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Journal Article
Journal Article
CELL, ISSN 0092-8674, 02/2003, Volume 112, Issue 3, pp. 391 - 401
In eukaryotes, DNA damage elicits a multifaceted response that includes cell cycle arrest, transcriptional activation of DNA repair genes, and, in... 
ALLOSTERIC REGULATION | IN-VITRO | CHECKPOINT PATHWAYS | POLYMERASE-ETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUTATOR PHENOTYPES | ESCHERICHIA-COLI | SMALL-SUBUNIT | CELL-CYCLE | CALF THYMUS | SACCHAROMYCES-CEREVISIAE | CELL BIOLOGY | Deoxyribonucleosides - genetics | Cell Cycle - genetics | Saccharomyces cerevisiae - genetics | Cell Survival - genetics | DNA Repair - radiation effects | Adenosine Triphosphate - genetics | Deoxyribonucleosides - metabolism | DNA Repair - genetics | Gene Expression Regulation, Fungal - drug effects | Feedback, Physiological - radiation effects | Adenosine Triphosphate - metabolism | DNA Damage - genetics | DNA - biosynthesis | Gene Expression Regulation, Fungal - genetics | DNA Damage - drug effects | Eukaryotic Cells - enzymology | Cell Survival - drug effects | DNA Repair - drug effects | Mutation - radiation effects | Cell Cycle - radiation effects | Mutagens - pharmacology | Binding Sites - genetics | Saccharomyces cerevisiae Proteins - genetics | Mutation - genetics | Cell Survival - radiation effects | DNA - genetics | Gene Expression Regulation, Fungal - radiation effects | Mutation - drug effects | Feedback, Physiological - genetics | Ribonucleotide Reductases - metabolism | Saccharomyces cerevisiae Proteins - metabolism | Saccharomyces cerevisiae - enzymology | Feedback, Physiological - drug effects | Ribonucleotide Reductases - genetics | Cell Cycle - drug effects | DNA Damage - radiation effects | Physiological aspects | Metabolic regulation | Genetic aspects | Mechanical chemistry | Research | DNA damage
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 06/2017, Volume 312, Issue 6, pp. C749 - C764
Reactive oxygen species (ROS) derived from NADPH oxidase (NOX) and mitochondria play a critical role in growth factor-induced switch from a quiescent to an... 
Angiogenesis | Mitochondria | Reactive oxygen species | Vascular endothelial growth factor | NADPH oxidase | ACTIVATION | PHYSIOLOGY | PROTEIN | mitochondria | angiogenesis | OXYGEN SPECIES GENERATION | ENDOTHELIAL-CELL MIGRATION | CELL BIOLOGY | REACTIVE OXYGEN | vascular endothelial growth factor | reactive oxygen species | GROWTH | COMPARTMENTALIZATION | HYDROGEN-PEROXIDE | NEOVASCULARIZATION | Human Umbilical Vein Endothelial Cells | Neovascularization, Physiologic - drug effects | RNA, Small Interfering - genetics | Membrane Glycoproteins - metabolism | Humans | NADPH Oxidases - metabolism | Time-Lapse Imaging | Vascular Endothelial Growth Factor Receptor-2 - genetics | Membrane Glycoproteins - antagonists & inhibitors | Src Homology 2 Domain-Containing, Transforming Protein 1 - genetics | NADPH Oxidases - genetics | Phosphorylation - drug effects | Vascular Endothelial Growth Factor A - pharmacology | Oxidation-Reduction | Signal Transduction | Catalase - genetics | Neovascularization, Physiologic - genetics | NADPH Oxidases - antagonists & inhibitors | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | NADPH Oxidase 4 | NADPH Oxidase 2 | Hydrogen Peroxide - metabolism | Membrane Glycoproteins - genetics | Catalase - metabolism | Cell Movement - drug effects | Feedback, Physiological | Biosensing Techniques | Cell Proliferation - drug effects | Microscopy, Fluorescence | Src Homology 2 Domain-Containing, Transforming Protein 1 - metabolism | RNA, Small Interfering - metabolism | Oxidases
Journal Article
Journal Article