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Nature (London), ISSN 1476-4687, 2017, Volume 551, Issue 7678, pp. 115 - 118
Journal Article
American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 04/2008, Volume 294, Issue 4, pp. 1185 - 1196
Two recent, large whole-genome association studies (GWAS) in European populations have associated a ∼47-kb region that contains part of the FTO gene with high... 
Obesity | Adipose tissue | Hypothalamus | CUTL1 | CCAAT DISPLACEMENT PROTEIN | DNA-BINDING | PHYSIOLOGY | FTO GENE | CUT EXPRESSION | adipose tissue | DROSOPHILA | TRANSCRIPTION FACTOR | WING MARGIN | obesity | hypothalamus | FAT MASS | GENOME-WIDE ASSOCIATION | Bardet-Biedl Syndrome - metabolism | Homeodomain Proteins - metabolism | Humans | Alpha-Ketoglutarate-Dependent Dioxygenase FTO | Male | RNA, Messenger - metabolism | Obesity - genetics | Embryo, Mammalian - metabolism | Mixed Function Oxygenases | Adipose Tissue - metabolism | Transfection | Gene Expression Regulation, Developmental | Oxo-Acid-Lyases - genetics | Bardet-Biedl Syndrome - genetics | Fasting - metabolism | Oxo-Acid-Lyases - metabolism | Energy Metabolism - genetics | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Disease Models, Animal | Eating | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Leptin - genetics | Adiposity - genetics | Homeodomain Proteins - genetics | Obesity - metabolism | Proteins - genetics | Animals | Hypothalamus - metabolism | Proteins - metabolism | Adipocytes - metabolism | Adaptor Proteins, Signal Transducing - genetics | Hypothermia, Induced | Mice, Obese | Mice | Polymorphism, Single Nucleotide | Adaptor Proteins, Signal Transducing - metabolism
Journal Article
Nature (London), ISSN 1476-4687, 2010, Volume 464, Issue 7285, pp. 121 - 125
Journal Article
PloS one, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0173676
Autophagy is a catabolic mechanism to degrade cellular components to maintain cellular energy levels during starvation, a condition where PPAR alpha may be... 
INSULIN SIGNALING TRANSDUCTION | GLUCOSE-HOMEOSTASIS | FIBROBLAST-GROWTH-FACTOR-21 | DEACETYLATION | METABOLISM | PATHWAY | STEATOSIS | MULTIDISCIPLINARY SCIENCES | RESISTANCE | RECEPTORS | FOXO1 | TOR Serine-Threonine Kinases - metabolism | Autophagy-Related Protein 7 - metabolism | Fibroblast Growth Factors - genetics | Autophagy-Related Protein 5 - genetics | fas Receptor - metabolism | Autophagy - drug effects | Fibroblast Growth Factors - metabolism | TOR Serine-Threonine Kinases - genetics | Liver - drug effects | fas Receptor - genetics | Autophagy - genetics | Proto-Oncogene Proteins c-akt - metabolism | Forkhead Box Protein O1 - metabolism | Signal Transduction | Liver - metabolism | PPAR alpha - genetics | Ubiquitin-Conjugating Enzymes - genetics | Stearoyl-CoA Desaturase - genetics | Mice, Knockout | Triglycerides - metabolism | Sequestosome-1 Protein - genetics | Ubiquitin-Conjugating Enzymes - metabolism | Cysteine Endopeptidases - genetics | Autophagy-Related Protein 5 - metabolism | Beclin-1 - genetics | Stearoyl-CoA Desaturase - metabolism | Mice | PPAR alpha - metabolism | Autophagy-Related Proteins - antagonists & inhibitors | Blood Glucose - metabolism | Autophagy-Related Proteins - metabolism | Forkhead Box Protein O1 - genetics | Autophagy-Related Protein 5 - antagonists & inhibitors | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Fenofibrate - pharmacology | Cysteine Endopeptidases - metabolism | Autophagy-Related Proteins - genetics | Sequestosome-1 Protein - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Autophagy-Related Protein 7 - antagonists & inhibitors | Mice, Inbred C57BL | Autophagy-Related Protein 7 - genetics | Gene Expression Regulation - drug