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1. Full Text Distinct effects of human glioblastoma immunoregulatory molecules programmed cell death ligand-1 (PDL-1) and indoleamine 2,3-dioxygenase (IDO) on tumour-specific T cell functions
by Avril, Tony and Saikali, Stéphan and Vauleon, Elodie and Jary, Anne and Hamlat, Abderrahmane and De Tayrac, Marie and Mosser, Jean and Quillien, Véronique
Journal of Neuroimmunology, ISSN 0165-5728, 2010, Volume 225, Issue 1, pp. 22 - 33
Abstract Immunotherapy is a promising new treatment for patients suffering from glioma, in particular glioblastoma multiforme (GBM). However, tumour cells use... 
Neurology | Allergy and Immunology | IDO | Immunomodulation | Immunotherapy | Glioblastoma | TGF-β | PDL-1 | T cells | GLIOMA-CELLS | FAS LIGAND | HLA-G EXPRESSION | PHASE-I/II TRIAL | IMMUNE ESCAPE | lmmunotherapy | IMMUNOLOGY | NEUROSCIENCES | MALIGNANT GLIOMA | TGF-beta | GROWTH-FACTOR-BETA | INTERFERON-GAMMA | CENTRAL-NERVOUS-SYSTEM | Indoleamine-Pyrrole 2,3,-Dioxygenase - metabolism | Fas Ligand Protein - metabolism | Flow Cytometry - methods | Transforming Growth Factor beta2 - metabolism | Humans | HLA Antigens - genetics | Interferon-gamma - metabolism | Indoleamine-Pyrrole 2,3,-Dioxygenase - pharmacology | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Antigens, CD - metabolism | Histocompatibility Antigens Class I - metabolism | T-Lymphocytes - drug effects | Antigens, Neoplasm - metabolism | Cytotoxicity, Immunologic - drug effects | B7-H1 Antigen | Oligonucleotide Array Sequence Analysis - methods | Gene Expression Profiling - methods | Lectins - pharmacology | Histocompatibility Antigens Class I - genetics | HLA-G Antigens | HLA Antigens - metabolism | Gene Expression Regulation - drug effects | Glioblastoma - immunology | Glioblastoma - pathology | Cell Line, Tumor | Antigens, CD - pharmacology | MART-1 Antigen | T-Lymphocytes - immunology | Cell Proliferation - drug effects | HLA-A2 Antigen - metabolism | Cytotoxicity, Immunologic - immunology | Fas Ligand Protein - genetics | Interferon-gamma - pharmacology | Cell research | Histocompatibility antigens | Messenger RNA | HLA histocompatibility antigens | Lectins | Biochemistry | Transforming growth factors | Glioblastoma multiforme | Apoptosis | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Histocompatibility Antigens Class I | Gene Expression Profiling | Life Sciences | Flow Cytometry | Immunology | Neoplasm Proteins | Transforming Growth Factor beta2 | Interferon-gamma | Antigens, Neoplasm | T-Lymphocytes | Gene Expression Regulation | Fas Ligand Protein | HLA-A2 Antigen | Indoleamine-Pyrrole 2,3,-Dioxygenase | HLA Antigens | RNA, Messenger | Antigens, CD | Cytotoxicity, Immunologic | Cancer
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2. Full Text Persistent antigen at vaccination sites induces tumor-specific CD8+ T cell sequestration, dysfunction and deletion
by Hailemichael, Yared and Dai, Zhimin and Jaffarzad, Nina and Ye, Yang and Medina, Miguel A and Huang, Xue-Fei and Dorta-Estremera, Stephanie M and Greeley, Nathaniel R and Nitti, Giovanni and Peng, Weiyi and Liu, Chengwen and Lou, Yanyan and Wang, Zhiqiang and Ma, Wencai and Rabinovich, Brian and Schluns, Kimberly S and Davis, Richard E and Hwu, Patrick and Overwijk, Willem W
Nature Medicine, ISSN 1078-8956, 04/2013, Volume 19, Issue 4, pp. 465 - 472
To understand why cancer vaccine-induced T cells often do not eradicate tumors, we studied immune responses in mice vaccinated with gp100 melanoma peptide in... 
