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21. Full Text Influence of prenatal hypoxia and postnatal hyperoxia on morphologic lung maturation in mice
by Schmiedl, Andreas and Roolfs, Torge and Tutdibi, Erol and Gortner, Ludwig and Monz, Dominik
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0175804
Background Oxygen supply as a lifesaving intervention is frequently used to treat preterm infants suffering additionally from possible prenatal or perinatal... 
BRONCHOPULMONARY DYSPLASIA | PULMONARY SURFACTANT | SURFACTANT PROTEIN-B | STEREOLOGICAL ESTIMATION | NEONATAL HYPEROXIA | PERINATAL INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | FETAL LUNG | ANIMAL-MODELS | ENDOTHELIAL GROWTH-FACTOR | Hyperoxia - pathology | Pregnancy | Lung - pathology | Animals | Hypoxia - pathology | Mice, Inbred C57BL | Female | Lung - embryology | Mice | Dysplasia | Care and treatment | Pregnant women | Infants (Premature) | Epithelial cells | Research | Health aspects | Pediatrics | Animal models | Parenchyma | Infants | Medical schools | Angiogenesis | Alterations | Rodents | Mathematical models | Alveoli | Oxygen | Maturation | Lung diseases | Septum | Inflammation | Surfactants | Wall thickness | Progeny | Offspring | Lungs | Hyperoxia | Morphology | Comparative studies | Hypoxia | Influence | Birth weight
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22. Full Text Maternal hypoxia alters matrix metalloproteinase expression patterns and causes cardiac remodeling in fetal and neonatal rats
by Tong, Wenni and Xue, Qin and Li, Yong and Zhang, Lubo
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2011, Volume 301, Issue 5, pp. H2113 - H2121
Tong W, Xue Q, Li Y, Zhang L. Maternal hypoxia alters matrix metalloproteinase expression patterns and causes cardiac remodeling in fetal and neonatal rats. Am... 
Tissue inhibitor of metalloproteinases | Cardiomy-ocyte proliferation | Collagen deposition | Hypertrophy | cardiomyocyte proliferation | EPSILON GENE REPRESSION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | COLLAGEN | hypertrophy | REPERFUSION INJURY | tissue inhibitor of metalloproteinases | PRENATAL HYPOXIA | collagen deposition | TISSUE INHIBITOR | PERIPHERAL VASCULAR DISEASE | EXTRACELLULAR-MATRIX | GROWTH RESTRICTION | ADULT MALE | HEART SUSCEPTIBILITY | Prenatal Exposure Delayed Effects | Cell Proliferation | Cardiomegaly - etiology | Cardiomegaly - pathology | Male | Maternal Exposure | Myocytes, Cardiac - enzymology | Ventricular Remodeling | Female | Disease Models, Animal | Animals, Newborn | Fibrillar Collagens - metabolism | Hypoxia - enzymology | Fetal Weight | Rats | Hypoxia - complications | Cardiomegaly - enzymology | Fetal Heart - enzymology | Gestational Age | Rats, Sprague-Dawley | Blotting, Western | Tissue Inhibitor of Metalloproteinases - metabolism | Fetal Heart - pathology | Pregnancy | Myocytes, Cardiac - pathology | Animals | Hypoxia - pathology | Matrix Metalloproteinases - metabolism | Integrative Cardiovascular Physiology and Pathophysiology
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23. Full Text Spatial Heterogeneity in Oligodendrocyte Lineage Maturation and Not Cerebral Blood Flow Predicts Fetal Ovine Periventricular White Matter Injury
by Riddle, Art and Ling Luo, Ning and Manese, Mario and Beardsley, Douglas J and Green, Lisa and Rorvik, Dawn A and Kelly, Katherine A and Barlow, Clyde H and Kelly, Jeffrey J and Hohimer, A. Roger and Back, Stephen A
Journal of Neuroscience, ISSN 0270-6474, 03/2006, Volume 26, Issue 11, pp. 3045 - 3055
Although periventricular white matter injury (PWMI) is the leading cause of chronic neurological disability and cerebral palsy in survivors of premature birth,... 
