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Seminars in immunopathology, ISSN 1863-2300, 2011, Volume 34, Issue 1, pp. 43 - 62
The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | ALZHEIMERS-DISEASE | IMMUNOLOGY | PATHOLOGY | GAMMA-C-DOMAIN | BINDING-SITE | CENTRAL-NERVOUS-SYSTEM | MULTIPLE-SCLEROSIS LESIONS | CRYSTAL-STRUCTURE | BLOOD-BRAIN-BARRIER | INTEGRIN ALPHA(M)BETA | PROTEASE-ACTIVATED RECEPTORS | PERIPHERAL ARTERIAL-DISEASE | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
European heart journal, ISSN 0195-668X, 08/2014, Volume 35, Issue 29, pp. 1917 - 1924
Epidemiological research over the last 50 years has discovered a plethora of biomarkers (including molecules, traits or other diseases) that associate with... 
Coronary artery disease | Mendelian randomization | C-REACTIVE PROTEIN | FUTURE MYOCARDIAL-INFARCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | ESTER TRANSFER PROTEIN | CARDIOVASCULAR EVENTS | SUBTILISIN/KEXIN TYPE 9 | ISCHEMIC-HEART-DISEASE | PHOSPHOLIPASE A | DENSITY-LIPOPROTEIN CHOLESTEROL | LAPLACE-TIMI 57 | GENOME-WIDE ASSOCIATION | Triglycerides - physiology | Cholesterol Ester Transfer Proteins - physiology | Humans | Obesity - genetics | Cholesterol, HDL - physiology | C-Reactive Protein - metabolism | Cholesterol Ester Transfer Proteins - genetics | Mendelian Randomization Analysis | Diabetes Complications - genetics | Genetic Pleiotropy - genetics | Hypertension - genetics | Phospholipases A2 - physiology | Telomere - genetics | Fibrinogen - physiology | Obesity - complications | Receptors, Interleukin-6 - genetics | Risk Factors | Linkage Disequilibrium - genetics | Lipoprotein(a) - physiology | C-Reactive Protein - genetics | Cholesterol, LDL - physiology | Cholesterol, HDL - genetics | Randomized Controlled Trials as Topic | Cholesterol, LDL - genetics | Serum Amyloid P-Component - genetics | Genetic Markers - genetics | Coronary Artery Disease - genetics | Hypertension - complications | Polymorphism, Single Nucleotide - genetics | Triglycerides - genetics | Lipoprotein(a) - genetics | Phospholipases A2 - genetics
Journal Article
by Sabater-Lleal, Maria and Huang, Jie and Chasman, Daniel and Naitza, Silvia and Dehghan, Abbas and Johnson, Andrew D and Teumer, Alexander and Reiner, Alex P and Folkersen, Lasse and Basu, Saonli and Rudnicka, Alicja R and Trompet, Stella and Mälarstig, Anders and Baumert, Jens and Bis, Joshua C and Guo, Xiuqing and Hottenga, Jouke J and Shin, So-Youn and Lopez, Lorna M and Lahti, Jari and Tanaka, Toshiko and Yanek, Lisa R and Oudot-Mellakh, Tiphaine and Wilson, James F and Navarro, Pau and Huffman, Jennifer E and Zemunik, Tatijana and Redline, Susan and Mehra, Reena and Pulanic, Drazen and Rudan, Igor and Wright, Alan F and Kolcic, Ivana and Polasek, Ozren and Wild, Sarah H and Campbell, Harry and Curb, J. David and Wallace, Robert and Liu, Simin and Eaton, Charles B and Becker, Diane M and Becker, Lewis C and Bandinelli, Stefania and Räikkönen, Katri and Widen, Elisabeth and Palotie, Aarno and Fornage, Myriam and Green, David and Gross, Myron and Davies, Gail and Harris, Sarah E and Liewald, David C and Starr, John M and Williams, Frances M.K and Grant, Peter J and Spector, Timothy D and Strawbridge, Rona J and Silveira, Angela and Sennblad, Bengt and Rivadeneira, Fernando and Uitterlinden, Andre G and Franco, Oscar H and Hofman, Albert and van Dongen, Jenny and Willemsen, Gonneke and Boomsma, Dorret I and Yao, Jie and Swords Jenny, Nancy and Haritunians, Talin and McKnight, Barbara and Lumley, Thomas and Taylor, Kent D and Rotter, Jerome I and Psaty, Bruce M and Peters, Annette and Gieger, Christian and Illig, Thomas and Grotevendt, Anne and Homuth, Georg and Völzke, Henry and Kocher, Thomas and Goel, Anuj and Franzosi, Maria Grazia and Seedorf, Udo and Clarke, Robert and Steri, Maristella and Tarasov, Kirill V and Sanna, Serena and Schlessinger, David and Stott, David J and Sattar, Naveed and Buckley, Brendan M and Rumley, Ann and Lowe, Gordon D and McArdle, Wendy L and Chen, Ming-Huei and Tofler, Geoffrey H and Song, Jaejoon and Boerwinkle, Eric and Folsom, Aaron R and ... and VTE Consortium and C4D Consortium and STROKE Consortium and Wellcome Trust Case Control Consortium 2 (WTCCC2) and CARDIoGRAM Consortium
Circulation (New York, N.Y.), ISSN 1524-4539, 2013, Volume 128, Issue 12, pp. 1310 - 1324
Journal Article
American journal of transplantation, ISSN 1600-6135, 03/2013, Volume 13, Issue 3, pp. 663 - 675
Atypical hemolytic and uremic syndrome (aHUS) is a severe disease strongly associated with genetic abnormalities in the complement alternative pathway... 
