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1. Full Text C-Myb+ Erythro-Myeloid Progenitor-Derived Fetal Monocytes Give Rise to Adult Tissue-Resident Macrophages
by Hoeffel, Guillaume and Chen, Jinmiao and Lavin, Yonit and Low, Donovan and Low, Ivy and Almeida, Francisca F and See, Peter and Beaudin, Anna E and Lum, Josephine and Forsberg, E. Camilla and Poidinger, Michael and Zolezzi, Francesca and Larbi, Anis and Ng, Lai Guan and Chan, Jerry K.Y and Greter, Melanie and Becher, Burkhard and Samokhvalov, Igor M and Merad, Miriam and Ginhoux, Florent
Immunity, ISSN 1074-7613, 04/2015, Volume 42, Issue 4, pp. 665 - 678
Although classified as hematopoietic cells, tissue-resident macrophages (MFs) arise from embryonic precursors that seed the tissues prior to birth to generate... 
CIRCULATING MONOCYTES | SYSTEM | MOUSE YOLK-SAC | GM-CSF | HEMATOPOIETIC STEM-CELLS | MYB | ALVEOLAR MACROPHAGES | STEADY-STATE | DIFFERENTIATION | IMMUNOLOGY | BLOOD | Skin - cytology | Embryo, Mammalian | Monocytes - cytology | Kidney - immunology | Monocytes - immunology | Lung - cytology | Yolk Sac - cytology | Cell Tracking | Aging - immunology | Hematopoietic Stem Cells - immunology | Liver - immunology | Microglia - immunology | Myeloid Progenitor Cells - immunology | Myeloid Progenitor Cells - cytology | Female | Fetus | Cell Differentiation | Macrophages - immunology | Skin - immunology | Yolk Sac - immunology | Biomarkers - metabolism | Microglia - cytology | Proto-Oncogene Proteins c-myb - immunology | Kidney - cytology | Macrophages - cytology | Proto-Oncogene Proteins c-myb - metabolism | Pregnancy | Animals | Hematopoietic Stem Cells - cytology | Liver - cytology | Mice | Primary Cell Culture | Lung - immunology | Cell Lineage - immunology | Flow cytometry | Rodents | Cloning | Stem cells | Homeostasis | Software | Skin | Gene expression | Embryos
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2. Reproductive immunology
by Olding, Lars B
1997, Current topics in microbiology and immunology, ISBN 9783540618881, Volume 222., viii, 219
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3. Full Text Neutralizing human antibodies prevent Zika virus replication and fetal disease in mice
by Sapparapu, Gopal and Fernandez, Estefania and Kose, Nurgun and Bin Cao, Cao and Fox, Julie M and Bombardi, Robin G and Zhao, Haiyan and Nelson, Christopher A and Bryan, Aubrey L and Barnes, Trevor and Davidson, Edgar and Mysorekar, Indira U and Fremont, Daved H and Doranz, Benjamin J and Diamond, Michael S and Crowe, James E
Nature, ISSN 0028-0836, 12/2016, Volume 540, Issue 7633, pp. 443 - 447
Zika virus (ZIKV) is an emerging mosquito-transmitted flavivirus that can cause severe disease, including congenital birth defects during pregnancy(1). To... 