Blood-brain | Ischemia | Oligodendrocyte | Cerebral blood flow | Injury | Neonatal prenatal | Development | Periventricular | White matter | Hypoxia-ischemia | Blood flow | PROGENITORS | hypoxia-ischemia | development | DEVELOPMENTAL NEUROPATHOLOGY | oligodendrocyte | blood-brain | SUBVENTRICULAR ZONE | NEUROSCIENCES | ischemia | white matter | periventricular | HEMORRHAGIC HYPOTENSION | SHEEP BRAIN | LEUKOMALACIA | SELECTIVE VULNERABILITY | cerebral blood flow | neonatal prenatal | injury | PERINATAL BRAIN-DAMAGE | PRETERM | blood flow | NEAR-TERM | Humans | Leukomalacia, Periventricular - pathology | Hypoxia-Ischemia, Brain - pathology | Microspheres | Leukomalacia, Periventricular - physiopathology | Female | Models, Animal | Imaging, Three-Dimensional | Infant, Newborn | Disease Susceptibility | Hypoxia-Ischemia, Brain - embryology | Reperfusion Injury - pathology | Leukomalacia, Periventricular - etiology | Cerebrovascular Circulation | Fetal Hypoxia - physiopathology | Gestational Age | Oligodendroglia - pathology | Pregnancy | Cell Lineage | Magnetic Resonance Imaging | Animals | Reperfusion Injury - embryology | Sheep | Apoptosis | Blood-Brain Barrier | Fetal Hypoxia - pathology | blood–brain
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24. Full Text Hypoxia during pregnancy in rats leads to early morphological changes of atherosclerosis in adult offspring
by Zhenhua Wang and Ziyang Huang and Guorong Lu and Ling Lin and Markus Ferrari
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 05/2009, Volume 296, Issue 5, pp. 1321 - 1328
Exposure to an adverse intrauterine environment increases the risk of cardiovascular disease later in adult life. However, the time course relationship between... 
Intimal thickness | Hyperlipidemia | Postnatal hypoxemia | Fetal programming | Sex | intimal thickness | NUTRIENT RESTRICTION | hyperlipidemia | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | GROWTH-RETARDATION | MATERNAL HYPOXIA | sex | fetal programming | CHICKEN-EMBRYO | BLOOD-PRESSURE | postnatal hypoxemia | PRENATAL HYPOXIA | VASCULAR FUNCTION | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | MYOGENIC TONE | CORONARY-HEART-DISEASE | Prenatal Exposure Delayed Effects | Hyperlipidemias - blood | Age Factors | Fetal Hypoxia - blood | Male | Atherosclerosis - etiology | Fetal Growth Retardation - etiology | Lipids - blood | Aging | Female | Fetal Hypoxia - complications | Aorta, Thoracic - pathology | Hyperlipidemias - complications | Disease Models, Animal | Atherosclerosis - pathology | Maternal-Fetal Exchange | Rats | Hypoxia - embryology | Hypoxia - complications | Carbon Dioxide - blood | Gestational Age | Rats, Sprague-Dawley | Oxygen - blood | Atherosclerosis - blood | Pregnancy | Hypoxia - blood | Animals | Tunica Intima - pathology | Hypoxia - pathology | Sex Factors | Hyperlipidemias - pathology | Fetal Hypoxia - pathology | Hydrogen-Ion Concentration
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25. Full Text Connexin hemichannel blockade improves outcomes in a model of fetal ischemia
by Davidson, Joanne O and Green, Colin R and B. Nicholson, Louise F and O'Carroll, Simon J and Fraser, Mhoyra and Bennet, Laura and Jan Gunn, Alistair
Annals of Neurology, ISSN 0364-5134, 01/2012, Volume 71, Issue 1, pp. 121 - 132
Objective: Connexin hemichannels can open during ischemia, resulting in loss of membrane potential, calcium influx, and release of glutamate. In this study, we... 
