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PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e14117
... EGFR is dominant for growth signaling, but not in cell lines where EGFR signaling is absent. A luciferase reporter containing... 
Fibroblast Growth Factor 7 - pharmacology | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Coculture Techniques | Humans | Lung Neoplasms - metabolism | Fibroblast Growth Factor 2 - pharmacology | Lung Neoplasms - pathology | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Fibroblasts - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Drug Resistance, Neoplasm - genetics | Signal Transduction - drug effects | Fibroblasts - drug effects | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Fibroblasts - cytology | Protein Kinase Inhibitors - pharmacology | Quinazolines - pharmacology | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Tyrosine | Phenols | Fibroblast growth factors | RNA | Analysis | Luciferase | Fibroblast growth factor | Biotechnology | Transcription | Gene regulation | Lung cancer | Biology | Kinases | Cell adhesion & migration | Morphogenesis | Epidermal growth factor | Fibroblast growth factor receptor 7 | Gefitinib | Protein-tyrosine kinase | Fibroblast growth factor receptor 2 | Fibroblast growth factor 2 | Epidermal growth factor receptors | Extracellular signal-regulated kinase | Non-small cell lung carcinoma | Gene expression | Src protein | Self sufficiency | Pulmonary hypertension | Studies | Signaling | Inhibitors | Monoclonal antibodies | Ligands | Mutation | Tumors | Fibroblast growth factor receptors
Journal Article
Sports Medicine, ISSN 0112-1642, 2003, Volume 33, Issue 5, pp. 381 - 394
.... Growth factors represent one of the most important of the molecular families involved in healing, and a considerable number of studies have been undertaken in an effort to elucidate their many functions... 
Tendon injuries | Connective tissue disorders | Growth factors, pharmacodynamics | Medicine & Public Health | Sports Medicine | SPORT SCIENCES | FACTOR-BETA | MEDIAL COLLATERAL LIGAMENT | MESSENGER-RNA | FACTOR-BB | RAT | TISSUE-REPAIR | FACTOR EXPRESSION | FACTOR-I | MODEL | BINDING-PROTEINS | Vascular Endothelial Growth Factor A | Insulin-Like Growth Factor I - pharmacology | Tendons - drug effects | Vascular Endothelial Growth Factors | Humans | Fibroblast Growth Factor 2 - pharmacology | Tendon Injuries - physiopathology | Endothelial Growth Factors - pharmacology | Intercellular Signaling Peptides and Proteins - physiology | Ligaments - physiopathology | Fibroblast Growth Factor 2 - physiology | Lymphokines - pharmacology | Wound Healing - drug effects | Endothelial Growth Factors - physiology | Ligaments - drug effects | Lymphokines - physiology | Transforming Growth Factor beta - physiology | Platelet-Derived Growth Factor - pharmacology | Transforming Growth Factor beta - pharmacology | Tendons - physiopathology | Animals | Insulin-Like Growth Factor I - physiology | Growth Substances - physiology | Intercellular Signaling Peptides and Proteins - pharmacology | Growth Substances - pharmacology | Wound Healing - physiology | Platelet-Derived Growth Factor - physiology
Journal Article
STEM CELLS, ISSN 1066-5099, 09/2007, Volume 25, Issue 9, pp. 2206 - 2214
We have utilized a serum‐ and stromal cell‐free “spin embryoid body (EB)” differentiation system to investigate the roles of four growth factors, bone morphogenetic protein 4 (BMP4... 