CANCER PATIENTS | CTL IMMUNITY | MEDICINE, RESEARCH & EXPERIMENTAL | LONG PEPTIDE VACCINE | TOLERANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HIGH-AVIDITY | MELANOMA PATIENTS | INDUCTION | CHRONIC VIRAL-INFECTION | CELL BIOLOGY | IMMUNE-RESPONSES | EXHAUSTION | Cancer Vaccines - pharmacology | Mice, Inbred C57BL | Interferon-gamma - physiology | Antigen Presentation - immunology | Fas Ligand Protein - physiology | Cancer Vaccines - immunology | Animals | Apoptosis - immunology | Melanoma, Experimental - immunology | CD8-Positive T-Lymphocytes - drug effects | Female | Mice | CD8-Positive T-Lymphocytes - immunology | Antigens | T cell receptors | Vaccines | Peptides | Tumors | Cancer
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3. Full Text Cancer-associated fibroblasts induce antigen-specific deletion of CD8 + T Cells to protect tumour cells
by Lakins, Matthew A and Ghorani, Ehsan and Munir, Hafsa and Martins, Carla P and Shields, Jacqueline D
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 948 - 9
Tumours have developed strategies to interfere with most steps required for anti-tumour immune responses. Although many populations contribute to anti-tumour... 
ACTIVATION | MELANOMA | DENDRITIC CELLS | INFILTRATING LYMPHOCYTES | MULTIDISCIPLINARY SCIENCES | STEADY-STATE | PD-1 | EXPRESSION | PROGRESSION | EXHAUSTION | CROSS-PRESENTATION | Fas Ligand Protein - metabolism | Antigens, Neoplasm - immunology | Cell Survival | Mice, Inbred C57BL | Programmed Cell Death 1 Ligand 2 Protein - metabolism | Cancer-Associated Fibroblasts - pathology | Animals | Cross-Priming - immunology | Proteolysis | Female | CD8-Positive T-Lymphocytes - immunology | Cytoprotection | Cytotoxicity, Immunologic | Antigens | Immune response | CD8 antigen | Crosstalk | Cytotoxicity | Lymphocytes T | FasL protein | Immune checkpoint | Clonal deletion | Lymphocytes | Stromal cells | Fibroblasts | Deletion | Tumors | Cancer
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4. Full Text IL-2 and antigen dose differentially regulate perforin- and FasL-mediated cytolytic activity in antigen specific CD4 + T cells
by Brown, Deborah M and Kamperschroer, Cris and Dilzer, Allison M and Roberts, Deborah M and Swain, Susan L
Cellular Immunology, ISSN 0008-8749, 2009, Volume 257, Issue 1, pp. 69 - 79
CD4 T cell effectors can promote survival against lethal influenza virus via perforin mediated cytolytic mechanisms; however, our understanding of how naïve... 
Cytotoxicity | IL-2 | CD4 subsets | Peptide dose | Perforin | VIRUS-INFECTION | DENDRITIC CELLS | SECRETING TH1 | CD8-T-CELL MEMORY | CD4-T-CELL HELP | IMMUNOLOGY | CUTTING EDGE | CELL BIOLOGY | IN-VIVO | GENE-EXPRESSION | TRANSCRIPTION FACTOR | INFLUENZA INFECTION | Fas Ligand Protein - metabolism | Perforin - genetics | Ovalbumin - immunology | Dendritic Cells - immunology | Granzymes - metabolism | Th2 Cells - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Fas Ligand Protein - immunology | CD4-Positive T-Lymphocytes - immunology | Th1 Cells - metabolism | T-Lymphocytes, Cytotoxic - drug effects | Interleukin-4 - pharmacology | Interleukin-2 - immunology | Peptide Fragments - immunology | Granzymes - immunology | Perforin - immunology | Dendritic Cells - metabolism | Cytokines - immunology | T-Lymphocytes, Cytotoxic - immunology | Cytokines - metabolism | Interleukin-4 - metabolism | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Mice, Transgenic | Hemagglutinins, Viral - immunology | Th2 Cells - metabolism | Mice, Knockout | Cell Differentiation - immunology | Interleukin-4 - immunology | Animals | T-Lymphocytes, Cytotoxic - metabolism | Cell Differentiation - drug effects | Interferon-gamma - immunology | Interleukin-2 - pharmacology | Antigens - immunology | Mice | Mice, Inbred BALB C | Perforin - metabolism | CD4-Positive T-Lymphocytes - drug effects | Cytotoxicity, Immunologic | peptide dose | perforin | cytotoxicity
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5. Fas signaling
by Wajant, Harald
2006, Medical Intelligence Unit, ISBN 0387321721, 160
"Fas Signaling" is focused on the signaling mechanisms and biology of the prototypic death receptor Fas, also called CD95 or APO-1. The chapters of this book... 
