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Seminars in immunopathology, ISSN 1863-2300, 2011, Volume 34, Issue 1, pp. 43 - 62
The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | ALZHEIMERS-DISEASE | IMMUNOLOGY | PATHOLOGY | GAMMA-C-DOMAIN | BINDING-SITE | CENTRAL-NERVOUS-SYSTEM | MULTIPLE-SCLEROSIS LESIONS | CRYSTAL-STRUCTURE | BLOOD-BRAIN-BARRIER | INTEGRIN ALPHA(M)BETA | PROTEASE-ACTIVATED RECEPTORS | PERIPHERAL ARTERIAL-DISEASE | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
International journal of cancer, ISSN 1097-0215, 2005, Volume 113, Issue 5, pp. 752 - 760
The role of platelets in tumor progression and metastasis has been recognized but the mechanism of their action remains unclear. Five human lung cancer cell... 
lung cancer | platelet microvesicles | metastasis | matrix metalloproteinases | exosomes | Matrix metalloproteinases | Metastasis | Platelet microvesicles | Exosomes | Lung cancer | SURVIVAL | VESICLES | MEMBRANE-TYPE-1 MATRIX-METALLOPROTEINASE | PROLIFERATION | ADHESION | ONCOLOGY | SURFACE | RHABDOMYOSARCOMA CELLS | GENERATION | ENDOTHELIAL MICROPARTICLES | HEMOSTASIS | Phosphorylation | Cell Proliferation | Humans | Lung Neoplasms - metabolism | Carcinoma, Lewis Lung - secondary | Lung Neoplasms - pathology | Metalloendopeptidases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Carcinoma, Lewis Lung - blood supply | Cyclin D2 | Neovascularization, Pathologic - pathology | Mitogen-Activated Protein Kinase Kinases - metabolism | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinases, Membrane-Associated | Cyclins - metabolism | Neoplasm Invasiveness - pathology | Bone Marrow - metabolism | Platelet Membrane Glycoproteins - metabolism | Female | Interleukin-8 - metabolism | Tumor Cells, Cultured | Lung Neoplasms - blood supply | Matrix Metalloproteinase 14 | Endothelial Cells - metabolism | Mice, Inbred C57BL | Umbilical Veins | Carcinoma, Lewis Lung - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Chemotaxis | Disease Progression | Hepatocyte Growth Factor - metabolism | Animals | Blood Platelets - metabolism | Endothelial Cells - cytology | Platelet Activation | Bone Marrow - pathology | Fibrinogen - metabolism | Mice | Neovascularization, Pathologic - metabolism
Journal Article
Journal Article
Journal of Biomechanics, ISSN 0021-9290, 2012, Volume 45, Issue 5, pp. 824 - 831
Abstract The elastic modulus of bioengineered materials has a strong influence on the phenotype of many cells including cardiomyocytes. On polyacrylamide (PAA)... 
Physical Medicine and Rehabilitation | Elastic modulus | Cardiac myocyte | Hyaluronic acid | Sarcomere | Mechanosensing | MERLIN | MATRIX | ENGINEERING, BIOMEDICAL | PHENOTYPE | BIOPHYSICS | GROWTH | SUBSTRATE STIFFNESS | CELL | CD44 | Biocompatible Materials - metabolism | Fibroblasts - physiology | Actins - metabolism | Extracellular Matrix - metabolism | Cell Culture Techniques - methods | Integrins - metabolism | Bioengineering - methods | Acrylic Resins - metabolism | Extracellular Matrix - physiology | Hydrogels - metabolism | Sarcomeres - physiology | Extracellular Matrix Proteins - metabolism | Fibroblasts - metabolism | Collagen Type I - metabolism | Receptor Cross-Talk | Tissue Engineering - methods | Myocytes, Cardiac - cytology | Sarcomeres - metabolism | Rats | Biomechanical Phenomena - physiology | Rats, Sprague-Dawley | Fibronectins - metabolism | Animals | Myocytes, Cardiac - physiology | Hyaluronic Acid - metabolism | Fibrinogen - metabolism | Myocytes, Cardiac - metabolism | Elastic Modulus - physiology | Fibroblasts - cytology | Gelatin - metabolism | Actins - physiology | Polyacrylamide | Integrins | Proteins | Genotype & phenotype | Heart attacks | Hydrogels | Rodents | Stem cells | Surface chemistry | Cardiomyocytes | Ligands | Molecular weight | mechanosensing | elastic modulus | cardiac myocyte | sarcomere | hyaluronic acid
Journal Article
Journal of the American College of Nutrition, ISSN 1541-1087, 2002, Volume 21, Issue 6, pp. 495 - 505
Among the fatty acids, it is the omega-3 polyunsaturated fatty acids (PUFA) which possess the most potent immunomodulatory activities, and among the omega-3... 