Fibroblast growth factor 2 | Bone morphogenetic protein 4 | Embryonic stem cells | Hematopoiesis | Vascular endothelial growth factor | Stem cell factor | stem cell factor | embryonic stem cells | MOUSE | COCULTURE | SPECIFICATION | MURINE | CELL & TISSUE ENGINEERING | CELL BIOLOGY | hematopoiesis | COMMITMENT | LETHALITY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | vascular endothelial growth factor | fibroblast growth factor 2 | MOVEMENT | HEMANGIOBLAST | STROMAL CELLS | HEMATOLOGY | bone morphogenetic protein 4 | MESODERM INDUCTION | Bone Morphogenetic Protein 4 | Embryonic Stem Cells - cytology | Bone Morphogenetic Proteins - physiology | Homeodomain Proteins - metabolism | Humans | Cells, Cultured | Fibroblast Growth Factor 2 - pharmacology | Primitive Streak - metabolism | Homeodomain Proteins - genetics | Hematopoiesis - drug effects | Drug Synergism | Animals | Cell Differentiation - drug effects | Gene Expression Regulation, Developmental | Stem Cell Factor - pharmacology | Hematopoiesis - physiology | Fibroblast Growth Factor 2 - physiology | Mice | Culture Media, Serum-Free - pharmacology | Bone Morphogenetic Proteins - pharmacology | Stem Cell Factor - physiology | Vascular Endothelial Growth Factor A - pharmacology | Vascular Endothelial Growth Factor A - physiology | Drug Combinations
Journal Article
PLoS medicine, ISSN 1549-1676, 2008, Volume 5, Issue 1, pp. e19 - 0138
Background Important support functions, including promotion of tumor growth, angiogenesis, and invasion, have been attributed to the different cell types populating the tumor stroma, i.e... 
HUMAN BREAST-CARCINOMA | MEDICINE, GENERAL & INTERNAL | TGF-BETA | GROWTH-FACTOR-RECEPTOR | SQUAMOUS CARCINOGENESIS | TYPE-16 TRANSGENIC MICE | FACTOR-BETA-RECEPTOR | CELL INVASION | MOUSE MODEL | TISSUE STROMA | CERVICAL-CANCER | Fibroblast Growth Factor 7 - physiology | Fibroblasts - enzymology | Uterine Cervical Neoplasms - blood supply | Humans | Carcinoma - virology | Neoplasm Proteins - antagonists & inhibitors | Antineoplastic Agents - therapeutic use | Receptors, Platelet-Derived Growth Factor - physiology | Proto-Oncogene Proteins c-kit - biosynthesis | Neovascularization, Pathologic - physiopathology | Neoplasm Proteins - genetics | Carcinoma - drug therapy | Estradiol - administration & dosage | Receptors, Platelet-Derived Growth Factor - genetics | Mice, Transgenic | Piperazines - therapeutic use | Uterine Cervical Neoplasms - physiopathology | Pyrimidines - pharmacology | Fibroblast Growth Factor 7 - biosynthesis | Piperazines - pharmacology | Carcinoma - physiopathology | Stromal Cells - secretion | Fibroblast Growth Factor 2 - genetics | Neovascularization, Pathologic - drug therapy | Fibroblasts - drug effects | Carcinoma - blood supply | Mice | Fibroblasts - secretion | Epithelial Cells - metabolism | Neoplasm Proteins - physiology | Platelet-Derived Growth Factor - genetics | Human papillomavirus 16 | Paracrine Communication - physiology | Stromal Cells - enzymology | Stromal Cells - drug effects | Female | Proto-Oncogene Proteins c-kit - genetics | Antineoplastic Agents - pharmacology | Fibroblast Growth Factor 2 - physiology | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Neoplasm Proteins - biosynthesis | Pericytes - metabolism | Fibroblast Growth Factor 2 - biosynthesis | Uterine Cervical Neoplasms - drug therapy | Mice, Inbred Strains | Imatinib Mesylate | Fibroblast Growth Factor 7 - genetics | Animals | Estradiol - toxicity | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Paracrine Communication - drug effects | Uterine Cervical Neoplasms - virology | Protein Kinase Inhibitors - pharmacology | Benzamides | Platelet-Derived Growth Factor - physiology | Platelet-Derived Growth Factor - antagonists & inhibitors | Proteins | Medical research | Human papillomavirus | Cervical cancer | Tumors | Index Medicus | Oncology | Gynecological | Chemotherapy | Cancer
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 45, pp. E4869 - E4877
... rhabdomyosarcoma and neuroendocrine breast carcinomas. Here we report on the use of a structure-based drug design to develop two selective, next-generation covalent FGFR inhibitors, the FGFR irreversible inhibitors 2 (FIIN-2) and 3 (FIIN-3... 