CD antigens | Clinical & internal medicine | Cellular signal transduction | Oncology | Molecular Medicine | Biomedicine | Immunology
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6. Full Text Self-recognition of CD1 by γ/δ T cells: Implications for innate immunity
by Spada, Franca M and Grant, Ethan P and Peters, Peter J and Sugita, Masahiko and Melián, Augustín and Leslie, David S and Lee, Hoi K and Van Donselaar, Elly and Hanson, Dennis A and Krensky, Alan M and Majdic, Otto and Porcelli, Steven A and Morita, Craig T and Brenner, Michael B
Journal of Experimental Medicine, ISSN 0022-1007, 03/2000, Volume 191, Issue 6, pp. 937 - 948
The specificity of immunoglobulins and alpha/beta T cell receptors (TCRs) provides a framework for the molecular basis of antigen recognition. Yet, evolution... 
T lymphocytes | CD1 | Cytolysis | Granulysin | T cell antigen receptors γ/δ | MYCOBACTERIUM-TUBERCULOSIS | LISTERIA-MONOCYTOGENES | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | NONPEPTIDE ANTIGENS | IMMUNOLOGY | ANTIGEN-PRESENTING MOLECULES | T cell antigen receptors gamma/delta | cytolysis | RECEPTOR-GAMMA-DELTA | PROTECTIVE ROLE | IN-VIVO | granulysin | MONOCLONAL-ANTIBODIES | LYMPHOCYTES-T | T-Lymphocyte Subsets - immunology | Cytotoxicity Tests, Immunologic | T-Lymphocyte Subsets - cytology | Anti-Infective Agents - metabolism | Humans | Molecular Sequence Data | Receptors, Antigen, T-Cell, gamma-delta - genetics | Th1 Cells - immunology | Gene Rearrangement, delta-Chain T-Cell Antigen Receptor | Th1 Cells - metabolism | Membrane Glycoproteins - physiology | Base Sequence | Pore Forming Cytotoxic Proteins | fas Receptor - physiology | Perforin | Amino Acid Sequence | Cell Line | Antigen-Presenting Cells - metabolism | Antigens, CD1 - biosynthesis | Receptors, Antigen, T-Cell, gamma-delta - metabolism | Lymphocyte Activation | Immunity, Innate | Antigen-Presenting Cells - immunology | Gene Rearrangement, gamma-Chain T-Cell Antigen Receptor | Cell Differentiation - immunology | Receptors, Antigen, T-Cell, gamma-delta - physiology | T-Lymphocyte Subsets - metabolism | Antigens, CD1 - metabolism | T-Lymphocyte Subsets - microbiology | Antigens, Differentiation, T-Lymphocyte - biosynthesis | Cytotoxicity, Immunologic | T cell antigen receptors γ | Original
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7. Full Text CD36 Is a Marker of Human Adipocyte Progenitors with Pronounced Adipogenic and Triglyceride Accumulation Potential
by Gao, Hui and Volat, Fanny and Sandhow, Lakshmi and Galitzky, Jean and Nguyen, Thuy and Esteve, David and Åström, Gaby and Mejhert, Niklas and Ledoux, Severine and Thalamas, Claire and Arner, Peter and Guillemot, Jean‐Claude and Qian, Hong and Rydén, Mikael and Bouloumié, Anne
STEM CELLS, ISSN 1066-5099, 07/2017, Volume 35, Issue 7, pp. 1799 - 1814
White adipose tissue (WAT) expands in part through adipogenesis, a process involving fat cell generation and fatty acid (FA) storage into triglycerides (TGs).... 
