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International Journal of Cancer, ISSN 0020-7136, 02/2005, Volume 113, Issue 5, pp. 752 - 760
The role of platelets in tumor progression and metastasis has been recognized but the mechanism of their action remains unclear. Five human lung cancer cell... 
lung cancer | platelet microvesicles | metastasis | matrix metalloproteinases | exosomes | Matrix metalloproteinases | Metastasis | Platelet microvesicles | Exosomes | Lung cancer | SURVIVAL | VESICLES | MEMBRANE-TYPE-1 MATRIX-METALLOPROTEINASE | ADHESION | IN-VITRO | ONCOLOGY | GROWTH | SURFACE | RHABDOMYOSARCOMA CELLS | GENERATION | ENDOTHELIAL MICROPARTICLES | Phosphorylation | Cell Proliferation | Humans | Lung Neoplasms - metabolism | Carcinoma, Lewis Lung - secondary | Lung Neoplasms - pathology | Metalloendopeptidases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Carcinoma, Lewis Lung - blood supply | Cyclin D2 | Neovascularization, Pathologic - pathology | Mitogen-Activated Protein Kinase Kinases - metabolism | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinases, Membrane-Associated | Cyclins - metabolism | Neoplasm Invasiveness - pathology | Bone Marrow - metabolism | Platelet Membrane Glycoproteins - metabolism | Female | Interleukin-8 - metabolism | Tumor Cells, Cultured | Lung Neoplasms - blood supply | Matrix Metalloproteinase 14 | Endothelial Cells - metabolism | Mice, Inbred C57BL | Umbilical Veins | Carcinoma, Lewis Lung - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Chemotaxis | Disease Progression | Hepatocyte Growth Factor - metabolism | Animals | Blood Platelets - metabolism | Endothelial Cells - cytology | Platelet Activation | Bone Marrow - pathology | Fibrinogen - metabolism | Mice | Neovascularization, Pathologic - metabolism | Index Medicus
Journal Article
Journal of Biomechanics, ISSN 0021-9290, 2011, Volume 45, Issue 5, pp. 824 - 831
Abstract The elastic modulus of bioengineered materials has a strong influence on the phenotype of many cells including cardiomyocytes. On polyacrylamide (PAA)... 
Physical Medicine and Rehabilitation | Elastic modulus | Cardiac myocyte | Hyaluronic acid | Sarcomere | Mechanosensing | SIGNALING PATHWAYS | SOFT MATERIALS | ENGINEERING, BIOMEDICAL | PHENOTYPE | HEART | BIOPHYSICS | INJECTABLE HYDROGEL | GROWTH | FIBRONECTIN | EXTRACELLULAR-MATRIX | SUBSTRATE STIFFNESS | CD44 | Biocompatible Materials - metabolism | Fibroblasts - physiology | Actins - metabolism | Extracellular Matrix - metabolism | Cell Culture Techniques - methods | Integrins - metabolism | Bioengineering - methods | Acrylic Resins - metabolism | Extracellular Matrix - physiology | Hydrogels - metabolism | Sarcomeres - physiology | Extracellular Matrix Proteins - metabolism | Fibroblasts - metabolism | Collagen Type I - metabolism | Receptor Cross-Talk | Tissue Engineering - methods | Myocytes, Cardiac - cytology | Sarcomeres - metabolism | Rats | Biomechanical Phenomena - physiology | Rats, Sprague-Dawley | Fibronectins - metabolism | Animals | Myocytes, Cardiac - physiology | Hyaluronic Acid - metabolism | Fibrinogen - metabolism | Myocytes, Cardiac - metabolism | Elastic Modulus - physiology | Fibroblasts - cytology | Gelatin - metabolism | Actins - physiology | Polyacrylamide | Integrins | Proteins | Genotype & phenotype | Heart attacks | Hydrogels | Rodents | Stem cells | Surface chemistry | Cardiomyocytes | Ligands | Molecular weight | Index Medicus | mechanosensing | elastic modulus | cardiac myocyte | sarcomere | hyaluronic acid
Journal Article
Journal Article
Advances in Experimental Medicine and Biology, ISSN 0065-2598, 06/2016, Volume 910, pp. 23 - 30
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 08/2005, Volume 83, Issue 4, pp. 364 - 374
Oval cells are facultative liver progenitor cells, which are invoked during chronic liver injury in order to replenish damaged hepatocytes and bile duct cells.... 