PROTECTION | MULTIDISCIPLINARY SCIENCES | WEST-NILE-VIRUS | ENHANCEMENT | CROSS-REACTIVITY | INFECTION | MONOCLONAL-ANTIBODIES | EXPOSURE | PREGNANCY | Americas | Antibody Specificity | Epitope Mapping | Humans | Protein Multimerization | Fetal Diseases - prevention & control | Viral Proteins - immunology | Antibodies, Monoclonal - therapeutic use | Male | Zika Virus Infection - virology | Antibodies, Neutralizing - immunology | Antibodies, Neutralizing - therapeutic use | Female | Fetus - virology | Antibodies, Monoclonal - chemistry | Antibodies, Monoclonal - immunology | Disease Models, Animal | Zika Virus - immunology | Zika Virus - growth & development | Antibodies, Viral - therapeutic use | Placenta - immunology | Viral Proteins - chemistry | Zika Virus Infection - pathology | Africa | Models, Molecular | Survival Rate | Virus Replication - immunology | Fetal Diseases - virology | Pregnancy | Animals | B-Lymphocytes - immunology | Asia | Antibodies, Neutralizing - chemistry | Fetal Diseases - immunology | Antibodies, Viral - chemistry | Viral Vaccines - chemistry | Zika Virus Infection - immunology | Antibodies, Viral - immunology | Fetus - immunology | Mice | Infectious Disease Transmission, Vertical - prevention & control | Placenta - virology | Viral Vaccines - immunology | Viral antibodies | Antibodies | Health aspects | Proteins | Immunoglobulins | Zika virus | Fetuses | Viral infections
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4. Full Text Human antibodies to the dengue virus E-dimer epitope have therapeutic activity against Zika virus infection
by Fernandez, Estefania and Dejnirattisai, Wanwisa and Cao, Bin and Scheaffer, Suzanne M and Supasa, Piyada and Wongwiwat, Wiyada and Esakky, Prabagaran and Drury, Andrea and Mongkolsapaya, Juthathip and Moley, Kelle H and Mysorekar, Indira U and Screaton, Gavin R and Diamond, Michael S
Nature Immunology, ISSN 1529-2908, 10/2017, Volume 18, Issue 11, pp. 1261 - 1269
The Zika virus (ZIKV) epidemic has resulted in congenital abnormalities in fetuses and neonates. Although some cross-reactive dengue virus (DENV)-specific... 
NEUTRALIZING ANTIBODIES | PROTECTION | STRUCTURAL BASIS | MALE SEXUAL TRANSMISSION | IN-VIVO | DISEASE | CROSS-REACTIVITY | MICE | BIRTH-DEFECTS | IMMUNOLOGY | DEPENDENT ENHANCEMENT | Dengue Virus - classification | Humans | Brain - virology | Cercopithecus aethiops | Protein Multimerization - immunology | Male | Neutralization Tests | Host-Pathogen Interactions - immunology | Epitopes - immunology | Zika Virus Infection - virology | Dengue Virus - immunology | Testis - immunology | Female | Fetus - virology | Vero Cells | Antibodies, Monoclonal - immunology | Zika Virus - immunology | Zika Virus - physiology | Cross Reactions - immunology | Viral Load - immunology | Pregnancy | Animals | Viral Envelope Proteins - chemistry | Zika Virus Infection - immunology | Antibodies, Viral - immunology | Fetus - immunology | Viral Envelope Proteins - immunology | Mice | Dengue Virus - metabolism | Brain - immunology | Testis - virology | Monoclonal antibodies | Dosage and administration | Research | Dengue viruses | Vector-borne diseases | Epidemics | Neonates | Cell culture | Immunoglobulins | Stability | Fetuses | Abnormalities | Dengue fever | Dengue | Viral diseases | Viruses | Infections | Zika virus | Lethality | Disease control | Receptors | Animal tissues | Testes
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5. Full Text Immunology of Pre-Eclampsia
by Redman, Christopher W.G and Sargent, Ian L
American Journal of Reproductive Immunology, ISSN 1046-7408, 2010, Volume 63, Issue 6, pp. 534 - 543
Pre-eclampsia develops in stages, only the last being the clinical illness. This is generated by a non-specific, systemic (vascular), inflammatory response,... 
NK cells | Immunoregulation | Placentation | HLA-C | Indoleamine 2,3-dioxygenase | Systemic inflammatory response | Regulatory T cells | NATURAL-KILLER-CELLS | PREGNANCY-INDUCED-HYPERTENSION | GENES INFLUENCE | INDOLEAMINE 2,3-DIOXYGENASE | placentation | PERIPHERAL-BLOOD | IMMUNOLOGY | 3-dioxygenase | TRYPTOPHAN CATABOLISM | indoleamine 2 | regulatory T cells | REPRODUCTIVE BIOLOGY | systemic inflammatory response | REGULATORY T-CELLS | immunoregulation | INDUCE PREECLAMPSIA | PLACENTAL OXIDATIVE STRESS | INFLAMMATORY RESPONSE | Oxidative Stress | Placenta - immunology | Humans | Inflammation | Pre-Eclampsia - pathology | T-Lymphocytes, Regulatory - immunology | Pregnancy | Trophoblasts - immunology | Animals | Placenta - pathology | Killer Cells, Natural - immunology | Female | Fetus - immunology | Mice | Pre-Eclampsia - immunology
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6. Full Text Functional Consequences of Neuromyelitis Optica-IgG Astrocyte Interactions on Blood-Brain Barrier Permeability and Granulocyte Recruitment
by Vincent, Thierry and Saikali, Philippe and Cayrol, Romain and Roth, Alejandro D and Bar-Or, Amit and Prat, Alexandre and Antel, Jack P
The Journal of Immunology, ISSN 0022-1767, 10/2008, Volume 181, Issue 8, pp. 5730 - 5737
Autoantibody neuromyelitis optica-IgG (NMO-IgG) recognizing aquaporin-4 (AQP4) is implicated as playing a central role in the physiopathology of NMO. The aim... 