HUMAN BRAIN | GAP-JUNCTIONAL HEMICHANNELS | MATTER INJURY | MIMETIC PEPTIDES | SPINAL-CORD-INJURY | CEREBRAL HYPOTHERMIA | VENTRICULAR MYOCYTES | CORTICAL ASTROCYTES | NEUROSCIENCES | CLINICAL NEUROLOGY | CELL-DEATH | PERINATAL ASPHYXIA | Neurons - pathology | Seizures - prevention & control | Brain Ischemia - metabolism | Male | Electroencephalography | Fetal Hypoxia - metabolism | Connexin 43 - physiology | Neurons - physiology | Sleep Stages - physiology | Female | Cell Survival - physiology | Disease Models, Animal | Gene Targeting | Peptide Fragments - administration & dosage | Treatment Outcome | Brain Ischemia - physiopathology | Brain Ischemia - prevention & control | Peptide Fragments - chemical synthesis | Pregnancy | Animals | Fetal Hypoxia - drug therapy | Sheep | Connexin 43 - antagonists & inhibitors | Peptide Fragments - therapeutic use | Fetal Hypoxia - pathology
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26. Full Text Association of Dll4/Notch and HIF-1a -VEGF Signaling in the Angiogenesis of Missed Abortion
by Fang, Yan and Yu, Shuang and Ma, Yuyan and Sun, Ping and Ma, Daoxin and Ji, Chunyan and Kong, Beihua
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e70667
Background: Dll4/Notch and HIF-1a-VEGF have been shown to play an important role during angiogenesis, but there are no data about their roles and association... 
DLL4 | RECURRENT MISCARRIAGE | TYROSINE KINASE | VEGF | MULTIDISCIPLINARY SCIENCES | EMBRYONIC LETHALITY | RECEPTOR | GROWTH-FACTOR | ORF VIRUS | HYPOXIA | EXPRESSION | Humans | Neovascularization, Pathologic | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Abortion, Missed - pathology | Case-Control Studies | Abortion, Induced | Hypoxia - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Adult | Female | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Chorionic Villi - metabolism | Fetal Death - pathology | Abortion, Missed - genetics | Pregnancy | Hypoxia - genetics | Abortion, Missed - metabolism | Chorionic Villi - pathology | Hypoxia - pathology | Adolescent | Chorionic Villi - blood supply | Fetal Death - metabolism | Immunohistochemistry | RNA | Miscarriage | Vascular endothelial growth factor | Abortion | Analysis | Hematology | Genes | Gynecology | Reverse transcription | Kinases | Obstetrics | Polymerase chain reaction | Angiogenesis | Signaling | Hypoxia-inducible factor 1a | Cell growth | Ultrasonic imaging | Hypoxia | Ligands | Aberration | Age
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27. Full Text Hippocampal NMDAR-Wnt-Catenin signaling disrupted with cognitive deficits in adolescent offspring exposed to prenatal hypoxia
by Wei, Bin and Li, Lingjun and He, Axin and Zhang, Yingying and Sun, Miao and Xu, Zhice
Brain Research, ISSN 0006-8993, 2015, Volume 1631, pp. 157 - 164
Abstract Prenatal hypoxia (PH) is one of the most common stresses on fetuses, and might lead to abnormal brain development. This work investigates whether PH... 
Neurology | Pregnancy | Hypoxia | NMDARs | Wnt | Catenin | Hippocampus | CELLS | RATS | BIRTH-WEIGHT | BETA-CATENIN | SYNAPTIC PLASTICITY | NEUROSCIENCES | CHILDHOOD NEURODEVELOPMENT | SPATIAL MEMORY | INTRAUTERINE GROWTH RESTRICTION | LONG-TERM POTENTIATION | FETAL | Prenatal Exposure Delayed Effects | Maze Learning - physiology | Signal Transduction | Learning Disorders | Down-Regulation | Receptors, N-Methyl-D-Aspartate - metabolism | Memory Disorders - metabolism | Rats | Cognition Disorders - metabolism | Male | Hippocampus - pathology | Random Allocation | Rats, Sprague-Dawley | beta Catenin - metabolism | Hypoxia - metabolism | Hippocampus - metabolism | Animals | Hypoxia - pathology | Female | Wnt Signaling Pathway | Methyl aspartate | Pregnant women | Aspartate
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28. Full Text A comparative analysis of human mesenchymal stem cell response to hypoxia in vitro : Implications to translational strategies
by Dionigi, Beatrice and Ahmed, Azra and Pennington, Elliot C and Zurakowski, David and Fauza, Dario O
Journal of Pediatric Surgery, ISSN 0022-3468, 2014, Volume 49, Issue 6, pp. 915 - 918
Abstract Purpose Mesenchymal stem cells (MSCs) are particularly valuable for structural tissue replacement. We compared the response to hypoxia among human... 
