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Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2009, Volume 119, Issue 3, pp. 512 - 523
Pulmonary hypertension (PH) is a progressive, lethal lung disease characterized by pulmonary artery SMC (PA-SMC) hyperplasia. leading to right-sided heart... 
MEDICINE, RESEARCH & EXPERIMENTAL | INHIBITION | SUBENDOTHELIAL EXTRACELLULAR-MATRIX | FACTOR-2 | GENE-EXPRESSION | RATS | MONOCROTALINE | FIBROBLAST-GROWTH-FACTOR | SMOOTH-MUSCLE-CELLS | LUNG-CANCER CELLS | FACTOR RECEPTOR | RNA, Small Interfering - genetics | Humans | Hypertension, Pulmonary - physiopathology | Fibroblast Growth Factor 1 - genetics | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Muscle, Smooth, Vascular - physiopathology | Hypertension, Pulmonary - prevention & control | Endothelium, Vascular - physiology | In Situ Hybridization | Cell Division | Fibroblast Growth Factor 1 - physiology | Fibroblast Growth Factor 2 - physiology | Muscle, Smooth, Vascular - physiology | Disease Models, Animal | RNA, Messenger - genetics | Cells, Cultured | Endothelium, Vascular - physiopathology | Gene Expression Regulation | Rats | Lung - physiopathology | Pulmonary Artery - physiopathology | Lung - physiology | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Disease Progression | Pulmonary Artery - physiology | Animals | Fibroblast Growth Factor 2 - genetics | Hypertension, Pulmonary - pathology | Physiological aspects | Development and progression | Fibroblast growth factors | Genetic aspects | Research | Pulmonary hypertension | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 10/2004, Volume 110, Issue 16, pp. 2436 - 2443
Background-Neointimal vascular smooth muscle cell (VSMC) proliferation is a primary cause of occlusive vascular disease, including atherosclerosis, restenosis... 
Restenosis | Gene therapy | Growth substances | restenosis | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROSIS | gene therapy | PROLIFERATION | CORONARY-ARTERIES | VASA VASORUM | ANGIOPLASTY | growth substances | DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | NEOVASCULARIZATION | ACCUMULATION | HEMATOLOGY | ENDOTHELIAL GROWTH-FACTOR | Angiogenesis Inducing Agents | Receptor, Fibroblast Growth Factor, Type 1 | Hyperplasia | Receptor Protein-Tyrosine Kinases - physiology | Myocytes, Smooth Muscle - pathology | Recombinant Fusion Proteins - physiology | Male | Fibroblast Growth Factor 1 - antagonists & inhibitors | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Receptors, Fibroblast Growth Factor - genetics | Fibroblast Growth Factor 1 - physiology | Models, Animal | Proteins - physiology | Rabbits | Antimicrobial Cationic Peptides - pharmacology | Solubility | Rats | Rats, Sprague-Dawley | Proteins - genetics | Muscle, Smooth, Vascular - pathology | Animals | Tunica Intima - pathology | Receptor Protein-Tyrosine Kinases - genetics | Carotid Artery Injuries - physiopathology | Catheterization - adverse effects | Proteins - pharmacology | Receptors, Fibroblast Growth Factor - physiology | Vasa Vasorum - pathology | Vascular Endothelial Growth Factor A - physiology | Neovascularization, Physiologic | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 11/2005, Volume 17, Issue 2, pp. 576 - 584
Journal Article
Journal Article
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 12/2006, Volume 45, Issue 12, pp. 934 - 942
Inappropriate fibroblast growth factor (FGF) signaling is involved in most tissue‐specific pathologies including cancer. Previously we showed that... 
growth factors | liver adenoma | hepatoma models | receptor switching | transgenic models | tyrosine kinases | hepatocellular carcinoma | Receptor switching | Transgenic models | Liver adenoma | Hepatoma models | Tyrosine kinases | Hepatocellular carcinoma | Growth factors | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | FIBROBLAST-GROWTH-FACTOR | PROLIFERATION | CANCER | REGULATOR | ONCOLOGY | AUTOCRINE | LIVER | HEPARAN-SULFATE | MICE | RECEPTORS | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Carcinoma, Hepatocellular - chemically induced | Transcriptional Activation | Fibroblast Growth Factors - genetics | Liver Neoplasms - chemically induced | RNA, Messenger - analysis | Hepatocytes - pathology | Cell Transformation, Neoplastic - chemically induced | Fibroblast Growth Factor 1 - genetics | Fibroblast Growth Factor 1 - antagonists & inhibitors | Hepatocytes - metabolism | RNA, Messenger - metabolism | Cell Transformation, Neoplastic - genetics | Carcinoma, Hepatocellular - genetics | Fibroblast Growth Factor 1 - physiology | Liver Neoplasms - pathology | Fibroblast Growth Factor 2 - physiology | Fibroblast Growth Factors - physiology | Gene Targeting | Liver Neoplasms - genetics | Mice, Transgenic | Carbon Tetrachloride - toxicity | Fibroblast Growth Factor 2 - antagonists & inhibitors | Animals | Fibroblast Growth Factor 2 - genetics | Receptor, Fibroblast Growth Factor, Type 4 - agonists | Carcinoma, Hepatocellular - pathology | Mice | Fibroblast Growth Factors - antagonists & inhibitors | Cell Transformation, Neoplastic - pathology | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e57284 - e57284
Fibroblast growth factor receptors (FGFRs) are activated by mutation and overexpressed in bladder cancers (BCs), and FGFR inhibitors are currently being... 
TRANSITIONAL-CELL CARCINOMA | MULTIDISCIPLINARY SCIENCES | MUTATION | KINASE | SENSITIVITY | MICE | INHIBITOR | FGFR3 | EXPRESSION | TUMORS | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Humans | Gene Expression Profiling | DNA Primers | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Receptor, Fibroblast Growth Factor, Type 1 - physiology | Blotting, Western | Cell Division - drug effects | Cell Division - physiology | Receptor, Fibroblast Growth Factor, Type 3 - physiology | Neoplasm Metastasis | Animals | RNA Interference | Mice, Nude | Base Sequence | Urinary Bladder Neoplasms - pathology | Cell Line, Tumor | Female | Mice | Mutation | Real-Time Polymerase Chain Reaction | RNA | Stem cells | Fibroblast growth factors | Genetic aspects | Metastasis | Health aspects | Bladder cancer | Vimentin | Cell proliferation | Fibroblast growth factor | Mesenchyme | Clinical trials | Bladder | Biology | mRNA | Kinases | E-cadherin | Urology | Lymph nodes | Metastases | Heterogeneity | Signal transduction | Receptors | Biological effects | Urinary bladder | Fibroblasts | Fibroblast growth factor receptor 1 | Growth factors | Deoxyribonucleic acid--DNA | Medical research | Tumor cells | RNA-mediated interference | Growth factor receptors | Gene expression | Patients | Studies | Inhibitors | Medical prognosis | Cell lines | Laboratory animals | Cancer | Fibroblast growth factor receptors | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2009, Volume 29, Issue 46, pp. 14571 - 14580
Journal Article