Adipose | Differentiation | Gene expression | Adipose stem cells | CD34 | Adipogenesis | STEM-CELLS | OBESE SUBJECTS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | HUMAN ADIPOSE-TISSUE | ONCOLOGY | HUMAN WHITE | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | CHAIN FATTY-ACIDS | SCAVENGER RECEPTOR CD36 | INSULIN-RESISTANCE | PROTEINS | HEMATOLOGY | PRECURSOR CELLS | EXPRESSION | Adipocytes, White - cytology | RNA, Small Interfering - genetics | Antigens, CD34 - metabolism | CD36 Antigens - genetics | Cell Proliferation | Leukocyte Common Antigens - metabolism | Humans | Middle Aged | Transcriptome | Adipose Tissue, White - metabolism | Gene Expression Profiling | Stem Cells - cytology | Stem Cells - metabolism | Adipose Tissue, White - cytology | Biological Transport | CD36 Antigens - metabolism | Antigens, Surface - metabolism | Adult | Female | Cell Differentiation | Single-Cell Analysis | Adipogenesis - genetics | Antigens, Surface - genetics | Triglycerides - metabolism | CD36 Antigens - antagonists & inhibitors | Leukocyte Common Antigens - genetics | Primary Cell Culture | Antigens, CD34 - genetics | Adipocytes, White - metabolism | RNA, Small Interfering - metabolism | Triglycerides | Fatty acids | Analysis | Stem cells | Cell proliferation | Femur | Adipose tissue | Fas antigen | Regional development | Adipocytes | Accumulation | CD45 antigen | Storage capacity | Attenuation | CD34 antigen | Complications | RNA-mediated interference | Cloning | CD36 antigen | Metabolism | Storage | Human performance | Stromal cells | Biomarkers | In vitro methods and tests
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8. Full Text Locally produced C5a binds to T cell expressed C5aR to enhance effector T-cell expansion by limiting antigen-induced apoptosis
by Lalli, Peter N and Strainic, Michael G and Yang, Min and Lin, Feng and Medof, M. Edward and Heeger, Peter S
Blood, ISSN 0006-4971, 09/2008, Volume 112, Issue 5, pp. 1759 - 1766
Our recent studies have shown that immune cell-produced complement provides costimulatory and survival signals to naive CD4(+) T cells. Whether these signals... 
SURVIVAL | IMMUNITY | ACTIVATION | VIRUS-INFECTION | IN-VIVO | DECAY-ACCELERATING FACTOR | FACTOR DAF | DEATH | DEFICIENT MICE | HEMATOLOGY | VIRAL-INFECTION | Receptor, Anaphylatoxin C5a - genetics | Complement Activation | CD55 Antigens - genetics | Complement C5a - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Complement C5a - biosynthesis | fas Receptor - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Mice, Inbred MRL lpr | T-Lymphocytes - metabolism | Isoenzymes - metabolism | Class Ib Phosphatidylinositol 3-Kinase | Complement C3 - genetics | Signal Transduction | Complement C3 - metabolism | Lymphocyte Activation | Mice, Inbred C57BL | Mice, Transgenic | Cell Survival - immunology | Antigen-Presenting Cells - immunology | Mice, Knockout | Animals | Apoptosis - immunology | T-Lymphocytes - cytology | Receptor, Anaphylatoxin C5a - deficiency | CD55 Antigens - metabolism | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | Receptor, Anaphylatoxin C5a - metabolism | Immunobiology
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9. Full Text Fas Receptor Expression in Germinal-Center B Cells Is Essential for T and B Lymphocyte Homeostasis
by Hao, Zhenyue and Duncan, Gordon S and Seagal, Jane and Su, Yu-Wen and Hong, Claire and Haight, Jillian and Chen, Nien-Jung and Elia, Andrew and Wakeham, Andrew and Li, Wanda Y and Liepa, Jennifer and Wood, Geoffrey A and Casola, Stefano and Rajewsky, Klaus and Mak, Tak W
Immunity, ISSN 1074-7613, 2008, Volume 29, Issue 4, pp. 615 - 627
Fas is highly expressed in activated and germinal center (GC) B cells but can potentially be inactivated by misguided somatic hypermutation. We employed... 
