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Molecular Neurobiology, ISSN 0893-7648, 03/2019, Volume 56, Issue 3, pp. 1770 - 1781
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 07/2018, Volume 115, Issue 29, p. 7605
Endocannabinoid signaling regulates feeding and metabolic processes and has been linked to obesity development. Several hormonal signals, such as... 
Glucocorticoids | Body weight | Hormones | Hypothalamus | High fat diet | Human populations | Energy | Ghrelin | Rodents | Eating behavior | Fatty-acid amide hydrolase | Hydrolase | Pharmacology | Metabolism | Fatty acids | Body weight gain | Feeding | Anandamide | Energy balance | Genetic variance | Signaling | Sensitivity | Food intake | Leptin | Signal processing | Mice | Polymorphism
Journal Article
Journal Article
Journal Article
The AAPS Journal, ISSN 1550-7416, 3/2009, Volume 11, Issue 1, pp. 39 - 44
The endogenous cannabinoid N-arachidonoyl ethanolamine (anandamide; AEA) produces most of its pharmacological effects by binding and activating CB1 and CB2... 
Biochemistry, general | Biotechnology | neuropathic pain | Biomedicine | CB 2 cannabinoid receptor | fatty acid amide hydrolase (FAAH) | Pharmacy | CB 1 cannabinoid receptor | inflammatory pain | Pharmacology/Toxicology | anandamide | endogenous cannabinoid | Fatty acid amide hydrolase (FAAH) | cannabinoid receptor | Endogenous cannabinoid | Inflammatory pain | Anandamide | Neuropathic pain | CB1 cannabinoid receptor | MULTIPLE MECHANISMS | ENDOCANNABINOID SYSTEM | CB2 cannabinoid receptor | TRANSPORT INHIBITOR AM404 | CB2 RECEPTORS | ACTIVATED-RECEPTOR-ALPHA | CANNABINOID RECEPTOR | PHARMACOLOGY & PHARMACY | PRIMARY SENSORY NEURONS | BRAIN | Inflammation - chemically induced | Analgesics - pharmacology | Peroxisome Proliferator-Activated Receptors - physiology | Humans | TRPV Cation Channels - drug effects | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Drug Delivery Systems | Receptors, Cannabinoid - drug effects | Receptors, Cannabinoid - metabolism | Receptors, Opioid - physiology | Arachidonic Acids - metabolism | Inflammation - drug therapy | Pain - drug therapy | Amidohydrolases - deficiency | Dronabinol - pharmacology | Drug Evaluation, Preclinical | Endocannabinoids | Disease Models, Animal | TRPV Cation Channels - physiology | Amidohydrolases - genetics | Rats | Amidohydrolases - antagonists & inhibitors | Analgesics - toxicity | Glycerides - metabolism | Polyunsaturated Alkamides - metabolism | Anti-Inflammatory Agents, Non-Steroidal - toxicity | Mice, Knockout | Animals | Pain - physiopathology | Amidohydrolases - physiology | Peroxisome Proliferator-Activated Receptors - drug effects | Mice | Dronabinol - toxicity | Receptors, Opioid - drug effects | Inflammation - physiopathology | Enzymes | Cannabinoids | Care and treatment | Pain | Analysis | Marijuana | Hydrolases | Inflammation | Universities and colleges | Ethanolamines | Fatty acids | Index Medicus
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 04/2012, Volume 165, Issue 8, pp. 2485 - 2496
BACKGROUND AND PURPOSE Inflammatory pain presents a problem of clinical relevance and often elicits allodynia, a condition in which non‐noxious stimuli are... 
Δ9‐tetrahydrocannabinol | CB2 | allodynia | fatty acid amide hydrolase | CB1 | inflammatory pain | endocannabinoids | anandamide | Allodynia | Fatty acid amide hydrolase | tetrahydrocannabinol | Inflammatory pain | Endocannabinoids | Anandamide | INVOLVEMENT | ALPHA | MODEL | NEUROPATHIC PAIN | CANNABINOID RECEPTOR | 9-tetrahydrocannabinol | INFLAMMATION | PHARMACOLOGY | PHARMACOLOGY & PHARMACY | MODULATION | ANALGESIA | Hyperalgesia - chemically induced | Inflammation - chemically induced | Spinal Cord - drug effects | Spinal Cord - metabolism | Male | Lipopolysaccharides | Brain - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Piperidines - pharmacology | Inflammation - drug therapy | Female | Amidohydrolases - deficiency | Pyridines - therapeutic use | Hyperalgesia - metabolism | Amidohydrolases - genetics | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Amidohydrolases - antagonists & inhibitors | Glycerides - metabolism | Polyunsaturated Alkamides - metabolism | Enzyme Inhibitors - therapeutic use | Mice, Knockout | Brain - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Hyperalgesia - drug therapy | Piperidines - therapeutic use | Peripheral Nervous System - metabolism | Mice | Pyridines - pharmacology | Peripheral Nervous System - drug effects | Index Medicus | Research Papers | Δ9-tetrahydrocannabinol | Themed Section
Journal Article
Frontiers in Pharmacology, ISSN 1663-9812, 10/2016, Volume 7, p. 370
This perspective was adapted from a Career Achievement Award talk given at the International Cannabinoid Research Society Symposium in Bukovina, Poland on June... 
Fatty acid binding protein (FABP) | Fatty acid amide hydrolase (FAAH) | Anandamide synthesis | FABP inhibitors | AEA | Anandamide transporter | FAAH inhibitors | Anandamide | Measurement | Chemical properties | Health aspects | Fatty acids | Fatty acid amide hydrolase | FABPs | FAAH | Fatty acid binding proteins
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 07/2018, Volume 115, Issue 29, pp. 7605 - 7610
Journal Article
Journal Article
Psychopharmacology, ISSN 0033-3158, 11/2017, Volume 234, Issue 21, pp. 3229 - 3240
Unlike other drugs of abuse, Δ9-tetrahydrocanabinol (THC) is generally aversive in rodent conditioned place preference models, but little is known about how... 
Neurosciences | Place conditioning | Biomedicine | Fatty acid amide hydrolase (FAAH) | Pharmacology/Toxicology | Psychiatry | Δ 9 -tetrahydrocanabinol (THC) | Stress | Fear conditioning |