acute phase response | cytokine | liver injury | inflammation | liver regeneration | Inflammation | Acute phase response | Liver regeneration | Cytokine | Liver injury | Liver - pathology | Apoptosis - drug effects | Cell Count | RNA, Messenger - analysis | Acute-Phase Proteins - genetics | Liver - injuries | Choline Deficiency - blood | Time Factors | Choline Deficiency - metabolism | Hepatitis - metabolism | Cytokines - genetics | Cytokines - immunology | B-Lymphocytes - cytology | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | Hepatitis - pathology | Choline Deficiency - pathology | Ethionine - pharmacology | Animals | Aspartate Aminotransferases - blood | T-Lymphocytes - cytology | Cell Proliferation - drug effects | Mice | Acute-Phase Proteins - biosynthesis | Cytokines - biosynthesis | Interleukin-18, metabolism | Liver, cytology | Antigens, CD45, metabolism | Liver, injuries | Comparative Study | Haptoglobins, metabolism | Male | Membrane Glycoproteins, metabolism | Choline Deficiency | Fibrinogen, metabolism | Tumor Necrosis Factor-alpha, metabolism | Orosomucoid, metabolism | Interleukin-6, metabolism | Interleukin-12, metabolism | Ethionine, pharmacology | RNA, Messenger, metabolism | Gene Expression | Antigens, CD3, metabolism | Interleukin-1, metabolism | Liver Regeneration, physiology | Antigens, CD4, metabolism | Transforming Growth Factor beta, metabolism | Antigens, Thy-1, metabolism | Liver, metabolism | Lymphotoxin, metabolism | Interferon Type II, metabolism | Antigens, CD, metabolism | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, pp. e0166886 - e0166886
Bronchopulmonary dysplasia (BPD) is a major cause of neonatal morbidity in premature infants, occurring as a result of arrested lung development combined with... 
BRONCHOPULMONARY DYSPLASIA | OXIDATIVE STRESS | NAD(P)H OXIDASE | PULMONARY-HYPERTENSION | PLEXIFORM LAYERS | OXYGEN-INDUCED RETINOPATHY | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | LUNG DEVELOPMENT | PRIMARY PREVENTION | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Retina - metabolism | Membrane Glycoproteins - metabolism | Vitreoretinopathy, Proliferative - etiology | Humans | NADPH Oxidases - metabolism | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | Retinal Ganglion Cells - metabolism | Oxygen - metabolism | Retinal Vessels - pathology | Retinitis - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hyperoxia - metabolism | NADPH Oxidases - genetics | Disease Models, Animal | Retinitis - pathology | Gene Expression | Retinal Vessels - metabolism | Vitreoretinopathy, Proliferative - pathology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vitreoretinopathy, Proliferative - metabolism | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Collagen - metabolism | Animals | Mice | Retina - pathology | Retinal Neovascularization - pathology | Retinitis - metabolism | Proteins | Dysplasia | Infants (Premature) | Analysis | Inflammation | Vascular endothelial growth factor | Retinal diseases | Neonates | Pediatrics | GTP-binding protein | Animal models | Retinopathy | Lung | Retina | Infants | Macrophages | Angiogenesis | Signal transduction | Newborn babies | Lymphocytes | Rodents | CYBB protein | Fibrinogen | Bioindicators | Age | Phenotypes | Oxygen | Wound healing | Lung diseases | Blood vessels | Morbidity | Medicine | Cyclin-dependent kinase inhibitor p21 | Ostomy | Detachment | Hyperoxia | Laboratory animals | Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 140, Issue 2, pp. 510 - 524.e3
Background In asthma remodeling airway smooth muscle cells (ASMCs) contribute to airway wall thickness through increased proliferation, migration, and... 