background diet | inflammation | IL-1 | cardiovascular disease and major depression autoimmune diseases | omega-6/omega-3 ratio | TNF | IL-6 | Inflammation | Autoimmune diseases | Cardiovascular disease and major depression autoimmune diseases | Background diet | Cardiovascular disease and major depression | Omega-6/omega-3 ratio | autoimmune diseases | C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | CORONARY-ARTERY DISEASE | DIETARY FISH-OIL | ULCERATIVE-COLITIS | MAJOR DEPRESSION | cardiovascular disease and major depression | TUMOR-NECROSIS-FACTOR | PLASMA-FIBRINOGEN CONCENTRATION | NUTRITION & DIETETICS | ACUTE MYOCARDIAL-INFARCTION | CARDIOVASCULAR RISK-FACTORS | Fatty Acids, Omega-6 | Asthma - metabolism | Depressive Disorder, Major - metabolism | Humans | Inflammatory Bowel Diseases - immunology | Autoimmune Diseases - diet therapy | Inflammatory Bowel Diseases - metabolism | Depressive Disorder, Major - immunology | Arthritis, Rheumatoid - metabolism | Asthma - diet therapy | Fatty Acids, Omega-3 - physiology | Inflammation - diet therapy | Inflammation - metabolism | Asthma - immunology | Psoriasis - metabolism | Anti-Inflammatory Agents - administration & dosage | Fish Oils - administration & dosage | Autoimmune Diseases - metabolism | Fatty Acids, Omega-3 - administration & dosage | Psoriasis - therapy | Psoriasis - immunology | Depressive Disorder, Major - diet therapy | Fatty Acids, Omega-3 - metabolism | Cardiovascular Diseases - metabolism | Eicosapentaenoic Acid - metabolism | Fatty Acids, Unsaturated - metabolism | Inflammatory Bowel Diseases - diet therapy | Arthritis, Rheumatoid - diet therapy | Cardiovascular Diseases - immunology | Animals | alpha-Linolenic Acid - metabolism | Arthritis, Rheumatoid - immunology | Fish Oils - chemistry | Cytokines - biosynthesis | Docosahexaenoic Acids - metabolism | Prostaglandins - metabolism
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 08/2005, Volume 83, Issue 4, pp. 364 - 374
Oval cells are facultative liver progenitor cells, which are invoked during chronic liver injury in order to replenish damaged hepatocytes and bile duct cells.... 
acute phase response | cytokine | liver injury | inflammation | liver regeneration | Inflammation | Acute phase response | Liver regeneration | Cytokine | Liver injury | Liver - pathology | Apoptosis - drug effects | Cell Count | RNA, Messenger - analysis | Acute-Phase Proteins - genetics | Liver - injuries | Choline Deficiency - blood | Time Factors | Choline Deficiency - metabolism | Hepatitis - metabolism | Cytokines - genetics | Cytokines - immunology | B-Lymphocytes - cytology | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | Hepatitis - pathology | Choline Deficiency - pathology | Ethionine - pharmacology | Animals | Aspartate Aminotransferases - blood | T-Lymphocytes - cytology | Cell Proliferation - drug effects | Mice | Acute-Phase Proteins - biosynthesis | Cytokines - biosynthesis | Interleukin-18, metabolism | Liver, cytology | Antigens, CD45, metabolism | Liver, injuries | Comparative Study | Haptoglobins, metabolism | Male | Membrane Glycoproteins, metabolism | Choline Deficiency | Fibrinogen, metabolism | Tumor Necrosis Factor-alpha, metabolism | Orosomucoid, metabolism | Interleukin-6, metabolism | Interleukin-12, metabolism | Ethionine, pharmacology | RNA, Messenger, metabolism | Gene Expression | Antigens, CD3, metabolism | Interleukin-1, metabolism | Liver Regeneration, physiology | Antigens, CD4, metabolism | Transforming Growth Factor beta, metabolism | Antigens, Thy-1, metabolism | Liver, metabolism | Lymphotoxin, metabolism | Interferon Type II, metabolism | Antigens, CD, metabolism
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2017, Volume 140, Issue 2, pp. 510 - 524.e3
Background In asthma remodeling airway smooth muscle cells (ASMCs) contribute to airway wall thickness through increased proliferation, migration, and... 
Allergy and Immunology | Airway wall remodeling | protein arginine methyltransferase 1 | extracellular signal-regulated kinase | primary airway smooth muscle cell | STAT1 | miR-19a | MIGRATION | LUNG | CYCLE REGULATION | STIMULATED PROLIFERATION | IMMUNOLOGY | FIBROBLASTS | BETA-AGONISTS | INDUCED PULMONARY INFLAMMATION | ALLERGY | BRONCHOCONSTRICTION | DISEASE | GROWTH FACTOR-BB | Asthma - metabolism | Lung - pathology | Collagen Type I - metabolism | Humans | Middle Aged | Cells, Cultured | Repressor Proteins - genetics | Male | MicroRNAs - metabolism | RNA, Messenger - metabolism | Protein-Arginine N-Methyltransferases - metabolism | Airway Remodeling | STAT1 Transcription Factor - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Fibrinogen - metabolism | Aged, 80 and over | Female | Aged | Lung - metabolism | Protein-Arginine N-Methyltransferases - genetics | Asthma - pathology | Myocytes, Smooth Muscle - metabolism | Repressor Proteins - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | MicroRNA | Arginine | Transferases | Stem cells | Smooth muscle | Molecular biology | Asthma | Immunohistochemistry | Fibronectins | Platelet-derived growth factor | Mitogens | Protein kinases | Cell proliferation | Collagen (type I) | Transcription | Leukocyte migration | Pathogenesis | Kinases | Remodeling | Fibronectin | Proteins | Respiratory tract | Genotype & phenotype | Cell growth | Cell cycle | Extracellular matrix | miRNA | Chronic obstructive pulmonary disease | Stat1 protein | Deposition | Lung diseases | Protein arginine methyltransferase | Extracellular signal-regulated kinase | Rats | MAP kinase | Inflammation | siRNA | Wall thickness | Ribonucleic acid--RNA | Patients | Platelet-derived growth factor BB | Polymerase chain reaction | Lungs | Protein kinase | Biopsy | MicroRNAs | Regulation | Immunofluorescence | Cell migration
Journal Article