Pediatrics | Surgery | Bone marrow mesenchymal stem cells | Adipose tissue mesenchymal stem cells | Tissue engineering | Regenerative medicine | Cord blood mesenchymal stem cells | Hypoxia | Amniotic mesenchymal stem cells | Mesenchymal stem cells | OXYGEN TENSION | SURGERY | PEDIATRICS | DIFFERENTIATION | BLOOD | Tissue Engineering - methods | Humans | Adipose Tissue - pathology | Amniotic Fluid - cytology | Bone Marrow Cells - pathology | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Hypoxia - embryology | Fetal Blood - cytology | Fetal Blood - metabolism | Gestational Age | Hypoxia - metabolism | Pregnancy | Adipose Tissue - metabolism | Hypoxia - pathology | Female | Cell Differentiation | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Mesenchymal Stromal Cells - pathology | Bone Marrow Cells - metabolism | Comparative analysis | Stem cells
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29. Full Text The importance of hypoxia and extra physiologic oxygen shock/stress for collection and processing of stem and progenitor cells to understand true physiology/pathology of these cells ex vivo
by Broxmeyer, Hal E and O’Leary, Heather A and Huang, Xinxin and Mantel, Charlie
Current Opinion in Hematology, ISSN 1065-6251, 07/2015, Volume 22, Issue 4, pp. 273 - 278
PURPOSE OF REVIEWHematopoietic stem (HSCs) and progenitor (HPCs) cells reside in a hypoxic (lowered oxygen tension) environment, in vivo. We review literature... 
Extra physiologic oxygen shock/stress and its mitigation | Hematopoietic stem and progenitor cells | Microenvironment | Oxygen tension | extra physiologic oxygen shock/stress and its mitigation | BONE-MARROW | microenvironment | MITOCHONDRIAL PERMEABILITY TRANSITION | CORD BLOOD TRANSPLANTATION | CYCLOPHILIN-D | IN-VITRO | CYCLOSPORINE-A | oxygen tension | UMBILICAL-CORD | REGULATORY NETWORKS | hematopoietic stem and progenitor cells | INNER-MEMBRANE | HEMATOLOGY | HEMATOPOIETIC STEM | Reactive Oxygen Species - metabolism | Oxidative Stress | Humans | MicroRNAs - metabolism | Cyclophilins - metabolism | Tumor Suppressor Protein p53 - genetics | Hypoxia - metabolism | Mitochondrial Membrane Transport Proteins - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cell Differentiation | Mitochondrial Membrane Transport Proteins - metabolism | Bone Marrow Cells - cytology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Gene Expression Regulation | Tumor Suppressor Protein p53 - metabolism | Cyclophilins - genetics | Hematopoietic Stem Cells - metabolism | Mitochondria - metabolism | Fetal Blood - cytology | Fetal Blood - metabolism | Hypoxia - genetics | Hypoxia - pathology | Hematopoietic Stem Cells - cytology | MicroRNAs - genetics | Blood Specimen Collection - methods | Bone Marrow Cells - metabolism
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30. Full Text Antenatal hypoxia induces epigenetic repression of glucocorticoid receptor and promotes ischemic-sensitive phenotype in the developing heart
by Xiong, Fuxia and Lin, Thant and Song, Minwoo and Ma, Qingyi and Martinez, Shannalee R and Lv, Juanxiu and MataGreenwood, Eugenia and Xiao, Daliao and Xu, Zhice and Zhang, Lubo
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2016, Volume 91, pp. 160 - 171
Abstract Large studies in humans and animals have demonstrated a clear association of an adverse intrauterine environment with an increased risk of... 