effects | Animals | Sterol Regulatory Element Binding Protein 1 - genetics | PPAR alpha - agonists | Ubiquitin-Conjugating Enzymes - antagonists & inhibitors | Beclin-1 - metabolism | Transcription factors | Adipose tissue | Liver | Body weight | AKT protein | Biochemistry | Glucose | Assaying | Proteins | Signal transduction | Temperature effects | Fibroblasts | Physiology | Acetylation | Inhibition | Growth factors | Hepatotoxicity | Activation analysis | Methanol | Starvation | Ethanol | AMP | Metabolism | Insulin | Fatty acids | Studies | Acetaminophen | Food intake | Weight reduction | Animal welfare | Circulation | Drugs | Biotechnology | Drug abuse | Laboratories | Centrifugation | Glass | Homeostasis | Activation | Biology | Kinases | AMP-activated protein kinase | Autophagy | Nutrient status | Rodents | Nutrients | Oxidation | Heart diseases | Age | Epinephrine | AKT1 protein | Fasting | Chloroform | Diabetes mellitus | Cardiomyocytes | Acclimatization | Triglycerides | Pharmacology | Nitrogen | Calories | Medicine | Nuclear fuels | Protein kinase | Insulin resistance | Diabetes
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 4, p. e62068
The NF-kappa B pathway plays an important role in chronic inflammatory and autoimmune diseases. Recently, NF-kappa B has also been suggested as an important... 
METABOLIC SYNDROME | OXIDATIVE STRESS | ACTIVATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | MESANGIAL CELLS | INDUCED INFLAMMATION | DIABETIC-NEPHROPATHY | BINDING-ACTIVITY | TRANSCRIPTION FACTOR | ADIPOSE-TISSUE | Albuminuria - complications | Diabetes Mellitus, Experimental - drug therapy | Liver - pathology | Triterpenes - pharmacology | Kidney - pathology | Glycated Hemoglobin A - metabolism | Triterpenes - therapeutic use | Kidney Glomerulus - physiopathology | Male | NF-kappa B - metabolism | Kidney Function Tests | Diabetes Mellitus, Experimental - blood | Adipose Tissue - metabolism | Albuminuria - physiopathology | Kidney - metabolism | Liver - drug effects | Lipids - blood | Inflammation Mediators - metabolism | Lipid Peroxidation - drug effects | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Diabetes Mellitus, Experimental - metabolism | Fatty Acids - metabolism | Kidney - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Albuminuria - pathology | NF-kappa B - antagonists & inhibitors | Kidney - drug effects | Podocytes - metabolism | Cytokines - metabolism | Kidney Glomerulus - drug effects | Liver - metabolism | Adipose Tissue - pathology | Cells, Cultured | Insulin Resistance | Kidney Glomerulus - pathology | Podocytes - pathology | Animals | Podocytes - drug effects | Mice | Oxidative Stress - drug effects | Adipose Tissue - drug effects | Transcription factors | Nephrology | Adipose tissue | Disease | Syngeneic grafts | Oxidative metabolism | Liver | Body weight | Neuropeptides | Adipocytes | Glucose | Animal tissues | Lipid metabolism | Obesity | Medical research | NF-κB protein | Cytokines | Internal medicine | Mortality | Abnormalities | Polyamide-imides | Metabolism | Insulin | Fatty acids | Inhibitors | Nephropathy | Weight reduction | Metabolic disorders | Structure-function relationships | Oxidative stress | Animal models | Homeostasis | Adiponectin | Oxidation resistance | Rodents | Collagen (type IV) | Creatinine | Urine | Hypertension | Excretion | Fasting | Kidneys | Diabetes mellitus | Organs | Inflammation | Medicine | Insulin resistance | Diabetes | Autoimmune diseases
Journal Article
Journal Article
Cell metabolism, ISSN 1550-4131, 2008, Volume 8, Issue 6, pp. 468 - 481
Obesity and nutrient homeostasis are linked by mechanisms that are not fully elucidated. Here we describe a secreted protein, adropin, encoded by a gene,... 