CELLIMMUNO | CLONAL SELECTION | GENE | AUTOIMMUNITY | IMMUNE-RESPONSES | LYMPHOPROLIFERATIVE SYNDROME | NEGATIVE SELECTION | CD28 | MICE | MUTATIONS | IMMUNOLOGY | ANTIGEN | Antigens, CD - immunology | Cell Proliferation | Germinal Center - immunology | fas Receptor - deficiency | Homeostasis | fas Receptor - metabolism | Antigens, CD - metabolism | CD28 Antigens - metabolism | fas Receptor - immunology | CD40 Antigens - metabolism | T-Lymphocytes - metabolism | Mice, Mutant Strains | Cell Differentiation | B-Lymphocytes - metabolism | Cytokines - blood | B7-1 Antigen - immunology | Lymphocyte Activation | Mice, Inbred C57BL | Cell Communication | B7-1 Antigen - metabolism | CD28 Antigens - immunology | Mice, Knockout | CTLA-4 Antigen | Animals | B-Lymphocytes - immunology | Germinal Center - metabolism | T-Lymphocytes - immunology | Mice | CD40 Antigens - immunology | Physiological aspects | Lymphocytes | Medical research | Antigens | Autoimmune diseases | Rodents | Immune system
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10. Full Text Patients with tuberculosis disease have mycobacterium tuberculosis-specific CD8 T cells with a pro-apoptotic phenotype and impaired proliferative capacity, which is not restored following treatment
by Day, Cheryl L and Moshi, Noella D and Abrahams, Deborah A and Van Rooyen, Michele and O'Rie, Terrence and De Kock, Marwou and Hanekom, Willem A
PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, p. e94949
CD8 T cells play a critical role in control of chronic viral infections; however, the role of these cells in containing persistent bacterial infections, such... 
LATENT INFECTION | CD127 EXPRESSION | NITRIC-OXIDE SYNTHASE | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | IN-VIVO | PULMONARY TUBERCULOSIS | ACTIVE DISEASE | VIRUS-INFECTIONS | CHRONIC VIRAL-INFECTION | ANTIMICROBIAL ACTIVITY | Tuberculosis, Pulmonary - microbiology | Cell Proliferation | Proto-Oncogene Proteins c-bcl-2 - immunology | Apoptosis - drug effects | Humans | Middle Aged | Antigens, Bacterial - immunology | Mycobacterium tuberculosis - immunology | Ki-67 Antigen - metabolism | Male | Tuberculosis, Pulmonary - drug therapy | Interleukin-7 Receptor alpha Subunit - immunology | Interferon-gamma - metabolism | fas Receptor - metabolism | Young Adult | Proto-Oncogene Proteins c-bcl-2 - metabolism | fas Receptor - immunology | Flow Cytometry | Ki-67 Antigen - immunology | Latent Tuberculosis - drug therapy | Mycobacterium tuberculosis - physiology | CD8-Positive T-Lymphocytes - metabolism | Adult | Female | Bacterial Proteins - immunology | Mycobacterium tuberculosis - metabolism | CD57 Antigens - metabolism | CD57 Antigens - immunology | CD8-Positive T-Lymphocytes - microbiology | Immunophenotyping | Treatment Outcome | Latent Tuberculosis - immunology | Tuberculosis, Pulmonary - immunology | Apoptosis - immunology | Interferon-gamma - immunology | Interleukin-7 Receptor alpha Subunit - metabolism | Adolescent | Bacterial Proteins - metabolism | Aged | CD8-Positive T-Lymphocytes - immunology | Latent Tuberculosis - microbiology | Antigens, Bacterial - metabolism | CD57 antigen | Senescence | Bcl-2 protein | CD8 antigen | Cytotoxicity | Infections | Lymphocytes T | Restoration | Lymphocytes | Bacteria | Pathogens | Antigens | Immunological memory | Memory cells | Preservation | Cytokines | CD95 antigen | Medical treatment | Lung diseases | Markers | Patients | CD4 antigen | Medicine | Observatories | Infectious diseases | Tuberculosis | ESAT-6 antigen | γ-Interferon | Interferon | Viral infections | Apoptosis