Structure-based drug design | Drug discovery | Cancer drug resistance | Kinase inhibitor | structure-based drug design | WILD-TYPE | MULTIDISCIPLINARY SCIENCES | BCR-ABL | GROWTH-FACTOR RECEPTORS | DRUG-RESISTANCE | kinase inhibitor | LUNG-CANCER | GENE FUSIONS | cancer drug resistance | SELECTIVE INHIBITOR | drug discovery | THERAPEUTIC TARGET | FACTOR RECEPTOR 4 | REGULATES PROLIFERATION | Receptor, Fibroblast Growth Factor, Type 4 - chemistry | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - chemistry | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Humans | ErbB Receptors - genetics | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Crystallography, X-Ray | Structure-Activity Relationship | Mutation, Missense | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Protein Kinase Inhibitors - chemistry | Neoplasms - genetics | Receptor, Fibroblast Growth Factor, Type 2 - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Binding Sites | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Neoplasms - enzymology | Antineoplastic Agents - chemistry | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Neoplasms - drug therapy | Drug Resistance, Neoplasm - genetics | ErbB Receptors - chemistry | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Receptor, Fibroblast Growth Factor, Type 1 - chemistry | Neoplasms - pathology | Receptor, Fibroblast Growth Factor, Type 4 - antagonists & inhibitors | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Amino Acid Substitution | Drug Resistance, Neoplasm - drug effects | Biological Sciences | PNAS Plus
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 23, Issue 3, pp. 443 - 453
Journal Article
Molecular and cellular biology, ISSN 0270-7306, 2003, Volume 23, Issue 1, pp. 14 - 25
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
ACTIVATION | PROTEIN-KINASE-B | GENE | PHOSPHATIDYLINOSITOL 3-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | MICE | DIFFERENTIATION | LIGAND | EXPRESSION | DROSOPHILA EYE | CELL BIOLOGY | Endothelium, Vascular - cytology | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Humans | Protein-Serine-Threonine Kinases | Fibroblast Growth Factor 2 - pharmacology | Cell Survival - genetics | Endothelium, Vascular - drug effects | Endothelial Growth Factors - pharmacology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Vascular Endothelial Growth Factor Receptor-2 - genetics | Phosphatidylinositol 3-Kinases - drug effects | Lymphokines - metabolism | Cell Survival - drug effects | Vascular Endothelial Growth Factor Receptor-2 - drug effects | Signal Transduction | Membrane Proteins - genetics | Enzyme Inhibitors - pharmacology | Solubility | Recombinant Proteins - chemistry | Cell Division - drug effects | Lymphokines - chemistry | Vascular Endothelial Growth Factor Receptor-1 - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | Transcription Factors | Vascular Endothelial Growth Factor A | Intercellular Signaling Peptides and Proteins - chemistry | Receptors, Cell Surface | Endothelial Growth Factors - chemistry | Phosphatidylinositol 3-Kinases - metabolism | Arteries - cytology | Endothelium, Vascular - physiology | Intercellular Signaling Peptides and Proteins - metabolism | Receptors, Notch | Arteries - physiology | Fibroblast Growth Factor 2 - metabolism | Flavonoids - pharmacology | Membrane Proteins - metabolism | Lymphokines - pharmacology | Vascular Endothelial Growth Factor Receptor-1 - genetics | Cell Division - genetics | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - drug effects | Cells, Cultured | Gene Expression Regulation | Intracellular Signaling Peptides and Proteins | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Proto-Oncogene Proteins - genetics | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Proto-Oncogene Proteins c-akt | Receptor, Notch1 | Neovascularization, Physiologic | Receptor, Notch4 | Cell Growth and Development
Journal Article
Gastric cancer : official journal of the International Gastric Cancer Association and the Japanese Gastric Cancer Association, ISSN 1436-3305, 2014, Volume 19, Issue 1, pp. 53 - 62
Up to 10 % of primary gastric cancers are characterized by FGFR2 amplification, and fibroblast growth factor receptor (FGFR... 