PLASMA-EXCHANGE | COMPLEMENT | MULTIPLE-SCLEROSIS | ENDOTHELIAL-CELLS | INJURY | AQUAPORIN-4 | ANTIBODY | WATER CHANNEL | IMMUNOLOGY | KNOCKOUT MICE | EXPRESSION | Immunoglobulin G - blood | Multiple Sclerosis - blood | Autoantibodies - blood | Humans | Middle Aged | Astrocytes - pathology | Complement System Proteins - immunology | Fetus - pathology | Male | Killer Cells, Natural - pathology | Neuromyelitis Optica - immunology | Cerebellum - immunology | Astrocytes - immunology | Immunoglobulin G - immunology | Aged, 80 and over | Killer Cells, Natural - immunology | Primates | Adult | Female | Granulocytes - pathology | Blood-Brain Barrier - immunology | Aquaporin 4 - immunology | Neuromyelitis Optica - pathology | Neuromyelitis Optica - blood | Permeability | Cell Degranulation - immunology | Cerebellum - blood supply | Cerebellum - pathology | Blood-Brain Barrier - pathology | Immunity, Cellular - immunology | Autoantibodies - immunology | Animals | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Fetus - immunology | Aged | Granulocytes - immunology
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7. Full Text Natural Killer Cells Promote Fetal Development through the Secretion of Growth-Promoting Factors
by Fu, Binqing and Zhou, Yonggang and Ni, Xiang and Tong, Xianhong and Xu, Xiuxiu and Dong, Zhongjun and Sun, Rui and Tian, Zhigang and Wei, Haiming
Immunity, ISSN 1074-7613, 12/2017, Volume 47, Issue 6, pp. 1100 - 1113.e6
Natural killer (NK) cells are present in large populations at the maternal-fetal interface during early pregnancy. However, the role of NK cells in fetal... 
growth-promoting factors | fetal growth restriction | maternal-fetal interface | aged pregnancy | tissue-resident NK | decidual NK cells | fetal development | INTERFACE | RESIDENT NK CELLS | INFLAMMATION | SENESCENCE | INNATE LYMPHOID-CELLS | DIFFERENTIATION | IMMUNOLOGY | IMMUNE TOLERANCE | HLA-G | EXPRESSION | PREGNANCY | Abortion, Habitual - pathology | Antigens, CD - immunology | Leukocyte Immunoglobulin-like Receptor B1 - genetics | Humans | Abortion, Habitual - immunology | Basic-Leucine Zipper Transcription Factors - immunology | Fetal Growth Retardation - genetics | T-Box Domain Proteins - immunology | Adoptive Transfer | Antigens, CD - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Leukocyte Immunoglobulin-like Receptor B1 - immunology | Gene Expression Regulation, Developmental | Abortion, Habitual - genetics | Killer Cells, Natural - immunology | Adult | Female | Fetus | HLA-G Antigens - immunology | HLA-G Antigens - genetics | Cytokines - genetics | Carrier Proteins - immunology | Cytokines - immunology | Killer Cells, Natural - transplantation | Cytokines - metabolism | Decidua - immunology | Signal Transduction | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Cellular Microenvironment | Fetal Growth Retardation - prevention & control | Decidua - pathology | Basic-Leucine Zipper Transcription Factors - genetics | T-Box Domain Proteins - genetics | Fetal Growth Retardation - pathology | Fetal Growth Retardation - immunology | Pregnancy | Carrier Proteins - genetics | Killer Cells, Natural - cytology | Animals | Carrier Proteins - metabolism | Fetal Development - immunology | Integrin alpha1 - genetics | Integrin alpha1 - immunology | Mice | Intercellular Signaling Peptides and Proteins - immunology | Killer cells | Histocompatibility antigens | Growth | HLA histocompatibility antigens | Osteoglycin | Secretion | Crosstalk | Fetuses | Pleiotrophin | Decidua | Adoptive transfer | Cells | Interfaces | Biomedical materials | Uterus | Biocompatibility | Histocompatibility antigen HLA | Natural killer cells
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8. Full Text Reproductive immunology
by Christiansen, Ole B
Molecular Immunology, ISSN 0161-5890, 08/2013, Volume 55, Issue 1, pp. 8 - 15
â–º Exaggerated inflammation characterizes pathological pregnancies. â–º Uterine NK cells interact with placental HLA-G and -C thereby modifying inflammation. â–º... 
