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Seminars in immunopathology, ISSN 1863-2300, 2011, Volume 34, Issue 1, pp. 43 - 62
The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | ALZHEIMERS-DISEASE | IMMUNOLOGY | PATHOLOGY | GAMMA-C-DOMAIN | BINDING-SITE | CENTRAL-NERVOUS-SYSTEM | MULTIPLE-SCLEROSIS LESIONS | CRYSTAL-STRUCTURE | BLOOD-BRAIN-BARRIER | INTEGRIN ALPHA(M)BETA | PROTEASE-ACTIVATED RECEPTORS | PERIPHERAL ARTERIAL-DISEASE | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
Critical care (London, England), ISSN 1364-8535, 12/2015, Volume 19, Issue 1, pp. 72 - 72
Knowing the pathophysiology of trauma-induced coagulopathy is important for the management of severely injured trauma patients. The aims of this review are to... 
THROMBIN GENERATION | ACTIVATED PROTEIN-C | MICROVASCULAR THROMBOSIS | ORGAN DYSFUNCTION SYNDROME | SOLUBLE THROMBOMODULIN ANTIGEN | INTERNATIONAL SOCIETY | EXTRACELLULAR HISTONES | FACTOR PATHWAY INHIBITOR | TISSUE FACTOR | INNATE IMMUNITY | CRITICAL CARE MEDICINE | Hemostasis - physiology | Humans | Thrombomodulin - physiology | Protein S - metabolism | Wounds and Injuries - metabolism | Shock, Traumatic - physiopathology | Immunity, Innate - physiology | HMGB1 Protein - metabolism | Lipoproteins - metabolism | Disseminated Intravascular Coagulation - metabolism | Shock, Traumatic - metabolism | Thrombosis - physiopathology | DNA, Mitochondrial - metabolism | Antithrombins - metabolism | Disseminated Intravascular Coagulation - diagnosis | Nucleosomes - metabolism | Wounds and Injuries - physiopathology | Thrombosis - metabolism | Animals | Disseminated Intravascular Coagulation - physiopathology | Wound Healing - physiology | Fibrinolysis - physiology | Histones - metabolism | Protein C - metabolism | Thrombin - metabolism | Fibrin | Immune response | Physiological aspects | Thrombin | Disseminated intravascular coagulation | Inflammation | Thrombosis | Blood clot | Endothelium | Pathogens | Anticoagulants | Cytokines | Multiple organ dysfunction syndrome | Neutrophils | Mitochondrial DNA | Trauma | Proteins | Signal transduction | Sepsis | Acidosis | Deoxyribonucleic acid--DNA | Review
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2013, Volume 288, Issue 10, pp. 6946 - 6956
Journal Article
Genes & development, ISSN 0890-9369, 2008, Volume 22, Issue 9, pp. 1231 - 1243
During vasculogenesis and angiogenesis, endothelial cell responses to growth factors are modulated by the compositional and mechanical properties of a... 
Angiogenesis | Extracellular matrix | Actomyosin | Endothelial cells | Fibronectin | VASCULAR DEVELOPMENT | endothelial cells | GROWTH-CONTROL | ASSEMBLY SITES | angiogenesis | MOUSE EMBRYOS | DEVELOPMENTAL BIOLOGY | CELL-CYCLE PROGRESSION | CELL BIOLOGY | IN-VITRO | fibronectin | actomyosin | BALLISTIC INTRACELLULAR NANORHEOLOGY | ENDOTHELIAL-CELLS | GENETICS & HEREDITY | EXTRACELLULAR MATRIX-CYTOSKELETON | extracellular matrix | Neovascularization, Physiologic - drug effects | Phosphorylation | Humans | Vinculin - metabolism | Extracellular Matrix - metabolism | Immunoblotting | Green Fluorescent Proteins - genetics | Hepatocyte Growth Factor - pharmacology | Recombinant Fusion Proteins - metabolism | Extracellular Matrix - chemistry | Transfection | Myosins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Cell Culture Techniques | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Green Fluorescent Proteins - metabolism | Collagen Type I - metabolism | Endothelial Cells - metabolism | Cells, Cultured | Cell Adhesion | Fibronectins - metabolism | Protein Folding | Microscopy, Confocal | Neovascularization, Physiologic - physiology | Vinculin - genetics | Fibronectins - chemistry | Mitogen-Activated Protein Kinase 3 - metabolism | Endothelial Cells - cytology | Cytoskeleton - metabolism | Recombinant Fusion Proteins - genetics | Cytoskeleton - physiology | Fibrin - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Fibronectins | Research | Neovascularization | Research Paper
Journal Article
Journal Article
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 2017, Volume 92, pp. 1103 - 1110
Abstract Background Schwann cells (SCs) play an indispensable role in the repair and regeneration of injured peripheral nerve. Curcumin can reduce SCs... 
