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Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 2, pp. 307 - 320
c-Jun-N-terminal kinase (JNK) is a mitogen-activated protein kinase family member that is activated by diverse stimuli, including cytokines (such as tumor... 
Gastroenterology and Hepatology | c-Jun | MAPK | Acetaminophen | TNF | Insulin Resistance | Hepatocellular Carcinoma | N-TERMINAL KINASE | JUN NH2-TERMINAL KINASE | ENDOPLASMIC-RETICULUM STRESS | TNF-INDUCED APOPTOSIS | KAPPA-B | MITOCHONDRIAL PERMEABILITY TRANSITION | HEPATOCYTE SURVIVAL | INSULIN-RESISTANCE | COMPENSATORY PROLIFERATION | GASTROENTEROLOGY & HEPATOLOGY | Acetaminophen - adverse effects | Liver Transplantation | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Metabolic Syndrome - metabolism | Liver - physiopathology | Metabolic Syndrome - drug therapy | MAP Kinase Signaling System | Carcinoma, Hepatocellular - drug therapy | Analgesics, Non-Narcotic - adverse effects | Cell Death | Biomarkers - metabolism | Liver Regeneration | Fatty Liver - metabolism | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Liver - metabolism | Liver Neoplasms - drug therapy | Fatty Liver - drug therapy | Animals | Chemical and Drug Induced Liver Injury - metabolism | Protein Kinase Inhibitors - therapeutic use | Chemical and Drug Induced Liver Injury - drug therapy | JNK Mitogen-Activated Protein Kinases - physiology | Liver Neoplasms - metabolism | Liver - physiology | Liver Diseases - drug therapy | Mice | Primary Graft Dysfunction - metabolism | Liver Diseases - physiopathology | Liver Diseases - metabolism | Carcinoma, Hepatocellular - metabolism | Enzymes | Medical colleges | Platelet-derived growth factor | Liver diseases | Isoenzymes | Genes | Liver | Development and progression | Transforming growth factors | Fatty acids | Liver cancer | Epidermal growth factor | Interleukins | DNA | Genetically modified organisms | Physiological aspects | Insulin resistance | Tumor proteins | Mitogens | Protein kinases | Tumors | Index Medicus | Abridged Index Medicus | acetaminophen | insulin resistance | hepatocellular carcinoma
Journal Article
Gut, ISSN 0017-5749, 03/2012, Volume 61, Issue 3, pp. 416 - 426
ObjectiveMonocyte chemoattractant protein-1 (MCP-1, CCL2), the primary ligand for chemokine receptor C–C chemokine receptor 2 (CCR2), is increased in livers of... 
FIBROSIS | STEATOSIS | INSULIN-RESISTANCE | INFLAMMATION | BONE-MARROW | DISEASE | MONOCYTE SUBSETS | MICE | SPIEGELMER | GASTROENTEROLOGY & HEPATOLOGY | CCR2 | Liver - pathology | Carbon Tetrachloride | Liver Cirrhosis, Experimental - etiology | Bone Marrow Cells - physiology | Male | Chemical and Drug Induced Liver Injury, Chronic - drug therapy | Liver Cirrhosis, Experimental - prevention & control | Non-alcoholic Fatty Liver Disease | Chemokine CCL2 - physiology | Bone Marrow Cells - drug effects | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury, Chronic - complications | Macrophages - physiology | Aptamers, Nucleotide - pharmacology | Drug Evaluation, Preclinical - methods | Acute Disease | Cytokines - metabolism | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Fatty Liver - prevention & control | Aptamers, Nucleotide - therapeutic use | Chemotaxis - drug effects | Chemical and Drug Induced Liver Injury, Chronic - pathology | Disease Progression | Fatty Liver - drug therapy | Animals | Chemical and Drug Induced Liver Injury - complications | Chemical and Drug Induced Liver Injury - drug therapy | Macrophages - drug effects | Mice | Chemokine CCL2 - antagonists & inhibitors | Fatty Liver - etiology | Care and treatment | Liver diseases | Fibrosis | Models | Research | Macrophages | Chemokine receptors | Studies | Cytokines | Polyethylene glycol | Rodents | Liver | Clinical trials | Tumor necrosis factor-TNF | Inflammation | Diabetes | Metabolic disorders | Chemokines | Index Medicus | Abridged Index Medicus
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 11/2006, Volume 355, Issue 22, pp. 2297 - 2307
Journal Article
Lancet, The, ISSN 0140-6736, 2010, Volume 376, Issue 9756, pp. 1916 - 1922
Journal Article
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