HUMDISEASE | PATHOGENESIS | OBESITY | INSULIN-RESISTANCE | FOOD-INTAKE | GLUCOSE | LIVER | ENDOCRINOLOGY & METABOLISM | RECEPTOR | MICE | ADIPOSE-TISSUE | MELANOCORTIN SYSTEM | CELL BIOLOGY | RNA, Small Interfering - genetics | Peptides | Benzoates - chemistry | Benzoates - metabolism | Humans | Leptin - metabolism | Adipose Tissue, White - metabolism | Molecular Sequence Data | Male | RNA, Messenger - metabolism | Obesity - genetics | Benzylamines - metabolism | DNA-Binding Proteins - metabolism | DNA-Binding Proteins - agonists | Proteins - secretion | RNA Interference | Blood Proteins - genetics | Base Sequence | Liver X Receptors | Female | Orphan Nuclear Receptors | Amino Acid Sequence | Proteins - physiology | Fasting | Fatty Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Blood Proteins - secretion | Lipid Metabolism | Mice, Transgenic | Receptors, Cytoplasmic and Nuclear - agonists | Obesity - metabolism | Proteins - genetics | Benzylamines - chemistry | Animals | Energy Metabolism | Blood Proteins - physiology | Adipose Tissue, Brown - metabolism | Mice | RNA, Small Interfering - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Obesity | Physiological aspects | Homeostasis | Biological apparatus and supplies | Insulin resistance | Neuropeptides | Glucose | Diabetes | Universities and colleges | Dextrose
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 2, p. e57915
...) WT and Gal-3 KO mice to investigate the role of Gal-3 in modulation of adiposity, glucose metabolism and inflammation... 
INDUCED GLOMERULAR INJURY | RECEPTOR-MEDIATED MECHANISMS | ACTIVATION | GALECTIN-3/AGE-RECEPTOR-3 KNOCKOUT MICE | LIPOXIDATION | MULTIDISCIPLINARY SCIENCES | FATTY LIVER | AGE-RECEPTOR | DIET-INDUCED OBESITY | INSULIN SENSITIVITY | EXPRESSION | Galectin 3 - metabolism | Glucose Tolerance Test - methods | Fibroblast Growth Factors - genetics | Monocytes - metabolism | PPAR gamma - metabolism | Obesity - genetics | Galectin 3 - deficiency | Fibroblast Growth Factors - metabolism | Inflammation - metabolism | Adiponectin - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Adiponectin - metabolism | Insulin - genetics | PPAR gamma - genetics | Liver - metabolism | Glucose - genetics | Lipase - metabolism | Transcription Factors - genetics | Galectin 3 - genetics | Adiposity - genetics | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Diet | Insulin Resistance - genetics | Glucose - metabolism | Inflammation - genetics | Mice, Obese | Adiposity - physiology | Mice | Lipase - genetics | Type 2 diabetes | Obesity | Glucose metabolism | Blood sugar monitoring | Liver | Physiological aspects | Inflammation | Glucose | Glucose tolerance tests | Insulin | Statistics | Dextrose | Fibroblast growth factor | Oxidative stress | Adipose tissue | Microflora | Adipocytes | Galectin-3 | Adiponectin | Proteins | Microbiota | Hyperglycemia | Rodents | Thrombocytosis | Age | Adducts | Heart failure | Medical research | Fasting | Cytokines | Anemia | Diabetes mellitus | Metabolism | Glucose tolerance | Nutrition research | Pathology | Monocytes | Acute phase substances | Antibiotics | Neutrophilia | Diabetes | Kinesiology
Journal Article