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Nature, ISSN 0028-0836, 09/2014, Volume 513, Issue 7518, pp. 436 - 439
Journal Article
Kidney International, ISSN 0085-2538, 01/2018, Volume 93, Issue 1, pp. 95 - 109
Inflammation plays a central role in the etiology of diabetic nephropathy, a global health issue. We observed a significant reduction in the renal expression... 
cytokines | fibrosis | inflammation | mesangial cells | diabetic nephropathy | PATHWAYS | ACTIVATION | JNK | KINASE | FIBROBLAST-GROWTH-FACTOR | PATHOGENESIS | OBESITY | INSULIN-RESISTANCE | UROLOGY & NEPHROLOGY | NF-KAPPA-B | EXPRESSION | Diabetes Mellitus, Experimental - drug therapy | Kidney - pathology | Rats, Wistar | Diabetic Nephropathies - etiology | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | Diabetes Mellitus, Type 1 - complications | Diabetic Nephropathies - blood | Diabetes Mellitus, Experimental - blood | Kidney - metabolism | Inflammation Mediators - metabolism | Diabetes Mellitus, Experimental - complications | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Cell Line | Diabetic Nephropathies - pathology | Kidney - drug effects | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Recombinant Proteins - pharmacology | Diabetes Mellitus, Type 1 - drug therapy | Blood Glucose - drug effects | Fibroblast Growth Factor 1 - blood | Diabetes Mellitus, Type 2 - blood | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Diabetes Mellitus, Type 1 - blood | Fibroblast Growth Factor 1 - pharmacology | Macrophages - drug effects | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2016, Volume 22, Issue 7, pp. 800 - 806
Type 2 diabetes (T2D) is among the most common and costly disorders worldwide(1). The goal of current medical management for T2D is to transiently ameliorate... 
MEDICINE, RESEARCH & EXPERIMENTAL | HOMEOSTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MODEL | SUPPRESSION | ASTROCYTES | BALANCE | CELL BIOLOGY | INSULIN | TANYCYTES | METABOLISM | INTRAVENOUS GLUCOSE-TOLERANCE | IN-VIVO | Body Composition | Ependymoglial Cells - metabolism | Injections, Intraventricular | Male | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | Neoplasm Proteins - metabolism | Deoxyglucose | Neoplasm Proteins - drug effects | Brain - metabolism | Adipose Tissue - metabolism | Receptor, Insulin - genetics | Diet, High-Fat | Muscle, Skeletal - drug effects | Myocardium - metabolism | Hypothalamus - drug effects | Diabetes Mellitus, Experimental - metabolism | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Glucose Tolerance Test | Heat-Shock Proteins - drug effects | Heat-Shock Proteins - metabolism | Liver - metabolism | Proto-Oncogene Proteins c-fos - metabolism | Rats | Ependymoglial Cells - drug effects | Receptor, Insulin - antagonists & inhibitors | Remission Induction | Blotting, Western | Proto-Oncogene Proteins c-fos - drug effects | Mice, Knockout | Blood Glucose - drug effects | Brain - drug effects | Hyperglycemia - metabolism | Rats, Zucker | Animals | Hypothalamus - metabolism | Hypothalamus - cytology | Fibroblast Growth Factor 1 - pharmacology | Heart - drug effects | Mice, Obese | Mice | Blood Glucose - metabolism | Adipose Tissue - drug effects | Forkhead Box Protein O1 - genetics | Carbon Radioisotopes | Type 2 diabetes | Molecular targeted therapy | Care and treatment | Innovations | Development and progression | Genetic aspects | Fibroblast growth factors | Health aspects | Fibroblasts | Diabetes | Drug therapy | Rodents | brain | diabetes | fibroblast growth factor
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 10/2015, Volume 16, Issue 10, pp. 24011 - 24031
Most anti-angiogenic therapies currently being evaluated target the vascular endothelial growth factor (VEGF) pathway; however, the tumor vasculature can... 
Angiogenesis | Akt | Melanoma | Ferulic acid | FGFR1 | CELLS | angiogenesis | BIOCHEMISTRY & MOLECULAR BIOLOGY | melanoma | PROLIFERATION | COMBINATION | CHEMISTRY, MULTIDISCIPLINARY | ferulic acid | SIGNALING PATHWAY | RESISTANCE | EXPRESSION | Human Umbilical Vein Endothelial Cells - metabolism | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Neovascularization, Pathologic - pathology | Matrix Metalloproteinase 9 - metabolism | Female | Antineoplastic Agents - pharmacology | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Angiogenesis Inhibitors - pharmacology | Rats | Melanoma - pathology | Enzyme Activation - drug effects | Chick Embryo | Rats, Sprague-Dawley | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Melanoma - drug therapy | Fibroblast Growth Factor 1 - pharmacology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Coumaric Acids - pharmacology | Photodynamic therapy | Acids | Neurons | Vascular endothelial growth factor
Journal Article
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 01/2009, Volume 296, Issue 1, pp. 121 - 131
Journal Article
Journal Article
Journal Article
Acta Biomaterialia, ISSN 1742-7061, 03/2014, Volume 10, Issue 3, pp. 1177 - 1186
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2010, Volume 107, Issue 52, pp. 22659 - 22664
Spinal astrocytes are coupled by connexin (Cx) gap junctions and express pannexin 1 (Px1) and purinergic receptors. Fibroblast growth factor 1 (FGF-1), which... 