Recurrent miscarriage | Inflammation | HLA-G | Mannose-binding lectin | HY antigens | NORMAL-PREGNANCY | SECONDARY RECURRENT MISCARRIAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | IMMUNOLOGY | RETROSPECTIVE CASE-CONTROL | INFLAMMATORY-BOWEL-DISEASE | SPONTANEOUS-ABORTION | REGULATORY T-CELLS | CLINICAL-IMPLICATIONS | CLASS-II ALLELES | KILLER NK CELLS | Maternal-Fetal Exchange - immunology | Pregnancy Complications - etiology | Humans | Male | Pregnancy | Animals | Immune Tolerance - immunology | Models, Biological | Female | Fetus - immunology | Models, Animal | Mice | Pregnancy Complications - immunology | Reproduction - immunology | Immunoglobulins | Histocompatibility antigens | Systemic lupus erythematosus | Gastrointestinal diseases | HLA histocompatibility antigens | Lectins | Sugars | Monosaccharides
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9. Full Text Viral infection of the placenta leads to fetal inflammation and sensitization to bacterial products predisposing to preterm labor
by Cardenas, Ingrid and Means, Robert E and Aldo, Paulomi and Koga, Kaori and Lang, Sabine M and Booth, Carmen and Manzur, Alejandro and Oyarzun, Enrique and Romero, Roberto and Mor, Gil
Journal of Immunology, ISSN 0022-1767, 07/2010, Volume 185, Issue 2, pp. 1248 - 1257
Pandemics pose a more significant threat to pregnant women than to the nonpregnant population and may have a detrimental effect on the well being of the fetus.... 
CELLS | DOUBLE-STRANDED-RNA | TRANSMISSION | ACTIVATION | ADENOASSOCIATED VIRUS | TOLL-LIKE RECEPTORS | MURINE GAMMAHERPESVIRUS-68 | TROPHOBLAST RESPONSES | IMMUNOLOGY | NF-KAPPA-B | PREGNANCY | Immunohistochemistry | Placenta Diseases - virology | NIH 3T3 Cells | Pregnancy Complications, Infectious - virology | Humans | Virus Diseases - virology | Rhadinovirus - immunology | Virus Diseases - complications | Host-Pathogen Interactions - immunology | Pregnancy Complications, Infectious - immunology | Bacterial Infections - complications | Bacterial Infections - immunology | Female | Fetus - virology | Obstetric Labor, Premature - etiology | Toll-Like Receptor 3 - genetics | Placenta Diseases - immunology | Pregnancy Complications, Infectious - microbiology | Rhadinovirus - physiology | Bacterial Infections - microbiology | Maternal-Fetal Exchange - immunology | Cell Line | Virus Diseases - immunology | Cytokines - metabolism | Placenta - immunology | Mice, Inbred C57BL | Cells, Cultured | Toll-Like Receptor 3 - immunology | Inflammation - immunology | Obstetric Labor, Premature - immunology | Toll-Like Receptor 3 - metabolism | Inflammation - etiology | Fetal Diseases - virology | Mice, Knockout | Pregnancy | Animals | Fetal Diseases - immunology | Fetus - immunology | Mice | Placenta - virology
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10. Full Text Intrathymic programming of effector fates in three molecularly distinct γδ T cell subtypes
by Narayan, Kavitha and Sylvia, Katelyn E and Malhotra, Nidhi and Yin, Catherine C and Martens, Gregory and Vallerskog, Therese and Kornfeld, Hardy and Xiong, Na and Cohen, Nadia R and Brenner, Michael B and Berg, Leslie J and Kang, Joonsoo and Zhou, Yan and Shinton, Susan A and Hardy, Richard R and Bezman, Natalie A and Sun, Joseph C and Kim, Charlie C and Lanier, Lewis L and Miller, Jennifer and Brown, Brian and Miller, Jennifer and Fletcher, Anne and Elpek, Kutlu and Bellemare-Pelletier, Angelique and Malhotra, Deepali and Turley, Shannon and Sylvia, Katelyn and Gazit, Roi and Garrison, Brian and Rossi, Derrick J and Jojic, Vladimir and Koller, Daphne and Jianu, Radu and Laidlaw, David and Costello, James and Collins, Jim and Cohen, Nadia and Brennan, Patrick and Brenner, Michael and Shay, Tal and Regev, Aviv and Kim, Francis and Rao, Tata Nageswara and Wagers, Amy and Gautier, Emmanuel L and Jakubzick, Claudia and Randolph, Gwendalyn J and Monach, Paul and Best, Adam J and Knell, Jamie and Goldrath, Ananda and Heng, Tracy and Kreslavsky, Taras and Painter, Michio and Mathis, Diane and Benoist, Christophe and Immunological Genome Project Consortium and The Immunological Genome Project Consortium
Nature Immunology, ISSN 1529-2908, 05/2012, Volume 13, Issue 5, pp. 511 - 518
Innate γδ T cells function in the early phase of immune responses. Although innate γδ T cells have often been studied as one homogenous population, they can be... 