Cardiovascular | DNA methylation | Hypoxia | Fetal programming | Glucocorticoid receptor | CARDIOMYOCYTE HYPERTROPHY | CARDIAC & CARDIOVASCULAR SYSTEMS | NEONATAL-RATS | CELL BIOLOGY | 11-BETA-HYDROXYSTEROID DEHYDROGENASE | PKC-EPSILON GENE | PRENATAL HYPOXIA | ACUTE MYOCARDIAL-INFARCTION | FETAL-RAT HEART | TRANSCRIPTION FACTOR | ENDOTHELIAL GROWTH-FACTOR | Oxygen - pharmacology | Hypoxia - drug therapy | Receptors, Glucocorticoid - antagonists & inhibitors | Exons | Epigenesis, Genetic | Male | Sp1 Transcription Factor - metabolism | Receptors, Glucocorticoid - metabolism | Maternal Exposure | Hypoxia - metabolism | Myocardial Reperfusion Injury - pathology | Female | Binding Sites | Myocardial Reperfusion Injury - genetics | Animals, Newborn | Promoter Regions, Genetic | Response Elements | Rats | Azacitidine - analogs & derivatives | Rats, Sprague-Dawley | Pregnancy | Azacitidine - pharmacology | Myocardial Reperfusion Injury - metabolism | Hypoxia - genetics | Phenotype | Animals | Sp1 Transcription Factor - genetics | Hypoxia - pathology | Receptors, Glucocorticoid - genetics | Protein Binding | DNA Methylation - drug effects | Myocardial Reperfusion Injury - prevention & control | Epigenetic inheritance | Genetic aspects | Corticosteroids | Methylation | Steroids | hypoxia | glucocorticoid receptor
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31. Full Text Systemic G-CSF attenuates cerebral inflammation and hypomyelination but does not reduce seizure burden in preterm sheep exposed to global hypoxia–ischemia
by Jellema, Reint K and Lima Passos, Valéria and Ophelders, Daan R.M.G and Wolfs, Tim G.A.M and Zwanenburg, Alex and De Munter, Stephanie and Nikiforou, Maria and Collins, Jennifer J.P and Kuypers, Elke and Bos, Gerard M.J and Steinbusch, Harry W and Vanderlocht, Joris and Andriessen, Peter and Germeraad, Wilfred T.V and Kramer, Boris W
Experimental Neurology, ISSN 0014-4886, 12/2013, Volume 250, pp. 293 - 303
Hypoxic–ischemic encephalopathy (HIE) is common in preterm infants, but currently no curative therapy is available. Cell-based therapy has a great potential in... 
Neuroprotection | Stem cells | White matter injury | Hypoxic–ischemic encephalopathy | G-CSF | Preterm | Sheep model | Hypoxic-ischemic encephalopathy | FUNCTIONAL RECOVERY | COLONY-STIMULATING FACTOR | WHITE-MATTER | BRAIN-INJURY | BONE-MARROW | RECOVERY ENHANCEMENT | NEUROSCIENCES | LATE OLIGODENDROCYTE PROGENITORS | SELECTIVE VULNERABILITY | STEM-CELL FACTOR | GROWTH-FACTOR | Immunohistochemistry | Nerve Fibers, Myelinated - drug effects | Encephalitis - pathology | Electroencephalography | Hypoxia-Ischemia, Brain - pathology | Hypoxia-Ischemia, Brain - complications | Granulocyte Colony-Stimulating Factor - pharmacology | Animals | Flow Cytometry | Neuroprotective Agents - pharmacology | Seizures - etiology | Electrocardiography | Fetus | Sheep | Hematopoietic Stem Cell Mobilization | Fetal Hypoxia - complications | Disease Models, Animal | Encephalitis - etiology | Fetal Hypoxia - pathology | Brain | Ischemia | Analysis | Inflammation | Seizures (Medicine) | Hematopoietic stem cells | Myelin proteins | Injuries
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