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11. Full Text Most do, but some do not: CD4+CD25- T cells, but not CD4+CD25+ treg cells, are cytolytic when redirected by a chimeric antigen receptor (CAR)
by Hombach, Andreas A and Abken, Hinrich
Cancers, ISSN 2072-6694, 09/2017, Volume 9, Issue 9, p. 112
Evidences are accumulating that CD4(+) T cells can physiologically mediate antigen specific target cell lysis. By circumventing major histocompatibility... 
CD4 | Chimeric antigen receptor | CAR | T cell | Treg cell | ACTIVATION | PERFORIN | chimeric antigen receptor | IL-2 | SUPPRESSION | FASL | ONCOLOGY | ESTABLISHED MELANOMA | INFECTION | EXPRESSION | GRANZYME-B | LYMPHOCYTES | Cytolysis | Antigens | Fas antigen | CD95 antigen | CD8 antigen | T cell receptors | Lymphocytes T | CD28 antigen | CD4 antigen | Cell activation | Granzyme B | Major histocompatibility complex | FasL protein | Interleukin 2 | Lymphocytes | Lysis | Interleukin 10 | CD25 antigen | Perforin | Histocompatibility
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12. Full Text CD3+CD8+NKG2D+ T Lymphocytes Induce Apoptosis and Necroptosis in HLA‐Negative Cells via FasL–Fas Interaction
by Ivanova, Olga K and Sharapova, Tatiana N and Romanova, Elena A and Soshnikova, Natalia V and Sashchenko, Lidia P and Yashin, Denis V
Journal of Cellular Biochemistry, ISSN 0730-2312, 10/2017, Volume 118, Issue 10, pp. 3359 - 3366
ABSTRACT An important problem in cellular immunology is to identify new populations of cytotoxic lymphocytes capable of killing tumor cells that have lost... 
APOPTOSIS | NKG2D | NECROPTOSIS | FasL | CYTOTOXITY | Fas | HUMAN LYMPHOCYTES | NKG2D LIGANDS | COMPLEX | HSP70 | KILLER-CELLS | PROTEIN TAG7 | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | TUMOR-CELLS | CELL BIOLOGY | IMMUNORECEPTOR | EXPRESSION | CYTOTOXICITY | NK Cell Lectin-Like Receptor Subfamily K | CD3 Complex | Humans | Fas Ligand Protein - immunology | fas Receptor - immunology | Animals | Apoptosis - immunology | Neoplasms - immunology | K562 Cells | Mice | HLA Antigens | CD8-Positive T-Lymphocytes - immunology | Immunity, Cellular | Heat shock proteins | Antigens | T cells | Apoptosis | Tumor necrosis factor receptors | Cell culture | Fas antigen | CD8 antigen | Cytotoxicity | Lymphocytes T | Machinery | Proteins | Cell activation | Immunology | Interleukin 2 | Lymphocytes | CD25 antigen | Killing | Antigen presentation | Tumor cells | CD95 antigen | Mortality | Hsp70 protein | CD3 antigen | Switching | CD4 antigen | Major histocompatibility complex | FasL protein | Cell death | Histocompatibility antigen HLA | Cancer
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