T-Lymphocyte Subsets - immunology | Age Factors | T-Lymphocyte Subsets - cytology | CD24 Antigen - metabolism | Interleukin-17 - immunology | Mycobacterium tuberculosis - immunology | Receptors, Antigen, T-Cell, gamma-delta - genetics | Transcriptome - immunology | Interferon-gamma - metabolism | Cell Differentiation - genetics | Flow Cytometry | Thymus Gland - metabolism | Transcription Factors - immunology | Mycobacterium tuberculosis - metabolism | Principal Component Analysis | Mice, Inbred C57BL | Thymus Gland - cytology | Receptors, Antigen, T-Cell, gamma-delta - immunology | Transcriptome - genetics | Fetus - cytology | Transcription Factors - metabolism | Cell Differentiation - immunology | Interleukin-17 - metabolism | Models, Immunological | Receptors, Antigen, T-Cell, gamma-delta - classification | Animals | T-Lymphocyte Subsets - metabolism | Interferon-gamma - immunology | Fetus - immunology | Thymus Gland - immunology | Mice | CD24 Antigen - immunology | Cell Lineage - immunology
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11. Full Text HLA-G expressing DC-10 and CD4+ T cells accumulate in human decidua during pregnancy
by Amodio, Giada and Mugione, Alessandra and Sanchez, Ana Maria and ViganĂ², Paola and Candiani, Massimo and Somigliana, Edgardo and Roncarolo, Maria Grazia and Panina-Bordignon, Paola and Gregori, Silvia
Human Immunology, ISSN 0198-8859, 2012, Volume 74, Issue 4, pp. 406 - 411
Abstract Multiple mechanisms underlie the surprising willingness of mothers to tolerate the semi-allogeneic fetal tissues during pregnancy. Chief among these... 
Allergy and Immunology | NATURAL-KILLER-CELLS | DENDRITIC CELLS | REGULATORY CELLS | INTERFACE | GESTATION | SPONTANEOUS-ABORTION | EMBRYO IMPLANTATION | PHENOTYPE | PERIPHERAL-BLOOD | IMMUNOLOGY | SUBSET | Antigens, CD - immunology | Cell Adhesion Molecules - genetics | Decidua - cytology | Dendritic Cells - immunology | Humans | Lectins, C-Type - immunology | Cell Adhesion Molecules - immunology | Lectins, C-Type - genetics | Antigens, CD - genetics | Pregnancy Trimester, First | T-Lymphocytes, Regulatory - immunology | Gene Expression - immunology | T-Lymphocytes, Regulatory - cytology | Female | Fetus | HLA-G Antigens - immunology | HLA-G Antigens - genetics | Decidua - immunology | Antigens, Surface - genetics | Antigens, CD1 | Immunophenotyping | Glycoproteins | Histocompatibility, Maternal-Fetal | Receptors, Cell Surface - immunology | Pregnancy | Interleukin-10 - genetics | Immune Tolerance - genetics | Dendritic Cells - cytology | Antigens, Surface - immunology | Interleukin-10 - immunology | Receptors, Cell Surface - genetics
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