Allergy and Immunology | Airway wall remodeling | protein arginine methyltransferase 1 | extracellular signal-regulated kinase | primary airway smooth muscle cell | STAT1 | miR-19a | MIGRATION | LUNG | CYCLE REGULATION | STIMULATED PROLIFERATION | IMMUNOLOGY | FIBROBLASTS | BETA-AGONISTS | INDUCED PULMONARY INFLAMMATION | ALLERGY | BRONCHOCONSTRICTION | DISEASE | GROWTH FACTOR-BB | Asthma - metabolism | Lung - pathology | Collagen Type I - metabolism | Humans | Middle Aged | Cells, Cultured | Repressor Proteins - genetics | Male | MicroRNAs - metabolism | RNA, Messenger - metabolism | Protein-Arginine N-Methyltransferases - metabolism | Airway Remodeling | STAT1 Transcription Factor - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Fibrinogen - metabolism | Aged, 80 and over | Female | Aged | Lung - metabolism | Protein-Arginine N-Methyltransferases - genetics | Asthma - pathology | Myocytes, Smooth Muscle - metabolism | Repressor Proteins - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | MicroRNA | Arginine | Transferases | Stem cells | Smooth muscle | Molecular biology | Asthma | Immunohistochemistry | Fibronectins | Platelet-derived growth factor | Mitogens | Protein kinases | Cell proliferation | Collagen (type I) | Transcription | Leukocyte migration | Pathogenesis | Lung | Kinases | Remodeling | Fibronectin | Proteins | Respiratory tract | Genotype & phenotype | Cell growth | Cell cycle | Extracellular matrix | Chronic obstructive pulmonary disease | Stat1 protein | Deposition | Lung diseases | MiRNA | Protein arginine methyltransferase | Extracellular signal-regulated kinase | Rats | MAP kinase | Inflammation | siRNA | Wall thickness | Ribonucleic acid--RNA | Patients | Platelet-derived growth factor BB | Polymerase chain reaction | Lungs | Protein kinase | Biopsy | MicroRNAs | Regulation | Immunofluorescence | Cell migration | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Matrix Biology, ISSN 0945-053X, 10/2010, Volume 29, Issue 8, pp. 668 - 677
Tendon-like tissue generated from stem cells has the potential to replace tendons and ligaments lost through injury and disease. However, thus far, no... 
Fibrin | Signaling | Differentiation | Forces | Collagen | Chondrocytes - cytology | Transforming Growth Factor beta3 - genetics | Gene Expression - genetics | Humans | Adipocytes - cytology | Monocyte-Macrophage Precursor Cells - metabolism | Transforming Growth Factor beta3 - metabolism | Antigens, CD - metabolism | Young Adult | Mesenchymal Stromal Cells - cytology | Tendons - cytology | Collagen Type I - genetics | Fibrillar Collagens - ultrastructure | Smad2 Protein - genetics | Gels - metabolism | Stromal Cells - drug effects | Adult | Bone Marrow Cells - drug effects | Osteoblasts - cytology | Phosphorylation - drug effects | Cell Differentiation - physiology | Chondrocytes - metabolism | Tendons - embryology | Transforming Growth Factor beta3 - pharmacology | Fibrillar Collagens - metabolism | Mesenchymal Stromal Cells - drug effects | Bone Marrow Cells - cytology | Stromal Cells - metabolism | Tendons - metabolism | Smad2 Protein - metabolism | Mesenchymal Stromal Cells - metabolism | Transforming Growth Factor beta3 - antagonists & inhibitors | Monocyte-Macrophage Precursor Cells - drug effects | Extracellular Matrix - ultrastructure | Signal Transduction - drug effects | Cell Differentiation - drug effects | Adipocytes - metabolism | Fibrinogen - metabolism | Monocyte-Macrophage Precursor Cells - cytology | Signal Transduction - physiology | Integrin beta Chains - genetics | Thrombin - metabolism | Fibrin - metabolism | Osteoblasts - metabolism | Bone Marrow Cells - metabolism | Stromal Cells - cytology | Gels - pharmacology | Index Medicus | BM-MNC, Bone marrow-derived mononuclear cell | MSC, Mesenchymal | marrow stromal stem cell
Journal Article