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Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 269 - 11
Resistance towards VEGF-centered anti-angiogenic therapy still represents a substantial clinical challenge. We report here that mast cells alter the... 
BREAST-CANCER | NEOADJUVANT CHEMOTHERAPY | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | C-KIT | EXTRACELLULAR-MATRIX | HEPARAN-SULFATE PROTEOGLYCANS | TUMOR ANGIOGENESIS | VASCULAR-PERMEABILITY | ENDOTHELIAL GROWTH-FACTOR | Fibroblast Growth Factor 2 - drug effects | Human Umbilical Vein Endothelial Cells | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Granzymes - metabolism | Extracellular Matrix - metabolism | Vascular Endothelial Growth Factor A - metabolism | Fibroblast Growth Factor 1 - drug effects | Cromolyn Sodium - pharmacology | Fibroblast Growth Factor 2 - metabolism | Vascular Endothelial Growth Factor A - drug effects | Anti-Asthmatic Agents - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - drug effects | Endothelial Cells - metabolism | Extracellular Matrix - drug effects | Fibroblast Growth Factor 1 - metabolism | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Neoplasms - blood supply | Vitronectin - metabolism | Mast Cells - drug effects | Granulocyte-Macrophage Colony-Stimulating Factor - drug effects | Neoplasms - drug therapy | Animals | Cell Line, Tumor | Mast Cells - secretion | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | Laminin - metabolism | Endothelial Cells - drug effects | Fibroblast growth factor 2 | Therapy | Fibroblast growth factor | Fibroblast growth factor 1 | Granulocyte-macrophage colony-stimulating factor | Secretion | Endothelial cells | Histamine | Angiogenesis | Granzyme B | Antiangiogenics | Mast cells | Vascular endothelial growth factor
Journal Article
Nature, ISSN 0028-0836, 05/2017, Volume 545, Issue 7653, pp. 224 - 241
Blood and lymphatic vasculatures are intimately involved in tissue oxygenation and fluid homeostasis maintenance. Assembly of these vascular networks involves... 
GENE-EXPRESSION DATA | MAINTENANCE | MULTIDISCIPLINARY SCIENCES | MOUSE | LYMPHANGIOGENESIS | ENDOTHELIAL-CELL METABOLISM | MECHANISMS | GROWTH-FACTOR | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Cell Proliferation | Lymphatic Vessels - cytology | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Proto-Oncogene Proteins c-myc - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Fibroblast Growth Factors - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - deficiency | Lymphatic Vessels - metabolism | Animals | Receptor, Fibroblast Growth Factor, Type 3 - deficiency | Endothelial Cells - cytology | Glycolysis | Female | Mice | Hexokinase - metabolism | Lymphangiogenesis | Neovascularization, Physiologic | Cell Movement | Fibroblast growth factors | Metabolism | Observations | Health aspects | Cell proliferation | Fibroblast growth factor | Leukocyte migration | c-Myc protein | Homeostasis | Biology | Myc protein | Kinases | Blood | Defects | Angiogenesis | Control | Cell growth | Metabolites | Rodents | Fibroblast growth factor receptor 1 | Vascular endothelial growth factor | Growth factors | Medical research | Enzymes | Grants | Hexokinase | Endothelial cells | Endothelium | Signaling | Oxygenation | Cell migration | Fibroblast growth factor receptors
Journal Article
Science, ISSN 0036-8075, 9/2012, Volume 337, Issue 6099, pp. 1231 - 1235
The brain tumor glioblastoma multiforme (GBM) is among the most lethal forms of human cancer. Here, we report that a small subset of GBMs (3.1%; 3 of 97 tumors... 