Internal Medicine | Medical Education | Erk1/2 | Curcumin | Akt | Autophagy | Schwann cells | Myelinization | SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | FUNCTIONAL RECOVERY | REGENERATION | SPINAL-CORD | MODEL | CRUSH | INHIBITION | SIGNALING PATHWAY | DISEASE | PHARMACOLOGY & PHARMACY | Phosphorylation | Sciatic Nerve - injuries | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Myelin Sheath - drug effects | Male | Autophagy - drug effects | Myelin Sheath - metabolism | Sciatic Nerve - metabolism | Neuroprotective Agents - pharmacology | Sciatic Nerve - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Sciatic Neuropathy - drug therapy | Sequestosome-1 Protein - metabolism | Disease Models, Animal | Schwann Cells - drug effects | Cell Line | Myelin Sheath - pathology | Sciatic Nerve - pathology | Curcumin - pharmacology | Sciatic Neuropathy - pathology | S100 Proteins - metabolism | Schwann Cells - pathology | Schwann Cells - metabolism | Rats, Sprague-Dawley | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Nerve Regeneration - drug effects | Sciatic Neuropathy - physiopathology | Beclin-1 - metabolism | Fibrin - metabolism | Myelin Proteins - metabolism | Sciatic Neuropathy - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Apoptosis | Immunohistochemistry | Fibrin
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 40, pp. 14430 - 14435
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 11/2017, Volume 117, Issue 11, pp. 2125 - 2134
Abstract Objective  Alveolar macrophages play a key role in the development and resolution of acute respiratory distress syndrome (ARDS), modulating the... 
Blood Cells, Inflammation and Infection | Acute respiratory distress syndrome | Acute lung injury | Heparin | Alveolarmacrophages | Anti-coagulants | ANTICOAGULANTS | alveolar macrophages | STIMULATED HUMAN MONOCYTES | RESOLUTION | RANDOMIZED CONTROLLED-TRIAL | anti-coagulants | acute lung injury | RESPIRATORY-DISTRESS-SYNDROME | heparin | acute respiratory distress syndrome | ANTITHROMBIN-III | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | TISSUE FACTOR | EPIDEMIOLOGY | SEVERE SEPSIS | Tumor Necrosis Factor-alpha - metabolism | Macrophages, Alveolar - pathology | Anticoagulants - administration & dosage | Fibrin Fibrinogen Degradation Products - metabolism | Pneumonia - pathology | Pneumonia - prevention & control | Heparin - administration & dosage | Male | NF-kappa B - metabolism | Acute Lung Injury - blood | Lipopolysaccharides | Pneumonia - chemically induced | Inflammation Mediators - metabolism | Pulmonary Edema - prevention & control | Anti-Inflammatory Agents - administration & dosage | Pneumonia - blood | Pulmonary Edema - pathology | Acute Lung Injury - drug therapy | Lung - metabolism | Interleukin-6 - metabolism | Pulmonary Edema - metabolism | Neutrophil Infiltration - drug effects | Disease Models, Animal | Peptide Hydrolases - metabolism | Lung - pathology | Administration, Inhalation | Acute Lung Injury - pathology | Rats, Sprague-Dawley | Animals | Acute Lung Injury - chemically induced | Blood Coagulation - drug effects | Signal Transduction - drug effects | Macrophages, Alveolar - drug effects | Lung - drug effects | Antithrombin III - metabolism | Macrophages, Alveolar - metabolism | Thromboplastin - metabolism | Transforming Growth Factor beta - metabolism | Nebulizers and Vaporizers
Journal Article
Journal Article