Acute rejection | endothelial cell damage | mutations and polymorphisms in complement alternative pathway | thrombotic microangiopathy | atypical hemolytic and uremic syndrome | renal transplantation | SURGERY | FACTOR-B | HUS | FACTOR-I | FACTOR-H-AUTOANTIBODIES | TRANSPLANTATION | KIDNEY-TRANSPLANTATION | MUTATIONS | CD46 | PREDISPOSE | MEMBRANE COFACTOR PROTEIN | Recurrence | Genetic Testing | Prognosis | Humans | Middle Aged | Male | Young Adult | Hemolytic-Uremic Syndrome - genetics | Complement Factor B - genetics | Adult | Complement System Proteins - genetics | Female | Hemolytic-Uremic Syndrome - therapy | Retrospective Studies | Complement C3 - genetics | Biomarkers - metabolism | Risk Factors | Biomarkers - analysis | Kidney Transplantation | Mutation - genetics | Fibrinogen - genetics | Graft Survival - genetics | Adolescent | Membrane Cofactor Protein - genetics | Aged | Complement Factor H - genetics | Graft Rejection - genetics | Atypical Hemolytic Uremic Syndrome | Medical research | Kidneys | Genes | Organ transplant recipients | Medicine, Experimental | Genetic research | Transplantation | Clinical trials | Mutation | Transplants & implants | Drug therapy | Biological Markers | Complement C3 | Life Sciences | Immunology | Fibrinogen | Hemolytic-Uremic Syndrome | Antigens, CD46 | Graft Survival | Complement Factor H | Complement System Proteins | Complement Factor B | Graft Rejection
Journal Article
PLoS genetics, ISSN 1553-7404, 2013, Volume 9, Issue 4, p. e1003444
Journal Article
American journal of respiratory and critical care medicine, ISSN 1535-4970, 2012, Volume 186, Issue 12, pp. 1238 - 1247
Journal Article
Journal of Assisted Reproduction and Genetics, ISSN 1058-0468, 1/2016, Volume 33, Issue 1, pp. 67 - 73
Journal Article
PloS one, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, p. e0180365
Introduction Genetic constitution and inflammation both contribute to development of coronary artery disease (CAD... 
C-REACTIVE PROTEIN | HEART-DISEASE | LDL-CHOLESTEROL | MYOCARDIAL-INFARCTION | MULTIDISCIPLINARY SCIENCES | LOCI | MYELOID-RELATED PROTEIN-14 | RISK SCORES | APOE | MYELOID-RELATED-PROTEIN-8/14 | GENOME-WIDE ASSOCIATION | Humans | Middle Aged | Male | Genetic Loci | Coronary Artery Disease - blood | C-Reactive Protein - metabolism | Interleukin-6 - blood | Membrane Transport Proteins - genetics | Coronary Artery Disease - pathology | Aged, 80 and over | Adult | Female | Complement C3 - genetics | Biomarkers - metabolism | Gene Expression | Cross-Sectional Studies | Interleukin-6 - genetics | Membrane Transport Proteins - blood | Complement C3 - metabolism | Risk Factors | C-Reactive Protein - genetics | Inflammation | Leukocyte L1 Antigen Complex - blood | Fibrinogen - genetics | Apolipoproteins E - blood | Coronary Artery Disease - diagnosis | Apolipoproteins E - genetics | Alleles | Coronary Artery Disease - genetics | Fibrinogen - metabolism | Leukocyte L1 Antigen Complex - genetics | Aged | Polymorphism, Single Nucleotide | Development and progression | Genetic aspects | Research | Biological markers | Coronary heart disease | C-reactive protein | Heart attacks | Interleukin | Cardiovascular disease | Genomes | Single-nucleotide polymorphism | Interleukin 6 | Proteins | Apolipoprotein E | Fibrinogen | Cardiology | Heart diseases | Constitution | Cytokines | Coronary artery | Metabolism | Gene expression | Coronary artery disease | Patients | Meta-analysis | Medicine | Studies | Sensitivity | Hospitals | Coronary vessels | Biomarkers | Clinical medicine | Acute coronary syndromes | Health risk assessment
Journal Article