Exons | Neurons | Genes | REPORTS | Stem cells | Aneuploidy | Chromosomes | Cells | Tumors | Daughter cells | Cancer | ANEUPLOIDY | SELECTIVE INHIBITOR | POTENT | MULTIDISCIPLINARY SCIENCES | CANCER | RECEPTOR TYROSINE KINASE | DISCOVERY | CHROMOSOMAL INSTABILITY | FAMILY | Microtubule-Associated Proteins - chemistry | Neoplasm Transplantation | Translocation, Genetic | Oncogene Proteins, Fusion - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - chemistry | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Mitosis | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - antagonists & inhibitors | Fetal Proteins - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Brain Neoplasms - metabolism | Oncogene Proteins, Fusion - chemistry | Spindle Apparatus - metabolism | Glioblastoma - genetics | Oncogene Fusion | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Nuclear Proteins - genetics | Chromosomal Instability | Pyrazoles - pharmacology | Protein Structure, Tertiary | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Enzyme Inhibitors - pharmacology | Brain Neoplasms - genetics | Nuclear Proteins - metabolism | Pyrimidines - pharmacology | Nuclear Proteins - chemistry | Piperazines - pharmacology | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Xenograft Model Antitumor Assays | Animals | Cell Transformation, Neoplastic | Oncogene Proteins, Fusion - genetics | Fetal Proteins - genetics | Mice | Receptor, Fibroblast Growth Factor, Type 1 - chemistry | Fetal Proteins - chemistry | Physiological aspects | Development and progression | Fibroblast growth factors | Genetic aspects | Research | Health aspects | Glioblastoma multiforme | Proteins | Kinases | Brain cancer | Genomics | Pharmaceutical sciences
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 04/2010, Volume 223, Issue 1, pp. 84 - 93
Common in vitro protocols for chondrogenesis of mesenchymal stem cells (MSCs) induce an inadequate, hypertrophic differentiation cascade reminiscent of... 
PHYSIOLOGY | HORMONE-RELATED PEPTIDE | AUTOLOGOUS CHONDROCYTE IMPLANTATION | ENDOCHONDRAL BONE-FORMATION | GENE-EXPRESSION | PARATHYROID-HORMONE | INDIAN HEDGEHOG | ATDC5 IN-VITRO | STROMAL CELLS | VITRO CARTILAGE FORMATION | ADIPOSE-TISSUE | CELL BIOLOGY | Immunohistochemistry | Staining and Labeling - methods | Coloring Agents | Humans | Alkaline Phosphatase - metabolism | RNA, Messenger - metabolism | Alcian Blue | Chondrocytes - transplantation | Intercellular Signaling Peptides and Proteins - metabolism | Bone Morphogenetic Proteins - metabolism | Time Factors | Cell Shape | Cell Differentiation | Chondrogenesis | Chondrocytes - metabolism | Biomarkers - metabolism | Recombinant Proteins - metabolism | Chondrocytes - pathology | Peptide Fragments - metabolism | Matrix Metalloproteinase 13 - genetics | Cells, Cultured | Fibroblast Growth Factor 1 - metabolism | Mesenchymal Stromal Cells - metabolism | Collagen Type X - genetics | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Parathyroid Hormone-Related Protein - metabolism | Collagen Type II - genetics | Phenotype | Animals | Receptor, Parathyroid Hormone, Type 1 - genetics | Mice | Mesenchymal Stromal Cells - pathology | Transforming Growth Factor beta - metabolism | Insulin-Like Growth Factor I - metabolism | Mesenchymal Stem Cell Transplantation | Hypertrophy
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 11/2014, Volume 111, Issue 45, pp. E4869 - E4877
The human FGF receptors (FGFRs) play critical roles in various human cancers, and several FGFR inhibitors are currently under clinical investigation.... 
Structure-based drug design | Drug discovery | Cancer drug resistance | Kinase inhibitor | structure-based drug design | WILD-TYPE | MULTIDISCIPLINARY SCIENCES | BCR-ABL | GROWTH-FACTOR RECEPTORS | DRUG-RESISTANCE | kinase inhibitor | LUNG-CANCER | GENE FUSIONS | cancer drug resistance | SELECTIVE INHIBITOR | drug discovery | THERAPEUTIC TARGET | FACTOR RECEPTOR 4 | REGULATES PROLIFERATION | Receptor, Fibroblast Growth Factor, Type 4 - chemistry | Receptor, Epidermal Growth Factor - genetics | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - chemistry | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Crystallography, X-Ray | Structure-Activity Relationship | Mutation, Missense | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Protein Kinase Inhibitors - chemistry | Receptor, Epidermal Growth Factor - metabolism | Neoplasms - genetics | Receptor, Fibroblast Growth Factor, Type 2 - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Binding Sites | Neoplasms - enzymology | Antineoplastic Agents - chemistry | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Receptor, Epidermal Growth Factor - chemistry | Neoplasms - drug therapy | Drug Resistance, Neoplasm - genetics | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Receptor, Fibroblast Growth Factor, Type 1 - chemistry | Neoplasms - pathology | Receptor, Fibroblast Growth Factor, Type 4 - antagonists & inhibitors | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Amino Acid Substitution | Drug Resistance, Neoplasm - drug effects | Biological Sciences | PNAS Plus
Journal Article
Diabetes, ISSN 0012-1797, 12/2016, Volume 65, Issue 12, pp. 3649 - 3659
Journal Article