Receptors | Spinal cord | Dyes | Astrocytes | Neurons | Gap junctions | Small interfering RNA | Fluorescence | Connexins | Fibroblast growth factors | Astroglia | Connexon | Neurodegeneration | Pannexon | Growth factor | astroglia | CELLS | GAP-JUNCTION CHANNELS | ACTIVATION | connexon | MULTIDISCIPLINARY SCIENCES | neurodegeneration | FIBROBLAST-GROWTH-FACTOR | PROLIFERATION | growth factor | pannexon | IMPROVES RECOVERY | RECEPTORS | CORD-INJURY | BRAIN | COMMUNICATION | Gap Junctions - metabolism | Adenosine Triphosphate - secretion | Botulinum Toxins, Type A - pharmacology | RNA Interference | Time Factors | Spinal Cord - cytology | Connexin 43 - genetics | Animals, Newborn | Astrocytes - cytology | Astrocytes - drug effects | Connexin 43 - metabolism | Cells, Cultured | Connexins - genetics | Rats | Nerve Tissue Proteins - genetics | Blotting, Western | Connexins - metabolism | Mice, Knockout | Nerve Tissue Proteins - metabolism | Neurotoxins - pharmacology | Animals | Cell Membrane Permeability - drug effects | Fibroblast Growth Factor 1 - pharmacology | Mice | Gap Junctions - drug effects | Microscopy, Fluorescence | Astrocytes - metabolism | Junctional complexes (Epithelium) | Physiological aspects | Cell junctions | Spinal cord injuries | Research | Health aspects | Adenosine triphosphate | Index Medicus | Biological Sciences
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 02/2004, Volume 94, Issue 3, pp. 316 - 323
ABSTRACT—Fibroblast growth factor-2 (FGF2) activates the extracellular signal–regulated kinases 1 and 2 (ERK1/2) through its specific receptors. Interaction of... 
Growth factors | Fibroblast growth factors | MIGRATION | CARDIAC & CARDIOVASCULAR SYSTEMS | growth factors | SYNDECAN-4 | PHOSPHORYLATION | PROTEIN-KINASE | fibroblast growth factors | FACTOR BINDING | INHIBITION | ENDOTHELIAL-CELLS | IN-VIVO | PERIPHERAL VASCULAR DISEASE | MUSCLE-CELL-PROLIFERATION | HEMATOLOGY | MODULATION | Receptor, Fibroblast Growth Factor, Type 1 | Receptor Protein-Tyrosine Kinases - physiology | Fibroblast Growth Factor 2 - pharmacology | Heparitin Sulfate - metabolism | Polysaccharide-Lyases - pharmacology | Fibroblast Growth Factors - metabolism | Receptors, Fibroblast Growth Factor - genetics | Fibroblast Growth Factor 2 - metabolism | DNA - biosynthesis | Receptors, Cell Surface - physiology | Endothelial Cells - physiology | CHO Cells | DNA - drug effects | Cell Line | Cricetinae | Intercellular Signaling Peptides and Proteins | Fibroblast Growth Factor 1 - metabolism | Receptors, Cell Surface - metabolism | Thionucleosides - pharmacology | Epidermal Growth Factor - metabolism | Fibroblast Growth Factors - pharmacology | Enzyme Activation - drug effects | Cell Movement - drug effects | Heparin-binding EGF-like Growth Factor | Animals | Fibroblast Growth Factor 10 | Receptor Protein-Tyrosine Kinases - genetics | Heparan Sulfate Proteoglycans - metabolism | Receptors, Fibroblast Growth Factor - metabolism | Fibroblast Growth Factor 1 - pharmacology | Growth Substances - pharmacology | Protein Binding | Deoxyadenosines - pharmacology | Epidermal Growth Factor - pharmacology | Mutation | Receptors, Fibroblast Growth Factor - physiology | Endothelial Cells - enzymology | Mitogen-Activated Protein Kinase 3 | Endothelial Cells - drug effects | Growth Substances - metabolism | Heparan Sulfate Proteoglycans - physiology | Mitogen-Activated Protein Kinase 1 - metabolism | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 05/2006, Volume 97, Issue 3, pp. 687 - 696
Astrocytes may modulate the survival of motor neurons in amyotrophic lateral sclerosis (ALS). We have previously shown that fibroblast growth factor‐1 (FGF‐1)... 
motor neurons | astrocytes | nuclear factor erythroid 2‐related factor 2 | p75 neurotrophin receptor | amyotrophic lateral sclerosis | glutathione | Motor neurons | Amyotrophic lateral sclerosis | Astrocytes | Glutathione | Nuclear factor erythroid 2-related factor 2 | Propionates - pharmacology | Embryo, Mammalian | Glutathione - metabolism | Nitrites - metabolism | Methionine - pharmacology | Glial Fibrillary Acidic Protein - metabolism | Hydroquinones - pharmacology | Quinolines - pharmacology | RNA, Messenger - biosynthesis | Glutathione - biosynthesis | Drug Interactions | Transfection - methods | Methionine - analogs & derivatives | Reverse Transcriptase Polymerase Chain Reaction - methods | Urea - analogs & derivatives | Spinal Cord - cytology | Receptor, Nerve Growth Factor - metabolism | Animals, Newborn | Astrocytes - drug effects | Cell Survival - drug effects | Blotting, Western - methods | Motor Neurons - physiology | Animals, Genetically Modified | Cells, Cultured | Nerve Growth Factor - pharmacology | Rats | Antioxidants - pharmacology | Pyrimidines - pharmacology | Coculture Techniques - methods | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Nitrates - metabolism | Animals | Cell Count - methods | NF-E2-Related Factor 2 - metabolism | Fibroblast Growth Factor 1 - pharmacology | Urea - pharmacology | Astrocytes - metabolism | Nitric Oxide Synthase Type II - metabolism | Superoxide Dismutase | Neurosciences | Inhibitor drugs | Gene expression | Neurons | Spine | Motor ability
Journal Article