AZD4547 | Medicine & Public Health | Gastroenterology | BGJ398 | Abdominal Surgery | Oncology | Cancer Research | Epithelial–mesenchymal transition | Drug resistance | Surgical Oncology | Fibroblast growth factor receptor | Fibroblast growth factor receptor inhibitor | TGF-BETA | SENSITIVITY | E-CADHERIN | ANTITUMOR-ACTIVITY | EMT | LINES | Epithelial-mesenchymal transition | POTENT | ONCOLOGY | GROWTH | MUTATIONS | GASTROENTEROLOGY & HEPATOLOGY | EXPOSURE | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Receptors, Fibroblast Growth Factor - antagonists & inhibitors | Epithelial-Mesenchymal Transition - drug effects | Stomach Neoplasms - pathology | Pyrimidines - pharmacology | Stomach Neoplasms - drug therapy | Piperazines - pharmacology | Isoxazoles - pharmacology | Resorcinols - pharmacology | Triazoles - pharmacology | Cell Line, Tumor | Receptor, Fibroblast Growth Factor, Type 2 - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Oxazoles - pharmacology | Phenylurea Compounds - pharmacology | Protein Kinase Inhibitors - pharmacology | Molecular Targeted Therapy - methods | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Transforming Growth Factor beta1 - pharmacology | Drug Resistance, Neoplasm - drug effects | Pyrazoles - pharmacology | Proteins | Stem cells | Heat shock proteins | Stomach cancer | Growth factors | Cancer | Original
Journal Article
Stem cells (Dayton, Ohio), ISSN 1549-4918, 2006, Volume 24, Issue 11, pp. 2412 - 2419
... from adult tissues have proven to be multipotent in vitro and have been successfully used for tissue repair in vivo [ 1 , 2 ]. Much attention has been paid... 
Fibroblast growth factor | Long‐term expansion | Adult stem cells | Self‐renewal | Adipogenesis | Self-renewal | Long-term expansion | PROGENITOR CELLS | SONIC HEDGEHOG | TISSUE | FIBROBLAST-GROWTH-FACTOR | self-renewal | adult stem cells | REPLICATIVE SENESCENCE | CELL & TISSUE ENGINEERING | CELL BIOLOGY | EX-VIVO EXPANSION | long-term expansion | PROMOTES PROLIFERATION | fibroblast growth factor | adipogenesis | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | STROMAL CELLS | HEMATOLOGY | EXPRESSION | SUPPRESSES OSTEOGENIC DIFFERENTIATION | Phosphorylation | Nitriles - pharmacology | Humans | Multipotent Stem Cells - metabolism | Fibroblast Growth Factor 2 - pharmacology | Adipose Tissue - cytology | Child, Preschool | Adipocytes - cytology | Male | Autocrine Communication - drug effects | Adipocytes - drug effects | Adipose Tissue - metabolism | Multipotent Stem Cells - drug effects | Cell Shape | Fibroblast Growth Factor 2 - metabolism | Colony-Forming Units Assay | MAP Kinase Kinase 1 - antagonists & inhibitors | Butadienes - pharmacology | Receptors, Fibroblast Growth Factor - antagonists & inhibitors | MAP Kinase Kinase 1 - metabolism | Pyrimidines - pharmacology | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Cell Differentiation - drug effects | Adipocytes - metabolism | Receptors, Fibroblast Growth Factor - metabolism | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Adipose Tissue - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article