrointestinal Tract - microbiology | Escherichia coli Proteins - metabolism | Escherichia coli Infections - metabolism | Fas-Associated Death Domain Protein - chemistry | N-Acetylglucosaminyltransferases - metabolism | Fas Ligand Protein - antagonists & inhibitors | Animals | TNF Receptor-Associated Death Domain Protein - chemistry | Escherichia coli Infections - pathology | Virulence Factors - metabolism | Mice | Enzyme Activation | HeLa Cells | Cell receptors | Bacterial infections | Pathogenic microorganisms | Microbiology | Physiological aspects | Research | Stomach diseases | Proteins | Yeast | Microscopy | Rodents | Infections | Kinases | Apoptosis | EPEC | apoptosis | caspase-8 | death domain
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2016, Volume 291, Issue 3, pp. 1221 - 1234
Journal Article
Cancer Letters, ISSN 0304-3835, 2010, Volume 298, Issue 1, pp. 88 - 98
Abstract Fas/APO-1/CD95, a member of the tumor necrosis factor (TNF) receptor superfamily, is a potential anti-cancer factor as it can induce apoptosis in... 
Hematology, Oncology and Palliative Medicine | Bcl-2 | Fas-mediated apoptosis | Cryptotanshinone | MAPK | DU145 prostate cancer cells | Apoptosis | ACTIVATED PROTEIN-KINASE | PANCREATIC-CANCER | DEATH | INDUCTION | DIABETES-MELLITUS | SALVIA-MILTIORRHIZA | INHIBITION | ONCOLOGY | RESISTANCE | TANSHINONE-IIA | Apoptosis - drug effects | Humans | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Fas Ligand Protein - immunology | Prostatic Neoplasms - immunology | MAP Kinase Kinase 4 - metabolism | Fas Ligand Protein - agonists | Antineoplastic Agents - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Fas Ligand Protein - biosynthesis | Prostatic Neoplasms - drug therapy | Cell Survival - drug effects | Prostatic Neoplasms - pathology | Jurkat Cells | Proto-Oncogene Proteins c-bcl-2 - biosynthesis | Drug Synergism | Phenanthrenes - pharmacology | Fas Ligand Protein - antagonists & inhibitors | Animals | Apoptosis - immunology | MAP Kinase Signaling System - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Mice | HeLa Cells | Chemotherapy | Cancer cells | Universities and colleges | Protein kinases | Cancer | Tumors | Proteins | Signal transduction | Obesity | Cloning | Homeostasis | Diabetes | Kinases | Prostate cancer | Cancer therapies | Chromatography | Molecular weight | Index Medicus
Journal Article
Science Signaling, ISSN 1937-9145, 2012, Volume 5, Issue 255, pp. ra93 - ra93
As a catalytically inactive homolog of caspase-8, a proapoptotic initiator caspase, c-FLIP blocks apoptosis by binding to and inhibiting caspase-8. The... 
INDUCED CELL-DEATH | CHRONIC INTESTINAL INFLAMMATION | JNK ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | COMPENSATORY PROLIFERATION | IKK-BETA | TNF-ALPHA | MEDIATED APOPTOSIS | NF-KAPPA-B | INDEPENDENT PATHWAY | CELL BIOLOGY | Organ Specificity - drug effects | Tumor Necrosis Factor-alpha - metabolism | Fas Ligand Protein - metabolism | Liver - pathology | Protein Binding - genetics | Caspase 8 | Colitis - genetics | Tumor Necrosis Factor-alpha - genetics | Homeostasis | Colitis - pathology | Hepatocytes - pathology | NF-kappa B - metabolism | Intestines - metabolism | Hepatocytes - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Necrosis - pathology | Organ Specificity - genetics | Mice, Mutant Strains | Hepatitis - genetics | Protein Binding - drug effects | Hepatitis - metabolism | CASP8 and FADD-Like Apoptosis Regulating Protein - metabolism | Intestines - pathology | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Liver - metabolism | Hepatitis - pathology | Antibodies, Neutralizing - pharmacology | Necrosis - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | Fas Ligand Protein - antagonists & inhibitors | Animals | NF-kappa B - genetics | Colitis - metabolism | Mice | Necrosis - genetics | Fas Ligand Protein - genetics | TNF-Related Apoptosis-Inducing Ligand - genetics | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Apoptosis
Journal Article